In 2007 we added a blog (known at the time as the FFG journal) to the site. Over time it's developed from "here's what I've been playing" and "there's a new game coming out" to longer pieces and features. To prevent the latter getting buried in the mists of time, we decided to link to them here.
Obviously this is an entirely subjective process, and one with fairly loose criteria, but the rest of the blog is still there to be searched should you wish to do so.
A selection of multi-part features.
The trend for retro emulation boxes set us wondering what games from the DOS era we'd bundle with a mini PC.
The trend for retro emulation boxes set us wondering what games from the DOS era we'd bundle with a mini PC. Which then set us wondering what games from the Windows 95 era we'd inclde in a follow-up bundle.
Rik used to provide an end of year update on site activity, on the basis that it might be interesting to someone, somewhere. It's a thought only recently abandoned, in the face of dwindling review numbers.
A four-part series in which Rik attempts to detail the joys of driving the less glamorous cars racing games have offered up over the years. (He drives a Honda Civic, with a dent in, so he should know).
Stoo outlines his struggles with various gaming baddies, mainly in the world of RPGs.
To celebrate 10 years of FFG, Stoo, Rik (and Jo) got together to re-evaluate and discuss some of the games covered on the site in its earlier years: Stunts, Puma World Football '98, Deus Ex and Broken Sword.
A regular series where we look back on gaming experiences that have left a particularly strong impression on us over the years, for good reasons or bad.
A reasonably detailed look back at our favourite gaming magazine, albeit an incomplete and subjective one.
We once had the Cupboard of Shame, where all the boxed games we bought but didn't play used to live, and now we have the Vault of Regret, in which we can store not only boxed games, but digital purchases, and sundry other gaming regrets and embarrassments. Here we write about all the games we should have played but haven't, or that we have played but didn't enjoy, among other things.
From the site's 15-year anniversary. Rik traces a path through early games and reviews in this series, in an attempt to say something about his personal history with games and this site, and how on earth we're still going somehow.
Any blog posts of sufficient significance to be given the slightly elevated status of 'article'.
A couple of brief pieces acknowledging the site's 10-year anniversary.
The 90s and 00s featured some fairly, er, 'edgy' ads. Here are some quite bad ones Rik found while raking through old copies of PC Zone.
Rik takes a look back at his own history with the venerable management series.
At one stage Rik developed an unhealthy obsession with CSI games. The best ones are on the Nintendo DS, though, and we can't write about them here. Or can we?
Rik and Stoo play through Bioware's space epic and indulge in a spoiler-tastic discussion. It's excluded from the review list for that reason, but now you've been warned, you can read it here.
Prompted by one of the big review sites dropping scores, Rik took a look back at some of the different approaches taken by magazines over the years.
After Stoo pointed out that there were lots of reviews of similar-sounding Need for Speed games on the site, Rik tried to put them into some sort of context.
Rik laments the end of his long-running multiplayer Pro Evolution Soccer rivalry.
Stoo used to like flight sims, until they got too complicated: here, he shares some memories of some of his 90s favourites.
After listening to rather too much of Radio 4's farming-based soap-opera, Rik imagines a game based on young Tom's obsession with ready-meals.
A few pieces we wrote pre-2007. Needless to say these are all pretty old, but we've retained them for posterity.
Our original 'brief history' features were among the first non-review items on FFG, and as such were written some time ago. We've made periodic attempts to update them but most stop in the early 00s. Hopefully they still serve some purpose.
Our first attempt at paying tribute to PC Zone, back when it was still a going concern.
A listicle featuring some of our favourite gaming bad guys.
A listicle featuring our favourite gravelly-voiced heroes.


text:
Motorcyclist killed in Motueka crash
16 Nov, 2012 8:30pm Quick Read
Man killed in drag race
Tiny council's debt at $100m
17 Nov, 2012 5:30am
Bruce Rogan is leading the rates revolt over the sewage system. Photo / Steven McNicholl
By: Andrew Laxon
News Editor, NZ Herald
andrew.laxon@nzherald.co.nz
A small council on the verge of bankruptcy now faces a potential debt of about $100 million - equal to more than $7000 for every ratepayer.
The Kaipara District Council is preparing for the possibility it will have to pay back about $19 million of illegally set rates, on top of the $79 million debt it has run up in the past four years.
Elected councillors resigned in August to make way for Government-appointed commissioners, who are trying to keep the Northland council financially afloat and stop the spread of a rates strike which affects much of the district.
The strikers' main grievance is that the council secretly agreed to double the cost of an unpopular sewage treatment scheme in Mangawhai, which has created most of the debt.
Commissioners chairman John Robertson said the council was unlikely to have to pay back the $19 million and he expected more people would start paying rates as the commissioners got the district on track.
However the council's latest financial records show:
*It expects to lose about $2.2 million, equivalent to 12 per cent of last year's rates revenue, from the rates strike.
*It had to borrow $4.5 million on its overdraft to pay the bills in the past few months as it waited for more rates payments.
*The cost of the controversial sew- age system has climbed from $58 million to $62 million, as interest has added to the bill.
The council's annual report says it is highly likely that $17.3 million of rates would be invalidated if ratepayers took their case to the High Court, as Mangawhai Ratepayers and Residents Association chairman Bruce Rogan has indicated.
Battle over golf course plan
20 Oct, 2012 5:30am
Coastal town's revolt
3 Nov, 2012 3:32pm
Kaipara rates rebellion grows
3 Nov, 2012 5:30am
Joel Cayford: Prudent councils shouldn't bank on growth to pay bills
The report estimates the council would have to pay out $19 million, allowing for likely court costs, which "would have an impact on the council's financial position".
It says if the council has to borrow the extra money, that will push its debt to 232 per cent of annual revenue, close to its self-imposed 250 per cent limit. (The council's current 180 per cent debt-to-revenue ratio has already been compared unfavourably to Greece's 160 per cent.)
Mr Robertson said he thought a legal challenge was unlikely and stressed the report only listed the payout as a contingent liability, which might not happen. If a challenge did succeed he believed the council would have to reset the invalid rates, which it had the power to do without asking the Government.
He said it was not unusual for councils to use overdraft facilities to pay expenses if they did not have enough cash from rates or other revenue. However the council was moving away from borrowing to pay for everyday services and was reducing the amount of interest capitalised on to the cost of the sewage scheme.
"The reaction I get in the community is very clear - they're pleased the commissioners have been appointed and they just want us to fix the council up. There are a few people that are not of that mind but by far the majority are paying rates."
Local government finance and policy commentator Larry Mitchell said the council's decision to include the $19 million payout risk could have been influenced by Audit New Zealand, which highlights the risk in its report on the council's financial statements. The auditor's report also draws attention to an Auditor-General's inquiry into the sewerage scheme and that the council depends mainly on its bankers to continue operating.
Mr Mitchell, who produces a rating guide to council performance, disagreed with Mr Robertson's view that the council could restrike the rates if it lost a legal challenge.
"If they lost a judicial review, the implications are that they couldn't do that ... They'd be better off from the council's perspective to go straight to special legislation without running the risk. If the courts found the rates illegal, it would be pushing it uphill to expect parliamentarians to correct the legality."
Mr Mitchell also disagreed that it was normal council practice to borrow large sums of money to cover shortfalls in revenue."It's an accounting and financial management no-no to be borrowing to pay for the housekeeping."
Kaipara council
$100m potential debt
$7184 for every ratepayer
1100 ratepayers on strike
$2.2 million shortfall expected
Man beaten up in midday gang attack
Police assure the public the incident was isolated and not indicative of gang tensions.

