You will be shown a claim related to the COVID-19 pandemic, and the abstract of a biomedical research paper. Each sentence from the abstract will be on a separate line. Your task is to return a JSON object with two fields:

- "verdict": The fact-checking verdict. If the information in the abstract supports the claim, write "SUPPORT". If the abstract contradicts the claim, write "CONTRADICT".
- "evidence": An array of sentences providing evidence for the verdict. Please copy all relevant sentences verbatim from the abstract.

For instance, if the model were given the claim "the COVID vaccine is safe for healthy adults", the output might be
{
  "verdict": "SUPPORT",
  "evidence": ["Our findings indicate that the risks of severe COVID vaccine side effects are low for healthy adults."]
}

Claim: Use of convalescent serum reduces severity of covid-19 in nonhuman modelss

Abstract:
SARS-CoV-2 coronavirus is responsible for Covid-19 pandemic.
In the early phase of infection, the single-strand positive RNA genome is translated into non-structural proteins (NSP).
One of the first proteins produced during viral infection, NSP1, binds to the host ribosome and blocks the mRNA entry channel.
This triggers translation inhibition of cellular translation.
In spite of the presence of NSP1 on the ribosome, viral translation proceeds however.
The molecular mechanism of the so-called viral evasion to NSP1 inhibition remains elusive.
Here, we confirm that viral translation is maintained in the presence of NSP1.
The evasion to NSP1-inhibition is mediated by the cis-acting RNA hairpin SL1 in the 5'UTR of SARS-CoV-2.
NSP1-evasion can be transferred on a reporter transcript by SL1 transplantation.
The apical part of SL1 is only required for viral translation.
We show that NSP1 remains bound on the ribosome during viral translation.
We suggest that the interaction between NSP1 and SL1 frees the mRNA accommodation channel while maintaining NSP1 bound to the ribosome.
Thus, NSP1 acts as a ribosome gatekeeper, shutting down host translation or fostering SARS-CoV-2 translation depending on the presence of the SL1 5'UTR hairpin.
SL1 is also present and necessary for translation of sub-genomic RNAs in the late phase of the infectious program.
Consequently, therapeutic strategies targeting SL1 should affect viral translation at early and late stages of infection.
Therefore, SL1 might be seen as a genuine 'Achille heel' of the virus.
{"verdict": "CONTRADICT", "evidence": ["One of the first proteins produced during viral infection, NSP1, binds to the host ribosome and blocks the mRNA entry channel.", "In spite of the presence of NSP1 on the ribosome, viral translation proceeds however.", "We show that NSP1 remains bound on the ribosome during viral translation."]}