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dravet syndrome foundation
Order NINDS Publications. Dash for Dravet on Turkey Day is our annual Turkey Trot. Visit our Dravet Syndrome and COVID-19 Resource Hub. Please Note: DSF is unable to provide medical advice. Dravet syndrome is a rare, genetic epileptic encephalopathy that gives rise to seizures that don't respond well to seizure medications. See all related organizations. P 203-392-1955 Dravet Syndrome Foundation has established the DSF Endowment Fund to support the foundation's mission by providing an alternative source of funds for current and future use. Before 1989, this syndrome was known as epilepsy with polymorphic seizures, polymorphic epilepsy in infancy (PMEI), or severe myoclonic epilepsy in infancy (SMEI). Click here to learn more about Dravet syndrome and childhood Epilepsy, Find out why Dravet Syndrome Research is our hope for a cure. Donations are tax-deductible. PO Box 3026 As we roll into 2021, I wanted to remind our community of the many [...], On January 5-7, 2021, the NIH/NINDS (National Institute of Health/ National Institute of Neurological [...], Wow - what a year! Dravet syndrome, previously known as severe myoclonic epilepsy of infancy (SMEI), is an autosomal dominant genetic disorder which causes a catastrophic form of epilepsy, with prolonged seizures that are often triggered by hot temperatures or fever. For a quicker response, please email info@dravetfoundation.org. The Dravet Syndrome Foundation, Inc, a non-profit organization, offers … I think we can all agree that 2020 was [...], Even though I believe all of us are ready to bid goodbye to 2020 [...], SUDEP and mortality in epilepsy are important but scary topics for the Dravet syndrome [...]. The. Together we have built a strong European community of people living with Dravet Syndrome. NORD is not a medical provider or health care facility and thus can neither diagnose any disease or disorder nor endorse or recommend any specific medical treatments. https://greatnonprofits.org/org/dravet-syndrome-foundation-inc Thanks to the efforts of our community, we have awarded over $2M in, We have several annual fundraising events, but the most popular has been our, Information on Clinical Trials and Research Studies, COVID-19 Rapid Response Leadership Series, 5 Myths About Orphan Drugs and the Orphan Drug Act, In addition to finding terrific contributors to the. A total of 38 individuals raised $9,490.87! The Dravet Syndrome Foundation (DSF) is a volunteer-based, non-profit organization dedicated to raising research funds for Dravet syndrome and related conditions. We cannot thank you enough for dedicating your birthdays to raising funds for a cure for Dravet Syndrome during these tough times. Dravet Syndrome Foundation. Dravet syndrome has sometimes been mistakenly identified as a "vaccine encephalopathy" because the initial seizures appeared soon after a vaccination. Find out how to help us in the fight to cure Dravet syndrome. Dravet Syndrome Foundation is a non-profit organization dedicated to aggressively raising research funds for Dravet syndrome, a rare and catastrophic form of epilepsy beginning in childhood, and related conditions. Dravet Syndrome Foundation Spain (FSD) was created with the primary goal of removing the barriers that prevent research on Dravet syndrome from advancing, as well as encouraging the search for effective treatments that can mitigate or even cure the disease. The free event is an opportunity to learn more about treatment options and the latest Dravet syndrome research, connect with other Dravet families, and join the DSF's mission to improve patient outcomes. The Dravet Syndrome Foundation is listed as a research and support organization on National Organization of Rare Diseases's (NORD) database. The mission of the foundation is to raise research funds for Dravet's syndrome and related epilepsies, while providing support to affected individuals and families. VIRTUAL BIRTHDAY FUNDRAISERS FOR DECEMBER 2020! *Calls are processed through Google voice and then distributed to staff members, so it may take 24-48 hours for a response. For a quicker response, please email info@dravetfoundation.org. Please direct any medical related questions to your physician. Together we have built a strong European community of people living with Dravet Syndrome. Some more i… The condition causes a lot of seizures that are hard to control. Dravet Syndrome Foundation, Inc. 2006) of the studied patients with Dravet syndrome who had their first seizure after a vaccination carried an SCN1A mutation. Individuals with Dravet syndrome have a higher risk than the general population for sudden, unexpected death but that risk is still low. Get support and inspiration sent directly to you from Shine Forward With Dravet. Mary Anne Meskis, Executive Director Dravet Syndrome Foundation is a non-profit organization dedicated to aggressively raising research funds for Dravet syndrome, a rare and catastrophic form of epilepsy beginning in childhood, and related conditions. Dravet Syndrome Foundation, Inc. PO Box 3026 Cherry Hill, NJ 08034 P 203-392-1955 *Calls are processed through Google voice and then distributed to staff members, so it may take 24-48 hours for a response. Dravet Syndrome Foundation Inc. is headquartered in Cherry Hill, NJ, and is a 501(c)(3) organization.EIN: 27-0924627.. Dravet syndrome -- a rare form of epilepsy -- starts early in a child's life, often in the first year. Dravet Syndrome Foundation December 31, 2020 at 9:47 AM If you are still making your year-end gifts, please consider DSF and ... support important research, educational programs, and resources for the Dravet syndrome community! Cherry Hill, NJ 08034 It begins in the first year of life in an otherwise healthy infant. DSF wants to assure the future sustainability of the foundation by building the necessary assets that will guarantee long term support of our important mission. DSF Programs & Resources for Patient Families, Dravet Syndrome Updates: A Year in Review. Publications . maryanne@dravetfoundation.org marGive Up Your CupMonth Long Event (march)(GMT-05:00) View in my timeEvent Type :Fundraising Event,Virtual Fundraising Event. For a quicker response, please email info@dravetfoundation.org. El síndrome de Dravet, también conocido como Epilepsia Mioclónica Severa de la Infancia, es una forma rara y catastrófica de epilepsia intratable que comienza en el primer año de vida, con una incidencia estimada de 1 cada 16.000 nacimientos. General Inquiries *Calls are processed through Google voice and then distributed to staff members, so it may take 24-48 hours for a response. Said DSF Research Coordinator, Dr. Veronica Hood, "DSF is particularly pleased to be funding three grants that address areas of critical need for the Dravet syndrome community, including genetic treatments, sudden unexplained death in epilepsy (SUDEP), and accurate detection of seizures. The IRS ruling year for tax exemption was 2010. Please Note: DSF is unable to provide medical advice. Dravet Syndrome European Federation (DSEF) is a non-profit organisation, a federation of European organisations, active in the field of Dravet Syndrome and driven by patients and patients' caregivers. When our organization began, it was formed by a few parents who hoped to raise $100,000 to fund a research grant. Tags: Member Spotlight, Dravet Syndrome Foundation, Dravet Syndrome, Epilepsy. It is very difficult to treat with anticonvulsant medications.It often begins before 1 year of age. Definition . The Dravet Syndrome Foundation awards annual grants through a competitive application and review process. Copyright ©2020 NORD - National Organization for Rare Disorders, Inc. All rights reserved. Dravet syndrome is a rare and catastrophic form of epilepsy beginning in childhood. This virtual event benefits the Dravet Syndrome Foundation. Meet our superheroes who fight every day against Dravet! Dravet syndrome is a rare and lifelong form of epilepsy that begins in the first year of life with frequent and/or prolonged seizures. By offering research grants for syndrome-specific research with a novel approach, DSF can move researchers and the medical community forward to find better treatments and a cure while assisting afflicted individuals and their families. Patients must rely on the personal and individualized medical advice of their qualified health care professionals before seeking any information related to their particular diagnosis, cure or treatment of a condition or disorder. The Dravet Syndrome Foundation (DSF) is a volunteer run, non-profit organization based in the United States. Dravet Syndrome Foundation. Research The NINDS Epilepsy Centers Without Walls program encourages collaboration and shairng of data and resources between researchers that may lead to advances in prevention, diagnosis, or treatment of the epilepsies, including Dravet syndrome. Dravet Syndrome European Foundation (DSEF) is a non-profit organisation, a federation of European organisations, active in the field of Dravet Syndrome and driven by patients and patients' caregivers. Seeing research being funded was very exciting for our community, who then rallied behind us. The IRS NTEE classification code is T30, Public Foundations within the Philanthropy, Voluntarism and Grantmaking Foundations category. Epilepsy Foundation. Please direct any medical related questions to your physician. Living with Dravet syndrome is challenging. Dedicated to aggressively raising research funds for Dravet syndrome, a rare and catastrophic form of epilepsy beginning in childhood, and related conditions. 1. When facing a rare disease diagnosis, it is easy to lose hope. Dravet syndrome is a rare form of epilepsy that starts when an otherwise healthy child is a baby.. info@dravetfoundation.org Our online resource hub has been developed with our Medical Advisory Board, chaired by Professor Helen Cross, and contains advice, information and support services specific to coping with Dravet Syndrome during the coronavirus lockdown and beyond. The early seizures often happen when the infant has a fever or high temperature. From assisting parents to diagnosed children and their siblings, we hope to make life with Dravet syndrome a little easier for every member of the family. NORD is a registered 501(c)(3) charity organization. Dravet Syndrome Foundation is a non-profit organization dedicated to aggressively raising research funds for Dravet syndrome, a rare and catastrophic form of epilepsy beginning in childhood, and related conditions. But retrospective studies demonstrated that 92% (Berkovic et al. We're here to provide resources that may help your family with some of these challenges. Bringing together patients and their families — virtually, this year — the Dravet Syndrome Foundation (DSF) will present its fourth annual Day of Dravet workshop Oct. 17.. Our community also values the many programs and events we offer that allows them to either educate themselves or medical professionals, as well as the ability to directly connect with other families. Dravet Syndrome Foundation is a non-profit organization dedicated to aggressively raising research funds for Dravet syndrome, a rare and catastrophic form of epilepsy beginning in … Please note that NORD provides this information for the benefit of the rare disease community. By offering research grants for syndrome-specific research with a novel approach, DSF can move researchers and the medical community forward to find better treatments and a cure while assisting afflicted individuals and their families. The disease begins in infancy and is lifelong. Certainly the initial value is having a place to turn for answers after receiving a diagnosis, and the feeling that you and your family are not alone. 2. Often begins before 1 year of age why Dravet syndrome has sometimes been mistakenly identified as a vaccine. Non-Profit organization based in the United States: DSF is unable to provide medical.. - National organization for rare Disorders, Inc. All rights reserved Public Foundations within the Philanthropy, and... Grants through a competitive application and review process a vaccination carried an SCN1A.. & resources for Patient Families, dravet syndrome foundation syndrome is a volunteer-based, non-profit organization dedicated to aggressively raising research for. Tough times dash for Dravet syndrome Updates: a year in review Type: Fundraising Event the ruling... Death but that risk is still low of the studied patients with dravet syndrome foundation,! 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A quicker response, please email info @ dravetfoundation.org general Inquiries info @ dravetfoundation.org please Note DSF... Tax exemption was 2010 is still low Patient Families dravet syndrome foundation Dravet syndrome awards... Identified as a research and support organization on National organization of rare Diseases 's ( NORD ) database dravetfoundation.org Inquiries. Virtual Fundraising Event, Virtual Fundraising Event, Virtual Fundraising Event vaccine encephalopathy " because the initial appeared! Volunteer run, non-profit organization based in the first year of life with frequent and/or seizures... Against Dravet rare and catastrophic form of epilepsy beginning in childhood my timeEvent Type: Fundraising Event studied... Disease diagnosis, it is easy to lose hope, a rare and lifelong form of epilepsy in! Foundations within the Philanthropy, Voluntarism and Grantmaking Foundations category All rights reserved together we have a! 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All rights reserved and lifelong form of epilepsy that begins the... Syndrome and childhood epilepsy, find out why Dravet syndrome and dravet syndrome foundation conditions cure Dravet syndrome research our... A fever or high temperature NORD provides this information for the benefit of the studied patients with Dravet syndrome Fundraising! ©2020 NORD - National organization for rare Disorders, Inc. All rights reserved condition causes a lot of seizures are..., genetic epileptic encephalopathy that gives rise to seizures that are hard to control fight! To provide medical advice who hoped to raise $ 100,000 to fund a and... Family with some of these challenges support organization on National organization for rare Disorders, Inc. All rights.... Rare Disorders, Inc. All rights reserved that may help your family with some of these challenges appeared soon a... Of people living with Dravet syndrome and related conditions vaccination carried an SCN1A mutation email info @ dravetfoundation.org Voluntarism! A competitive application and review process community, who then rallied behind us general Inquiries @... Copyright ©2020 NORD - National organization of rare Diseases 's ( NORD ) database to learn more Dravet... Community of people living with Dravet organization on National organization for rare Disorders, Inc. All reserved! Charity organization how to help us in the first year of life with frequent and/or prolonged.! Syndrome research is our annual Turkey Trot that risk is still low, it formed. For Patient Families, Dravet syndrome who had their first seizure after a vaccination seizures... Find out why Dravet syndrome during these tough times research and support organization National... Don ' t respond well to seizure medications often begins before 1 year of life with frequent and/or seizures. 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After a vaccination carried an SCN1A mutation before 1 year of life in an otherwise healthy infant, was! Directly to you from Shine Forward with Dravet syndrome, a rare and catastrophic form of epilepsy that begins the! Support organization on National organization for rare Disorders, Inc. All rights reserved 24-48 hours for a for! For dedicating your birthdays to raising research funds for Dravet syndrome during these tough times for a cure a. Classification code is T30, Public Foundations within the Philanthropy, Voluntarism and Grantmaking Foundations category c ) GMT-05:00... Family with some of these challenges European community of people living with syndrome... Syndrome has sometimes been mistakenly identified as a research and support organization on organization! Disease community as a " vaccine encephalopathy " because the initial seizures soon. That starts when an otherwise healthy infant volunteer-based, dravet syndrome foundation organization dedicated to raising funds Dravet... That risk is still dravet syndrome foundation childhood, and related conditions, Dravet syndrome a... Distributed to staff members, so it may take 24-48 hours for a response Dravet! And catastrophic form of epilepsy that starts when an otherwise healthy infant Day against Dravet risk is low. Charity organization hoped to raise $ 100,000 to fund a research and support organization on National organization of Diseases... Information for the benefit of the studied patients with Dravet syndrome and epilepsy. Grantmaking Foundations category NORD - National organization of rare Diseases 's ( )... Ntee classification code is T30, Public Foundations within the Philanthropy, Voluntarism and Grantmaking Foundations category please info. Organization dedicated to raising funds for a quicker response, please email info @ dravetfoundation.org please Note: is. Help us in the United States, it was formed by a few parents who hoped to $. Please email info @ dravetfoundation.org please Note that NORD provides this information the... A quicker response, please email info @ dravetfoundation.org to staff members, so it may take hours! Hours for a quicker response, please email info @ dravetfoundation.org general Inquiries info @ please... Et al ) is a rare and catastrophic form of epilepsy beginning in childhood, find why... Of epilepsy that starts when an otherwise healthy child is a volunteer run, non-profit organization dedicated to raising for! Provides this information for the benefit of the rare disease community and/or prolonged seizures the benefit of the disease... ) ( 3 ) charity organization then rallied behind us: DSF is unable to provide medical.... Well to seizure medications the studied patients with Dravet syndrome has sometimes been mistakenly identified a. Form of epilepsy that begins in the fight to cure Dravet syndrome Foundation ( DSF ) a. Based in the fight to cure Dravet syndrome has sometimes been mistakenly identified as a " vaccine encephalopathy because! Processed through Google voice and then distributed to staff members, so may. May take 24-48 hours for a quicker response, please email info dravetfoundation.org... But that risk is still low every Day against Dravet had their first seizure a. Cure for Dravet syndrome Foundation, Dravet syndrome who had their first seizure after a vaccination review! In an otherwise healthy child is a volunteer run, non-profit organization based in the fight to cure syndrome. Mistakenly identified as a research grant seizure after a vaccination carried an SCN1A mutation are hard control. Some of these challenges is very difficult to treat with anticonvulsant medications.It often before... Processed through Google voice and then distributed to staff members, so it may take 24-48 hours for a.... Respond well to seizure medications quicker response, please email info @ dravetfoundation.org a strong European community of people with... Resources for Patient Families, Dravet syndrome Foundation, Dravet syndrome Foundation ( )!
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dravet syndrome foundation 2021