redpajama_set_name: RedPajamaCommonCrawl


text:
Our Other Platypus
27.3.2017 by Shoshana Rapley
Image: Nolan White
Most Australians are comfortable with the knowledge of our native marsupials they acquire around primary school age. Most Australians could comfortably name a species or two of kangaroo and wallaby. Most would be able to recall a time when a possum became a nuisance. And at a pinch, some may be able to distinguish monotremes from marsupials.
But if you ask an Australian about rodents, the response you are likely to get is some form of 'yuck'. Rodent usually equals introduced critters with a penchant for growing into plague proportions and spreading infection. It is true, we have five species of introduced rodents: House Mouse, Black Rat, Brown Rat, Pacific Rat, and (fun fact) the Five-lined Palm Squirrel, a zoo escapee species established in Perth. Chances are, however, that you would be surprised if I told you we have nearly 50 species of native rodents – more than 50 if you count the extinct critters. For instance, the Spinifex Hopping Mouse lives in elaborate burrow systems in close family groups, and is an incredibly sophisticated desert specialist species. And for an extinct example, the White-footed Rabbit-rat – 'gnar-ruck' to the Eora people – was a kitten-sized, squirrel-like Conilurus species which used its semi-prehensile black and white tail to craft grass nests in among the eucalypts from Adelaide to Sydney. All 47 extant species are uniquely adapted to their Australian ecosystem homes, and contribute to the ecosystem functioning and biodiversity of this wide brown land.
I recently shared a piece on Facebook about the Rakali, the largest species of native rodent. This post was met with two comments. The first read, 'one of Australia's most underrated mammals.' Thumbs up, totally agree. The second said something to the extent of, 'this is disgusting, I can barely type this response because I don't want to look at it.' Wot?
Well, my friends, it's time to buckle up, because I am about to challenge the latter sentiment with some non-alternative facts.
The Rakali (also known as 'water-rat') is a top aquatic predator, akin to an otter, which deftly hunts for fish, amphibians, crustaceans, and occasionally land mammals. They are gorgeously well adapted to their environment with webbed feet, sensitive whiskers for detecting prey and a water-resistant coat in golden-orange and charcoal coloration with a white-tipped tail.
This lovely coat meant that the Rakali was hunted towards the point of extinction for their pelts, particularly in the 1930s when importation of overseas furs was banned. In 1943, the Rakali was protected from hunting. Human activities, however, continue to have a detrimental impact on the Rakali. Many drown in yabby and crayfish traps, and others are actively killed by fishermen who perceive the Rakali as a pest that will steal their bait or catch. Anecdotally, they have been known to steal pet food from suburban backyards, as well as fish and chip scraps from lake edges. Misinformed local beach and lake goers try to injure Rakali, falsely thinking they are righteously removing a rat from the area.
Rakali are seen fairly regularly in Lake Burley Griffin, particularly at the eastern inlet of the Molonglo River, as well as in Lake Ginninderra. I can recall one particular half-moon Friday night, eating a Yarra kebab on the edge of the lake, with a Rakali spreading ripples across the pensive surface, as it leisurely paddled through reflections of street lights above.
Despite the manufactured and degraded water quality of our water systems, Rakali continue to persist in the Canberra region. The introduced Common Carp makes up an estimated 90 percent of the fish biomass in some regions of the Murray-Darling Basin. Carp have many impacts on their environment – reducing water visibility, promoting algal blooms, contributing to erosion and spreading diseases – but they also impacts predators like Rakali and Platypus by competitively feeding on invertebrates . Additionally, Carp reduce native fish numbers, but this effect is poorly understood.
Not very much is known about the Rakali, as they are a shy, solitary species. In an ABC news interview, Dr Melody Serena from the Australian Platypus Conservancy said, 'we are relying heavily on feedback from the public about whether water rats have disappeared from their local area … They are charming little animals and ecologically very important.' As a top predator, the Rakali controls aquatic populations – in particular crustaceans like yabbies – which in high numbers would threaten the structural integrity of mud banks with their burrowing. Professor Peter Banks from the University of Sydney is currently investigating how Rakali supress the invasive Black Rat population by outcompeting the feral species. Incredibly, the Rakali is also known to eat Cane Toads by flipping them onto their belly to avoid contact with the toxic glands.
So, keep an eye out around Canberra's lakes next time you go for a jog, cycle, early morning adventure or late night snack run. You might just see the cryptically gorgeous Rakali ottering about.
If you see the Rakali, please send a detailed description and location information to the Australian Platypus Conservancy at platypus.apc@westnet.com.au or check out their Facebook page, Australian Platypus Conservancy (Official).
What if Animals did the Talking?
6.4.2017 by Heloise Duce
A Slice of the Pie
6.4.2017 by Liam Osawa
Things Thought in Showers Rarely Happen the Way You Want
6.4.2017 by Mahalia Crawshaw
Foreign Film Fights: Sub vs Dub
6.4.2017 by Caroline Hendy


It is great to see sea trout in the river in better numbers than for some years. Cortachy, about 6 miles upstream of Finavon, has been catching lots of sea trout, and every one of them of course passed through Finavon's pools. But high water has meant that the big shoals have moved upriver. At Inshewan (3 miles upriver) Colin Gibb tells me that there are also good numbers of fish, both salmon and sea trout.
We are now awaiting the arrival of the grilse, hoping that the nets don't take too many of them before good numbers can get upriver. We are still catching the odd salmon and, with a mid July total of 33 salmon and 64 sea trout we are ready to welcome in the summer and autumn salmon and grilse.
We were told by the BBC that this was going to be a long hot summer. So far it has been a cool rather damp one. If the water continues to flow at a good level into August and September we can expect to see good numbers of fish. Here's hoping!


text:
Epidemiological Perspective
Cholera: The Epidemic Power of Vibrio cholerae
By Jeremy Akers, Danny Mays, Marc Siegel, MD, Visiting Focus On Haiti Writer
Cholera patients in Haiti. Source: Rapadoo Observateur
This is the first in a series of posts on cholera and its impact on Haiti. The first post is a discussion of the disease itself, and serves as an examination of medical and epidemiological factors that enabled cholera to be carried by Nepalese UN peacekeepers and spread throughout Haiti with deadly force.
Haiti's cholera epidemic began in October of 2010. Since then, researchers have been investigating the origin of the infection, as it had not been seen on the island of Hispaniola in over a century. Scientists have performed biochemical tests and genetic analysis on the bacteria in Haiti, tracing the current outbreak to a strain of Vibrio cholerae that is widespread in cholera-endemic southern Asia. The CDC found evidence that human waste from a UN peacekeeping base staffed by Nepalese officers was contaminating tributaries of the Artibonite River that flowed through many of the tent cities housing displaced Haitians after the earthquake. Journalists and the public put two and two together, and outrage has ensued, and legal action is being taken against the UN on behalf of affected Haitians.
Vibrio cholerae are microscopic bacteria that live and reproduce in water. They are resilient and can survive inclement conditions by attaching to the surface of other organisms and by forming protective biofilms. Cholera epidemics occur when the V. cholerae bacteria contaminate community water sources or food. Haiti's inadequate infrastructure for water and sanitation, poor hygiene practice, and heavy rainfall create a conducive platform for a cholera epidemic.
Since 2010, cholera has infected over 660,000 Haitians and killed more than 8,100. The disease is not usually transmitted directly from person-to-person (like the flu). The common process of transmission–the "fecal-oral" route–is as follows: V. cholerae are excreted in the stool of an infected person and, where proper sanitation is lacking, contaminate the water supply or food. Contaminated water is ingested and many of the bacteria are killed by stomach acid. However, if enough V. cholerae are ingested, some will reach the intestines and produce toxins that cause massive intestinal secretion of water and electrolytes leading to profuse watery diarrhea and vomiting. This is the disease we call "cholera."
Once in the intestines, V. cholerae reproduce rapidly to the point where billions can be excreted in every milliliter of diarrhea. With a daily volume of diarrhea reaching up to 20 liters, there is enormous potential to contaminate the environment. If untreated, this level of fluid loss will lead to dehydration and serious electrolyte imbalances causing muscle cramps, heart arrhythmias, kidney failure, and often death. Essentially, cholera is a disease of fluid loss through diarrhea and vomiting that leads to dehydration with potentially grave physical consequences and, in the right context, it is easy to transmit and can result in epidemics.
V. cholerae infections can be mild, mimicking Traveler's diarrhea, or completely asymptomatic. This is one of cholera's most powerful means to cause deadly epidemics because, even in the absence of overt symptoms, bacteria are still shed in the stool. If infected people have mild or absent symptoms, they are more likely to travel and take fewer hygiene precautions. Cholera often spreads geographically via human movement. In a globalizing world, there is unprecedented opportunity for pathogens to be introduced into vulnerable geographic regions where the local population lacks preexisting immunity. Along these lines, recent research has proven that the movement of UN troops from Nepal was almost certainly the avenue by which cholera came to Haiti.
Cholera is a somewhat ironic disease in that the source of the infection is also the basis of the treatment: a simple solution of water and electrolytes known as "ORS" (oral rehydration salts). It is essentially GatoradeTM. Antibiotics may be of some benefit in severe cases, but the most important intervention is restoration of water and electrolyte balance in the body using ORS. If an infected person is treated promptly and appropriately, the mortality rate is about 1%. If proper treatment is not provided in time, mortality can reach 60%. Given the simplicity of the treatment, management of a cholera outbreak "should" be feasible. As the death toll in Haiti continues to climb, clearly there is more to the story.
Saha, D. et al. Hunter's Tropical Medicine and Emerging Infectious Diseases. Copyright © 2013, Elsevier Inc.
Webber, R. Communicable Diseases: A Global Perspective. Copyright © 2012, CABI, Wallingford, UK


text:
Patrick Hunt (* 1951 in Kalifornien) ist ein US-amerikanischer Archäologe.

Forschung
Hunt leitet seit 1994 das Alpine Archaeology Project der Stanford University. Im Jahre 1996 fand er am Pass Fenêtre de Ferret den Steinbruch für den Tempel des Jupiter am Grossen Sankt Bernhard. Im Jahre 2003 fand er einen Hort von römischen Silbermünzen in den Schweizer Alpen.

