You will be shown a scientific claim, and the abstract of a biomedical research paper. Each sentence from the abstract will be on a separate line. Your task is to return a JSON object with two fields:

- "verdict": The fact-checking verdict. If the information in the abstract supports the claim, write "SUPPORT". If the abstract contradicts the claim, write "CONTRADICT". If the abstract does not provide enough information to arrive at a verdict, write "NEI" (for "not enough information").
- "evidence": An array of sentences providing evidence for the verdict. Please copy all relevant sentences verbatim from the abstract. If the verdict was "NEI", then return an empty array.

For instance, if the model were given the claim "smoking causes cancer", the output might be
{
  "verdict": "SUPPORT",
  "evidence": ["The results of our meta-analysis provide overwhelming support that cigarette smoking is a risk cause for lung cancer."]
}

Your response should not include any text other than the json.

Claim: Mutations in RIM1 decrease levels of IME1 RNA.

Abstract:
In the yeast Saccharomyces cerevisiae, genetic studies suggest that the RIM1 gene encodes a positive regulator of meiosis.
rim1 mutations cause reduced expression of IME1, which is required for expression of many meiotic genes, and thus lead to a partial defect in meiosis and spore formation.
We report the sequence of RIM1 and functional analysis of its coding region.
The RIM1 gene product (RIM1) contains three regions similar to C2H2 zinc fingers.
Serine substitutions for cysteine in each of the putative zinc fingers abolish RIM1 function.
The carboxyl-terminus of RIM1 is enriched in acidic amino acids and is required for full RIM1 activity.
RIM1 also contains two putative cAMP-dependent protein kinase (cAPK) phosphorylation sites.
At one site, substitution of alanine for serine does not affect RIM1 activity; at the other site, this substitution impairs activity.
This analysis of RIM1 suggests that the protein may function as a transcriptional activator.
We have used the cloned RIM1 gene to create a complete rim1 deletion.
This null allele, like previously isolated rim1 mutations, causes a partial meiotic defect.
In addition to RIM1, maximum IME1 expression requires the MCK1 and IME4 gene products.
Defects associated with rim1, mck1, and ime4 mutations in expression of a meiotic reporter gene (ime2-lacZ) and in sporulation are additive.
These findings suggest that RIM1 acts independently of MCK1 and IME4 to stimulate IME1 expression.
{"verdict": "SUPPORT", "evidence": ["rim1 mutations cause reduced expression of IME1, which is required for expression of many meiotic genes, and thus lead to a partial defect in meiosis and spore formation.", "These findings suggest that RIM1 acts independently of MCK1 and IME4 to stimulate IME1 expression."]}