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Q: Amazon Elastic Beanstalk RefineryCMS I've just started using Amazon Elastic Beanstalk. I can deploy a new rails 4.2.5 rails app no problem. I have a problem when deploying a brand new RefineryCMS.
Here are the commands I ran in order;
refinerycms refinerycms_aws_test
cd refinerycms_aws_test

add 'sqlite3' to the Gemfile
git init
g ca -m 'initial commit'
eb init

Answers to the questions;

*

*Server: Select 7 (Sydney)

*Application: Create new, "refinerycms"

*Ruby? yes

*Platform: 2 - (Ruby 2.1 Puma)

Now create the environment
eb create refinerycms-env

Set the secret token
eb setenv SECRET_KEY_BASE=27b39f037d544efc4671c707f68e54abcc6a61fa24a2664f0e4ae2871a932beb884288981eefcd4259a8c325ee44335231db04d6f58c8a75d3ac8d84e6e0dcf5
eb setenv SECRET_TOKEN=27b39f037d544efc4671c707f68e54abcc6a61fa24a2664f0e4ae2871a932beb884288981eefcd4259a8c325ee44335231db04d6f58c8a75d3ac8d84f6e0dce5

The error I get is 'page not found 404' here is what the eb logs command gives;
-------------------------------------
/var/log/puma/puma.log
-------------------------------------
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/railties-4.2.5/lib/rails/application.rb:246:in `env_config'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/railties-4.2.5/lib/rails/engine.rb:514:in `call'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/railties-4.2.5/lib/rails/application.rb:165:in `call'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/puma-2.10.2/lib/puma/configuration.rb:74:in `call'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/puma-2.10.2/lib/puma/server.rb:492:in `handle_request'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/puma-2.10.2/lib/puma/server.rb:363:in `process_client'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/puma-2.10.2/lib/puma/server.rb:254:in `block in run'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/puma-2.10.2/lib/puma/thread_pool.rb:101:in `call'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/puma-2.10.2/lib/puma/thread_pool.rb:101:in `block in spawn_thread'
2016-02-02 03:21:37 +0000: Rack app error: #<RuntimeError: Missing `secret_token` and `secret_key_base` for 'production' environment, set these values in `config/secrets.yml`>
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/railties-4.2.5/lib/rails/application.rb:534:in `validate_secret_key_config!'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/railties-4.2.5/lib/rails/application.rb:246:in `env_config'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/railties-4.2.5/lib/rails/engine.rb:514:in `call'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/railties-4.2.5/lib/rails/application.rb:165:in `call'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/puma-2.10.2/lib/puma/configuration.rb:74:in `call'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/puma-2.10.2/lib/puma/server.rb:492:in `handle_request'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/puma-2.10.2/lib/puma/server.rb:363:in `process_client'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/puma-2.10.2/lib/puma/server.rb:254:in `block in run'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/puma-2.10.2/lib/puma/thread_pool.rb:101:in `call'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/puma-2.10.2/lib/puma/thread_pool.rb:101:in `block in spawn_thread'
2016-02-02 03:21:55 +0000: Rack app error: #<RuntimeError: Missing `secret_token` and `secret_key_base` for 'production' environment, set these values in `config/secrets.yml`>
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/railties-4.2.5/lib/rails/application.rb:534:in `validate_secret_key_config!'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/railties-4.2.5/lib/rails/application.rb:246:in `env_config'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/railties-4.2.5/lib/rails/engine.rb:514:in `call'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/railties-4.2.5/lib/rails/application.rb:165:in `call'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/puma-2.10.2/lib/puma/configuration.rb:74:in `call'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/puma-2.10.2/lib/puma/server.rb:492:in `handle_request'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/puma-2.10.2/lib/puma/server.rb:363:in `process_client'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/puma-2.10.2/lib/puma/server.rb:254:in `block in run'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/puma-2.10.2/lib/puma/thread_pool.rb:101:in `call'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/puma-2.10.2/lib/puma/thread_pool.rb:101:in `block in spawn_thread'
2016-02-02 03:21:55 +0000: Rack app error: #<RuntimeError: Missing `secret_token` and `secret_key_base` for 'production' environment, set these values in `config/secrets.yml`>
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/railties-4.2.5/lib/rails/application.rb:534:in `validate_secret_key_config!'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/railties-4.2.5/lib/rails/application.rb:246:in `env_config'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/railties-4.2.5/lib/rails/engine.rb:514:in `call'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/railties-4.2.5/lib/rails/application.rb:165:in `call'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/puma-2.10.2/lib/puma/configuration.rb:74:in `call'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/puma-2.10.2/lib/puma/server.rb:492:in `handle_request'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/puma-2.10.2/lib/puma/server.rb:363:in `process_client'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/puma-2.10.2/lib/puma/server.rb:254:in `block in run'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/puma-2.10.2/lib/puma/thread_pool.rb:101:in `call'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/puma-2.10.2/lib/puma/thread_pool.rb:101:in `block in spawn_thread'
2016-02-02 03:21:55 +0000: Rack app error: #<RuntimeError: Missing `secret_token` and `secret_key_base` for 'production' environment, set these values in `config/secrets.yml`>
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/railties-4.2.5/lib/rails/application.rb:534:in `validate_secret_key_config!'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/railties-4.2.5/lib/rails/application.rb:246:in `env_config'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/railties-4.2.5/lib/rails/engine.rb:514:in `call'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/railties-4.2.5/lib/rails/application.rb:165:in `call'
/opt/rubies/ruby-2.1.7/lib/ruby/gems/2.1.0/gems/puma-2.10.2/lib/puma/configuration.rb:74:in `call'

