text:
"These electronic 'skull-zappers' are designed to invade the mind and short circuit its synapses... in the hands of government technicians, it may be used to disorient entire crowds, or to manipulate individuals into self-destructive acts. It's a terrifying weapon."
-- Dr Emery Horvath, Physics Professor at Harvard University
Targeted Justice is the world's leading information resource for Targeted Individuals. If we have helped you, please consider a donation of $19 per month. (That's less than your expensive cup of coffee, each week.) https://www.patreon.com/TargetedJustice
Directed Energy Weapons are real.
Textbook examples from National Defense University (NDU).
https://ndupress.ndu.edu/Portals/68/Documents/Books/CTBSP-Exports/Effects-of-Directed-Energy-Weapons.pdf?ver=2017-06-16-125220-170
"Directed Energy Weapons are Real . . . And Disruptive," 2019.
By Lt General Henry "Trey" Obering, III (ret. - USAF)
https://ndupress.ndu.edu/Portals/68/Documents/prism/prism_8-3/prism_8-3.pdf
David M. Sowders et al., "High Power Microwave (HPM) and Ultrawideband (UWB): A Primer on High Power RF," PL-TR-95-1111, Special Report, Phillips Laboratory, March 1996.
Eileen Walling, "High Power Microwaves, Strategic and Operational Implications for Warfare," Occasional Paper no. 11, Air War College Center for Strategy and Technology, May 2000.
The earliest, well-documented Targeted Individual case that we are aware of - Norman C. Rabin, for 34 years. He is 59, and has been attacked with Directed Energy Weapons (DEW) since December 1985, and V2K since Oct 15, 1990.
Update on microwave frequencies used on the population, from Schriever Space Force Base. Thanks to Armondo D.
V2K: 450 - 700 MHz [Subliminal messages at population] U.S. Patent 4,877,027 (intermittent)
Scanning from Cell Towers: 500 - 580 MHz, 5 - 10 MHz spacing, X-Y-Z planes (continuous)
Remote Neural Monitoring (RNM): 600 - 800 MHz, U.S. Patent 3,951,134 (continuous)
Satellite tracking (Lockheed GPS): 3200 - 3750 MHz, U.S. Patent 4,384,293A (continuous)
Satellite Vircator Weapon (L3Harris): 3920 - 3935 MHz, U.S. Patent 4,345,220 (intermittent)
Lockheed Martin GPS Satellite tracking is done at 3600 - 3750 MHz. The Vircator (Patent 4345220) operates at 3920 - 3935 MHz. Note the graphs show there is almost zero background noise in 2016. The U.S. Space Force has started adding artificial background noise to these frequency ranges, to hide their criminal activity.
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Technology & Technical information:
Targeting Technology
There are 4 main technologies being used in the targeting:
(1) Smart Meters
(2) Microwave Satellites
(3) Cell Towers
(4) Drones
(5) [Portable units are usually fake.] They cannot carry the massive electrical load for a weapon - which is approx. 20,000 volts and 2000 amps.
The smart meters are readily controlled from a remote location, which allows the government criminals to harass an individual with plausible deniability. This is one of the first steps taken against a new Targeted Individual. The microwave signal is transmitted thru all of the household wiring. If you are exposed to microwaves coming from all directions, the first possible solution - is to switch off some of the circuit breakers at the breaker box. This can make a safe area in your house, i.e, shut off all the breakers that go to a back bedroom.
(2) Satellites
The first microwave attack satellites were launched under President Reagan's Star Wars program (SDI) in 1987. These satellites were designed to disable ICBM nuclear missiles during the ascent phase. Tracking frequencies used are 3600 - 3750 MHz, and the attack signal is 3920 - 3935 MHz. FCC frequency allocation tables, confirm the use of these frequencies as Satellite-To-Ground signals. These satellites and frequencies are now being used against Targeted Individuals. The tracking signal has an accuracy better than 1 centimeter, and the GPS coordinates can be relayed to a cell tower or drone for additional targeting. These tracking signals generally come from a satellite that is almost directly overhead - Lockheed Martin builds these GPS satellites in Littleton, Colorado. (NavStar satellite).
Fig 1. Satellite Operator Stations at Peterson AFB. Fig 2. Vircator - Microwave satellite weapon
Fig 3. Headquarters - Air Force Space Command (AFSPC);
Peterson Air Force Base,
Colorado Springs, Colorado, U.S.A.
The satellites are operated and controlled from Peterson Air Force Base and Schreiver Air Force Base, near Colorado Springs, Colorado, under the Air Force Space Command. There are hundreds of Air Force personnel that have been trained to control the satellites and "push the button" to hit a person on the ground with microwave beam pulses. They know exactly what they are doing to people, and they know it is probably illegal. Some of these Air Force personnel have probably tried to become whistleblowers. This may explain the alarming suicide rate in Colorado Springs - some of these incidents were actually murders or staged suicides. The people that "push the button" are usually young, in their 20's and 30's. They are enlisted personnel and their rank is typically an Airman or Senior Airman. They hold a Top Secret security clearance, TS/SCI (Sensitive Compartmented Information), and follow the orders of their commanding officers. In the case of the Targeted Individuals program, it is called an Unacknowledged Special Access Program (USAP), which means that even the President, will not be informed about the program (this is illegal). All USAP's are illegal and a violation of the U.S. Constitution. People that work on these programs, have their lives and families threatened to keep it secret. The irony about working within a USAP - is that, if a person reveals their existence, they cannot be charged with a crime, because the program itself is illegal. For a discussion on this, see Dr Steven Greer's latest book and movie - "Unacknowledged."
How do we know that Air Force Space Command is behind these satellite attacks? The ability to precisely track, millions of people, requires a massive network of satellites using the latest technology in positioning, navigation, and timing. Literally, every person in the United States is being tracked with a microwave pulse to the top-back part of their head - that's 327 million people, being tracked every second. There is only one organization in the world that has that capability - the U.S. Air Force Space Command. This statement from the Air Force website is basically an admission of guilt:
"AFSPC (Air Force Space Command) is the sole provider of positioning, navigation, and timing, as well as the bulk of satellite communications to the warfighting community. Lastly, AFSPC serves as the Air Force's Executive Agent for space. In this role, AFSPC is the advocate for space capabilities and systems for all unified commands and military services, and collectively provides space capabilities the US needs today and in the future."
http://www.afspc.af.mil/About-Us/Heritage/2000-2007/
Watch this 5 minute video by Dr Bill Deagle, MD, about Schriever Air Force Base:
https://youtu.be/BzzKACyw7jc
"The underground facilities at Schriever Air Force Base have more computing power, than all of the rest of the planet put together, by a very large order of magnitude."
Digital beam-forming technologies have been installed on many cell tower antennas in urban areas. The antennas consume large amounts of electricity to act as a weapon. The 5G technology allows the weapons to fire, millions of times each second from a single antenna. Typical frequencies used are 2400 MHz and 2550 MHz, although it can vary from 2300 - 2900 MHz. With 5G, the frequencies will be much higher.
There are two primary drones in use by the military services against civilians in the United States. The MQ-9 Reaper (and its variants) are manufactured by General Atomics in Poway, California. This drone is operated by U.S. Air Force personnel. The other primary drone is the RQ-7 Shadow (and its variants) manufactured by AAI Corporation, in Hunt Valley, Maryland. This drone is primarily operated by U.S. Army personnel. Many thousands of both drones have been ordered for the military. The largest drone training facility is located near Fort Huachuca, Arizona. We know these are the drones that are being used, because TI's have identified them from numerous images, after seeing them in the air. There is also the payload requirement - smaller drones cannot lift the 30 to 60 pounds of equipment that are needed to perform a microwave attack. The MQ-9 Reaper is operated from many Air Force Bases in the United States. In Houston, Texas, they are operated from a hangar at Ellington Air Force Base and used to target civilians. Also note, the Drones in Houston were probably used to initiate multiple Plant-Factory fires, during 2018 - 2019.
MQ-1 Predator used for microwave targeting by Air Force personnel.
RQ-7 Shadow used for microwave targeting by Army personnel.
Specifications for the MQ-1. Note the payload.
Specifications for RQ-7.
V2K (Microwave Hearing)
[thanks to Mark at NewWorldWar.org]
Dr. Frey, in his 1962 report, Human Auditory System Response to Modulated Electromagnetic Energy, which appeared in the Journal of Applied Psychology; Dr. Frey described how microwave hearing was demonstrated using a microwave transmitter that projected sound several hundred feet.
The transmitter used pulse-modulated waves at extremely low average power levels. The transmission was immediate. The system was wireless and receiverless, and the sounds were even heard by the deaf. Then in the mid 1970s, Dr. Sharp helped to develop microwave hearing technology for DARPA by conducting research at the Walter Reed Army Institute of Research (WRAIR). In 1973 Dr. Sharp proved that the correct modulation of microwave energy could result in the wireless and receiverless transmission of audible speech.
In 1976 it was reported by the Los Angeles Herald-Examiner that the Soviets were also conducting extensive research into microwave hearing that was brought to the attention of the US Defense Intelligence Agency. Words which appeared to be originating within a person's skull, they reported, could be induced by microwaves. The technology was to be used for antipersonnel purposes.
The October 1998 US Patent 4877027—Hearing System, mentioned that sound could be induced in the head of an individual using microwaves in the range of about 100 MHz to 10 GHz. The waves consist of frequency modulated bursts lasting from about 500 nanoseconds to 100 microseconds that create a sense of hearing in the individual whose head is targeted. It is effective regardless of a person's hearing ability.
Others patents pertaining to microwave hearing, such as the August 1989, US Patent 4858612—Hearing Device, and the July of 2003, US Patent 6587729—Apparatus for Audibly Communicating Speech Using the Radio Frequency Hearing Effect, are based on similar scientific principles.
In July of 2008, New Scientist announced that Sierra Nevada Corporation planned to build a microwave gun able to project beams of sounds directly into people's heads. The weapon, which is based on the microwave audio effect, will be used by the military. Wired's February 2008 story, Pentagon Report: Nonlethal Weapons Could Target Brain, Mimic Schizophrenia, described how microwave weapons which create voices in people's heads are not only possible, but have already been demonstrated.
Most researchers from the mid 1960s into the early 1990s have concluded that bone conduction (thermal expansion) is the physical mechanism that allows for microwave hearing. The detection of microwave hearing is the same in humans as it is in other mammals. Bone conduction is just a secondary pathway for sound transmission in humans and most other mammals. Bone conduction works basically in the following way: Upon hitting the skull and interacting with the soft tissue in the head, a microwave signal in the range of about 2.4 MHz to 10 GHz is down-converted to an acoustic frequency of about 5 kHz.
electromegnaticweapons.pdf
U.S. Electromagnetic Weapons and Human Rights. Peter Phillips, 2006.
(We apologize for obvious errors, the government criminals are hacking and altering our web site.)
Our Main Message:
1) More than 200,000 Targeted Individuals in the U.S., are being tracked and attacked with High-Powered Microwave (HPM) weapons, from satellites and cell towers, 24/7. These weapons are controlled from Schriever Air Force Base in Colorado. See our pictures of microwave burns as proof: https://www.targetedjustice.com/microwave-burns1.html This is the same weapon system, that was used on the U.S. Diplomats in Cuba.
Many famous people are Targeted Individuals: Ed Snowden, Julian Assange, and William Binney.
Also, Roseanne Barr, Van Morrison, and John McAfee. They are hit with painful microwave attacks every day.
2) Most people in the world, are being precisely tracked by microwave pulses using Lockheed GPS satellites, operated by the U.S. Space Force, Squadron 2, at Schriever Air Force Base - Colorado Springs. This is the largest satellite operations facility in the world. (No, this is not Russia or China. It is funded and controlled by the CIA.)
3) Panel antennas on cell towers are part of a "global distributed weapon system." The antennas use digital beamforming to create microwave "bullets," that can be very painful, and even lethal.
4) Subliminal messages are directed through Cell Towers, at everyone, 24/7. Schriever AFB is the control source. U.S. Patent 4,877,027. Senators, Congressmen, Judges, and even the President, are being hit with specific subliminal messages to affect their decisions.
5) Approximately 1/3 of the global population is "highly suggestible" and easily manipulated by these subliminal messages, because they are effectively in a Deep Trance, even while their eyes are open. These people can be commanded to say or do, almost anything. Many secondary personalities are also involved.
6) The CIA controls and funds this illegal program, with direction from the Council on Foreign Relations (CFR). The program is based on MK-Ultra mind control technology, and the FBI's illegal COINTELPRO.
7) Many people can feel the satellite-tracking microwave pulse, on the top-back part of your head – about one pulse each second. Many people can hear the cell tower tracking "popcorn," by cupping your hands over your ears for 60 seconds.
8) This article quotes NSA data - as of 2019, there are 200,000 Targeted Individuals in the United States, and more than 5.5 million worldwide. The FBI calls these people "Targets," or "Non-Investigative Subjects (NIS)."
https://www.nytimes.com/2021/04/26/us/politics/fbi-fisa-surveillance.html
Latest Videos from Targeted Justice:
https://odysee.com/@shadowcitizen:d/targetjustice:3
https://www.youtube.com/watch?v=zNDswN3nnIA
Here is the handout for talking to Legislators, Lawyers, family members, etc.
Harmonics of the Lighthouse Frequency & Laser wavelengths. This diagram shows how the Lighthouse Frequency is used as a weapon. Harmonic numbers 256 & 512 are being used by the USAF & US Space Force. Highlighted in yellow are the harmonics which are very close to known laser frequencies. By utilizing the Expanding Neutral Point - rapid aging occurs at the target. Note the overhead view of the World Trade Center on 9/11 - cookie cutter holes that go thru steel and concrete.
We are still early in this process.
We understand you have many questions and suggestions. Please understand that we are very early in this process and are developing the requirements for presenting our case. Your support and patience are requested. This is a large effort and we are working diligently to achieve a successful outcome.