Im Rahmen der Hannibal Expedition 2007-2008, die von der National Geographic Society finanziell unterstützt wird, suchte Hunt Überreste der Alpenüberquerung Hannibals 218 v. Chr. mit seinem Heer und 37 Elefanten während des Zweiten Punischer Krieges. Hunt untersuchte bislang 25 Alpenpässe und favorisiert unter ihnen den Col de Clapier.

Bisher brach sich Hunt bei mehreren alpinen Unfällen 30 Knochen; darunter eine Fraktur des Beins 2002 während lichenologischer Untersuchungen oberhalb von Bourg-Saint-Pierre in den Walliser Alpen und seitdem einige Hand- und Fußknochen. Er ist Mitglied der Royal Geographical Society seit 1989. In jüngeren Publikation beschäftigte er sich unter anderen mit Rembrandt van Rijn und Michelangelo Merisi da Caravaggio.

Werkauswahl
Monografien

 Provenance, Weathering and Technology of Selected Archaeological Basalts and Andesites. Ph.D. Dissertation, Institute of Archaeology, UCL, University of London, 1991.
 Caravaggio (Life & Times). 2004, ISBN 978-1904341734
 Rembrandt: His Life in Art. Ariel Books, New York, 2006, ISBN 0976316285
 Alpine Archaeology. 2007, ISBN 978-1934269008
 Ten Discoveries That Rewrote History. 2007, ISBN 978-0452288775

Weblinks
 Offizielle Webseite
 Persönliche Website
 In the Alps, hunting for Hannibal's trail. Stanford Report. May 16, 2007

Klassischer Archäologe
US-Amerikaner
Geboren 1951
Mann

redpajama_set_name: RedPajamaWikipedia


Realtors, Integrabond and the Rental Prop brand.
You are welcome to contact me directly (franz@holm.co.za) should you have any questions. However, Nicolette (reception@holm.co.za) may be contacted regarding the levies, accounts, budgets and financial reports and Pieter (admin@holm.co.za) regarding maintenance, security, gardens service, etc.
For emergencies, please do not hesitate to contact our after hours (24 hour service) mobile number, 083 658 2253.


This stylish and comfortable e-bike turns heads and floats over bumps all the way to the beach.
This stylish and comfortable e-bike turns heads and floats over bumps all the way to the beach. The Cadence is all about striking, smooth styling and uncompromised comfort. Schwalbe puncture proof tyres go with amazing full length front shocks your arms will never feel a pothole again. All eZeebike owners love their rides, but there's nothing like the smiles on our Cadence riders.


text:
Q: Uncaught TypeError: Cannot read property 'dispatchEvent' of null I am getting the above error when I run my function. The goal is when a user enters a number in a  search box it should zoom to that number in the visualization. Below is my code -
function zoom1() {
 var input1 = document.getElementById("myInput1").value; //value from searchbox
  console.log("input from searchbox :"+input1);

 d3.json("intervals.json", function(alldata)  // entering json file  to look for oid
 {
    // console.log("all data from json"+alldata);
    var i=0;
    for (i = 0; i < alldata.records.length; i++)  //for loop for getting the "oid"  alldata.records.length;
    {

      conceptid1 = alldata.records[i].eag;  //saving all the oid in conceptid
      console.log("conceptid1: "+conceptid1);
      var conceptid2 = conceptid1.toString();
      console.log("conceptid2: "+conceptid2);
      if (conceptid2  === input1) //if the user input1 matches conceptid2
      {
         console.log("inside if conceptid2:"+conceptid2);

         console.log(document.getElementById(conceptid2).dispatchEvent(new Event('click'))); // zoom
      }

    }

 });
}

A: Can you share the console log for conceptid2?
By the way, use ES6 syntax. It's a newer and better way to write JS. Here is your code written in ES6:
const zoom = () => {
    let value = document.querySelector("#myInput1").value;
    console.log(`Input ${value}`);

    d3.json("intervals.json", (alldata) => {
        const { records } = alldata;

        for (let i = 0; i < records.length; ++i) {

            const conceptId = records[i].eag.toString();

            console.log(`Concept ID: ${conceptId}`);

            if (conceptId  === value) {
                // Your stuff
            }
        }
    });
}

Anyways didn't want to come on too hard but I hope this helps :)

redpajama_set_name: RedPajamaStackExchange


Rent vs Own Calculator – To purchase a property or to simply rent it has become a central question for many people up and down the country.
Many people simply do not understand the critical factors that they need to take into consideration in order to arrive at a sound decision. Broadly speaking, there are two main reasons why one would need to tackle this question – the first, and most salient reason, is the personal and emotional factors. The second factor, however, focuses on the financial issue, which by and far, has greater input into the decision making process. It's these financial factors that will be considered in this piece that should give you, the prospective tenant or owner, some degree of clarification on where you stand.
The very first step one has to take is to determine whether or not it's possible to purchase a home in the first place. This may seem like very obvious advice on the face of it, however, once one examines it further, we find that many people inaccurately understand the stability or otherwise of their finances. This can lead to bad decisions. The factors you need to take into consideration are: whether you can afford the down payment (which can be as much as 20% of the value of the home), as well as knowing if you can afford closing costs (as much as 5%). These values will massively exceed that of the renting deposit and initial payments and thus play a crucial role in the decision making process.
For the sake of argument, let's assume you take up residence and decide to purchase a new home. You need to factor in the maintenance costs that will inevitably crop up. These maintenance costs will include fittings and furnishings as well as the expected structural faults that may arise. In addition, it's worth emphasising that nothing ever lasts forever and you will have regular costs in the overall maintenance of your house. If you compare this to renting, the landlord covers the cost of the maintenance while you're almost guaranteed a fixed payment each month. Financial experts have ascertained that one should not exceed 28% of their gross pay on mortgage repayments and overall monthly debt repayments should not exceed 36%.
Thus, you can clearly see the turbulent wave of financial costs really hits hard when one decides to purchase a home. However, it needs to be noted that these are short-term consequences that we have hitherto analysed. In order to fully analyse your situation as effectively as possible, it's necessary to engage in a long-term cost-benefit analysis; only this way can a more mature decision be determined. Most proponents of purchasing a home will point to things such as the investment value of the home as well as the ability to built equity while availing of tax breaks. However, upon further long-term analysis, we can see that the situation is not as rosy as these proponents like to conclude.
Let's start by looking at equity. Proponents often cite that money spent renting is money wasted because you're not building an equity base. However, this is overly simplistic. It's important to recognise that equity takes time to build and the focus of mortgage repayments in the initial years is on interest repayments. In addition, if you choose to move home after only a few years, then you may have accrued very little equity and, upon selling, you may end up incurring a loss. With respect to tax breaks, it's worth highlighting that tax breaks only apply where the amount of your itemised deductions is greater than your standard deductions. Ultimately, depending on your finances, you may be better off taking the standard deduction and therefore would have gained no tax benefits.
Proponents of purchasing homes also, as briefly mentioned earlier, point to long-term investment opportunities. While it's certainly true that price appreciation can lead to selling at a substantial profit to subsidise a lifestyle at less expensive homes, it's equally worth noting that there are no guarantees that your home will appreciate over time. Indeed, there are many areas across the country where homeowners have failed to sell their homes at equal to or greater than the initial purchase price. This, of course, depends on many varying and interdependent present economic factors, but we should not lose the overall sight of the argument that it swings both ways – appreciation as well as depreciation.
To conclude, the argument as to whether you should rent or purchase is much more complex than it is at face value. You need to do your homework with respect to all the aforementioned financial factors that will impact on your decision. In addition, there is much assistance you can gain via the web such as the use of calculators that help to determine the benefits of a range of different financial variables. In addition, these calculators can be used to evaluate your investment opportunities and whether or not it's worth taking the risk in purchasing a home. Whatever your decision, try to ensure it's based on solid arguments and not solely driven by the personal factors that were mentioned at the beginning of this article.
Call us today at 424 225 2167 for details. One of our mortgage professionals will help you get the best possible loan solution for your situation. We'll be with you every step of the process and not hand you off to someone else.


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Planning to convert half of the basement and build an extension. Advice and obtaining planning permission are required.