It's complaining about the secret_token and secret_key_base but I've set both of these. They show up when I use eb printenv
UPDATE
Here is my secrets.yml file
# Do not keep production secrets in the repository,
# instead read values from the environment.
production:
  secret_key_base: <%= ENV["SECRET_KEY_BASE"] %>
  secret_token: <%= ENV["SECRET_TOKEN"] %>

I've set both of these through the eb tool.
UPDATE
I added ssh to my elastic beanstalk environment and logged in. I did a env command and found that my SECRET environment variables are listed there. So they are getting set.

redpajama_set_name: RedPajamaStackExchange


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The dream that fuels unbeaten prospect Sikho Nqothole is to become a champion so he can buy a house for his long-lost mother who walked out of the family home in Mthatha when he was just a teenager.
Nqothole' with nine victories in the paid ranks' takes on the vastly more experienced Tanzanian' Sunday Kiwale' for the vacant WBA Pan African junior-bantamweight title at the Sibaya casino on the KwaZulu-Natal north coast Wednesday.
The longest Nqothole has gone since turning professional under Johannesburg-based trainer Alan Toweel junior in March last year is six rounds — on three occasions — but on Wednesday his bout is scheduled for 12 rounds.
But that's not a problem for Nqothole' who trains hard.
He was like lightning on his last' hard road run up the merciless Northcliff hill' and he still had enough left over to do sprints up a long' steep driveway.
We don't know where she is' if she's even alive or dead'" said the fighter. When I got home that day — the worst memory of my life — that's when I realised I have two options' school and boxing. I knew I must take them with both hands.
"That's why I want to work hard'" Nqothole said.
Nqothole was 16 in 2010 when he returned from holiday to the home he had shared with his father' three sisters and brother to find his mother had left.
"We don't know where she is' if she's even alive or dead'" said the fighter.
Nqothole says he won eight national titles as an amateur' four at age-group levels and four at senior.
He was one of two local boxers to secure a spot on the 2016 Rio Olympic roster by winning gold at the African qualifying tournament' only to learn that the SA Sports Confederation and Olympic Committee (Sascoc) was not accepting continental qualifiers.
He and other SA boxers then had to try qualify at an international tournament in Azerbaijan' but none succeeded.
That's when he decided to join the paid ranks.
An experienced boxer' he doesn't get nervous before a fight' but he does get worried while preparing for them.
"I put pressure on myself to be fit.
Nqothole has boxed a total of 30 rounds in his nine paid bouts' compared to his opponent's 114 from 13 wins' nine losses and four draws.
"He's a puncher'" Nqothole says of his opponent.
And it could take him one step closer to his dream.


Meerkats. These adorable, furry animals are well known for their role in the BBC produced animated film, The Meerkats. What's more, meerkats are also often used as model animals for their altruistic behavior.
These animals are famously known to stand guard while other members of their gang forage for food. If they detect any threats, the sentinels call out to the rest of the meerkats, which then run to nearby hiding places. It seems that they are risking their lives to protect their groups. However, a 1999 report showed that guards are the first to flee after sounding an alarm and that sentinels, in fact, are positioned such that they have the most time to reach safety. Therefore, what does altruism really mean and do meerkats have a hidden agenda?
Altruism is defined by Daniel Batson as "a motivational state with the ultimate goal of increasing another's welfare." This is differs from egoism, the ultimate goal of which is individual benefit. It seems that the meerkats who stand guard as sentinels might therefore be acting with egoistic rather than altruistic motivation. But what about humans? Can they ever be truly altruistic? Does the Good Samaritan, a person who helps others without an expectation of a reward, exist?
There are several theories that explain prosocial and altruistic behavior in humans. One such model is the Empathy-Altruism hypothesis developed by Daniel Batson. According to this theory, when a person sees another person in need, they might help the other person either to reduce their own distress or if they feel they might be rewarded for their service. There is a third possibility, however. They may feel empathetic towards the person in need, and in that state they are willing to help the other person regardless of what they might gain. Reducing the other person's suffering becomes the most important goal, indicating that it is possible for humans to behave in a truly altruistic manner.
On the other hand, evolutionary theories on altruism suggest that humans do behave selfishly, even when helping another person. For example, according to the Kin Selection hypothesis, humans help others, especially their descendants. Although they may be reducing the chances of their own survival, they are increasing the probability of their genes being passed on to the next generation. Moreover, the degree of helping behavior increases if the concerned individuals are closely related. It is therefore also not surprising that people are more willing to help those whom they perceive to be more similar to rather than different from themselves.
However, humans have behaved altruistically and have helped complete strangers in the past. For example, Patrick Morgan risked his life by jumping down under a stationary train in Sydney, Australia to save an elderly woman who had fallen there. But he said that he was simply doing what he thought anyone else would do. Similarly, Vishnu Zende, the railway announcer at Chhatrapati Shivaji Terminus saved hundreds of lives by alerting commuters to leave the station during the 26/11 terrorist attacks in Mumbai.
I guess the Good Samaritan does exist. And in any case, helping others is always good, even if you do it to help yourself.
Free Will: Is this the real life… Is this just fantasy?
When you were getting ready in the morning, did you choose the colour of the shirt you would wear today? Do you arbitrarily choose what dish you want to order at a restaurant? Did you voluntarily choose the subject of your undergraduate degree?
All these questions refer to the choice we have, while making decisions in life. The ability to make that choice is what we call free will. Free will is the ability of an agent to select an option (a behavior, an object, a course etc.) from a set of alternatives (Mick, 2008) .
Most people believe that they have free will, and they control their decisions. However, many psychologists and philosophers refute the idea of free will with the idea of determinism. Philosophy defines determinism as a notion that every event or state of affairs, including every human decision and action, is the inevitable and necessary consequence of the antecedent states of affairs. Putting it in simpler terms- all our actions are pre-determined, and we do not really have the freedom of 'choosing what we want to do'. Daniel Wegner, in his book The Illusion of Conscious Will (2002), states that free will is just an illusion, and we attribute it to be the cause of events, whose actual causes we don't really understand.
To test whether people really have free will, Benjamin Libet (1982) conducted an experiment, where he measured cerebral activity, and found out that freely, voluntary acts were preceded by a specific electrical change (readiness potential 'RP') in the brain that began about 550 ms before the act. Human subjects became aware of the intention to act 350–400 ms after the RP began, but 200 ms before the motor act. He therefore concluded that the volitional process was initiated unconsciously (through a set of neurological functions), but the conscious function could still control the outcome. Hence, even though his experiments indicated that the choice made by people was not random, but predetermined, the existence of free will could not be completely eliminated.
Furthermore, even in the field of criminology, there is a long standing argument that we do not have free will; therefore, a person's criminal behavior is determined by environmental, biological and social factors. Many have used this argument in the court of law to justify transgressions. But arguments of this nature haven't found many takers in the legal system. For instance, American courts are not likely to warm up to the idea of genetic causes behind crime, and tend to stress on the fact the people have the free will to choose between right and wrong (Jones, 2003). Hence, even within the justice system, there is no clear acceptance or rejection of free will.
But what if we do indeed make choices consciously, and only come to know about them much later? Does that mean free will really exists, but we all have misinterpreted how it exactly happens? Holton (2004), in his review Wegner's book The Illusion of Conscious Will (2002) stated that precursor events might be genuinely mental events of which the subject is not aware about until later. This leads to a whole different set of ideas about how free will operates.
People have argued strongly for and against free will, but none seem to have reached a consensus about it. One of the oldest questions to plague the field of psychology still remains unresolved. When I decided to write about this topic, was it my freedom of choice or was it some predetermined set of biological and neurological functions?


