You will be shown a question, followed by excerpts from biomedical research papers. Please answer the question based on the provided context. Do not include any text in your response other than the answer.

Question: What is the effect of TRH on myocardial contractility?

Context: Acute intravenous administration of TRH to rats with ischemic cardiomyopathy caused a significant increase in heart rate, mean arterial pressure, cardiac output, stroke volume, and cardiac contractility
TRH can enhance cardiomyocyte contractility in vivo.
TRH in the range of 0.1-10 mumol/l was found to exert a positive inotropic effect on cardiac contractility
Thyrotropin-releasing hormone (TRH) improved mean arterial pressure (MAP) and myocardial contractility (dp/dtmax, -dp/dtmax, Vpm, and Vmax)
TRH improves cardiac contractility, cardiac output, and hemodynamics
Thyrotropin-releasing hormone (TRH) could improve mean arterial pressure (MAP), myocardial contractile parameters (+/- dp/dtmax, Vpm and Vmax)
TRH increased the contractile force of muscles dose-dependently without changing the time course of contraction
TRH potentiated the response of contractile force to increasing extracellular Ca2+ concentration.
TRH has a positive inotropic effect at least partly due to an increase in the slow inward Ca2+ current
Thus, TRH improves cardiac contractility, cardiac output, and hemodynamics during hemorrhagic shock.
TRH improves myocardial contractility