GI Labs is proud to display the most recent issue of our newsletter, GI Review.
In this issue of GI Review, we hear from Dr. Nick Bellissimo of Ryerson University, about new research examining appetite and energy imbalances in children. Nutrition News summarizes recent developments in clinical nutrition research, and Industry Briefs presents relevant market research data about consumer attitudes and purchasing patterns related to the healthy food industry.
This month, we interviewed Dr. Nick Bellissimo, Associate Professor in the School of Nutrition at Ryerson University, whose research specializes in appetite and energy imbalances in children.
What are some of the factors that contribute to overeating and obesity in children?
Where do we begin? One of the things we know is that over the last 40 years, we have moved away from traditional foods to highly processed foods which may or may not be as satiating as other foods that were previously available. In kids, the picture is quite complex and many factors interact to determine a child's growth trajectory, including screen-time exposure, physical activity and snacking. Our goal is to understand how diet, exercise and environment interact to shape a child's eating behaviors and body weight.
What are some of the strategies you are researching to address overeating and obesity in children?
We examine both energy intake and expenditure. For energy intake, we study how the macronutrient composition of a meal or snack affects appetite, food intake suppression and metabolic profiles in children. Our goal is to understand how meals and snacks can be redesigned to tide people over to their next meal so that they are less likely to reach for less satiating snacks that are high in calories and poor in nutrients. We can also monitor the effects of meal composition on resting metabolism and basal metabolic rate, using indirect calorimetry. We also routinely determine how meal composition affects the thermic effect of food (energy required to digest and process food). We can also measure non-exercise activity thermogenesis, which is heat production from daily movement using research grade accelerometers in free-living conditions.
One thing we see is that if you change one component on one side of the equation, there is often (within days or weeks) compensation on the other side of the equation. For example, if you are trying to lose weight and you exercise more, your body often adjusts by eating more. We try to look at the overall picture and investigate if there is a way to manipulate the physiological systems and the regulatory pathways to impact eating behaviours to promote the achievement or maintenance of healthier body weights. From a food science perspective, we often use reverse engineering to understand why some food and food components are more satiating than other. We then use this information to develop new and improved food products to enhance satiety or control excursion in blood glucose.
What types of specialized equipment do you use in your metabolic research?
We have the Bod Pod, for measuring body composition using the same principles as underwater weighing, but using air displacement.
We use indirect calorimetery to measure energy expenditure at rest, during exercise, and in response to meal digestion and processing.
We have an exercise physiology unit, where we can perform time trials to assess the effect of food and food components on exercise performance, in addition to assessing aerobic fitness.
In 2017, we are expanding our research program to include a suite of food science tools and instruments to monitor food digestion in vitro, as well as new tools to monitor physiological hormones in vivo.
In research performed at GI Labs examining a randomized, cross-over study of 38 healthy subjects, Dr. Thomas Wolever, Dr. Alexandra Jenkins, and colleagues demonstrate that oatmeal elicits significantly lower glycemic responses than matched servings of Cream of Rice, even when sugar was added to the oatmeal, demonstrating that oatmeal minimizes postprandial glycemia.
A meta-analysis published by GI Labs President Dr. Thomas Wolever and colleagues demonstrated that Glycemic Index values do not differ when measured in Caucasians versus non-Caucasians. Reviewing 1288 trials validated that, with the possible exception of rice, GI values remain consistent across people with different ethnicities.
In research performed at GI Labs, Dr. Thomas Wolever and colleagues investigated whether oat β-glucan mixed in water and consumed before a meal would have an effect on postprandial glucose levels. Previous studies suggested that viscous fibers may need to be taken with the meal in order to have a lowering effect on glucose levels. The results of the study demonstrate that oat bran can be used as an effective preload to consume before a meal in order to manage postprandial glucose levels..
Research from the University of Sussex demonstrated that when certain odours were associated with creamy and thick foods (which are expected to be satiating), those odours themselves became associated with satiety and feelings of fullness. This finding holds potential for the development of weight-loss products or other products that promote satiety.
With more than half of Americans partaking in regular exercise and with exercise a first choice option in disease prevention, consumer interest in sports nutrition products is growing. Key priorities for consumers include foods that are easy to consume on the go, products that promote recovery post-exercise, and products that provide long-lasting energy and endurance.
According to a recent survey of 7000 people across Europe, the Middle East, and Africa, almost half of the respondents (45%) were worried about getting the right amount of nutrition. Weight is the top concern among adults aged 18-50. Respondents in Italy, France and Egypt are most concerned about protecting themselves against disease later in life.
According to recent market research data from the Hartman Group, consumers associate high quality food and beverages with better nutrition, further reinforcing the connection between "premium" and "healthy" in food products.
Dr. Thomas Wolever, President of GI Labs, and Dr. Alexandra Jenkins, Director of Research, will be attending as well as presenting their research.
GI Labs will once again be exhibiting at this major event for the food industry. Come visit us at booth #4564!


text:
Q: Collection of Illustrated Horror Stories I've been trying to find a collection horror stories that I used to have in the 90s when I was a small child.
I don't think they had it at school. It was definitely not Scary Stories to Tell in the Dark. I also read those and enjoyed them immensely.
It had large full page color illustrations of the stories that were terrifying and quite disturbing to me as a child.
It had several short stories and was a large, thin, book; significantly larger than legal paper but I'm not sure how large since I was so young when I had the book. All the illustrations were in full color, I think.
There are two stories from it I remember. I'm not sure about the details but the overall plot I'm sure of.
One was about a man who wants to book passage on a passenger ship, but the cabins are all full. Somehow he finds out there is one free cabin. The crew tires to dissuade him from using it and say they never book it because it's haunted or something. He insists, saying he's not afraid, and is allowed to sleep in the one empty cabin. He starts getting scared at night and hears some sound or something. Maybe he heard knocking but there wasn't anyone at the door. Anyway he winds up in bed with the porthole window open and a dead man who had drowned crawls in through the porthole to sleep in bed.
There was a truly terrifying illustration of the man lying in bed with the drowned corpse crawling in through the window.
The other story I don't remember well at all except for the illustration.
There was some man who was being hanged for something, and they hang him but he doesn't die. His corpse keeps twitching and starts jerkily moving and chases the people who hung him.
I mostly just remember the illustration of a corpse with a grotesquely long neck, broken and disjointed and splattered with blood, jerkily staggering after a crowd of people.
I'm pretty sure this was a children's book of some kind. I must have been 6-8 when I had it, and while it was pretty gruesome and dark it seemed to be humorous or at least didn't take itself seriously. The illustrations were almost cartoony, not like the charcoal realism of Scary Stories to Tell in the Dark.

A: Could it have been Mostly Ghostly by Steve Zorn? It's a collection of classic horror tales adapted for children, and it does include an illustrated version of The Upper Berth. It also contains Ambrose Bierce's A Creature of Habit which has a walking hanged man.
Here's its illustration for The Upper Berth:

Also, a summary of A Creature of Habit specifically mentions the wobbling head and the rope still attached to the body, which is grabbed onto by some of the townspeople.


text:
'''
A logistic regression learning algorithm example using TensorFlow library.
This example is using the MNIST database of handwritten digits (http://yann.lecun.com/exdb/mnist/)

Author: Aymeric Damien
Project: https://github.com/aymericdamien/TensorFlow-Examples/
'''

# Import MINST data
import input_data
mnist = input_data.read_data_sets("/tmp/data/", one_hot=True)

import tensorflow as tf

# Parameters
learning_rate = 0.01
training_epochs = 25
batch_size = 100
display_step = 1

# tf Graph Input
x = tf.placeholder("float", [None, 784]) # mnist data image of shape 28*28=784
y = tf.placeholder("float", [None, 10]) # 0-9 digits recognition => 10 classes

# Create model

# Set model weights
W = tf.Variable(tf.zeros([784, 10]))
b = tf.Variable(tf.zeros([10]))

# Construct model
activation = tf.nn.softmax(tf.matmul(x, W) + b) # Softmax

# Minimize error using cross entropy
cost = -tf.reduce_sum(y*tf.log(activation)) # Cross entropy
optimizer = tf.train.GradientDescentOptimizer(learning_rate).minimize(cost) # Gradient Descent

# Initializing the variables
init = tf.initialize_all_variables()

# Launch the graph
with tf.Session() as sess:
    sess.run(init)

    # Training cycle
    for epoch in range(training_epochs):
        avg_cost = 0.
        total_batch = int(mnist.train.num_examples/batch_size)
        # Loop over all batches
        for i in range(total_batch):
            batch_xs, batch_ys = mnist.train.next_batch(batch_size)
            # Fit training using batch data
            sess.run(optimizer, feed_dict={x: batch_xs, y: batch_ys})
            # Compute average loss
            avg_cost += sess.run(cost, feed_dict={x: batch_xs, y: batch_ys})/total_batch
        # Display logs per epoch step
        if epoch % display_step == 0:
            print "Epoch:", '%04d' % (epoch+1), "cost=", "{:.9f}".format(avg_cost)

    print "Optimization Finished!"