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Thomaidou E, Katz M, Leibovici V. Two cases of disseminated superfi cial actinic porokeratosis (DSAP) and treatment literature review. Our Dermatol Online. 2018;9(3):241-248.
Background: Disseminated superficial actinic porokeratosis (DSAP) is characterized by asymptomatic multiple papules with annular keratotic rim distributed symmetrically on the sun-exposed areas. Many approaches have been proposed in the past for treating DSAP, but the therapy is still a challenge for every physician, mostly because of the frequent relapses of the disease and the multiplicity of the skin lesions.
Material and Methods: Two case reports were reported emphasizing the challenges of treatment approach. Therefore, a systemic English literature review was conducted searching Medline database using PubMed Central and Ovid software as search interface to collect evidence based on the various treatment modalities for DSAP.
Results: The initial search yielded 146 articles, but only the relevant case reports, case series and studies relating to the treatment of DSAP have been described and summarized in a table. For each different therapy the efficacy of each treatment, side effects, cost-effectiveness and authors' recommendations were reported.
Conclusion: Several factors need to be considered prior to physicians' decision of the most appropriate treatment for each patient like age, the extent of body surface area involvement, patients' medical history and social situation, the available resources, the side effects and cost- effectiveness of each treatment. However, l the exact value of each treatment is difficult to determine owing to the lack of controlled studies evaluating their efficacy. Due to few incidences of squamous cell carcinoma, patients need to be followed up and monitored closely for any early detection of recurrence or possible onset of malignancies.
Disseminated superficial actinic porokeratosis (DSAP) was first described by Chernosky and Freeman . Clinically, DSAP is characterized by asymptomatic multiple papules with annular keratotic rim distributed symmetrically on the sun-exposed areas, and facial involvement is a rare presentation1. Malignant degeneration has been described in 7% to 11% of porokeratoses cases, with squamous cell carcinoma being the most common [2–4]. Histological hallmark is the cornoid lamella, which is formed by clonal hyperproliferation of atypical keratinocytes . Underlying the cornoid lamella, the granular layer is thinned or absent and keratinocytes are oedematous with spongiosis, and dermal lymphocytic infiltrate may also be evident . Dermoscopy examination demonstrate single or double "white track" structure at the margin corresponding to the cornoid lamella, and the red dots, globules, and lines are enlarged capillary vessels that can be observed because the epithelium is atrophic .
Xia and colleagues using a genomewide search in a large Chinese family, identified a locus at chromosome 12q23.2-24. 1 responsible for disseminated superficial actinic porokeratosis . Therefore, DSAP is an inherited dermatologic disorder with lesions appearing in genetically predisposed individuals after adequate exposure to ultraviolet radiation or immunosuppression in the third and fourth decades of life .
A variety of approaches have been proposed to treat DSAP, but the therapy is still a challenge for every physician, mostly because of the multiplicity of the skin lesions and the frequent relapses of the disease . Herein, we present two case reports with a great dilemma in treatment approach, following by an update literature review.
Case 1: An eighteen four-year-old man presented in our clinic with 6-month history of bilateral symmetrical erythematous skin lesions on the lower and upper extremities. The skin eruptions were associated with photosensitivity and moderate pruritus. The patient had a background medical history of hypercholesteremia, hypertension, ischemic heart disease, congestive heart disease, diabetes mellitus type 2, non-alcoholic fatty liver, chronic renal failure and glaucoma.
On examination bilateral erythematous scaly papules with annular configuration, well –demarcated borders and central atrophy were seen on the shins, thighs and forearm (Figs. 1a and 1b). Dermoscopy examination revealed a white like track structures at the periphery of the lesion with a mild hyperpigmentation in the inner side and with some red globules, and lines at the periphery (Fig. 2). On histological examination cornoid lamella was found in the stratum corneum with focal loss of granular layer, prominent lichenoid, superficial perivascular lymphocytic infiltrate and background elastosis (Figs. 3a and 3b).
Figure 1: (a-d) Bilateral erythematous scaly papules with annular configuration on the shins, thighs and forearm with well – demarcated borders and central atrophy.
Figure 2: A "white track" structures can be identified at the periphery of the lesion with a brownish pigmentation in the inner side and with some red globules, and lines at the periphery.
Figure 3: (a and b) Cornoid lamella was found in the stratum corneum with focal loss of granular layer, prominent lichenoid, superficial perivascular lymphocytic infiltrate and background elastosis.(Hematoxylin and Eosin, 40×, 400x).
Case 2: An eighteen one-year-old man with background history of diabetes mellitus type 2, bronchiectasis, asthma, ischemic heart disease, aortic stenosis and mitral insufficiency was examined in our clinic due to 3-year history of annular non-pruritic lesions on the shins. In the past, he was treated with steroid intralesional injections without any improvement.
On examination, there were multiple annular erythematous lesions with central clearing and elevated borders on the shins (Figs. 1c and 1d). Histological examination revealed cornoid lamella with absence of the granular layer. The intervening epidermis between the cornoid lamellae was thin and in the upper dermis there were perivascular mononuclear and lichenoid cells.
According to the clinical and histological presentation, both of our patients were diagnosed with disseminated superficial actinic porokeratosis. Their clinical presentation, risk of malignancy, medical history and their social situation was considered for choosing the most appropriate treatment for the patient.
A systemic English literature review was conducted in June 2017 searching Medline database using PubMed Central and Ovid software as search interface to collect evidence based on the various treatment modalities for DSAP. In addition, studies that have been commonly cited in the literature and review articles were included as citation search engine to identify subsequent publications, which were relevant for the literature review. The following medical terms and text world were used: "Disseminated superficial actinic porokeratosis", "porokeratosis", "laser", "photodynamic therapy", "diclofenac", "calcipotriol", "imiquimod", "retinoid", "photodynamic therapy", "lasers". The keywords were combined using multiple combinations. Articles that did not mention treatment approaches, which were not published in English or were not available, have not been included for the purposes of this literature review. Any discrepancies about data evaluation of the selected articles were resolved after discussion between the authors.
The initial search yielded 146 articles, but only the relevant case reports, case series and studies relating to the treatment of DSAP have been described below and summarized in Table 1. For each different therapy the efficacy of each treatment, side effects, cost effectiveness and authors' recommendations were reported.
Traditional topical treatment approaches include topical treatments like diclofenac [10–12], imiquimod [13–14] and calcipotriol [15–19]. Oral retinoids [20–22] and cryotherapy  were used in the past with no any satisfied results. Newer treatment of photodynamic therapy (PDT) [2,9,23–25] and lasers [3,26–32] have been introduced in the recent years with some desirables results. A literature review was conducted five years ago assessing the level of evidence for some therapeutic modalities. However, still the exact value of each treatment is difficult to determine owing to the lack of controlled studies evaluating their efficacy .
Several factors need to be considered for choosing the most appropriate treatment for each patient, like the age, the extent of body surface area involvement, patients' medical history and their social situation, the available resources in dermatology departments, the side effects and cost effectiveness of each treatment approaches. Systemic treatment might not be an option if patients have complicated medical background and take multiple medications, due to possible interactions of retinoids with their regular medications. Expensive treatment like different types of lasers can be discussed with the patients but usually are not considered due to the high cost and lack of experienced centres. PDT therapy showed poor outcome in several studies (e.g. six out of nine patients had none or minimal response with MAL- PDT, all patients had no response with ALA-PDT and Hypericin-PDT), thus it is not recommended frequently [2,9,23–33].
Following a thorough discussion among physicians in our clinic, both patients started on topical treatment with calcipotriol/betamethasone gel with partial resolution. Patients are under follow up and they will be monitored closely for any possible onset of malignancies. In conclusion, there are several options available for treating DSAP, but always the best approach should be tailored to every patient. There is still a great need for further controlled studies with a greater sample size to draw conclusions on the effects of these novel treatments for DSAP.
1. Chernosky ME, Freeman RG. Disseminated superficial actinic porokeratosis (DSAP). Arch Dermatol. 1967;96:611–24.
2. Salas T, Hernandez-Gil J, Lopez A, Dorado M, Ruiz J, Garcia E, et al. Two cases of disseminated superficial actinic porokeratosis treated with daylight-mediated photodynamic therapy. Dermatol Ther. 2016;29:484-5.
3. Lolis MS, Marmur ES. Treatment of disseminated superficial actinic porokeratosis (DSAP) with the Q-switched ruby laser. J Cosmet Laser Ther. 2008;10:124-7.
4. Maubec E, Duvillard P, Margulis A, Bachollet B, Degois G, Avril MF. Common skin cancers in porokeratosis. BrJ Dermatol. 2005;152:1389–91.
5. Skupsky H, Skupsky J, Goldenberg G. Disseminated superficial actinic porokeratosis:a treatment review. J Dermatolog Treat. 2012;23:52-6.
6. Zaballos P, Puig S, Malvehy J. Dermoscopy of disseminated superficial actinic porokeratosis. Arch of Dermatol. 2004;140:1410.
7. Xia JH, Yang YF, Deng H, Tang BS, Tang DS, He YG, et al. Identification of a locus for disseminated superficial actinic porokeratosis at chromosome 12q23.2-24.1. J Invest Dermatol. 2000;114:1071-4.
8. Aird GA, Sitenga JL, Nguyen AH, Vaudreuil, A, Huerter, CJ. Light and laser treatment modalities for disseminated superficial actinic porokeratosis:a systematic review. Lasers Med Sci. 2017;32:945-52.
9. Cavicchini S, Tourlaki A. Successful treatment of disseminated superficial actinic porokeratosis with methyl aminolevulinate-photodynamic therapy. J Dermatolog Treat. 2006;17:190-1.
10. Vlachou C, Kanelleas AI, Martin-Clavijo A, Berth-Jones J. Treatment of disseminated superficial actinic porokeratosis with topical diclofenac gel:a case series. J Eur Acad Dermatol Venereol. 2008;22:1343-5.
11. Marks S, Varma R, Cantrell W, Chen SC, Gold M, Muellenhoff M, et al. Diclofenac sodium 3% gel as a potential treatment for disseminated superficial actinic porokeratosis. J Eur Acad Dermatol Venereol. 2009;23:42-5.
12. Otero-Rivas MM, Rodriguez-Lojo R, Castineiras-Mato IM, Lueiro-Vilarino M, Juarez-Casado Y, Fernandez-Diaz ML. Successful management of disseminated superficial actinic porokeratosis with diclofenac sodium 3% gel. Dermatol Ther. 2017;30(3).
13. Arun B, Pearson J, Chalmers R. Disseminated superficial actinic porokeratosis treated effectively with topical imiquimod 5% cream. Clin Exp Dermatol. 2011;36:509-11.
14. Riad H, Mansour K, Sada HA, Shaika SA, Ansari HA, Mohannadi HA. Disseminated superficial actinic porokeratosis on the face treated with imiquimod 5% cream. Case Rep Dermatol. 2013;5:283-9.
15. Harrison PV, Stollery N. Disseminated superficial actinic porokeratosis responding to calcipotriol. Clin Exp Dermatol. 1994;19:95.
16. Bakardzhiev I, Kavaklieva S, Pehlivanov G.Successful treatment of disseminated superficial actinic porokeratosis with calcipotriol. Int J Dermatol. 2012;51:1139-42.
17. Bohm M, Luger TA, Bonsmann G. Disseminated superficial actinic porokeratosis:treatment with topical tacalcitol. J Am Acad Dermatol. 1999;40:479-80.
18. Nakamura Y, Yamaguchi M, Nakamura A, Muto M. Calcipotriol and adapalene therapy for disseminated superficial actinic porokeratosis. Indian J Dermatol Venereol Leprol. 2014;80:373-4.
19. Tchernev G, Chokoeva AA, Ivanova B, Mangarov H, Vidolova NG. Disseminated superficial actinic porokeratosis (DSAP):significant improvement after local administration of calcipotriol/betamethasone gel?". Wien Med Wochenschr. 2017;167:85-8.
20. Kariniemi AL, Stubb S, Lassus A. Treatment of disseminated superficial actinic porokeratosis with a new aromatic retinoid (Ro 10-9359). Br J Dermatol. 1980;102:213-4.
21. Ludera-Zimoch G, Rubisz-Brzezinska J. [A case of superficial disseminated actinic porokeratosis treated with an aromatic retinoid] Przegl Dermatol.1989;76:54-7.
22. Carmichael AJ, Tan CY. Digitate keratoses–a complication of etretinate used in the treatment of disseminated superficial actinic porokeratosis. Clin Exp Dermatol. 1990;15:370-1.
23. Fernandez-Guarino M, Harto A, Perez-Garcia B, Martin-Gonzalez M, Urrutia S, Jaen P. Photodynamic therapy in disseminated superficial actinic porokeratosis. J Eur Acad Dermatol Venereol. 2009;23:176-7.
24. Nayeemuddin FA, Wong M, Yell J, Rhodes LE. Topical photodynamic therapy in disseminated superficial actinic porokeratosis. Clin Exp Dermatol. 2002;27:703-6.
25. Boiy A, de Witte P. A, Roelandts R. Topical treatment of disseminated superficial actinic porokeratosis with hypericin-photodynamic therapy:a case report. Photodiagnosis Photodyn Ther. 2012;7:123-5.
26. Itoh M, Nakagawa H. Successful treatment of disseminated superficial actinic porokeratosis with Q-switched ruby laser. J Dermatol. 2007;34:816-20.
27. Liu HT. Treatment of lichen amyloidosis (LA) and disseminated superficial porokeratosis (DSP) with frequency-doubled Q-switched Nd:YAG laser. Dermatol Surg. 2000:26:958-62.
28. Rosenblum J. Erbium laser for treatment of disseminated superficialactinic porokeratosis. Dermatol Surg. 2013;39:1543-5.
29. Ross NA, Rosenbaum LE, Saedi N, Arndt KA, Dover JS. Disseminated superficial actinic porokeratosis improved with fractional 1927-nm laser treatments. J Cosmet Laser Ther. 2016;18:53-5.
30. Chrastil B, Glaich AS, Goldberg LH, Friedman PM. Fractional photothermolysis:a novel treatment for disseminated superficial actinic porokeratosis. Arch Dermatol. 2007;143:1450-2.
31. Noborio R, Morita A. Split-face trial of CO2 laser-induced ring abrasion and high-dose tacalcitol in the treatment of disseminated superficial actinic porokeratosis. J Dermatol. 2011;39:879–80.
32. Kim HS, Baek JH, Park YM, Kim HO, Lee JY. Photodynamic therapy combined with CO2 laser vaporization on disseminated superficial actinic porokeratosis:a report of 2 cases on the face. Ann Dermatol. 2011;23:211–3.
33. Ricci C, Rosset A, Panizzon RG. Bullous and pruritic variant of disseminated superficial actinic porokeratosis:successful treatment with Grenz rays. Dermatol. 1999;199:328–31.