text:
Wike, Fayemi, El-Rufai, Obaseki, Others To Receive COVID-19 Vaccine On Live TV
Members of the Nigeria Governors' Forum (NGF) have accepted to take the COVID-19 vaccine on live television to encourage Nigerians to accept the vaccine.
Naija News reports that Governor Kayode Fayemi of Ekiti State confirmed it when speaking to State House correspondents after a meeting with President Muhammadu Buhari on Friday.
Fayemi, chairman of the NGF, said he and other Nigerian governors had accepted to do so.
"Absolutely," Fayemi answered when asked if governors will also take the vaccines on live television. "We too will like to demonstrate to our citizens that we believe that vaccines would work."
"Don't forget, we have a lot of experience on this. Governors Forum managed the polio vaccines administration in the country and we have garnered a lot of experience," he said.
This online Newspaper earlier reported that the federal government has said provision would be made for a live broadcast of President Buhari and Vice President Yemi Osinbajo receiving the COVID-19 vaccine.
This was made known by the executive director and chief executive officer, National Primary Health Care Development Agency (NPHCDA), Faisal Shuaib.
"We will like to see a situation where Mr. president, the vice-president, the SGF, critical leaders come and take the vaccine in the full glare of the public to demonstrate that these vaccines are safe. So we have to make provisions for those," he said.
Nigeria is expecting 100,000 doses of the Pfizer/BioNTech vaccine before the end of January.
Related Topics:coronavirusCOVID-19COVID-19 VaccineHeadlinesKayode FayemiMuhammadu BuhariNigeria GovernorsNyesom WikeYemi Osinbajo
COVID-19: FG Suspends Screening Of 5,000 Successful Candidates For NDLEA Job
NIN To SIM Cards: Pantami Reveals Simple Ways To Get Linking Done
Top Nigerian Newspaper Headlines For Today, Saturday, 15th January, 2022
'Election Go Humble You' – Nigerians React As Osinbajo Posts Video On Tik Tok

redpajama_set_name: RedPajamaCommonCrawl


text:
Q: Android store API Keys in secret I have an Android app public on the Github. Even if it is public I have some values like API keys that I don't want other people to see.
I've seen people storing them on gradle.properties but I have that file visible also.
I've see that the gitignore command will not commit that file at all in case I update it.
Question: Which is the best way to achieve this for Android projects and keep the project public?

A: There is a file called local.properties at the project root level which could be used to hold the api keys. It shouldn't be committed to source control.

redpajama_set_name: RedPajamaStackExchange


text:
About CU
Where We're Going
Cumberland University Board of Trust
Cumberland University Leadership
For 180 years, Cumberland University has been committed to preparing students for a lifetime of learning, leadership, and growth. Throughout our long history, we've continued to evolve and rise. From the very ashes of the Civil War, we have expanded our campus, broadened our academic programs, added new living and learning facilities, and graduated some of the most respected and influential leaders in our state and nation's histories.
1842 – Cumberland University is founded.
1847 – The School of Law is founded, it is the first in Tennessee and west of the Appalachian Mountains.
1861-1865 – Civil War
1866 – All departments of the university are in operation in various locations of Lebanon.
1892 – Cumberland moves to its present location
1896 – Memorial Hall is completed
1942 – Cumberland celebrates 100th anniversary
1942 – Almost 850k Soldiers from 25 US Army divisions, making up the Red and Blue Armies, were training for World War II. Headquartered at Cumberland University.
1946 – The Tennessee Baptist Convention assumed control of the University, ending a century of operation under the auspices of the Presbyterian Church.
1951 – The Tennessee Baptists close the College of Arts and Sciences and operate only the School of Law.
1956 – The Board of Trust secure an amendment to the Charter and change Cumberland to a private, independent corporation. The College of the Arts and Sciences is reopened as a two-year junior college, known as Cumberland College of Tennessee.
1961 – The Law School is sold to Samford University (then Howard College) in Birmingham, Alabama, after 114 Years.
1982 – The Board of Trust expands the academic programs of the junior college returning Cumberland to a four-year, degree institution. It resumed the old name of Cumberland University. It spent 28 years as a junior school.
1984 – First Phoenix Ball is held.
1986 – The first class of Baccalaureate Degrees graduates.
1995 – MBA program begins.
1997 – First class of MBA students graduate.
2015 – Dr. Paul C. Stumb is named the 26th President of CU.
2016 – Cumberland University changes the nickname of its athletic program from "Bulldogs" to "Phoenix," stating that, "For more than 150 years, the Phoenix has personified the spirit of Cumberland University."
2017 – Cumberland University rises to record enrollment.
What is new at CU
Getting Organized for the Semester
Most students start the semester off with good intentions of staying organized during the semester. However, after a few weeks have passed and assignments begin piling up, those intentions get pushed to the side. Here
Phoenix Feature: Chris Banos, Inaugural Recipient of the Pay It Forward Scholarship
At Cumberland, 9 out of 10 students receive tuition assistance, either in the form of grants or scholarships. Chris Banos, a junior studying business and coaching administration, is the inaugural recipient of the Pay It
Cumberland University Announces Fall 2021 Dean's List
Cumberland University congratulates the following 703 students for their outstanding accomplishment and inclusion on the 2021 Fall Dean's List. This honor is reserved for students who have achieved a GPA of 3.5 or higher on


What the heck is "Zeolite"?
Synthetic turf, pool filtration, odor elimination—what do these have in common? Zeolite—a fancy sounding name for a simple class of naturally occurring minerals consisting of hydrated aluminosilicates of sodium, potassium, calcium and barium. Zeolites have some special properties that make them very useful—they are in a class of microporous solids known as molecular sieves—because they have a negative charge and absorb positively charge molecules.
It means… when you spill your Seattle craft microbrew beer on the synthetic turf Transblue installed in your back yard—the zeolite infill in the synthetic turf will absorb the beer odors—and when it rains (or when you rinse with water) it will all rinse away—resetting your zeolite infill to be ready for the next spill or malodorous liquid!
Zeolite is what differentiates our synthetic turf from less-experienced installers—it makes our pools cleaner, it's a huge win for our clients!