    # Test model
    correct_prediction = tf.equal(tf.argmax(activation, 1), tf.argmax(y, 1))
    # Calculate accuracy
    accuracy = tf.reduce_mean(tf.cast(correct_prediction, "float"))
    print "Accuracy:", accuracy.eval({x: mnist.test.images, y: mnist.test.labels})

redpajama_set_name: RedPajamaGithub


Just off England's sun-kissed south coast lies the idyllic green island we call the Isle of Wight. A brief ferry ride across the waves will bring you to this charming place, whose striped cliff sands and rolling hills make it one of the country's favourite holiday destinations. Here's our guide to the 10 best things to see and do in England's Isle of Wight.
These three impressive spikes of chalk are around 30 metres high and pierce the Isle of Wight's seascape just next to Alum Bay. Perched shyly behind the third of these jagged mounds is the red- and white-striped Needles Lighthouse, too. There was once a fourth shard, known as Lot's Wife, which fell into the sea in 1764 and had the sharp, column-like appearance of a needle, which is where the remaining three shapes got their charming name. Take a boat trip for a closer look at these unique, natural works of art.
The craggy cliffs at the Isle of Wight's dreamy Alum Bay are streaked with pretty shades of pink, yellow and gold. Ride the chair lift to gaze at these unique colours or check them out as you wander the beach below. The stripes were caused by the shifting sea bed which was eroded almost 70 million years ago. Around 10 million years later, the bedrock moved, pushing the sedimentary layers upward, creating the unique pattern we see today.
This peculiar hexagonal tower, otherwise known as St Catherine's Oratory, could be straight out of a fairy tale. It is the remains of England's only surviving medieval lighthouse, dating back to 1314. The story goes that wealthy landowner Walter de Godeton came into possession of some stolen wine (174 casks, to be exact) which had been on its way to France in a ship that ran aground. The Catholic Church was unimpressed, so as penance, Walter built a lighthouse where a priest would pray for sailors wrecked on the treacherous rocks below.
A leisurely wander up Ryde Pier allows you to gaze back across the sea to mainland Portsmouth. There are terrific sandy beaches all along the town's coast, and a boutique shop or two in which to while away some hours browsing for the perfect souvenir. There is also the Appley Tower, built as a garden folly in 1875, whose crenelations make for a perfectly photogenic snapshot of the English coast.
There's plenty of action to enjoy on the Isle of Wight, from cycling to horseback riding. But if speed is your thing, then Wight Karting makes a brilliant day out. With racing for groups and families, there's a full range of packages to choose from. The all-weather track is 500 metres long and is open all year long.
According to legend, St Catherine once competed with the Devil to see who should be in charge of the Isle of Wight. The Longstone at Mottistone is said to be a sandstone pillar, which St Catherine threw down in the contest. It's four metres tall and two metres wide, suggesting Catherine was not only a saint, but had some serious upper body strength, too. The stone is, in truth, an ancient burial mound and can be incorporated into a scenic country walk.
Aside from its fabulous festivals, the Isle of Wight also boasts a cracking live music scene the rest of the year round. There is the much-loved Shanklin Theatre, which hosts orchestra recitals and toe-tapping musicals in equal measure. If theatre's not your thing, head into any pub on this lovely island and you'll not wait long to hear of an open mic event or mini-gig from local musicians.
From surfing to kayaking, swimming to windsurfing, there is something for every mermaid (and merman) over on the Isle of Wight. Try Wight Waters for paddle boarding and sailing, too. There really is nothing like a bracing splash in the chilly channel to blow away those pesky cobwebs.
Whether it's smooth jazz, club classics, live poetry or yoga workshops that float your ferry, you can catch one across from the mainland to one of this island's renowned music and arts festivals. There's the legendary Bestival or the sparkling Isle of Wight Festival, as well as the quirky Eklectika which showcases electronic, dance and pop acts big and small.
Perfect for long, windswept dog walks or even a horseback ride, the beaches on the Isle of Wight are many and beautiful. From the sweeping curve of Whitecliff Bay to the pleasant Englishness of Sundown Pier and the tumbledown charm of Steephill Cove, there is fresh air and crashing waves galore on this pretty island. But the long sweep of Sandown Beach has a particular charm. With craggy cliffs looming in the distance and the waves lapping gently at the shore, this is one of the island's most pleasant spots for an afternoon stroll or seaside picnic, when the sun is high.


Planning for retirement? Congress recently eliminated three popular strategies that affect planning for Social Security retirement benefits. These new changes could mean a loss in potential retirement income. These changes go into effect on May 1st so there is still time to use these strategies if you qualify and act quickly. The suspended strategies, which are explained below, enabled retirees to increase their ultimate retirement benefits. In some cases, those retirees and their spouses or dependents already utilizing these planning strategies will be grandfathered in. Some of these strategies are still available on a limited basis.
An individual will receive 100% of his or her benefits if they file at full retirement age. If you were born between 1943 and 1954, your full retirement age is 66. For those born between 1955 and 1959, full retirement age gradually increases until it reaches age 67 for anyone born in 1960 or later.
An individual may claim social security benefits as early as age 62. However, if an individual claims a benefit before full retirement age, the monthly payment could be reduced by up to 25%. Similarly, an individual who defers payments passed full retirement age will receive an increase in monthly payments of eight percent annually. This annual increase is only available up to age 70, the maximum retirement age.
A person may claim benefits based upon his or her own earnings record or a spousal benefit. Spousal benefits are based upon the earnings record of a spouse or, possibly, a former spouse. The spousal benefit could equal up to one-half of the spouse's benefit at full retirement age, and the spouse must be eligible for or receiving benefits.
File & Suspend Strategy- allowed a spouse to file for social security benefits at full retirement age and immediately suspend the benefits. This gave the spouse access to spousal benefits, while the deferred benefits of the first spouse grew annually up to age 70.
The Restricted Application- allowed for a person of full retirement age to collect spousal benefits of their retirement age partner, while deferring their own full benefits, which would grow annually up to age 70.
Retroactive Lump Sum- allowed for retirement age individuals who file and suspended their benefits, to un-suspend and collect a lump sum retroactive payment at any time prior to 70. The new law still permits un-suspending, but trades the lump sum retroactive payment for deferred retirement credits.
It is important to understand your options and to start planning for social security early.


Simon of Cyrenne helped Jesus carry His cross on the way to the crucifixion. In this new ministry we, as a parish, support and help families experiencing the loss of a loved one. We want our parishioners to know that we are there for them in such a difficult time.
Every member of a family can participate in one way or another. Children can make and send sympathy cards. Teenagers can babysit or do any needed errands around the house. Adults can cook meals, visit during the wake and after, run errands, etc. All members can pray for the deceased as well as for the family.
Sign up forms can be found in the back of both churches. If you are interested, please read and consider carefully what you can do for the bereaved families of our parish. You can place your completed form in the collection basket or drop them off at the rectory. If you have any questions, please contact Marge Pike at 401-722-7605.
St. John Paul II Simon Ministry would like to invite anyone grieving from the loss of a loved on to attend our newly formed bereavement group. Beginning on Tuesday, July 19, 2016. We will be meeting on the first and third Tuesday of each month at 7:00 pm in St. Cecilia Sacristy. Please use the "Adoration Chapel" door on the Newport Avenue side of the church. Come and hear the stories of people who are going through the same kind of loss you are. Sharing is optional and never mandated.


When asked to write a piece on representation for Spine, I immediately wondered why I was asked? In my opinion, plenty has been written on the subject. Frankly, I'm often wrangled into similar conversations with my publishing peers. These are always awkward, so did I really want to write a piece on representation in book covers? Call it explanation-fatigue, but I really did hesitate.
Some people can only feel satiated by power. And they feel most powerful when they can take power by diminishing others. When silencing or bullying others, they convince themselves of their supremacy. And one of the most effective disguises of their true harmful intentions is humor.
Some Things Covered: What Fight?
One morning Eric Wilder sent me an email at 9:00 am. He asked if I had seen a new Guardian article about UK book cover design. Did I mention it was at 9:00 am? I'm not a rational person at 9:00 am, so I knew he had to be serious.
He sent the article, but I was mystified by his urgency, till I read the thing. It was extremely familiar. Some version of this article comes around every so often. To make a long story short, it claimed UK cover design was the envy of the world, and had always been better than US design. And Eric wanted me to pen a rebuttal. I read the piece and sent him my response. I wasn't interested in getting in the middle of the old rehashed nationalistic argument.
Some Things Covered: Why Purple?
Eric and I have been talking about collaborating for a while. My interest was piqued when a few months ago he sent me an email asking me if I would write semi-regularly for Spine. But I can't tell you the immediate panic that came over me. I mean yes, I've been writing my blog for a while. Yes, I wanted to write different kinds of things for different kinds of audiences. But how would it work? So it's funny that when I finally committed to writing something, Eric decides that purple covers were interesting in that week. Because it's a color I literally feel nothing about.