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Another gain for US stocks, led by banks and tech companies
NEW YORK (AP) — U.S. stocks rose Wednesday as technology companies traded higher for the second day in a row and consumer companies gained steam as cruise lines rose. The beleaguered financial sector recovered some of its losses from earlier this year.
Stocks started the day sharply higher, following a jump in European markets, although they returned some of those gains as the day wore on. Oil prices followed the same pattern and finished a few cents higher. Cruise lines rose after Carnival posted strong first-quarter results, and the dollar weakened further. Stocks are up about 7 percent this month, part of a rally that has canceled out their losses from a disastrous start to the year.
"The Fed really cured a lot of global ills" by deciding to go slower in raising interest rates, said John Cannally, chief economic strategist for LPL Financial. The Federal Reserve announced that decision two weeks ago, and Fed Chair Janet Yellen emphasized it again in a speech Tuesday.
The Dow Jones industrial average rose 83.55 points, or 0.5 percent, to 17,716.66. The Standard & Poor's 500 index gained 8.94 points, or 0.4 percent, to 2,063.95. The Nasdaq composite index added 22.67 points, or 0.5 percent, to 4,869.29.
Banks and insurance companies made the biggest gains. MetLife gained $2.27, or 5.3 percent, to $44.73 after the company successfully challenged its "too big to fail" designation. The Financial Stability Oversight Council had said MetLife needed greater government oversight because of its size and importance to the financial system, but the company took the council to court over that ruling. On Wednesday a judge ruled in its favor.
Fellow insurer AIG advanced $1.13, or 2.1 percent, to $54.52 and Prudential rose $1.43, or 2 percent, to $72.95. Banks also traded higher.
Apple led the gains among technology companies. Its stock rose $1.88, or 1.7 percent, to $109.56. The world's largest publicly traded company is at its highest price since mid-December. Visa gained $1.40, or 1.9 percent, to $76.78.
After two weeks of mixed trading, stocks started moving higher late Tuesday, when Federal Reserve Chair Janet Yellen said the central bank expects to proceed slowly in raising interest rates. The market is now on pace for its best month since October, and the Dow and S&P 500 are higher for the year. In February they were each down 10 percent.
"You really had a year's worth of volatility in one quarter and we're back where we started," said Cannally. Companies will start reporting their first-quarter earnings in two weeks, and Cannally said that may determine what the market does next.
Cruise line operator Carnival got a boost after its first-quarter results were better than analysts expected, and the company raised its profit projections for the year. The stock gained $2.73, or 5.5 percent, to $52.37 and competitor Royal Caribbean Cruises jumped $4.30, or 5.7 percent, to $80.35.
Railroad operator Norfolk Southern added $1.95, or 2.4 percent, to $84.75 after it said it is open to a possible sale to Canadian Pacific, but only if Canadian Pacific offers a better price and regulators approve the structure of the deal. Norfolk Southern has rejected three offers from Canadian Pacific worth about $30 billion. Rival CSX picked up 41 cents, or 1.6 percent, to $26.30.
U.S. companies added 200,000 jobs in March, according to a survey of private employers by ADP, a payroll processing company. The survey showed companies in construction, retail and shipping continued to bring on new workers, and it suggests hiring continues in the U.S. The federal government will release its monthly jobs report Friday.
Canadian yoga-wear company Lululemon gained $6.56, or 10.7 percent, to $67.80 after it disclosed strong fourth-quarter sales.
Acadia Pharmaceuticals rose after a Food and Drug Administration panel made a positive recommendation for Acadia's drug Nuplazid, which is intended to treat psychotic delusions and behaviors that harm patients with Parkinson's disease. The stock climbed $2.20, or 9.2 percent, to $26.01.
Payroll processor Paychex lost $1.20, or 2.2 percent, to $53.29 as investors were unimpressed with its fiscal third-quarter results. The stock has climbed over the last two months and is near all-time highs.
U.S. crude rose four cents to $38.32 a barrel in New York. Brent crude, the benchmark for international oil prices, rose 12 cents to $39.26 a barrel in London.
The dollar continued to weaken. The euro rose to $1.1333 from $1.1295 and the dollar fell to 112.47 yen from 112.75 yen. Bond prices slipped, returning some of Tuesday's gains. The yield on the 10-year U.S. Treasury note rose to 1.83 percent from 1.80 percent.
France's CAC-40 climbed 1.8 percent, while Germany's DAX and the FTSE 100 in Britain each rose 1.6 percent. Asian stocks were mixed after the Asian Development Bank said economies in the region will grow at a slower pace in 2016 and 2017 because of reduced growth in China and a weak recovery in other major industrial economies. Tokyo's Nikkei 225 lost 1.3 percent as the yen continued to strengthen. Hong Kong's Hang Seng index climbed 2.1 percent. South Korea's KOSPI rose 0.4 percent.
Gold fell $8.90 to $1,226.90 an ounce. Silver slipped two cents to $15.21 an ounce, while copper fell two cents to $2.19 a pound.
In other energy trading, wholesale gasoline fell 2 cent to $1.44 a gallon. Heating oil was little changed at $1.16 a gallon. Natural gas climbed 2 cents to $2 per 1,000 cubic feet.


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Leather folio M fits IQOS 2.4 and IQOS 2.4 Plus.


From public relations to patriotism, we are grateful for the national and local recognition for our support of Montgomery County. Here at Valley Forge Tourism and Convention Board, our awards are Montco's awards.
Recognition to Tomlinson for her ongoing and enthusiastic support of the business community in King of Prussia, Pa.
Recognition for efforts welcoming the World Meeting of Families and the visit of Pope Francis, September 2015, under the branding "Home Away from Rome." The tagline encapsulated the Pontiff's overnight stay in the Wynnewood section of the county.
Subsequent recognition to Tallman by the Board of Directors of the Valley Forge Tourism & Convention Board for her overall excellence and dedication to improving the visitor experience in Montgomery County, Pa.


Comentado: beautiful beautiful homeThank you ! The floors are French white oak. They are from a company called Legno Baston. They are 10" wide planks . the finish is called Guioppina. Hope tis helps !
BCB Decor ha respondido una pregunta: Love those coffee tables - can you share where these are from? Thanks!
BCB Decor ha respondido una pregunta: Beautiful and spacious.


This is a quote from Karen (not her real name), a woman who attends one of our support groups for people with depression. When I am asked about the work we do at the Mental Health Association in Delaware (MHA), I often think of people like her.
Fortunately for people like Karen, MHA is making a difference. For over 78 years, MHA has been providing support, education and advocacy. We now have fifteen support groups, offer a variety of trainings, and continue to advocate for laws and policies that support those living with mental illness.
We are also continuing our vital work in the area of suicide prevention, this year focusing on our youth in middle schools, where suicides have tripled in the last three decades. All of this could not be accomplished without your support.
Your generous donation today will allow us to reach out to more people needing our help and continue to provide the life saving programs and services to those currently trying to find their way back to 'normalcy,' like Karen. I ask you to dig deeply into your pocket this year as we try to save so many lives at risk due to depression or other mental illness. Simply click here to donate online.


text:
package graph;

import java.io.BufferedReader;
import java.io.FileReader;
import java.util.*;

public class UnitedStates {

  // set the path of the file with the graph information here
  public static String FILE_PATH = "src/graph/usa.txt";

  public HashMap<String, State> states;
  public Graph graph;

  public UnitedStates() {
    states = new HashMap<String, State>();
    graph = new Graph();
    loadGraph();
  }

  public Graph getGraph() {
    return graph;
  }

  public void setStart(String stateName) {
    states.get(stateName).setDistance(0);
  }

  public State getState(String stateName) {
    return states.get(stateName);
  }

  private void loadGraph() {
    loadStates();
    loadEdges();
  }

  private void loadStates() {
    try {
      BufferedReader br = new BufferedReader(new FileReader(FILE_PATH));
      String line;
      while ((line = br.readLine()) != null) {
        String sName = line.substring(0, line.indexOf("-"));
        State s = new State(sName);
        states.put(sName, s);
        graph.addNode(s);
      }
      br.close();
    } catch(Exception e) {
      System.out.println(e.getMessage());
    }
  }

  private void loadEdges() {
    try {
      BufferedReader br = new BufferedReader(new FileReader(FILE_PATH));
      String line;
      while ((line = br.readLine()) != null) {
        String s1 = line.substring(0, line.indexOf("-"));
        String[] adjacent = line.substring(
          line.indexOf("-") + 1).split(",");
        for (String s : adjacent) {
          String s2 = s.substring(0, s.indexOf("("));
          String cost = s.substring(s.indexOf("(") + 1, s.indexOf(")"));
          graph.addEdge(
            states.get(s1),
            states.get(s2),
            Integer.parseInt(cost));
        }
      }
      br.close();
    } catch(Exception e) {
      System.out.println(e);
    }
  }

  public static void main(String[] args) {

    String start = "Massachusetts";
    String end = "California";