Chennai is an IT hub where you will find most of the IT and telecom companies here, dependant on large data and its security. Our company is one of the foremost Data recovery company in Chennai. We not only offer data recovery services in Chennai but also provide data security, Data privacy and other types of hard disk recovery services in Chennai.
The data storage devices like hard disks, diskettes, USB flash, tapes, RAID/NAS/M SATA volumes etc are a very important part of any company which is running well. Imagine losing data if any of these devices starts misbehaving; sounds very scary but it can happen because life is uncertain and so are these devices. However every problem has a solution, the solution of losing data is to hire the best data recovery services in Chennai and that's what we are. We aim to deliver best data recovery, Hard disk recovery, crash drive recovery, disaster recovery, server and files recovery. We basically work on all types of disaster data recovery services.
We as one of the best data recovery company very well understand that the entire working of an IT and Telecom Companies depends on the data and servers. What if any system loses the data due to damage of the hard disk or any other reason, the entire working system will be affected and the results could be worse than you would have ever thought. However we can't stop the mishap but yes, we can assure you a guaranteed HDD recovery and other types of data recovery at a very nominal data recovery cost.
Our company works transparently on the "No Data, No cost" basis. We value our customers and their hard earned money and this is the reason we offer a free evaluation of the data losses. Once we are confident that we will be able to recover your data to the maximum limit then only we work further. This way our valuable customers have to pay only for the amount of data recovered and not the amount of time we worked for. Isn't that too fair? Yes of it is. If we are unable to recover your data you won't have to pay any data recovery cost to us, because customer satisfaction and their data recovery is more valuable to us then money.
We have teamed up with the most skilled professionals who are ready to serve you throughout the week. You just need to call up at our customer helpline number and leave the rest to us.
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Manchester's property market is stimulated by the Northern Powerhouse Initiative, the High Speed Rail project, and a number of regeneration developments within the city. A recent report ranked Manchester as the fastest growing city across 20 cities in the UK and reported that its property market last year reached a growth rate of 6.6%. With a lagging British pound and Manchester's strong economic growth and investment potential, now is the best opportunity to invest into the city's property market.
Golden Emperor Properties hosted a Manchester Property Investment Seminar and launched the latest residence, Crescent, crafted by one of Manchester's most renowned developer, Salboy International. Nearby finance district New Bailey and digital hub MediaCity, the seminar was widely supported by investors during the 2-day seminar event.
Photo Above: Golden Emperor Properties Managing Director, Mr. Terence Chan discusses the latest trend of Manchester's property market at the seminar over the weekend.
Photo Above:The 2-day seminar received a wide amount of support from investors.


text:
package cz.helmisek.evtalibrary.builder;

import android.databinding.ViewDataBinding;
import android.support.annotation.LayoutRes;

import java.util.ArrayList;
import java.util.List;

import cz.helmisek.evtalibrary.adapter.entity.AdapterViewType;
import cz.helmisek.evtalibrary.adapter.viewholder.BaseViewHolder;

/**
 * Data builder class basically allows developer to create custom viewType based list of data using this builder.
 *
 * It is recommended to use enum ordinals as viewTypes.
 */
public class AdapterDataBuilder
{

	private List<AdapterViewType> mViewTypeList = new ArrayList<>();
	private List<AdapterViewType> mAdapterViewTypes = new ArrayList<>();

	/**
	 * Add single viewType item without any provided data.
	 *
	 * @param viewTypeId - ViewType ID to determine which type is going to handle your new item
	 */
	public AdapterDataBuilder addViewType(int viewTypeId, @LayoutRes int layoutId,
										  Class<? extends BaseViewHolder> viewHolderClazz, Class<? extends ViewDataBinding> bindingClazz)
	{
		mViewTypeList.add(new AdapterViewType(viewTypeId, layoutId, viewHolderClazz, bindingClazz));
		return this;
	}

	/**
	 * Add list of viewType items without provided data entities.
	 *
	 * @param viewTypeId - ViewType ID to determine which type is going to handle your new item
	 * @param content - List consisting custom type data entities to be provided for adapter
	 */
	public <T extends Object> AdapterDataBuilder addViewTypeItemList(int viewTypeId, List<T> content)
	{
		mAdapterViewTypes.addAll(ViewAdapterTypeItemFactory.addViewTypeItemList(mViewTypeList, viewTypeId, content));
		return this;
	}

	/**
	 * Add single viewType item with custom type provided data entity.
	 *
	 * @param viewTypeId    - ViewType ID to determine which type is going to handle your new item
	 * @param singleContent - Custom type data entity to be provided for this item
	 */
	public <T extends Object> AdapterDataBuilder addViewTypeItem(int viewTypeId, T singleContent)
	{

		mAdapterViewTypes.addAll(ViewAdapterTypeItemFactory.addViewTypeItem(mViewTypeList, viewTypeId, singleContent));
		return this;
	}

	/**
	 * Add single viewType item without any provided data.
	 *
	 * @param viewTypeId - ViewType ID to determine which type is going to handle your new item
	 */
	public <T extends Object> AdapterDataBuilder addViewTypeItem(int viewTypeId)
	{

		mAdapterViewTypes.addAll(ViewAdapterTypeItemFactory.addViewTypeItem(mViewTypeList, viewTypeId));
		return this;
	}

	/**
	 * A build method.
	 *
	 * @return list implementation of type {@link AdapterViewType}.
	 */
	public List<AdapterViewType> build()
	{
		return this.mAdapterViewTypes;
	}
}

redpajama_set_name: RedPajamaGithub


"Kidding" drew 443,000 viewers in its linear debut. When adding in the three replays that aired the same night, that grows to 787,000. Showtime also made the premiere available on digital platforms ten days prior to the linear launch, with the premium cabler claiming an additionl 360,000 viewers watched in that time. As such, "Kidding's" total premiere haul stands at approximately 1.2 million.
Kepts it 0.1 rating, but was down in total viewers from 443,000 to 290,000.
Stayed again outside of the top 25, but held its previous 0.1 rating and stable in audience now with 303,000.
That's solid. I'm glad. It's not as good as I hoped for, considering it's a major movie star in the main role, but it's higher than 'Dying Up Here'. So if more seasons were planned, we're safe. It will be interesting to see if ratings go up, down or pretty much stay the same in the coming weeks. Overall the audience reviews were good to very good, but certainly not great. I always think it's kinda silly judging on series based on one episode only, though.
Episode 2 was down in total viewers from 443,000 to 290,000. Lets hope it stay there and can increase from that.
Those numbers aren't impressive, though. Which is a shame. On the other hand: Few days ago I was reading this Flemish articles about ratings and how live ratings are less and less important. These days a large number of people are recording shows on their digibox and watching everything at once when the final episode is out. Also 'on demand' numbers are increasing around that time. Times have changed. I'm guilty of that too.
Saw the numbers for last Sunday. Bit up, not enough. This show just isn't doing that well, unfortunately. It hurts my heart, cause I really think it's good. Very good. Considering it's such a big star in the main role, this should be way up. Luckily ShowTime isn't that dependable of adds, so there's a good chance more will come.
Down again. The lowest so far. All we can do is enjoying it for the time it's around, I guess.
A -29,18% drop once again. Only 0.199 M viewers.
It hurts me to see this happening and I don't like to say this, but these numbers are not just mediocre, they are bad. Unfortunately. To me, this feels like something that would be a hit on Netflix. ShowTime just ain't working for Kidding.
Yea, we are lower than some of the episodes for "I'm Dying Up Here". Really not good.
How do you read the contract Jim has with Showtime grinchy steve?
"Now, after 20 years, he signs a five years deal to be back on TV to play the character of Mr. Pickles on Kidding."
TNPihl wrote: Yea, we are lower than some of the episodes for "I'm Dying Up Here". Really not good.
It's a fact that he signed a contract for five years, but it's nowhere confirmed that it's for Kidding only. (unless I'm mistaken?) Not that we know of. I'm quite sure that, as often is the case, it will be included he plays the character for five years as long as the show is a succes. But also that they can cancel when it's not. That contract can include his work as a producer, or maybe a comic special. It's all a wild guess, but common.
Maybe he had to let go 'Dying Up Here' to get 'Kidding'? Maybe he gets to keep'Kidding' if he does other things too for the network. All possible.
The "good" news is that ShowTime is less dependable on live-ratings. For example 'The Crazy aones' was considered a flop for Robin Williams. But those numbers were way, waaaay higher. So, it all depends what network it is on. For ShowTime, this might be enough to go through.
@veysel. Thanks for sharing, just saw it too. Good news. And proof ratings aren't everything on ShowTime. Great they extended the chance for audiences to pick up on this. The second season will be crucial.
I think the main reason why Showtime picked up Kidding for another season is because they believe more people will discover the show in this hiatus, based on the strong critical response and recommendations from friends, family etc.
Down again. The lowest again and again. 166,000 viewers for episode 6. I can not imagine it getting even lower.
It's a good thing that they have more than a million viewers (not live) through other channels. That's what I meant with 'times changed'. Apparently people do watch it, just not live. Cause those numbers are just terrible. Good thing live ratings aren't that important for ShowTime, so it seems. Otherwise 'Kidding' would have been cancelled already.
I watch what i want and if i dont have access, I wait until i do.