Your gazebo adds an elegant oasis to your back yard setting. It gives you shade, a relaxing place to enjoy a beautiful summer evening and a comfortable seat to watch your kids play or take in a beautiful sunset. What do you give back to your gazebo? Anything? What should you give back? Protection. You can get this protection from Plastics & Signs of Ottawa. We offer the best custom plastic fabrication in Ottawa, ON.
Polyurethane plastic windows are a great addition to a gazebo. They seal the gazebo's windows tightly, ensuring proper insulation. They retain warmth, creating an outdoor haven even during Ottawa's harsh winters. They also protect your gazebo in rainy weather. Spring storms can easily blow rain and debris into a gazebo. Plastic windows will keep your gazebo drier and cleaner, and provide a dry place where you can sit and watch the rainstorm.
Acrylic and plexiglass window coverings are another option for great insulation on your gazebo. They will keep out the frigid winter air, allowing you to sip your hot cocoa from the confines of your insulated gazebo. These plastic coverings serve a secondary purpose as well. In addition to creating a winter haven, they protect your gazebo from snow and ice. They will help keep the outside elements… outside.
Once you have added great protection to your gazebo through plexiglass insulation and coverings, it is important to maintain it with regular cleaning. Sweep up any debris, clear away cobwebs and check for any insect nests and carefully remove them.
Do a thorough inspection of your gazebo at least once per year. The fall season is a good time for this, to ensure your gazebo is ready to endure winter. Make sure you have completed an overall cleaning. Look for any signs of needed repair. Is it sturdy? Are all joints still strong? Does anything need reinforcement or refastening? Are there any signs of termite or other infestation?
Are there any signs of mold or mildew? Algae and other growths can spread quickly and cause damage to your gazebo. It's also not aesthetically pleasing or aromatically pleasant. Prompt removal of any mold, mildew, or algae is important to maintain your gazebo. Many products are available that will kill these unsightly growths, including outdoor sprays, bleach and vinegar products.
With your plastic windows in place and regular cleaning and inspections, your gazebo interior will remain well-protected and maintained. However, after a few seasons, the exterior may need some renewing. Sometimes, a fresh coat of paint can make your gazebo feel like new.
The first step to protecting your gazebo is plexiglass. Getting this in place will help extend the life of your gazebo and make it more functional year-round. For custom plastic fabrication in Ottawa, ON, call Plastics & Signs of Ottawa. We are ready to meet all your custom plastic needs and keep you (and your gazebo) protected.
Out of sight, out of mind. If the customer doesn't see your product, they won't buy it. Your task is to get your product in their sights. How can you ensure they see it? The answer is a custom plastic display. The proper display can make or break your product's success. Plastics and Signs of Ottawa can make sure you get the best merchandising through custom plastic fabrication in Ottawa, ON.
Whether you will be displaying products on countertops, in storefront windows, on store shelves, or elsewhere, you need a custom plastic display that showcases your merchandise in the best possible way. It is important your display follows the ABC's (and DEF's) of merchandising.
Accessibility: Is your product easy to reach? Can customers see it? Does the display hold merchandise properly? Is it a good fit so that potential buyers can easily access whatever you have on display? A customized plastic display is best to ensure your products are getting the best exposure.
Benefit: The right custom plastic display case can help you drive sales. Through proper display, you can push new products and increase your bottom line. But, this is only possible if the case is right for you and your merchandise. Don't settle for something that doesn't maximize your merchandising potential. The sales benefits are well-worth investing in a display case built with your needs in mind.
Conservation: Retail and commercial space are prime. You don't want to waist an inch. For best ROI, maximize your space. Use as little space as possible with the use of a custom plastic display. Wasted space means lost opportunities to promote additional products or achieve add-on sales. Making the best use of your space is essential to boost revenue. By designing something for your specific product and the desired space, you will get the best results from your display.
Draw: Is the display aesthetically pleasing? If it doesn't draw the customer's gaze, it's not doing its job. It is essential to choose a display that is eye-catching, inviting, and appealing. Is must draw the customer to it, engage them, and make them want to pluck from its shelves. By designing a plastic display case specific to your marketing needs, you will get the best draw for your merchandise.
Ease: For the benefit of both you and your customer, your display should be easy to use. Cumbersome, hard to stock, unstable, or unnecessarily complex displays only make your job harder. You want sales to be easier, not more difficult, to achieve. In choosing a plastic display case, be sure to select a design that makes merchandising as simple as possible.
Fresh: Keeping things fresh and exciting keeps customers interested and encourages them to keep coming back. Regularly switching out your displays retains their interest. Showcasing the same products or displaying them in the same manner over a long period of time can cause them to eventually go unnoticed. Use fresh displays and custom arrangements to ensure nothing you have to offer is ever overlooked.
If you're ready to apply these ABC's to your next display, contact Plastics and Signs of Ottawa. We will help you get the best return on custom plastic fabrication in Ottawa, ON, with display cases designed with you in mind.


Ariana Grande has apologised to her ex-boyfriend Ricky Alvarez as she gave him the "worst" line in her new song 'thank u, next'.
Ariana Grande has apologised to her ex-boyfriend Ricky Alvarez for giving him the "worst" line in 'thank u, next'.
The 25-year-old singer name-checked her ex-boyfriends - Pete Davidson, Mac Miller, Big Sean and Ricky - on her new song and joked with her former flame about the line she gave him.
In the track, she sings: "Wrote some songs about Ricky / Now I listen and laugh."
And taking to Instagram and responding to Ricky's post, which was a video of him listening to the song, she wrote: "HAHAHAHAHHAHAHAH IM SORRY U GOT THE WORST LINE. It was meant v lovingly (sic)."
In the new song, she sings: "I met someone else, we havin' better discussions. I know they say I move on too fast, but this one gon' last 'cause her name is Ari, and I'm so good with that."


text:
A REFLECTION-BASED RF SOURCE LOCALIZATION ALGORITH
Saeid Haidari, Hadi Moradi, Mahmoud Shahabadi, S.M. Mehdi Dehghan
RSSI, reflection, NLOS, RF source localization, UAV, optimal pair
In this paper, an RF source localization approach, in non-line of sight condition, using reflected signals, is introduced in which received signal strength indicator and angle of arrival (AOA) observations are used. Furthermore, the optimal positions of the receiver to increase the accuracy of the localization are discussed. In the proposed approach, a reflected signal is modelled by three parameters, i.e., distance between the RF source and the receiver via the reflector, the reflection angle, and the AOA. These parameters are used to determine a locus for the RF source location. Then, by using at least two reflected signals to give two loci for the RF source location, the location of the RF source is determined. Furthermore, the optimal locus of one reflection with respect to the other one is determined. The approach has been simulated, and the results show better accuracy of localization for optimal pairs compared to the other pairs, which decreases the search time.


text:
<?php

namespace Oro\Bundle\EntityExtendBundle\EventListener;

use Oro\Bundle\EntityExtendBundle\Migration\RefreshExtendCacheMigration;
use Oro\Bundle\EntityConfigBundle\Tools\CommandExecutor;
use Oro\Bundle\MigrationBundle\Event\PostMigrationEvent;

class RefreshExtendCachePostUpMigrationListener
{
    /**
     * @var CommandExecutor
     */
    protected $commandExecutor;

    /**
     * @param CommandExecutor $commandExecutor
     */
    public function __construct(CommandExecutor $commandExecutor)
    {
        $this->commandExecutor = $commandExecutor;
    }

    /**
     * POST UP event handler
     *
     * @param PostMigrationEvent $event
     */
    public function onPostUp(PostMigrationEvent $event)
    {
        $event->addMigration(
            new RefreshExtendCacheMigration($this->commandExecutor)
        );
    }
}


I had my ACL reconstruction surgery using ST+G tendons 6 years ago. As of right now G tendon has completely regrown and attached to Pes Anserinus by itself, while ST is still dangling inside my thigh (when I contract my ST muscle I can visually see the muscle belly gathering next to by buttock).
So, the question is: can harvested Semitendinosus be attached back to Pes Anserinus surgically?
I'm having a revision surgery soon and I was wondering if I should ask my doctor to try attaching it back to original spot.
If you have any thoughts about it, they would be greatly appreciated!


What if one day, all of a sudden, your life turned upside down? So much so that your identity was shaken to the core. Events, experiences, expectations, certainty, and uncertainty define life as we know it; or life as we cannot dare to envision it.
Berit Hessing is joining the upcoming TEDx event, to tell a story very close to her heart. One that challenged her, made her and her loved ones reconsider and restructure their entire realities. She will narrate how a family illness reshaped her life, for the better.
"This experience made me more secure. It showed me how to take what comes to me, and use the moment".
However certain one might be about life situations, a bad or a good surprise might always be around the corner. Berit will voice a lesson she learned the hard way. That any circumstances, be them dramatic, life changing, challenging or chaotic, they can always teach you something remarkable.
"Don't be afraid of circumstances in life. They could be a gift, if you just take it in".
This August, join Berit in her journey through the unexpected, the uncertain, and the serendipitous discoveries of life; life as we don't know it.