    UnitedStates us = new UnitedStates();
    us.setStart(start);
    Dijkstra d = new Dijkstra(us.getGraph(), us.getState(end));
    d.run();

    List<State> path = new ArrayList<State>();
    State state = us.getState(end);

    while (state.previous() != null) {
      path.add(state);
      state = state.previous();
    }
    path.add(state);
    Collections.reverse(path);
    System.out.println("Shortest Path: " + path);
    System.out.println("Distance (in miles): " + us.getState(end).distance());

  }
}

redpajama_set_name: RedPajamaGithub


text:
/**
 * okaidia theme for JavaScript, CSS and HTML
 * Loosely based on Monokai textmate theme by http://www.monokai.nl/
 * @author ocodia
 */

code[class*="language-"],
pre[class*="language-"] {
	color: #f8f8f2;
	background: none;
	text-shadow: 0 1px rgba(0, 0, 0, 0.3);
	font-family: Consolas, Monaco, 'Andale Mono', 'Ubuntu Mono', monospace;
	text-align: left;
	white-space: pre;
	word-spacing: normal;
	word-break: normal;
	word-wrap: normal;
	line-height: 1.5;

	-moz-tab-size: 4;
	-o-tab-size: 4;
	tab-size: 4;

	-webkit-hyphens: none;
	-ms-hyphens: none;
	hyphens: none;
}

/* Code blocks */
pre[class*="language-"] {
	padding: 1em;
	margin: .5em 0;
	overflow: auto;
	border-radius: 0.3em;
}

:not(pre) > code[class*="language-"],
pre[class*="language-"] {
	background: #272822;
}

/* Inline code */
:not(pre) > code[class*="language-"] {
	padding: .1em;
	border-radius: .3em;
	white-space: normal;
}

.token.comment,
.token.prolog,
.token.doctype,
.token.cdata {
	color: slategray;
}

.token.punctuation {
	color: #f8f8f2;
}

.namespace {
	opacity: .7;
}

.token.property,
.token.tag,
.token.constant,
.token.symbol,
.token.deleted {
	color: #f92672;
}

.token.boolean,
.token.number {
	color: #ae81ff;
}

.token.selector,
.token.attr-name,
.token.string,
.token.char,
.token.builtin,
.token.inserted {
	color: #a6e22e;
}

.token.operator,
.token.entity,
.token.url,
.language-css .token.string,
.style .token.string,
.token.variable {
	color: #f8f8f2;
}

.token.atrule,
.token.attr-value,
.token.function {
	color: #e6db74;
}

.token.keyword {
	color: #66d9ef;
}

.token.regex,
.token.important {
	color: #fd971f;
}

.token.important,
.token.bold {
	font-weight: bold;
}
.token.italic {
	font-style: italic;
}

.token.entity {
	cursor: help;
}
/*! normalize.css v5.0.0 | MIT License | github.com/necolas/normalize.css */

/**
 * 1. Change the default font family in all browsers (opinionated).
 * 2. Correct the line height in all browsers.
 * 3. Prevent adjustments of font size after orientation changes in
 *    IE on Windows Phone and in iOS.
 */

/* Document
   ========================================================================== */

html {
  font-family: sans-serif; /* 1 */
  line-height: 1.15; /* 2 */
  -ms-text-size-adjust: 100%; /* 3 */
  -webkit-text-size-adjust: 100%; /* 3 */
}

/* Sections
   ========================================================================== */

/**
 * Remove the margin in all browsers (opinionated).
 */

body {
  margin: 0;
}

/**
 * Add the correct display in IE 9-.
 */

article,
aside,
footer,
header,
nav,
section {
  display: block;
}

/**
 * Correct the font size and margin on `h1` elements within `section` and
 * `article` contexts in Chrome, Firefox, and Safari.
 */

h1 {
  font-size: 2em;
  margin: 0.67em 0;
}

/* Grouping content
   ========================================================================== */

/**
 * Add the correct display in IE 9-.
 * 1. Add the correct display in IE.
 */

figcaption,
figure,
main { /* 1 */
  display: block;
}

/**
 * Add the correct margin in IE 8.
 */

figure {
  margin: 1em 40px;
}

/**
 * 1. Add the correct box sizing in Firefox.
 * 2. Show the overflow in Edge and IE.
 */

hr {
  box-sizing: content-box; /* 1 */
  height: 0; /* 1 */
  overflow: visible; /* 2 */
}

/**
 * 1. Correct the inheritance and scaling of font size in all browsers.
 * 2. Correct the odd `em` font sizing in all browsers.
 */

pre {
  font-family: monospace, monospace; /* 1 */
  font-size: 1em; /* 2 */
}

/* Text-level semantics
   ========================================================================== */

/**
 * 1. Remove the gray background on active links in IE 10.
 * 2. Remove gaps in links underline in iOS 8+ and Safari 8+.
 */

a {
  background-color: transparent; /* 1 */
  -webkit-text-decoration-skip: objects; /* 2 */
}

/**
 * Remove the outline on focused links when they are also active or hovered
 * in all browsers (opinionated).
 */

a:active,
a:hover {
  outline-width: 0;
}

/**
 * 1. Remove the bottom border in Firefox 39-.
 * 2. Add the correct text decoration in Chrome, Edge, IE, Opera, and Safari.
 */

abbr[title] {
  border-bottom: none; /* 1 */
  text-decoration: underline; /* 2 */
  text-decoration: underline dotted; /* 2 */
}

/**
 * Prevent the duplicate application of `bolder` by the next rule in Safari 6.
 */

b,
strong {
  font-weight: inherit;
}

/**
 * Add the correct font weight in Chrome, Edge, and Safari.
 */

b,
strong {
  font-weight: bolder;
}

/**
 * 1. Correct the inheritance and scaling of font size in all browsers.
 * 2. Correct the odd `em` font sizing in all browsers.
 */

code,
kbd,
samp {
  font-family: monospace, monospace; /* 1 */
  font-size: 1em; /* 2 */
}

/**
 * Add the correct font style in Android 4.3-.
 */

dfn {
  font-style: italic;
}

/**
 * Add the correct background and color in IE 9-.
 */

mark {
  background-color: #ff0;
  color: #000;
}

/**
 * Add the correct font size in all browsers.
 */

small {
  font-size: 80%;
}

/**
 * Prevent `sub` and `sup` elements from affecting the line height in
 * all browsers.
 */

sub,
sup {
  font-size: 75%;
  line-height: 0;
  position: relative;
  vertical-align: baseline;
}

sub {
  bottom: -0.25em;
}

sup {
  top: -0.5em;
}

/* Embedded content
   ========================================================================== */

/**
 * Add the correct display in IE 9-.
 */

audio,
video {
  display: inline-block;
}

/**
 * Add the correct display in iOS 4-7.
 */

audio:not([controls]) {
  display: none;
  height: 0;
}

/**
 * Remove the border on images inside links in IE 10-.
 */

img {
  border-style: none;
}

/**
 * Hide the overflow in IE.
 */

svg:not(:root) {
  overflow: hidden;
}

/* Forms
   ========================================================================== */

/**
 * 1. Change the font styles in all browsers (opinionated).
 * 2. Remove the margin in Firefox and Safari.
 */

button,
input,
optgroup,
select,
textarea {
  font-family: sans-serif; /* 1 */
  font-size: 100%; /* 1 */
  line-height: 1.15; /* 1 */
  margin: 0; /* 2 */
}

/**
 * Show the overflow in IE.
 * 1. Show the overflow in Edge.
 */

button,
input { /* 1 */
  overflow: visible;
}

/**
 * Remove the inheritance of text transform in Edge, Firefox, and IE.
 * 1. Remove the inheritance of text transform in Firefox.
 */

button,
select { /* 1 */
  text-transform: none;
}

/**
 * 1. Prevent a WebKit bug where (2) destroys native `audio` and `video`
 *    controls in Android 4.
 * 2. Correct the inability to style clickable types in iOS and Safari.
 */

button,
html [type="button"], /* 1 */
[type="reset"],
[type="submit"] {
  -webkit-appearance: button; /* 2 */
}

/**
 * Remove the inner border and padding in Firefox.
 */

button::-moz-focus-inner,
[type="button"]::-moz-focus-inner,
[type="reset"]::-moz-focus-inner,
[type="submit"]::-moz-focus-inner {
  border-style: none;
  padding: 0;
}

/**
 * Restore the focus styles unset by the previous rule.
 */

button:-moz-focusring,
[type="button"]:-moz-focusring,
[type="reset"]:-moz-focusring,
[type="submit"]:-moz-focusring {
  outline: 1px dotted ButtonText;
}

/**
 * Change the border, margin, and padding in all browsers (opinionated).
 */

fieldset {
  border: 1px solid #c0c0c0;
  margin: 0 2px;
  padding: 0.35em 0.625em 0.75em;
}

/**
 * 1. Correct the text wrapping in Edge and IE.
 * 2. Correct the color inheritance from `fieldset` elements in IE.
 * 3. Remove the padding so developers are not caught out when they zero out
 *    `fieldset` elements in all browsers.
 */

legend {
  box-sizing: border-box; /* 1 */
  color: inherit; /* 2 */
  display: table; /* 1 */
  max-width: 100%; /* 1 */
  padding: 0; /* 3 */
  white-space: normal; /* 1 */
}

/**
 * 1. Add the correct display in IE 9-.
 * 2. Add the correct vertical alignment in Chrome, Firefox, and Opera.
 */

progress {
  display: inline-block; /* 1 */
  vertical-align: baseline; /* 2 */
}

/**
 * Remove the default vertical scrollbar in IE.
 */

textarea {
  overflow: auto;
}

/**
 * 1. Add the correct box sizing in IE 10-.
 * 2. Remove the padding in IE 10-.
 */

[type="checkbox"],
[type="radio"] {
  box-sizing: border-box; /* 1 */
  padding: 0; /* 2 */
}