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PUSDFOUNDATION
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Strong business and community partnerships are fostering meaningful experiences for the students of the Poway Unified School District. With business and community support, we can continue to implement the many programs and activities that develop the necessary skills to prepare students for their future.
College And Career Fair
The annual College and Career Fair is coordinated by Palomar Council PTA, CTE staff and the PUSD Foundation. Students and parents had the opportunity to speak to university, college and technical school representatives. Over 125 colleges were represented at this fair at Rancho Bernardo High School last Fall. In addition, students and parents had the opportunity to attend several presentations on topics such as financial aid, college preparation, certifications, internships, and workshops about different college and career options.
Hour of Code takes place each year during Computer Science Education Week in December. More than 90 Hewlett Packard employees volunteered at six local school districts in San Diego County, with the majority of employees volunteering in the Poway Unified School District. Volunteers assisted 1,070 students at fifteen PUSD schools, with a focus on elementary and middle school students. HP Rancho Bernardo site employees have always had a strong interest in helping local students during the Hour of Code week.. The Hour of Code activities started with an introduction to computer science, which is designed to demystify "coding" and to show that anybody can learn the basics and to broaden participation in the field of computer science.
Northrop Grumman Space Camp
Northrop Grumman's Space Camp program provides teachers and middle school students with an opportunity to visit Space Camp in Huntsville Alabama. The goal of Space Camp is to inspire and motivate the next generation of explorers, scientists, teachers, and engineers. The Northrop Grumman sponsorship provided life-changing experiences for these middle school students who have the potential to excel, and with the right support, inspire them to dream big.
The Space Academy for Educators program provides teachers an immersive learning environment where they learn new ways to present science, math, technology and engineering concepts in their classrooms. At Space Camp, the classroom becomes a space outpost and an educational environment for the space mission they have been given. The goal for teachers is to enhance the quality of instruction in the classroom by improving the way teachers present STEM concepts.
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Poway High School's Team Spyder the FIRST (For Inspiration and Recognition of Science and Technology) Chairman's Award, the highest and most coveted award at a Regional and the team that best represents a model for other teams to emulate and best embodies the purpose and goals of FIRST. "The Chairman's award is presented to the team judged to have the most significant measurable impact of its partnerships among its participants and community over a sustained period, not just a single build season. The winner is able to demonstrate progress towards FIRST's mission of transforming our culture. The recipient team will be invited to a FIRST Championship where it will compete for the Chairman's award against winners from other qualifying events." FIRST was founded in 1989 to inspire young people's interest and participation in science and technology.
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Clean eating is fast learning to be a very popular movements; from fitness center fanatics to working moms, many have implemented this style of eating to improve their lifestyle, bodies and basic wellbeing. Dietary supplement: in each serving: 1/8 tsp of calcium Canine Necessities - 1 capsule of Krill essential oil for kids (Mercola), 1 capsule of Spirugreen (Mercola House animals), one pump of Ubiquinol for domestic pets (Mercola), 1/8 tsp of Prozyme, 1 chewable tablet of Joint Health (Springtime) , 1/8 tsp of Natural and organic Multi-Vitamin (Canine Essentials).
Around 65% of individuals 85 years or more mature suffer from the health of diverticulitis - contamination of the top intestine. Tackling diverticulitis normally starts with controlling the intestines with healthy bacterias, within probiotic beverages. An oft-cited analysis was done by Francis M. Pottenger, which still left little question to the value of enzyme-rich natural foods for felines.
Diet can have a big effect on PCOS. It is one of the main natural solutions to help PCOS and its effect on fertility. To learn more about how to consume specifically for PCOS read the PCOS Fertility Guide. If you feel that your dog is likely to be pretty hungry on such a light diet, you are probably right. commercial dog foods are laden with preservatives, colors (dyes), and sodium. They may have additives to make the food flavor better so that the puppies will overeat.
Regardless I do not feel that vegans (instead of all people who eat completely vegetarian diets) care and attention much about the dietary plan health argument. Without a vegan myself, my impression is the fact that vegans argue that people should avoid all canine products inside our lives for ethical and environmental reasons, and their diet/health debate is simply a balanced completely vegetarian diet plan is healthy. They don't seem to dispute that it is the perfect diet.
All our cleanliness products are carefully selected to contain only 100 % natural ingredients rather than severe chemicals. Following are guidelines for feeding a uncooked or grilled homemade diet to healthy puppies. No single kind of food, such as chicken, should ever make up more than half the diet. You might not apply legal terms or technological procedures that restrict others from doing anything permitted here.


Today looks like a good day to get Happy Hour(s) back on track @rionexiiiseattle 5-7 everyday!! Over 14 of your favorite dishes to choose from. 10 cocktail choices. $20 bottles of wine. Dollar off all beers. Come eat & drink it all.