text:
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Welcome to the NetFortris blog. Here you'll find a plethora of topics covering the telecommunications industry, including VoIP, collaboration, innovation, networking, and security. You can also find NetFortris company news, partner news, and press releases here.
Who Says a 6-year-old Can't Run a Company?
Watch, if you will, a few seconds of this video. What do you see? We don't know about you, but what we witness is the emergence of some real corporate diplomacy.
The child on the left is clearly the more aggressive negotiator of the two, using all tactics at her disposal - from a meaningful nudge to an enterprising bite - to convince the other one to agree to the business deal, whatever that may be. For her part, the child on the right can be considered the more reserved negotiator, evincing a calculated nonchalance that clearly makes her all the more appealing of a company partner. By the end of the video the two seem to be moving toward some kind of agreement that merges their disparate approaches to business.
If this scenario sounds far-fetched to you - if you'd wager that what you saw was merely baby talk - then you've never met the following CEOs. Today we present you with a list of some of the youngest business leaders out there:
Lizzie Marie Likness: The next time you see a kid selling lemonade or food from a stand, consider that they might be the next Rachel Ray, or, as it happens, Likness. At the age of six, Marie was just a girl who needed to pay for horseback riding lessons, according to her website. But where some kids would simply whine until their parents shelled out the green, Likness turned to business to generate the capital, setting up her own food stand at a local farmers market. Her selection of healthy foods was a smashing success, and soon she was looking beyond the farmers market and at the corporate world at large. Today Likness is rapidly becoming the next TV food personality, with her own online show and an array of appearances on programs like Rachel Ray.
Her answer was to found Empower Orphans, a group aimed at carrying out projects that directly improve the lives of orphaned children. One such project, for instance, involved the establishing of a library at Feltonville School in Philadelphia. The project, which provided the school with 3,000 children's books, functioned to improve literacy within the institution.
Neha Gupta: We don't know about you, but at age nine we were hardly thinking about ways to get involved in charitable work. It wasn't until later that we learned the importance and value of giving back. But that was a lesson that Neha Gupta learned very early on, and by nine years old she found herself concerned about the dire condition of India's orphaned children and wanted to do something to change it.
Nick D'Aloisio: We live in an age when we want all the text we read to be condensed into the dimensions of an iPhone screen. At age 17, D'Aloisio - a British tech whiz with a clear enterprising streak - sold an app that condenses news stories to Yahoo for a cool $30 million, according to estimates discussed in The New York Times. Oh, and did we mention that he actually developed the app at age 15? His app - which uses an algorithm to extract the most salient points from long-form stories and present them in short form - attracted the attention of investors like Wendi Murdoch, Yoko Ono, and (as if this guy's cool factor couldn't be upped any more) Ashton Kutcher.
Here at Fonality, we know that anyone can helm a successful business provided they have the motivation and the tools. We can help you meet the needs of that latter category with our unified communications offerings.
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There are around 100 museums in Gujarat. This is about one third of all the tourist places listed in this state. Top cities in Gujarat with popular museums are Ahmedabad, Vadodara, Rajkot, Diu and Bhuj. The major types of museums listed here are history museums and arts museums. Then there is also science museums listed in Gujarat. You may find a list of all categories of Gujarat attractions on this page to choose from.
Visit the top museums in Gujarat. Click on a museum bellow to find the detailed information.


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Barrett's esophagus is a condition in which there is an abnormal (metaplastic) change in the mucosal cells lining the lower portion of the esophagus, from stratified squamous epithelium to simple columnar epithelium with interspersed goblet cells that are normally present only in the small intestine and large intestine. This change is considered to be a premalignant condition because it is associated with a high incidence of further transition to esophageal adenocarcinoma, an often-deadly cancer.

The main cause of Barrett's esophagus is thought to be an adaptation to chronic acid exposure from reflux esophagitis. Barrett's esophagus is diagnosed by endoscopy: observing the characteristic appearance of this condition by direct inspection of the lower esophagus; followed by microscopic examination of tissue from the affected area obtained from biopsy. The cells of Barrett's esophagus are classified into four categories: nondysplastic, low-grade dysplasia, high-grade dysplasia, and frank carcinoma. High-grade dysplasia and early stages of adenocarcinoma may be treated by endoscopic resection or radiofrequency ablation. Later stages of adenocarcinoma may be treated with surgical resection or palliation. Those with nondysplastic or low-grade dysplasia are managed by annual observation with endoscopy, or treatment with radiofrequency ablation. In high-grade dysplasia, the risk of developing cancer might be at 10% per patient-year or greater.

The incidence of esophageal adenocarcinoma has increased substantially in the Western world in recent years. The condition is found in 5–15% of patients who seek medical care for heartburn (gastroesophageal reflux disease, or GERD), although a large subgroup of patients with Barrett's esophagus are asymptomatic. The condition is named after surgeon Norman Barrett (1903–1979) even though the condition was originally described by Philip Rowland Allison in 1946.

Signs and symptoms
The change from normal to premalignant cells indicate Barrett's esophagus does not cause any particular symptoms. Barrett's esophagus, however, is associated with these symptoms:
 frequent and longstanding heartburn
 trouble swallowing (dysphagia)
 vomiting blood (hematemesis)
 pain under the sternum where the esophagus meets the stomach
 pain when swallowing (odynophagia), which can lead to unintentional weight loss

The risk of developing Barrett's esophagus is increased by central obesity (vs. peripheral obesity). The exact mechanism is unclear. The difference in distribution of fat among men (more central) and women (more peripheral) may explain the increased risk in males.

Pathophysiology

Barrett's esophagus occurs due to chronic inflammation.  The principal cause of chronic inflammation is gastroesophageal reflux disease, GERD (UK: GORD).  In this disease, acidic stomach, bile, and small intestine and pancreatic contents cause damage to the cells of the lower esophagus. In turn, this provokes an advantage for cells more resistant to these noxious stimuli in particular HOXA13-expressing stem cells that are characterised by distal (intestinal) characteristics and outcompete the normal squamous cells.

This mechanism also explains the selection of HER2/neu (also called ERBB2) and the overexpressing (lineage-addicted) cancer cells during the process of carcinogenesis, and the efficacy of targeted therapy against the Her-2 receptor with trastuzumab (Herceptin) in the treatment of adenocarcinomas at the gastroesophageal junction.

Researchers are unable to predict who with heartburn will develop Barrett's esophagus. While no relationship exists between the severity of heartburn and the development of Barrett's esophagus, a relationship does exist between chronic heartburn and the development of Barrett's esophagus. Sometimes, people with Barrett's esophagus have no heartburn symptoms at all.

Some anecdotal evidence indicates those with the eating disorder bulimia are more likely to develop Barrett's esophagus because bulimia can cause severe acid reflux, and because purging also floods the esophagus with acid. However, a link between bulimia and Barrett's esophagus remains unproven.

During episodes of reflux, bile acids enter the esophagus, and this may be an important factor in carcinogenesis.  Individuals with GERD and BE are exposed to high concentrations of deoxycholic acid that has cytotoxic effects and can cause DNA damage.

Diagnosis

Both macroscopic (from endoscopy) and microscopic positive findings are required to make a diagnosis. Barrett's esophagus is marked by the presence of columnar epithelia in the lower esophagus, replacing the normal squamous cell epithelium—an example of metaplasia. The secretory columnar epithelium may be more able to withstand the erosive action of the gastric secretions; however, this metaplasia confers an increased risk of adenocarcinoma.

Screening

Screening endoscopy is recommended among males over the age of 60 who have reflux symptoms that are of long duration and not controllable with treatment. Among those not expected to live more than five years screening is not recommended.

The Seattle protocol is used commonly in endoscopy to obtain endoscopic biopsies for screening, taken every 1 to 2 cm from the gastroesophageal junction.

Since the COVID-19 pandemic In Scotland, the local NHS started using a swallowable sponge (Cytosponge) in hospitals to collect cell samples for diagnosis. Preliminary studies have shown this diagnostic test to be a useful tool for screening people with heartburn symptoms and improved diagnosis.

Intestinal metaplasia
The presence of goblet cells, called intestinal metaplasia, is necessary to make a diagnosis of Barrett's esophagus. This frequently occurs in the presence of other metaplastic columnar cells, but only the presence of goblet cells is diagnostic. The metaplasia is grossly visible through a gastroscope, but biopsy specimens must be examined under a microscope to determine whether cells are gastric or colonic in nature. Colonic metaplasia is usually identified by finding goblet cells in the epithelium and is necessary for the true diagnosis.

Many histologic mimics of Barrett's esophagus are known (i.e. goblet cells occurring in the transitional epithelium of normal esophageal submucosal gland ducts, "pseudogoblet cells" in which abundant foveolar [gastric] type mucin simulates the acid mucin true goblet cells).  Assessment of relationship to submucosal glands and transitional-type epithelium with examination of multiple levels through the tissue may allow the pathologist to reliably distinguish between goblet cells of submucosal gland ducts and true Barrett's esophagus (specialized columnar metaplasia). The histochemical stain Alcian blue pH 2.5 is also frequently used to distinguish true intestinal-type mucins from their histologic mimics. Recently, immunohistochemical analysis with antibodies to CDX-2 (specific for mid and hindgut intestinal derivation) has also been used to identify true intestinal-type metaplastic cells. The protein AGR2 is elevated in Barrett's esophagus and can be used as a biomarker for distinguishing Barrett epithelium from normal esophageal epithelium.