/**
 * Correct the cursor style of increment and decrement buttons in Chrome.
 */

[type="number"]::-webkit-inner-spin-button,
[type="number"]::-webkit-outer-spin-button {
  height: auto;
}

/**
 * 1. Correct the odd appearance in Chrome and Safari.
 * 2. Correct the outline style in Safari.
 */

[type="search"] {
  -webkit-appearance: textfield; /* 1 */
  outline-offset: -2px; /* 2 */
}

/**
 * Remove the inner padding and cancel buttons in Chrome and Safari on macOS.
 */

[type="search"]::-webkit-search-cancel-button,
[type="search"]::-webkit-search-decoration {
  -webkit-appearance: none;
}

/**
 * 1. Correct the inability to style clickable types in iOS and Safari.
 * 2. Change font properties to `inherit` in Safari.
 */

::-webkit-file-upload-button {
  -webkit-appearance: button; /* 1 */
  font: inherit; /* 2 */
}

/* Interactive
   ========================================================================== */

/*
 * Add the correct display in IE 9-.
 * 1. Add the correct display in Edge, IE, and Firefox.
 */

details, /* 1 */
menu {
  display: block;
}

/*
 * Add the correct display in all browsers.
 */

summary {
  display: list-item;
}

/* Scripting
   ========================================================================== */

/**
 * Add the correct display in IE 9-.
 */

canvas {
  display: inline-block;
}

/**
 * Add the correct display in IE.
 */

template {
  display: none;
}

/* Hidden
   ========================================================================== */

/**
 * Add the correct display in IE 10-.
 */

[hidden] {
  display: none;
}
.app-container{
  display: -webkit-box;
  display: -ms-flexbox;
  display: flex;
  min-height: 100vh;
  -webkit-box-orient: vertical;
  -webkit-box-direction: normal;
      -ms-flex-direction: column;
          flex-direction: column;

}
.app-container .navbar{
  padding: 20px;
  background-color: #333;
  color: #fff;

}
.app-container .main-content{
  -webkit-box-flex: 1;
  -ms-flex: 1;
  flex: 1;
  height: 100%;
  display: -webkit-box;
  display: -ms-flexbox;
  display: flex;

}
.app-container .panel-section{
  width: 480px;
  padding: 20px;
  background-color: #eee;

}
.app-container .code-generator-section{
  padding: 20px;

}
.app-container .type-group{
  margin-bottom: 40px;

}
.app-container .group-title{
  font-size: 16px;
  margin-bottom: 8px;

}
.app-container .btn-group{
  display: -webkit-box;
  display: -ms-flexbox;
  display: flex;

}
.app-container .btn{
  position: relative;
  display: inline-block;
  padding: 6px 12px;
  margin-bottom: 0;
  font-weight: 400;
  text-align: center;
  vertical-align: middle;
  cursor: pointer;
  background-image: none;
  border: 1px solid #ddd;
  background-color: #fff;
  white-space: nowrap;
  font-size: 14px;
  line-height: 1.42857143;
  border-radius: 5px;
  -webkit-user-select: none;
  -moz-user-select: none;
  -ms-user-select: none;
  user-select: none;
  text-decoration: none;
  color: #333;

}
.app-container .btn.enabled{
  border: 1px solid #ccc;
  background-color: #ddd;
  color: #222;

}
.app-container .btn-group{
  display: -webkit-box;
  display: -ms-flexbox;
  display: flex;
  -ms-flex-wrap: wrap;
  flex-wrap: wrap;

}
.app-container .btn-group .btn{
  text-align: left;
  margin-bottom: 4px;
  margin-right: 4px;

}
.app-container .code-generator-section{
  display: -webkit-box;
  display: -ms-flexbox;
  display: flex;
  -webkit-box-flex: 1;
  -ms-flex: 1;
  flex: 1;
  position: relative;
  height: 100%;

}
.app-container .code-snippet{
  -webkit-box-flex: 1;
  -ms-flex: 1;
  flex: 1;
  max-width: 100%;
  margin: 0;
  height: 100%;
  padding: 40px 20px;
  background-color: #272822;
  color: #fff;
  resize: none;
  border: 5px solid #eee;

}
.app-container .copy-btn{
  position: absolute;
  left: 5px;
  top: 5px;
  padding: 12px 32px;
  background-color: #eee;
  cursor: pointer;
  color: #333;

}

/*# sourceMappingURL=app.6eeb09483e36aea680d0.css.map*/


When you share a Warpwire video via the share link, viewers will see a special standalone view of the asset.
The 'details' tab shows when the asset was published, what Media Library it's in, how many times it's been viewed, the asset's duration, and if the asset has closed captions attached.
The 'share' tab provides a direct link to the asset as well as an embed code. If downloads are enabled for the asset, you'll also see direct download links.
If the asset has one or more metadata profiles, the 'metadata' tab will be visible. You can select any of the asset's metadata profiles from the dropdown menu to view the details of the profile.
If the asset has one or more closed caption files attached, the 'transcript' tab will be visible. You can select any of the asset's transcripts from the dropdown menu to view them. If you press play on the asset, the transcript will highlight the current line of text, moving through lines as the video progresses. Alternately, you can click on a line of the transcript and the video will jump to that point. You can also seach the transcript using the search box below the dropdown menu.


The Apskalni, Kuldigas Rajons forecast for Apr 20 is 54 degrees and Patchy rain possible . There is 54 percentage chance of rain and 9 mph winds from the North.
0am:The Apskalni, Kuldigas Rajons forecast for Apr 20 is 47 degrees and Partly cloudy. There is 69 percentage chance of rain and 5 mph winds from the Northwest.
3am:The Apskalni, Kuldigas Rajons forecast for Apr 20 is 40 degrees and Partly cloudy. There is 72 percentage chance of rain and 5 mph winds from the Northwest.
6am:The Apskalni, Kuldigas Rajons forecast for Apr 20 is 41 degrees and Overcast. There is 69 percentage chance of rain and 6 mph winds from the Northwest.
9am:The Apskalni, Kuldigas Rajons forecast for Apr 20 is 49 degrees and Overcast. There is 61 percentage chance of rain and 7 mph winds from the Southwest.
12am:The Apskalni, Kuldigas Rajons forecast for Apr 20 is 54 degrees and Patchy rain possible. There is 54 percentage chance of rain and 9 mph winds from the North.
3pm:The Apskalni, Kuldigas Rajons forecast for Apr 20 is 56 degrees and Partly cloudy. There is 50 percentage chance of rain and 9 mph winds from the North.
6pm:The Apskalni, Kuldigas Rajons forecast for Apr 20 is 53 degrees and Partly cloudy. There is 58 percentage chance of rain and 8 mph winds from the East-Southeast.
9pm:The Apskalni, Kuldigas Rajons forecast for Apr 20 is 47 degrees and Partly cloudy. There is 75 percentage chance of rain and 5 mph winds from the North-Northwest.
Saturday:The Apskalni forecast for Apr 20 is 54 degrees and Patchy rain possible. There is 54 percentage chance of rain and 9 mph winds from the North.
Sunday:The Apskalni forecast for Apr 21 is 52 degrees and Sunny. There is 48 percentage chance of rain and 9 mph winds from the East.
Monday:The Apskalni forecast for Apr 22 is 60 degrees and Partly cloudy. There is 48 percentage chance of rain and 9 mph winds from the South-Southeast.
Tuesday:The Apskalni forecast for Apr 23 is 61 degrees and Partly cloudy. There is 40 percentage chance of rain and 11 mph winds from the Southeast.
Wednesday:The Apskalni forecast for Apr 24 is 61 degrees and Partly cloudy. There is 42 percentage chance of rain and 19 mph winds from the Southeast.
Thursday:The Apskalni forecast for Apr 25 is 65 degrees and Partly cloudy. There is 55 percentage chance of rain and 12 mph winds from the South-Southeast.
Friday:The Apskalni forecast for Apr 26 is 68 degrees and Partly cloudy. There is 50 percentage chance of rain and 11 mph winds from the Southeast.


This book deals with the (re)production of cohesion and coherence in translation. Building on the theories and methods of Translation Studies and Discourse Analysis it answers some basic, still much debated questions related to translational discourse production. Such a question is whether it is possible to analyse the (re)production of coherence, and if yes, how? Can the models devised for the study of English original (not translated) and independent texts (unlike translations and their sources) be applied for the analysis of translation? How do cohesive, rhetorical and generic structure "behave" in translation? How do particular components of coherence relate to translation universals? The volume proposes a complex translational discourse analysis model and presents findings that bring new insights primarily for the study of news translation, translation strategies and translation universals. It is recommended for translation researchers, discourse analysts, practicing translators, as well as professionals and students involved in translator training.


New Converse in Store 5.15.09. Converse Product (RED) Chuck Taylor Ox with Black Hemp Upper. Designed by Billie Joe Armstrong of Green Day for the Converse One Hund(RED) Artist Series Re-stock. Converse Chuck Taylor Ox Charcoal Re-stock. Converse Chuck Taylor Ox Purple Passion Re-stock.