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Activation, EGFR leads to Goblet cell metaplasia
Activation, EGFR
Goblet cell metaplasia
EGFR Activation Leading to Decreased Lung Function adjacent High Moderate Cataia Ives (send email) Under development: Not open for comment. Do not cite Under Development
human Homo sapiens Moderate NCBI
mouse Mus musculus Moderate NCBI
rat Rattus norvegicus Low NCBI
Mixed Moderate
All life stages Low
Airway epithelial injury can be caused by various inhalation exposures (e.g. cigarette smoke, sulfur dioxide, endotoxin, viruses). Subsequent tissue repair processes are thought to initiate the transdifferentiation process, whereby ciliated epithelial cells first dedifferentiate and then redifferentiate to goblet cells, without an apparent increase in the total number of epithelial cells resulting in goblet cell metaplasia (Lumsden et al., 1984; Shimizu et al., 1996; Reader et al., 2003). EGFR was shown to be a key player in this process in both murine and human airway epithelia (Tyner et al., 2006; Hao et al., 2011; Habibovic et al., 2016).
Goblet cell metaplasia was shown to occur following the activation of EGFR-mediated anti-apoptotic signaling in ciliated epithelial cells (Tyner et al., 2006). Subsequent stimulation by proinflammatory stimuli such as the Th2 cytokines interleukin (IL)-4 and IL-13 then promotes transdifferentiation of ciliated cells into goblet cells, thereby increasing the number of goblet cells ("second hit hypothesis") in mouse tracheal epithelium and airway epithelia of COPD patients (Curran and Cohn, 2010). In vitro, EGFR can be activated by ROS or IL-13 to lead to ciliated cell transdifferentiation. IL-13 stimulates transdifferentiation of ciliated epithelial cells to goblet cells through EGFR activation increasing MMP/ADAM activity and ERK/MAPK activation (Casalino-Matsuda et al., 2006; Yoshisue and Hasegawa, 2004; Tyner et al., 2006).
Two studies showed EGFR involvement in a decrease in goblet cell and increase in ciliated cell numbers or cell-specific marker expression (Yoshisue and Hasegawa, 2004; Casalino-Matsuda et al., 2006). Other studies demonstrated ciliated cell transdifferentiation in response to ILß13 in an EGFR-dependent manner in a mouse viral infection model and mouse tracheal epithelial cells in vitro (Tyner et al., 2006), rat nasal epithelial cells (Lee et al., 2000), and human airway epithelial cells (Kim et al., 2002; Hao et al., 2011). The KER is therefore highly plausible.
Cigarette smoke exposure resulted in increased goblet cell numbers and extensive AB/PAS staining, which were considered signs of goblet cell metaplasia (the outcome of transdifferentiation), in the airways of Sprague-Dawley rats. These effects were greatly diminished in animals pre-treated daily with the EGFR kinase inhibitor BIBX 1522 (Takeyama et al., 2001). Similarly, plugging of F344 rat bronchi with agarose resulted in goblet cell metaplasia as evidenced by histology and increased AB/PAS staining, and the extent of airway remodeling was markedly lower in animals treated concurrently with BIBX 1522 (Lee et al., 2000). EGFR-dependent goblet cell metaplasia was also observed in primary human bronchial epithelila cells exposed to ROS (Casalino-Matsuda et al., 2006), nasal epithelial cells of asthmatics and lungs of patients with diffuse panbronchiolitis that exhibited goblet cell metaplasia and concomitant EGFR expression or activation (Habibovic et al., 2016; Kim et al., 2004).
It is not well known how transdifferentiation of ciliated cells occurs in humans. Under normal conditions, lung epithelial cells (except basal cells) are terminally differentiated (Donnelly et al., 1982; Breuer et al., 1990; Rawlins and Hogan, 2008), and which signals initiate the dedifferentiation/redifferentiation process is not well understood. The available evidence is indirect or correlative. It also is not in agreement with other studies, which showed that ciliated cells do not give rise to goblet cells during airway remodeling in rodents and humans and with studies that provide evidence for increased goblet cell proliferation and goblet cell hyperplasia (Pardo-Sargenta et al., 2013; Hays et al., 2006; Lawson et al., 2002; Tesfaigzi et al., 2004; Taniguchi et al., 2011; Park et al., 2006; Turner et al., 2011).
A 30-min treatment of primary human bronchial epithelial cells at the air-liquid interface with 0.6 mM xanthine and 0.5 units xanthine oxidase resulted in a 2-fold increase in EGFR phosphorylation. Daily 30-min treatments of primary human bronchial epithelial cells at the air-liquid interface with 0.6 mM xanthine and 0.5 units xanthine oxidase for 3 days resulted in goblet cell metaplasia as evidenced by an increase in the numbers of MUC5AC-positive cells from 3.3 ± 1.2%to 21.6 ± 3.4%, a decrease in ciliated cell numbers, and increased MUC5AC protein expression (32.5 + 9.3% above PBS control). This effect could be inhibited by EGFR blockade with neutralizing antibodies (Casalino-Matsuda et al., 2006).
Cigarette smoke exposure at 8 cigarettes (nonfiltered cigarettes; 1.2 mg nicotine, 12 mg condensate) per day for 5 days markedly increased AB/PAS staining in airway epithelia of male Sprague-Dawley rats and goblet cell numbers from 40 ± 19 to 167 ± 19 cells/mm of epithelium, while decreasing the number of ciliated cells (not quantified). Treatment with the EGFR inhibitor BIBX1522 during exposure dose-dependently decreased goblet cell numbers, with a maximal decrease seen for 3 mg/kg inhibitor (51 ± 19 cells/mm epithelium) (Takeyama et al., 2001).
Intranasal insitillation of 0.1 mg LPS (E.coli 0111:B4) once a day for 3 consecutive days induced goblet cell metaplasia in the nasal epithelium (as judged by histopathology), with an approx. 50% increase in AB/PAS-stained epithelium compared to untreated controls. Intranasal insitllation of AG1478 1 hr after LPS instillation dose-dependently decreased the % AB/PAS-stained epithelium, with a maximal decrease seen at 10 mg/kg (Takezawa et al., 2016).
Induction of airway inflammation with 50 µg house dust mite (1.27 endotoxin units/mg) for 5 days/week for 3 weeks resulted in a 3-fold increase of pEGFR-positive cells in the bronchiolar epithelium of C57Bl/6 mice. Six-week treatment led to goblet cell metaplasia as evidenced by extensive AB staining and an approx. 10-fold increase in Clca3-positive cells in the animals' airways. Concomitant treatment with 100 mg/kg erlotinib six times a week for 6 weeks reduced the number of Clca3-positive cells by ca. 5-fold (Le Cras et al., 2011). Using the same model with a 3-week treatment demonstrated goblet cell metaplasia as judged by increased PAS staining in the airway epithelium and ca. 10-, 5-, and 4-fold increases in expression of goblet cell metaplasia-related genes Muc5ac, Clca1, and Postn, respectively (Habibovic et al., 2016).
Pyocyanin, a redox-active exotoxin of Pseudomonas aeruginosa, caused goblet cell metaplasia in C57Bl/6 mice after 3-week treatment (25 µg/day). PAS staining increased by ca. 30%; the percentage of Muc5ab-positive cell in bronchial epithelium increased 6.4-fold and in bronchiolar epithelium 11.4-fold. This was accompanied by increased EGFR phosphorylation coincident with AB/PAS staining. Moreover, 24-h pyocyanin treatment of H292 lung cancer cells and immortalized human bronchial epithelial 16-HBE cells with physiologically relevant concentrations from 1.3 to 25 µg/mL pyocyanin significantly increased MUC5B mRNA expression 3.8- to 13.4-fold and increased levels of pEGFR 11.8- to 18.3-fold (1.6 to 12.5 µg/mL pyocyanin) (Hao et al., 2012).
Male Sprague–Dawley rats that were exposed to 3 ppm acrolein for 6 h a day, for 12 days developed goblet cell metaplasia (as judged by histopathology), increasing the % AB/PAS-positive stained epithelium from ca. 5% (in air controls) to 35%. This was accompanied by a 1.6-fold change in EGFR phosphorylation, a nearly 15% increase in Muc5ac-positive stained cells, a ca. 3-fold increase in Muc5ac mRNA expression and a ca. 4-fold increase in protein expression (Chen et al., 2010).
Exposure of BALB/c mice to 1.0 ppm O3 , but not lower concentrations, for 3 h a day, for 7 days, caused goblet cell metaplasia in the bronchial epithelium (as judged by histopathology). Exposure to 0.25, 0.5, and 1.0 ppm O3 also increased levels of p-EGFR (Y1068) in the bronchial epithelium and lung tissues in a dose-dependent manner, with the maximal (an approx. 2-fold) increase over controls reached at 0.5 ppm (Feng et al., 2016).
Exposure of female Sprague-Dawley rats to wood smoke (40 g of China fir sawdust was smoldered) for 1 h four times per day, five days per week, for three months caused goblet cell metaplasia in the airways (as judged by histopathology), a 2-fold increase in Muc5ac gene expression, an increase in the % AB/PAS-positive stained epithelium from approx. 6% (air controls) to ca. 17%, an increase in Muc5ac-positive stained cells from approx. 5% (air controls) to ca. 25%. EGFR activation by wood smoke particle exposure was confirmed in vitro: Treatment of H292 lung cancer cells with wood smoke particulate matter (0.5 to 24 μg/mL) for 24 h increased MUC5AC gene and protein expression in a dose-dependent manner, with maximal increases of ca. 40-fold and 5-fold, respectively, seen for 8 μg/mL. Treatment of H292 lung cancer cells with 8 μg/mL wood smoke particulate matter for various times (2–36 h) increased MUC5AC gene and protein expression in a time-dependent manner, with significant increases starting at 24 h. Treatment of H292 lung cancer cells with 8 μg/mL wood smoke particulate matter for up to 24 h also increased EGFR phosphorylation in a time-dependent manner, with a significant and sustained increase (approx. 3-fold compared to control) seen from 1 h onwards. Pretreatment with 0.5 μg/mL neutralizing anti-EGFR antibody for 1 h completely abrogated the increases in EGFR phosphorylation and MUC5AC gene expression (Huang et al., 2017).
Exposure of male Sprague-Dawley rats to smoke from five cigarettes (2R4F, University of Kentucky) a day for 5 days resulted in goblet cell metaplasia in the airways (as judged by histopathology) and an approx. 70% increase in AB/PAS-stained epithelium. EGFR activation by cigarette smoke exposure was confirmed in vitro: Treatment of NCI-H292 lung cancer cells with cigarette smoke extract (2R4F cigarette smoke was withdrawn into a 35-mL polypropylene syringe at a rate of one puff/min and then bubbled slowly into 20 mL of RPMI 1640 medium containing 50mM HEPES buffer) for 15 min increased EGFR phosphorylation, and dose-dependently increased MUC5AC protein expression, with a significant increase starting from 3 puffs (ca. 130% over control) and a maximum increase of nearly 250% over control seen with 9 puffs (Lee et al., 2006).
Intratracheal instillation of LPS (P. aeruginosa serotype 10; 200 or 300 μg in 300 μL PBS) in male Sprague-Dawley rats caused goblet cell metaplasia in the airways, with 42.31 ± 3.36, 45.46 ± 2.24, and 63.13 ± 4.6% AB/PAS-positive staining at 3, 5, and 7 days after low-dose LPS instillation, respectively, and 71.6 ± 2.56% AB/PAS-positive staining at 7 days after high-dose LPS instillation. MUC5AC protein expression in the bronchial epithelium of the control and LPS groups (300 μg, 7 days post-instillation) were 5.46 ± 4.68 and 75.32 ± 4.53, respectively, and the mean % area of bronchiolar epithelium showing EGFR-positive staining in the LPS group was 24.54 ± 5.78% (compared to absent staining in the control) (Kim et al., 2004).
Instillation of agarose plugs (0.7-0.8 mm diameter, 4% agarose II) in Fischer rats caused a time-dependent increase in goblet cell area (by AB/PAS staining), which was detectable as early as 24 h and was greatest 72 h post-instillation. The AB/PAS-stained area increased from 0.1 ± 0.1% in control animals to 4.7 ± 1.4, 13.3 ± 0.7, and to 19.1 ± 0.7% at 24, 48, and 72 h post-instillation, respectively. Goblet cell numbers increased from 0 to 13.1 ± 5.6, 25.7 ± 15.0, and 51.5 ± 9.0 cells/mm basal lamina at 24, 48, and 72 h post-instillation, respectively. Treatment of the animals prior and after instillation with 80 mg/kg/day BIBX1522 resulted in a marked decrease in the AB/PAS-stained area (<5% at 72 h). Of note, the AB/PAS staining in the airway epithelia coincided with EGFR staining (Lee et al., 2000).
Two mouse studies demonstrated ciliated cell transdifferentiation and goblet metaplasia in response to viral infection and/or IL-13 treatment (Tyner et al., 2006; Fujisawa et al., 2008). Indirect evidence is also available from rat studies and studies on human cells and clinical samples.
Breuer, R., Zajicek, G., Christensen, T.G., Lucey, E.C., and Snider, G.L. (1990). Cell Kinetics of Normal Adult Hamster Bronchial Epithelium in the Steady State. Am. J. Respir. Cell Mol. Biol. 2, 51–58.
Casalino-Matsuda, S., Monzón, M., and Forteza, R. (2006). Epidermal Growth Factor Receptor Activation by Epidermal Growth Factor Mediates Oxidant-Induced Goblet Cell Metaplasia in Human Airway Epithelium. Am. J. Respir. Cell Mol. Biol. 34, 581–591.
Chen, Y.-J., Chen, P., Wang, H.-X., Wang, T., Chen, L., Wang, X., et al. (2010). Simvastatin attenuates acrolein-induced mucin production in rats: involvement of the Ras/extracellular signal-regulated kinase pathway. Intl. Immunopharmacol. 10, 685-693.
Curran, D.R., and Cohn, L. (2010). Advances in mucous cell metaplasia: a plug for mucus as a therapeutic focus in chronic airway disease. Am. J. Respir. Cell Mol. Biol. 42, 268-275.
Donnelly, G.M., Haack, D.G., and Heird, C.S. (1982). Tracheal epithelium: cell kinetics and differentiation in normal rat tissue. Cell Tissue Kinet. 15, 119–130.
Feng, F., Jin, Y., Duan, L., Yan, Z., Wang, S., Li, F., et al. (2016). Regulation of ozone-induced lung inflammation by the epidermal growth factor receptor in mice. Environ. Toxicol. 31, 2016-2027.
Fujisawa, T., Ide, K., Suda, T., Suzuki, K., Kuroishi, S., Chida, K., et al. (2008). Involvement of the p38 MAPK pathway in IL‐13‐induced mucous cell metaplasia in mouse tracheal epithelial cells. Respirology 13, 191-202.
Habibovic, A., Hristova, M., Heppner, D.E., Danyal, K., Ather, J.L., Janssen-Heininger, Y.M., Irvin, C.G., Poynter, M.E., Lundblad, L.K., and Dixon, A.E. (2016). DUOX1 mediates persistent epithelial EGFR activation, mucous cell metaplasia, and airway remodeling during allergic asthma. JCI Insight 1, e88811.
Hao, Y., Kuang, Z., Walling, B.E., Bhatia, S., Sivaguru, M., Chen, Y., Gaskins, H.R. and Lau, G.W. (2012). Pseudomonas aeruginosa pyocyanin causes airway goblet cell hyperplasia and metaplasia and mucus hypersecretion by inactivating the transcriptional factor FoxA2. Cell Microbiol. 14, 401-415.
Hays, S.R., and Fahy, J.V. (2006). Characterizing mucous cell remodeling in cystic fibrosis: relationship to neutrophils. Am. J. Respir. Crit. Care Med. 174, 1018-1024.
Huang, L., Pu, J., He, F., Liao, B., Hao, B., Hong, W., et al. (2017). Positive feedback of the amphiregulin-EGFR-ERK pathway mediates PM2.5 from wood smoke-induced MUC5AC expression in epithelial cells. Sci. Rep. 7, 11084.
Kim, S., Shim, J., Burgerl, P., Ueki, I., Dao-Pick, T., Tam, D., and Nadel, J. (2002). IL-13-induced Clara cell secretory protein expression in airway epithelium: role of EGFR signaling pathway. Am. J. Physiol. Lung Cell. Mol. Physiol. 283, L67–L75.
Kim, J.H., Lee, S.Y., Bak, S.M., Suh, I.B., Lee, S.Y., Shin, C., Shim, J.J., In, K.H., Kang, K.H., and Yoo, S.H. (2004). Effects of matrix metalloproteinase inhibitor on LPS-induced goblet cell metaplasia. Am. J. Physiol. Lung Cell. Mol. Physiol. 287, L127-L133.
Lawson, G.W., Van Winkle, L.S., Toskala, E., Senior, R.M., Parks, W.C., and Plopper, C.G. (2002). Mouse strain modulates the role of the ciliated cell in acute tracheobronchial airway injury-distal airways. Am. J. Pathol. 160, 315–327.
Le Cras, T.D., Acciani, T.H., Mushaben, E.M., Kramer, E.L., Pastura, P.A., Hardie, W.D., Korfhagen, T.R., Sivaprasad, U., Ericksen, M., Gibson, A.M. and Holtzman, M.J., 2010. Epithelial EGF receptor signaling mediates airway hyperreactivity and remodeling in a mouse model of chronic asthma. Am. J. Physiol. Lung Cell. Mol. Physiol. 300, L414-L421.
Lee, H.-M., Takeyama, K., Dabbagh, K., Lausier, J.A., Ueki, I.F., and Nadel, J.A. (2000). Agarose plug instillation causes goblet cell metaplasia by activating EGF receptors in rat airways. Am. J. Physiol. Lung Cell. Mol. Physiol. 278, L185-L192.
Lee, S.Y., Kang, E.J., Hur, G.Y., Jung, K.H., Jung, H.C., Lee, S.Y., et al. (2006). The inhibitory effects of rebamipide on cigarette smoke-induced airway mucin production. Respir. Med. 100, 503-511.
Lumsden, A.B., McLean, A., and Lamb, D. (1984). Goblet and Clara cells of human distal airways: evidence for smoking induced changes in their numbers. Thorax 39, 844-849.
Pardo-Saganta, A., Law, B.M., Gonzalez-Celeiro, M., Vinarsky, V., and Rajagopal, J. (2013). Ciliated cells of pseudostratified airway epithelium do not become mucous cells after ovalbumin challenge. Am. J. Respir. Cell Mol. Biol. 48, 364–373.
Park, K.-S., Wells, J.M., Zorn, A.M., Wert, S.E., Laubach, V.E., Fernandez, L.G., and Whitsett, J.A. (2006). Transdifferentiation of ciliated cells during repair of the respiratory epithelium. Am. J. Respir. Cell Mol. Biol. 34, 151–157.
Rawlins, E.L., and Hogan, B.L.M. (2008). Ciliated epithelial cell lifespan in the mouse trachea and lung. Am. J. Physiol. Lung Cell. Mol. Physiol. 295, L231–L234.
Reader, J.R., Tepper, J.S., Schelegle, E.S., Aldrich, M.C., Putney, L.F., Pfeiffer, J.W., and Hyde, D.M. (2003). Pathogenesis of mucous cell metaplasia in a murine asthma model. Am. J. Pathol. 162, 2069-2078.
Shim, J.J., Dabbagh, K., Ueki, I.F., Dao-Pick, T., Burgel, P.R., Takeyama, K., Tam, D.C., and Nadel, J.A. (2001). IL-13 induces mucin production by stimulating epidermal growth factor receptors and by activating neutrophils. Am. J. Physiol. Lung Cell. Mol. Physiol. 280, L134–L140.
Shimizu, T., Takahashi, Y., Kawaguchi, S., and Sakakura, Y. (1996). Hypertrophic and metaplastic changes of goblet cells in rat nasal epithelium induced by endotoxin. Am. J. Respir. Crit. Care Med. 153, 1412-1418.
Takezawa, K., Ogawa, T., Shimizu, S., and Shimizu, T. (2016). Epidermal growth factor receptor inhibitor AG1478 inhibits mucus hypersecretion in airway epithelium. Am. J. Rhinol. Allergy 30, e1-e6.
Taniguchi, K., Yamamoto, S., Aoki, S., Toda, S., Izuhara, K., and Hamasaki, Y. (2011). Epigen is induced during the interleukin-13–stimulated cell proliferation in murine primary airway epithelial cells. Exp. Lung Res. 37, 461-470.
Tesfaigzi, Y., Harris, J.F., Hotchkiss, J.A., and Harkema, J.R. (2004). DNA synthesis and Bcl-2 expression during development of mucous cell metaplasia in airway epithelium of rats exposed to LPS. Am. J. Physiol. Lung Cell. Mol. Physiol. 286, L268-L274.
Turner, J., Roger, J., Fitau, J., Combe, D., Giddings, J., Heeke, G.V., and Jones, C.E. (2011). Goblet cells are derived from a FOXJ1-expressing progenitor in a human airway epithelium. Am. J. Respir. Cell Mol. Biol. 44, 276–284.
Tyner, J., Tyner, E., Ide, K., Pelletier, M., Roswit, W., Morton, J., Battaile, J., Patel, A., Patterson, G., Castro, M., et al. (2006). Blocking airway mucous cell metaplasia by inhibiting EGFR antiapoptosis and IL-13 transdifferentiation signals. J. Clin. Invest. 116, 309–321.
Yoshisue, H., and Hasegawa, K. (2004). Effect of MMP/ADAM inhibitors on goblet cell hyperplasia in cultured human bronchial epithelial cells. Biosci. Biotechnol. Biochem. 68, 2024–2031.


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Football player's injury makes heart condition discussions prominent
Dave Dawson, Reporter
A vigil at the University of Cincinnati Medical Center was created for football player Damar Hamlin of the Buffalo Bills after he collapsed following a tackle during the game against the Cincinnati Bengals.
When Damar Hamlin slumped to the ground Monday during the Buffalo Bills' game against the Cincinnati Bengals, America's heart collectively skipped a beat.
Hamlin, a safety, suffered cardiac arrest and collapsed after making a tackle during the first quarter of the game. The drama was broadcast live as EMTs rushed to the field and started CPR. Hamlin, 24, was in critical condition Tuesday.
"His heartbeat was restored on the field, and he was transferred to the (University of Cincinnati) Medical Center for further testing and treatment," the Buffalo Bills' Twitter account said. "He is currently sedated and listed in critical condition."
Hamlin was hurt after tackling Cincinnati wide receiver Tee Higgins on a seemingly routine play that didn't appear unusually violent, the Associated Press reported. Hamlin quickly got to his feet, appeared to adjust his face mask with his right hand, and then fell backward about three seconds later and lay motionless.
Predictably, after such a widely viewed event, people took to social media to exchange some confusing and inaccurate views of what happened. First, a clarification of terminology is in order.
People often use the terms cardiac arrest, heart attack and heart failure interchangeably, but they're not the same, according to Dr. Fahmi Farah, a cardiologist and director of Bentley Heart in Fort Worth, Texas.
A big difference between the heart failure and cardiac arrest is the latter comes on suddenly and without warning, Farah said. Heart failure occurs gradually. A person can have it for years before they notice symptoms.
"A heart attack is when blood flow to the heart is blocked inside an artery and obstructs blood flow. When that happens, that part of the heart muscle dies. If not addressed quickly and correctly, that person can die," Farah said.
Sudden cardiac arrest is when the heart malfunctions and suddenly stops beating, Farah said. A heart attack is a "circulation" problem and sudden cardiac arrest is an "electrical" problem.
"Cardiac arrest can happen because of multiple things. Your heart is not working at that moment. Essentially, it means you're medically dead until revived by CPR," Farah said.
She said in Hamlin's case it was crucial to restart the heart and resume the process of normal pumping. Farah said the immediate response by EMTs improved chances for recovery.
Farah said the likeliest causes of cardiac arrest are congenital or from a sudden jolt.
"Cardiac arrest can happen for non-cardiac reasons. Not being able to breathe for some reason, or an electrolyte abnormality such as caused by heavy drinking, and the heart is not beating regularly or very well," Farah said.
"It could be structural, such as when a person is born with a thickened heart muscle. And they can be revealed in situations involving exercise when someone could go into arrythmia," Farah said. "Congenital heart defects are a reason young athletes have heart problems."
Farah said if the hit was strong enough – equivalent to getting hit when an airbag inflates – it can cause blood vessels to burst. Or it could cause blood vessels to constrict causing ventricular tachycardia, which is a heart rhythm problem caused by irregular electrical signals in the lower chambers of the heart.
"Typically, if it is one of those two things, then there are no warning signs. Usually it happens during sports," Farah said.
"It's one of the reasons why a person playing at a high level of competitive sports goes through a thorough cardiac exam. Only an echocardiogram would detect something like a congenital condition," Farah said.
An echocardiogram uses sound waves to produce images of your heart. This common test allows the doctor to see a heart beating.
According to the Centers for Disease Control, sudden cardiac arrest in young people is extremely rare. About 2,000 seemingly healthy people under 25 die in the United States each year from sudden cardiac arrest.
Signs of sudden cardiac arrest are immediate and drastic and include collapse, no pulse, and no breathing, according to the American Heart Association.
Symptoms of a heart attack include chest pain or discomfort; feeling weak, light-headed, or faint; pain or discomfort in the jaw, neck, or back; pain or discomfort in one or both arms or shoulders; and shortness of breath, the association said.
The heart association also suggest these lifestyle changes to reduce the risk of a heart attack including stop smoking; choose good nutrition; reduce high cholesterol; lower high blood pressure; be physically active every day; and aim for a healthy weight.
Dave Dawson


text:
Couple Likes Being 'Boxed In'
Fri May 20, 2016 - National Edition
The Blackwells say that there are many misconceptions about shipping container homes.
ONEONTA, Ala. (AP) - Upon meeting Bryan and Gwen Blackwell, it's easy see that the pair is accustomed to thinking outside the box. So, it's a little ironic that the couple lives in one.
Well, four to be exact.
The Alabama couple designed and built their 2000-square-foot home out of four 40-foot-long shipping containers that came from a port in Miami. They use a fifth container for storage.
When the construction industry started to tank around eight years ago, they decided to make a change. They sold the building that housed their steel construction business in Florida and decided to move to Oneonta, Alabama, in the summer of 2009 where they owned land.
Bryan moved the containers one by one from Fort Myers, Florida, also transporting the steel beams, and other materials needed for the house. All of the steel used in the home are leftovers from jobs they did back in Florida.
So why use containers? "It was just the next step in an adventure. Let's go from one thing to another. Every time, we'll try something a little more creative," said Gwen. "This was the next step."
Shipping container structures are incredibly strong, durable, and eco-friendly. The giant building blocks made perfect sense for their unique home.
"I've wanted to build one of these long before they ever started showing up on the internet, so we did it," said Bryan. "It's the strongest method of construction there is right now other than poured concrete."
They lived in a travel trailer for two years while they put the home together themselves. They moved in shortly after completing the necessities - the master bath, bedroom, and kitchen.
The Blackwells say that there are many misconceptions about shipping container homes. One they hear often is how inexpensive an elaborate shipping container build is. "They know you can buy a shipping container for cheap, but then they think that's the end of it. They don't realize how much work it takes to modify one and the expense that comes with it." said Bryan. "It's like buying a truckload of lumber and expecting that to be all of your home costs."
Doing it themselves allowed them the flexibility to do whatever they wanted and the time to work on it, think about it, and work on it some more while adding creative touches as they thought of them. They finished the rest a year later.
"I don't know if you can rush through something like this because we kind of ad-libbed as we went," said Gwen.
Take for instance some of the interior features. They repurposed discontinued ceiling tiles they bought from several area Lowes stores to cover bedroom and bathroom walls. Stained plywood trim frames doors in the home, most of which are recessed. Inexpensive clip lights wired together create industrial lighting in what they call the gallery, the home's main gathering space. They converted an old propane tank they got for free into a ceiling mounted fireplace that heats the entire space. The even used automotive parts pallets to make up parts of the gallery walls.
The home sits atop eight steel posts with footings buried in concrete so while it is not tornado proof - "You can't build anything that god can't tear up," said Bryan- it is storm resistant.
All of the touches come together to form a home that is as unique and creative as the couple who lovingly put it together. "It makes it more interesting don't you think?" said Gwen. "Why build it like everybody else? Where's the fun in that?"
Project Experiences


Although certainly not as common as Pterostylis nutans Nodding Greenhood that was featured in last month's newsletter, P. curta Blunt Greenhood is another species that forms quite large eye-catching colonies in the district.
The robust flowers with a blunt hood and a strongly twisted labellum distinguish this greenhood from all others.
It has a rosette of up to 6 stalked leaves and a flowering stem to 30cm tall bearing a solitary, erect to semi-nodding flower that is translucent white with green stripes. The brown, strongly twisted labellum has a raised central ridge and can be clearly seen through the wide frontal opening. The Aireys Inlet area is the best place to explore for this species, with some large colonies being found on residential blocks. The reserve next to the Aireys Inlet school has a small colony flowering at the present time.
There are many other species of greenhoods that you may find in flower. These include P. concinna Trim Greenhood, P. melagramma Tall Greenhood, P. nana Dwarf Greenhood, P. nutans Nodding Greenhood, and P.pedunculata Maroonhood.
The attractive brown and yellow Diuris pardina Leopard Orchids and the purple Acianthus caudatus Mayfly Orchids with their long slender sepals are in flower, as are the two species of Gnat Orchids Cyrtostylis reniformis and C. robusta. While most of the Helmet Orchids have finished flowering, you may still find some flowers of the Corybas incurvus Slaty Helmet Orchid as you walk the tracks. As we said last month it is a great time to be exploring in the field.
Photographs and descriptions of all of these orchids can be found in Orchids of the Anglesea District available from ANGAIR.