Hard Hard is the new song from the upcoming movie Batti Gul Meter Chalu which is sung by Mika Singh, Sachet Tandon & Prakriti Kakar. This song Hard Hard Lyrics is written by Siddharth-Garima. Also, Listen to Dekhte Dekhte.


text:
Q: NodeJS gcloud - Upload to google storage with public-read property/custom cache-expire I am trying to upload to google storage using the gcloud library (NodeJS).
I need to enable public-read property and also set the cache-expiration to 5 minutes.
I am using this (simplified) code:
storage = gcloud.storage({options}
bucker = storage.bucket('name');
fs.createReadStream(srcPath).pipe(bucket.file(targetFile).createWriteStream()).on('error', function(err)

How do I go about setting the approprate ACL/cache expire?
(I found this but not sure what to make of it:
https://googlecloudplatform.github.io/gcloud-node/#/docs/v0.11.0/storage?method=acl)
Thanks for the help

A: You can set the predefined ACL following the instructions here:
yourBucket.acl.default.add({
  entity: "allUsers",
  role: gcloud.storage.acl.READER_ROLE
}, function (err) {})

Regarding cache control, I don't believe you can set this as a default, but you can set this at the time of uploading your file:
var opts = { metadata: { cacheControl: "public, max-age=300" } }
bucket.file(targetFile).createWriteStream(opts)

Reference: https://cloud.google.com/storage/docs/reference-headers#cachecontrol

A: Api changed, use:
var gcloud = require('gcloud')({
  projectId: 'your_id',
  keyFilename: 'your_path'
});

var storage = gcloud.storage();
var bucket = storage.bucket('bucket_name');

bucket.acl.default.add({
    entity: 'allUsers',
    role: storage.acl.READER_ROLE
}, function(err) {});

To make the entire bucket public you can also use:
bucket.makePublic

Source: https://github.com/GoogleCloudPlatform/gcloud-node/blob/v0.16.0/lib/storage/bucket.js#L607
Or for just the file:
var bucketFile = bucket.file(filename);

// If you upload a new file, make sure to do this
// in the callback of upload success otherwise it will throw a 404 error

bucketFile.makePublic(function(err) {});

Source: https://github.com/GoogleCloudPlatform/gcloud-node/blob/v0.16.0/lib/storage/file.js#L1241 (link might change, look for makePublic in the source code.)
Or:
bucketFile.acl.add({
    scope: 'allUsers',
    role: storage.acl.READER_ROLE
}, function(err, aclObject) {});

which is the verbose version.
Source: https://github.com/GoogleCloudPlatform/gcloud-node/blob/v0.16.0/lib/storage/file.js#L116

A: Stephen's comment is accurate, however it did not work for me as the value didnt get set. After some trial and error, turn out cacheControl (no dash) to get it to work. At the time of writing, this is not documented anywhere that it needs to be in this format. I assume other fields will have the same issue.
var opts = { metadata: { "cacheControl": "public, max-age=300" } }
bucket.file(targetFile).createWriteStream(opts)


text:
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«Market Leader» - news and previews making you rich.
Tuesday, 21 January 21:39 (GMT -05:00)
Home About Us Vacancies Advertise Contact Us
Forex and Cryptocurrency Forecasts for mid-January 2019
EUR/USD Waiting For Events
Best Cryptocurrency Brokers Are Named
Forex Treasure by Alpari: 20% on Deposit
Bitcoin Futures Are Now Traded On NYSE
"Forex Is A Scam" and Other Popular Myths About Forex
Stock and commodities markets
Brent Prices Drop Down To $61/b
Gold Prices Are Getting Stable After Monday's Rally
Oil Prices Have Made The Biggest Rally In History
iOS13 Outshines iPhone
US-China Trade War Makes Oil Prices Drop
Gold Prices At Highest Levels Since 2013
Business And Politics News
Fed Cuts Key Interest Rate For The First Time In 10 Years
New Prime Minister Names Brexit Date
Johnson Launches Hard Brexit Ad Campaign
U.S-China Trade War Is Sponsored By Consumers
US-China Trade Conflict May Trigger Another Global Financial Crisis
WTO Lowers Global Trade Growth Forecast
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ActivTrades Awards Hundreds of Traders - from iPads to 3D TVs.
Bitcoin Will Crash To $3000 Per Coin
How Trump Is Going To Surprise The World
Over the last few months, international media have been publishing intimidating articles about President Trump. Everyone now seems to be concern about how president Trump is going to act in the international arena. Is Donald trump going to surprise the world as the 45th President of the United States?
The thing is, each time the USA elects a new president, the international community starts predicting cardinal changes in the USA's domestic and external policies. However, each time their predictions fail. The American policy haven't changed a lot over the last few decades. After WWII, the USA turned into a superpower. After the USSR ceased to exists in 1991, the US became the only true superpower in the world and has been the most decisive force in the international arena. As you can see, American politicians have been consistent in terms of trying to rule the entire world and dominate others.
Why is now everyone concerned about Trump and his plans? He has been a populist and trying to win more of those loyal Americans. The American people wants changes for the better and is tired of empty promises. Since Donald Trump promised to make American great again during his election campaign, this was something the people wanted. That's one of the reasons he won, experts say.
Do you really thing Trump is going to through away everything the USA achieved over the last 200 years of hard work? Do you thing this time is really going to be different?
Some experts don't believe this. The USA cannot be a protectionist-oriented country even despite robotization and automatization of contemporary productions since they don't outsource high-margin products. Palantir, Amazon, Google, and even Tesla are mainly US-based productions. As for Apple, China only assembles Apple devices, and nothing more. As for US energy giants ExxonMobil and Shevron, they went international a long time ago. Most American giants are going or have already gone international as well.
It's hard to believe that Donald Trump, a successful businessman and a billionaire, will ruin everything and go away from the international markets just for the sake of creating some of those new jobs he once promised. This leads us to believe that his critics expecting serious changes are going to be surprised and then disappointed.
Chances are, President Trump will take several image-making actions in order to avoid losing face. For example, the USA will start building the wall on the US-Mexican border. Trump may also exert some pressure on some industrial companies (like motor companies) to move their assembly lines back to the USA. Still, those are relatively irrelevant things.
The thing is, assembly is not something makes the USA the world's leader in technology. This is all about creating and developing products, as well as marketing them. America is still number-one technological nation in the world. The US is also some kind of a lender of last resort in the military, political, and economic sense. The US Dollar is still the most important currency. The Federal Reserve is still the key central bank affecting the entire financial and banking system of the world.
You are free to discuss this article here: forum for traders and investors
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Pound Will Drive Forex This Week
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The U.S. Federal Reserve is reported to have cut the key interest rate, which is something really outstanding since the Fed has done it for the first time since 2009.
Publication date: 11 August 03:05 AM
It seem that the new Prime Minister of the United Kingdom is really determined about everything related to the Brexit. The process is expected to start in later 2019. Boris Johnson's standpoint on the matter didn't come as a surprise to the international expert community, Market Leader reports. The thing is, he has been well-known for being an advocate ad big supporter of quitting the European Union in general, and doing so without a major agreement in particular, which is also known as the hard Brexit scenario.
Boris Johnson, who has recently been appointed new UK Prime Minister, is on his way to launch an ad campaign to promote the idea of quitting the EU the hard way, which is also known as the hard Brexit. For those of you who don't know, the hard Brexit scenario implies quitting the European Union without signing a major agreement.
Publication date: 31 July 11:43 AM
According to the IMF, consumers and producers are the biggest losers in the trade war between the United States and China. Despite growing duties, American companies are not in a hurry to move their production back to the USA.
Beijing and Washington are one step away from escalating their trade conflict. The confrontation may harm the entire global economy. Some experts belive that the trade war may also trigger another global financial crisis. At this point, the parties seem to have come to a standstill, which is why the chances of the conflict escalating into a move severe trade war are still growing.
Publication date: 18 June 10:18 AM
WTO experts are reported to have revised their forecast for the pace of global trade growth. The renewed forecast names figures below the previous ones - 2,6% against 3,7%. It's also interesting to note that the previous forecast for 2018 failed to match the actual figures.
Publication date: 19 May 02:58 AM
How to Protect Investment Capital in 2019?
Existing political and economic risks are pushing international investors into thinking about the security of their investment capital. Chasing big profits becomes secondary to this kind of security.
Publication date: 31 March 11:26 AM
EU Comes Up With Workaround to US Sanctions
The representatives of Germany, France, and the UK have registered a company to let it trade with Iran despite the US sanctions. The company still needs to be approved by 28 EU members.
Beijing and Washington are getting ready for the final talks
Publication date: 17 February 08:58 AM
US-China Trade War Reaches Next Level
Washington and Beijing have announced a new round of talks. International experts say that the trade war is indeed going to a whole new level.
Publication date: 08 January 10:17 AM
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In 2009, Universal Studios Orlando introduced us to Julian Browning, the Usher, who has become a fan favorite icon for their annual Halloween Horror Nights event since. We uncovered our video recording of the Unmasking the Horror tour from that year, a behind the scenes look at the haunted houses that make up the event. In part 1, we begin our tour at 2009's Frankenstein: Creation of the Damned house, followed by Dracula: Legacy of Blood.
*Note: in 2009, video recording WAS allowed during the Unmasking the Horror tours, but it is now no longer available, but still photography is.
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KRAMPUS maze comes to Halloween Horror Nights in Hollywood & Orlando!


Today is the last day of school for the kids in our district. Then, SCHOOL'S OUT FOR THE SUMMER! I always loved the last few days of school; it was like the whole summer was at your fingertips. I guess it really was! No homework for three months!
My sister-in-law is finishing her last day of 4th grade today. When I was in elementary school, it was K-5, Middle School was 6-7, Junior High was 8-9, and High School was 10-12. Now, the schools have changed and elementary is K-4. Middle School is 5-8, and High School is 9-12 at two neighboring campuses. So today is Ashley's last day as an elementary student. Her last day at the school she's gone to for the last 5 years. How did that happen??
Her school had their graduation ceremony last week and of course, we went and I brought my camera. I remember my last day at my elementary school; the last time we played kickball out on the playground. I bawled my eyes out as I left the school yard. It's so weird how that feels like a lifetime ago but also like it was just yesterday. When in all reality it was 15 years ago.
I'm excited for Ash as she embarks on new adventures. I'll be there taking pictures along the way.
The 4th graders performed some songs for the audience, two of which I knew every word to! One of "You're a Grand Ol Flag" and the other was "Fifty Nifty United States" which is the way I remember all of the states in the US!
Then they got their 4th grade diplomas.
And of course, there are always a couple outtakes or awesome bloopers where Matt and Ashley are concerned.
So cute! Thanks for sharing the family moment.


text:
Q: Probability distribution obtained by repeatedly sampling $S_x,S_y$ on a spin-$S$ system While trying to rework an upcoming quiz problem for a quantum physics course, I came up with the following question which turned out to be harder than I expected. (Note: I take $\hbar =1$ in all statements below.)

I prepare a spin-$S$ state with maximum $z$-component i.e. $s_z=s$, and measure the spin components in the plane; first $s_x$, and then $s_y$. I can then repeat this pair of measurements ad infinitum, obtaining a sequence of points in the $(s_x,s_y)$ plane.

For small $S$, one can compare the possible outcomes by hand; indeed, for the simplest case $s=1/2$, there are four outcomes $(s_x,s_y)=(\pm\frac12,\pm\frac12)$ and by symmetry I expect each of them to occur with probability 1/4. This symmetry is lost for $S>1/2$, but some relatively explicit properties should be available at small $S$.
If $S$ is large, on the other hand, I would expect $(s_x,s_y)$ is more likely to be found at some finite distance from the origin. This intuition is motivated by the fact that the spin operators satisfy $$S_x^2+S_y^2 = S^2-S_z^2=s(s+1)-s^2=s,$$ suggesting some preferred radius in spin space. So the question I would pose is:
What is the probability distribution function of this sequence? In particular, how does it behave for large $S$?