Recce "Squad" is a company within the paramilitary General Service Unit (GSU) organization. The headquarters of Recce Company is located in Ruiru. The principal function of the company is to support and reinforce the field companies during combat or security operations. The company can also be deployed independently to perform sensitive and delicate security operations. By virtue of their elite training and combat experience, the company can also be deployed into the theater of war as a wholly functional combat unit. Their specialist Crisis Response training has come in handy during hostage-barricade sieges such as the 2013 Westgate Shopping Mall attack and the Garissa attack (where the company was credited for ending the terrorist siege).
Recce company is composed of well-trained elite soldiers and as such they are highly regarded within the military fraternity. The soldiers are trained both within Kenya and overseas with specialist training taking place in Israel, United States and the United Kingdom. For that reason, the company is considered a specialist unit of Commandos comparable to their peers in the British SAS, Israel's Sayeret Matkal and America's Delta Force.
Recce company is made up of three units: Sky Marshal unit, Crisis response team and the Rendition operation team. Sky Marshal unit is a covert counter-terrorism unit whose officers are embedded on board commercial planes so as to avert, and if necessary, counter plane hijackings. The crisis response team serves as the standby unit that swiftly intervenes in situations which threatens to turn either unpredictable or volatile. The officers of the rendition operation team are exclusively trained in the US and Israel.
Officers of the Recce Company are equipped with the appropriate military equipments including a bullet proof vest with attached magazine pouches which protects the torso from high-velocity projectiles as well as provides carrying space for additional magazines. Recce Commandos are armed with M16 fully automatic (or their semi-automatic equivalent) rifles and additional pistols carried on thigh pistol holsters. The officers also wear sunglasses to conceal their gaze from the public while their chest- or shoulder-fixed portable police radios with attached earphones facilitates smooth communication in the field. For close-quarter combat, the commando is provided with an bayonet knife or a stubby spear-shaped weapon while the knee an shoulder pads provide comfort and protection during combat operations.
The Recce Squad recruits young, healthy and physically fit Kenyans during the nationwide National Police Service (NPS) recruitment drive. The selected candidates then attend basic GSU training at the GSU training school located in Embakasi. The best performers in academics and field operations thereafter undergo a 7 month specialist training in rescue operations, VIP protection, anti-terrorism, critical infrastructure protection, sky marshal and explosive handling. After the completion of their training, the cadets pass out and are deployed to either one of the three units.
I didn't know we had Sky Marshals.


Louise has really helped me with my acne, scarring, and all the other issues I was having. What she has done for me is life changing! Highly recommend!
I can not say enough about Louise Enkermann's services. She really knows what she is doing. She got me ready for my wedding by waxing my eyebrows beautifully, giving me a "acne facial" that was smooth for months and later she gave me an incredible facial. My skin and brows never looked better. I routinely go for waxing and I would not return to someone who wasn't doing it right (I'm very picky about my brows). Her bikini wax was amazing as well, it lasted for weeks. Massages are awesome. You really can't go wrong with any of her services. She's part of the reason the breakouts on my back cleared up so well. It's easy to book and you can go online to do it. Her pricing is fair as well. I love coming in to see Louise. I wouldn't trust anybody else at this point. Don't get offended if she tells you no about a service you want that day, it's for your own good! She'll ask questions and instruct you before she does anything, so you're in safe hands. She's the best.
If you're looking for a average fluff and puff facial, SHES not IT! She really makes a difference in your skin, from acne to spots. She makes your face not only feel wonderful but beautiful as well. I have very sensitive skin and the products she uses are awesome.
I have been a client of hers for over 10 years. Thank you Louise.
I have always assumed I had oily skin, because of oil on T-zone and constant acne, Louise informed me I had dry skin and to stop using my astringent and start moisturizing regularly… I have gotten maybe one zit a month and my skin has never looked better!! Also, best place to get a facial, wax or sugaring while you're in!!
I have been a client of Louise for many years. She is simply the best for all skincare needs in the metroplex. Louise is also ALWAYS up on the latest technologies, is kind, thoughtful, and extremely helpful. In addition, she has several hair removal options. I have always used her for waxing but she also offers threading and sugaring. And extremely clean and sanitary!
I have been a client for over 15 years and Louise is awesome. From massages to waxing she will take care of all your needs!


The 2019-20 Idaho big game hunting brochure is now available online. It contains seasons and rules for deer, elk, pronghorn, black bear, mountain lion and gray wolf, will be available in print at license vendors and Fish and Game offices by May 1, 2019.
Hunters should note that the Idaho Hunt Planner does not yet reflect the new seasons and rules, but will be updated by mid-April.
Applications for controlled hunts for deer, elk, pronghorn and fall black bear runs May 1 through June 5. Hunters are reminded that controlled hunt applications are not accepted via mail.


since I found a lot of answers regarding the question how to consume a web service within Unity. But now I have the scenario that I wanna realize a web service in Unity so that a web application on any server, for example in the cloud or in a network, could consume the web service in Unity.
1) Is it possible to realize a web service in Unity?
3) If no, is there any other possibility to realize anything similar?
A wild guess would be that - yes, you can do that, unless Unity is blocking the Ports or using a old C# version.
Have a look at https://support.microsoft.com/en-us/help/308359/how-to-write-a-simple-web-service-by-using-visual-c--net or just search for "c# web service" as it's more of a programming thing than anything else.
You could always just do a simple C# project instead (which is basically what servers run for the most part anyway) instead of having a Server run inside Unity unless that's your goal.


† Rate applies for new loans when you borrow up to 70% of the property value with an owner occupied, principal and interest repayment variable rate loan. Subject to change without notice.
Unless stated otherwise, all information on this page is provided by Macquarie Bank Limited ABN 46 008 583 542 AFSL and Australian Credit Licence 237502.
Finance provided by Macquarie Leasing Pty Limited ABN 38 002 674 982 (Australian Credit Licence No. 394925) to approved personal applicants (Macquarie staff and family only), subject to its credit assessment criteria. Standard terms and conditions apply and are available on application.
Information and interest rates are current as at 17 January 2019 and subject to change.
50c cash out reward (Macquarie debit MasterCards only): when you withdraw $100 or more in a single transaction at a point-of-sale (e.g. EFTPOS) terminal in Australia, we will credit $0.50 to your account. This offer applies for the first 200 eligible withdrawals per customer in a given financial year. These amounts may need to be included in income tax and social security calculations, depending on your circumstances. We recommend you seek independent advice if you need more information. From time-to-time we may change this offer or withdraw it. In circumstances where we reasonably determine that this offer is being used inappropriately we may decline to credit your account or require you to repay us an amount already credited to your account. This offer expires on 1 February 2018.
Purchase security cover is available where an eligible item is stolen or damaged within 90 days of making the purchase with your Macquarie Platinum Debit MasterCard® online, by phone, pressing the 'credit' button at an EFT terminal or tapping your card on a MasterCard® contactless terminal.
Wallet guard: provides a lump sum payment of up to $500 to replace valuables if your wallet or handbag is stolen. Terms, conditions, limits, exclusions and excesses apply. Cover provided by ACE Insurance Limited ABN 23 001 642 020, AFSL 239 687. Please refer to the Macquarie platinum debit card benefits for further information.
Emergency travel assistance: Macquarie has arranged for these services to be made available to cardholders by International SOS (Australasia) Pty Ltd ABN 83 052 247 104. These services are available 24 hours a day, seven days a week by calling +61 2 9372 2306. Eligibility criteria apply. You are liable to pay for any goods or services that you request. Please refer to the Macquarie platinum debit card benefits for further information.
Refer to section 9 (overdrawing your transaction account) of the Macquarie transaction and savings accounts terms and conditions for further information.
You must be a member of the Qantas Frequent Flyer program to earn and redeem Qantas Points. A joining fee usually applies, however MBL has arranged for this to be waived if you are a new applicant for a home loan that includes a flyer feature and you apply for membership online at qantas.com/joinffmacquarieflyer. Membership and the earning and redemption of Qantas Points are subject to the Qantas Frequent Flyer terms and conditions available at qantas.com/terms. Qantas Points, offered by Macquarie Bank Limited (MBL), accrue and will be credited to your Qantas Frequent Flyer account in accordance with your Home Loan terms and conditions. You may not earn Qantas Points if you are in arrears or default under your loan agreement, or during the construction period of a loan. Macquarie is not responsible for the administration of the Qantas Frequent Flyer program. Qantas Airways Limited remains at all times solely responsible for the administration of the Qantas Frequent Flyer program. Qantas has made no enquiries as to the accuracy of the Macquarie products or services described, and is not responsible for errors or omissions.
Flyer feature only available on Basic and Offset home loans. Terms, conditions and limitations apply.
Qantas has made no enquiries as to the accuracy of the Macquarie products or services described, and is not responsible for errors or omissions. Macquarie Bank Basic and Offset home loans are not Qantas products and are not offered or issued by Qantas but by Macquarie as Servicer.
25,000 Qantas Points on the third and fifth anniversaries of your loan, calculated from the day of settlement, as long as the outstanding loan balance is at least $150,000 at the time of the anniversary.
This offer is available for new Macquarie home loan applications only.
^^ The credit card product you are issued will be determined by your final approved credit limit. Annual fees are payable if the home loan is no longer held. Existing Macquarie credit card holders will retain their existing cards with the annual fee waived whilst the home loan is held. For information on credit card fees and charges, please visit Fees and charges.
** After the promotional period, the standard variable interest rate for purchases will apply, which is currently 15.95% pa for Macquarie RateSaver and 20.70% pa for Macquarie Platinum and Macquarie Black.
i $0 annual fee for the first year on any Macquarie Credit Card - primary or additional cardholder annual fees may be introduced or varied in future. Other fees and charges may apply.
2 The 0% pa interest rate applies to balances transferred with this offer for the first 14 months from the date your account is approved and then reverts to the standard variable interest rate for purchases, which is currently 20.70% pa for Macquarie Black Card, 20.70% pa for the Macquarie Platinum Card and 15.95% pa for the Macquarie RateSaver Card. Interest rates are subject to change and Macquarie Card Services credit criteria. Minimum repayments are required. Card repayments will be applied last to the repayment of transferred balances that are in the promotional interest rate period. For further details refer to the Balance Transfer Terms and Conditions.
3 Macquarie Reward points are earned on up to $200,000 of spend annually on the Macquarie Platinum Card and no points cap on the Macquarie Black Card. Macquarie Rewards are subject to the Macquarie Rewards Program Terms and Conditions. Macquarie Reward points are not earned on cash advances, balance transfers or other restricted transactions detailed in the Macquarie Rewards Program Terms and Conditions.
4 Concierge Services are provided by International SOS (Australasia) Pty Ltd, trading as Aspire Lifestyles. The booking must be made through the concierge service and the cost of transfer must be paid for with your Macquarie Black Card. The discount will be applied at the time of booking. If a booking is cancelled within 24 hours of the scheduled time, you will still be charged for the booking . Limousine travel is subject to availability.
5 Overseas Travel Insurance, Interstate Flight Inconvenience Insurance, Transport Accident Insurance, Purchase Security Insurance, Extended Warranty Insurance and Global Hire Car Excess Waiver are underwritten by Chubb Insurance Limited (ABN 23 001 642 020, AFSL No. 239687) (Chubb) and are subject to the terms, conditions and exclusions contained in the Macquarie Black Card policy of insurance or Macquarie Platinum Card policy of insurance between Macquarie Bank Limited (ABN 46 008 583 542 AFSL No. 237502) (MBL) and Chubb. This brochure doesn't take into account your objectives, financial situation or needs. It is important for you to read Macquarie Black Card Insurance Terms and Conditions available by calling 1300 150 300 and consider the appropriateness of that insurance in relation to your individual requirements.
6 Qantas Points are earned in accordance with and subject to the Macquarie Qantas Rewards Program Terms and Conditions, available at Macquarie Online Banking and macquarie.com.au/cards. You will earn 1 Qantas Point per $1 spent on eligible purchases on the Macquarie Platinum Card on up to $1,000 spend per statement period, then 0.5 Qantas Points per $1 spent on eligible purchases for the remainder of that statement period. Qantas Points are earned on up to an annual maximum of $200,000 of eligible spend on the Macquarie Platinum Card. You will earn 1 Qantas Point per $1 spent on eligible purchases on the Macquarie Black Card on up to $4,000 spend per statement period, then 0.5 Qantas Points per $1 spent on eligible purchases for the remainder of that statement period with no points cap. Qantas Points are not earned on cash advances, balance transfers or other restricted transactions detailed in the Macquarie Qantas Rewards Program Terms and Conditions. You must be a member of the Qantas Frequent Flyer program to earn and redeem Qantas Points. Complimentary membership is provided by Macquarie Bank Limited for new or existing Macquarie Platinum and Macquarie Black cardholders who are not already members of the Qantas Frequent Flyer Program and who apply for membership online at qantas.com/joinffmacquarie. This offer is non-transferable and not available in conjunction with any other offer. Qantas Frequent Flyer membership and each application is subject to approval by Qantas. Membership and the earning and redemption of Qantas Points are subject to the Qantas Frequent Flyer Program terms and conditions. Find out more about the Qantas Frequent Flyer Program at qantas.com/terms or call 13 11 31.
7 Applicable only if your account is paid in full by the due date shown each month (including any balance transfers but excluding any Interest Free Finance balances that are in the specified promotional period).
8 Visa's Zero Liability policy covers Australian and New Zealand-issued cards and does not apply to ATM transactions, transactions not processed by Visa or certain commercial card transactions. Cardholders should notify their issuer promptly of any unauthorised Visa use.
b The Qantas Frequent Flyer Loyalty Program is operated by Qantas Airways Limited ABN 16 009 661 901. Qantas Points only available to Qantas Frequent Flyer Members when consumer finance is obtained directly through Macquarie Leasing, and only up to a maximum of 100,000 points per finance contract. Not available where you have been referred or introduced to Macquarie Leasing by or through a motor vehicle dealer, introducer or third party, or otherwise in conjunction with any other offers, including any credit card payment options. Terms and conditions apply. The accumulation of Qantas Points may incur taxes for which the Qantas Frequent Flyer member may be liable. We recommend you consult your accountant or tax adviser to ensure you understand possible tax implications, for example fringe benefits tax (if applicable). Redemption of Qantas Points for flights and other awards may incur taxes, fees, levies and charges even where no ticket fare or other acquisition cost is applicable, and it is the responsibility of the Qantas Frequent Flyer Member to pay these costs.
c Vehicle Select have no influence on Macquarie Leasing's lending decisions and vehicle sourcing is independent of any application for credit.
d Bonus Qantas Points offer is only available to Qantas Frequent Flyer members that acquire and finance a vehicle directly through Macquarie Leasing.
e A business must be a Qantas Business Rewards Member to earn Qantas Points for business. A one-off join fee of $89.50 including GST normally applies, however this will be waived for Macquarie Leasing customers. Membership and Qantas Points are subject to the Qantas Business Rewards Terms and Conditions. Qantas Points for business are offered under the Macquarie Leasing Terms and Conditions. Qantas Points will be credited to the Members' Business Rewards account within 14 days of purchase. Members will earn 1 Qantas Point for every $1 financed. Any claims in relation to Qantas Points under this offer must be made directly to Macquarie Leasing by calling 1300 732 878.Qantas Points can only be earned by Qantas Business Rewards Members when commercial finance is obtained through Macquarie Leasing directly or when an eligible Macquarie Leasing finance contract is obtained through an approved intermediary of Macquarie Leasing. A maximum of 150,000 Qantas Points per finance contract obtained through Macquarie Leasing directly and a maximum of 100,000 Qantas Points per intermediary introduced finance contract can be earned. Not available in conjunction with any other offers, including credit card payment options. Not available in conjunction with any other offers. Only available to Qantas Business Rewards members when commercial finance is obtained directly through Macquarie Leasing.
f Streamlined approval process available to eligible applicants for commercial vehicle and equipment finance through Macquarie Leasing Pty Ltd. Conditions regarding asset types, age of assets and where assets are sourced apply.


text:
package lruCache;

/**
 * Design and implement a data structure for Least Recently Used (LRU) cache. It should support the following operations: get and set.
 * <p/>
 * get(key) - Get the value (will always be positive) of the key if the key exists in the cache, otherwise return -1.
 * set(key, value) - Set or insert the value if the key is not already present. When the cache reached its capacity, it should invalidate the least recently used item before inserting a new item.
 */
public class LRUCache {
    private java.util.LinkedHashMap<Integer, Integer> lruMap;

    public LRUCache(int capacity) {
        final int cap = capacity;
        lruMap = new java.util.LinkedHashMap<Integer, Integer>(cap + 1, .75F, true) {
            @Override
            protected boolean removeEldestEntry(java.util.Map.Entry<Integer, Integer> eldest) {
                return size() > cap;
            }
        };
    }

    public int get(int key) {
        if (lruMap.containsKey(key)) {
            return lruMap.get(key);
        } else {
            return -1;
        }
    }

    public void set(int key, int value) {
        lruMap.put(key, value);
    }
}


age 74, of Amberg, died unexpectedly Wednesday January 30, 2019. He was born February 9, 1944, in Iron Mountain; son of Robert and Cecilia (Gravel) Flaws. Jerry enjoyed gardening, fishing, and the outdoors. He walked many miles with his dog Sport to Dave's Falls.
Jerry is survived by his niece, Charlene Bruette; brother, Bob (Phyllis Townsend) Flaws; and many nieces, nephews, and extended family. He was preceded in death by his parents; his sister, Jalaine; and brother, Eugene.
There will not be a service for Jerry.


text:
bs11000.com
the World's first national standard for Collaborative Business relationships
Partner selection
Staying together
The ICW
BS11000 > About BS11000
About BS11000
Collaborative Business Relationships
In business, as in other walks of life, teamwork can pay real dividends. Companies that work together can often achieve much more in collaboration than they can alone. Working in partnership with other organisations allows knowledge, skills and resources to be shared effectively – helping all involved to meet mutual goals.
Collaborative relationships also lead to better cost and risk management, as well as levels of innovation not normally achieved in a typical client-supplier relationship, thus enhancing competitiveness and performance.
What is BS 11000?
Helping you and your partners work better together…
BS 11000, the successor to the PAS 11000 management system standard, is a British Standard that provides a framework specification for creating and managing collaborative business relationships. It's all about the best way for businesses to collaborate – how they can work together most effectively, developing and managing their interactions with each other for maximum benefit to all.
Using an eight stage approach, BS 11000 is designed to enable organisations of any size and sector to apply best practice principles to their own ways of working, to get the most out of business relationships . BS 11000 gives guidelines on increasing joint efficiency, transparency and understanding around governance processes. It also includes advice on assessing and monitoring these partnerships through a relationship management plan.
BS 11000 maps the key areas which all organisations should address. It is recognised that every relationship is different and that the degree of application will vary within the overall framework. However, the key issues will be common to most. It is these key factors that BS 11000 captures and thus provides a common and consistent foundation for collaboration.
Who should buy BS 11000?
Organisations working within a supply chain
Organisations managing a supply chain
Those who want to improve efficiency and partnership working.
BSi Shop – Buy the Standard
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Valley Line LRT - Questions?
Here's your direct route to staying informed during LRT construction!
TransEd Partners makes every effort to minimize construction impacts on affected properties and communities.
In the unlikely event that distress or damage occurs to your property as a result of work related to Valley Line construction, please visit TransEd Partners' website for information about their construction damage claims process.