The presence of intestinal metaplasia in Barrett's esophagus represents a marker for the progression of metaplasia towards dysplasia and eventually adenocarcinoma. This factor combined with two different immunohistochemical expression of p53, Her2 and p16 leads to two different genetic pathways that likely progress to dysplasia in Barrett's esophagus. Also intestinal metaplastic cells can be positive for CK 7+/CK20-.

Epithelial dysplasia
After the initial diagnosis of Barrett's esophagus is rendered, affected persons undergo annual surveillance to detect changes that indicate higher risk to progression to cancer:  development of epithelial dysplasia (or "intraepithelial neoplasia").
Among all metaplastic lesions, around 8% were associated with dysplasia. particularly a recent study demonstrated that dysplastic lesions were located mainly in the posterior wall of the esophagus.

Considerable variability is seen in assessment for dysplasia among pathologists. Recently, gastroenterology and GI pathology societies have recommended that any diagnosis of high-grade dysplasia in Barrett be confirmed by at least two fellowship-trained GI pathologists prior to definitive treatment for patients. For more accuracy and reproducibility, it is also recommended to follow international classification systems, such as the "Vienna classification" of gastrointestinal epithelial neoplasia (2000).

Management
Many people with Barrett's esophagus do not have dysplasia. Medical societies recommend that if a patient has Barrett's esophagus, and if the past two endoscopy and biopsy examinations have confirmed the absence of dysplasia, then the patient should not have another endoscopy within three years.

Endoscopic surveillance of people with Barrett's esophagus is often recommended, although little direct evidence supports this practice. Treatment options for high-grade dysplasia include surgical removal of the esophagus (esophagectomy) or endoscopic treatments such as endoscopic mucosal resection or ablation (destruction).

The risk of malignancy is highest in the United States in Caucasian men over fifty years of age with more than five years of symptoms. Current recommendations include routine endoscopy and biopsy (looking for dysplastic changes). Although in the past physicians have taken a watchful waiting approach, newly published research supports consideration of intervention for Barrett's esophagus. Balloon-based radiofrequency ablation, invented by Ganz, Stern, and Zelickson in 1999, is a new treatment modality for the treatment of Barrett's esophagus and dysplasia and has been the subject of numerous published clinical trials. The findings demonstrate radiofrequency ablation is at least 90% effective to completely clear Barrett's esophagus and dysplasia, with durability of up to five years and a favorable safety profile.

Anti-reflux surgery has not been proven to prevent esophageal cancer. However, the indication is that proton pump inhibitors are effective in limiting the progression of esophageal cancer. Laser treatment is used in severe dysplasia, while overt malignancy may require surgery, radiation therapy, or systemic chemotherapy. A recent five-year random-controlled trial has shown that photodynamic therapy using photofrin is statistically more effective in eliminating dysplastic growth areas than sole use of a proton pump inhibitor.

There is presently no reliable way to determine which patients with Barrett's esophagus will go on to develop esophageal cancer, although a recent study found the detection of three different genetic abnormalities was associated with as much as a 79% chance of developing cancer in six years.

Endoscopic mucosal resection has also been evaluated as a management technique. Additionally an operation known as a Nissen fundoplication can reduce the reflux of acid from the stomach into the esophagus.

In a variety of studies, nonsteroidal anti-inflammatory drugs (NSAIDS) such as low-dose aspirin (75–300 mg/day) have shown evidence of preventing esophageal cancer in people with Barrett's esophagus.

Prognosis

Barrett's esophagus is a premalignant condition, not a malignant one. Its malignant sequela, esophagogastric junctional adenocarcinoma, has a mortality rate of over 85%.  The risk of developing esophageal adenocarcinoma in people who have Barrett's esophagus has been estimated to be 6–7 per 1000 person-years, but a cohort study of 11,028 patients from Denmark published in 2011 showed an incidence of only 1.2 per 1000 person-years (5.1 per 1000 person-years in patients with dysplasia, 1.0 per 1000 person-years in patients without dysplasia).

The relative risk of esophageal adenocarcinoma is about ten times higher in those with Barrett's esophagus than the general population. Most patients with esophageal carcinoma survive less than one year.

Epidemiology
The incidence in the United States among Caucasian men is eight times the rate among Caucasian women and five times greater than African American men. Overall, the male to female ratio of Barrett's esophagus is 10:1. Several studies have estimated the prevalence of Barrett's esophagus in the general population to be 1.3% to 1.6% in two European populations (Italian and Swedish), and 3.6% in a Korean population.

History
The condition is named after Australian thoracic surgeon Norman Barrett (1903–1979), who in 1950 argued that "ulcers are found below the squamocolumnar junction ... represent gastric ulcers within 'a pouch of stomach ... drawn up by scar tissue into the mediastinum' ... representing an example of a 'congenital short esophagus. In contrast, Philip Rowland Allison and Alan Johnstone argued that the condition related to the "esophagus lined with gastric mucous membrane and not intra-thoracic stomach as Barrett mistakenly believed." Philip Allison, cardiothoracic surgeon and Chair of Surgery at the University of Oxford, suggested "calling the chronic peptic ulcer crater of the esophagus a 'Barrett's ulcer, but added this name did not imply agreement with "Barrett's description of an esophagus lined with gastric mucous membrane as stomach." Bani-Hani KE and Bani-Hani KR argue that the terminology and definition of Barrett's esophagus is surrounded by extraordinary confusion unlike most other medical conditions and that "[t]he use of the eponym 'Barrett's' to describe [the condition] is not justified from a historical point of view". Bani-Hani KE and Bani-Hani KR investigated the historical aspects of the condition and found they could establish "how little Norman Barrett had contributed to the core concept of this condition in comparison to the contributions of other investigators, particularly the contribution of Philip Allison".

A further association was made with adenocarcinoma in 1975.

References

External links

 Barrett's esophagus at National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK)
 Barrett's esophagus Video Overview  and Barrett's esophagus Health Information at Mayo Clinic

Esophagus disorders


I almost didn't read Free Food because it's long. 576 pages in print, and it was nearly 1,000 screens on my tablet. That's intimidating for a lady who mostly reads YA. I dug in anyway, and it was worth it.
The narrative is omniscient, but the action is centered on Casey Han, the first-born daughter of Korean-Americans living in Queens and running a dry cleaner with sewing services on the Upper East Side of Manhattan. Casey has just graduated from Princeton. She has no job, an inflexible father, and a white boyfriend. She is a high achiever and a big spender who has deferred her entry to the Columbia Law School to...she's not sure what. Her sister, Tina, is even more of a wünderkind: pre-med at Dartmouth, virgin, Korean med student boyfriend. Tina is also more agreeable.
I bookmarked a million pages and am intimidated by them, too, so I'll finish up this shite review from memory.
Trying to sustain oneself in one's twenties in NYC is hard. Casey's struggles are different than mine. She's got access to tons of money, but she hangs out with a finance crowd. An evening of drinks costs $50 minimum. Plus Casey's working class childhood and Princetonhood, or maybe an inborn proclivity, Casey likes to buy expensive things. She spends $4,000 on high end work clothes when she doesn't have a job, just because she's pissed at a girl she went to church with as a child. My blood pressure is rising even as I type. Did I mention the book takes place in the 1990s? $4,000 has the spending power of about $7,200 now. Aack.
Probably some people wouldn't read the book as a story about irresponsible spending. Some of the people in Casey's life admire it. I get that it could be a PTSD response--about physical violence or the trauma of being an immigrant scholarship student of color in the Ivy League, but it still gives me the chills. That's my own trauma speaking.
The novel is also about romantic relationships, family, friendship, mentors and proteges, and the old coming-of-age thing. Casey is believable, sympathetic, and surprising.


Latest Hollywood films DeadPool 2, The New Mutants, and Gambit have had their release dates changed. This is part of the major changes relating to the new upcoming Hollywood movies. Those who were looking forward to watching DeadPool 2 should be more excited because the release date has been set to be May 18, instead of June 1, 2018.
According to Hollywood gossips, the changes are expected to ease competition among the new Hollywood movies. In fact, the changes put DeadPool 2 ahead of the other latest Hollywood movies because its release date was fast tracked. The film is one of those eagerly anticipated around the mid of this year.
Although there are other competitive Hollywood releases like the Avengers: Infinity War set to be released early in the same month of May, DeadPool 2 stands out as the most awaited movie. It could be due to the publicity that made the movie go viral after its first teaser trailer hit the market towards the end of last year.
The film's new date seems to be strategically chosen to coincide with the boom summertime ticket sales. It will be competing for the season's boom with Sony's Slender Man. This is one of the horror movies that released its trailer recently. The controversial reception that the Slender Man had received confirms to the fans that DeadPool 2 will steal the show immediately after its release.
Another 21st Century Fox studio movie that is expected to come after DeadPool 2 is the Solo: A star Wars Story, the bigger box office story. It is scheduled to come a week after DeadPool hits the theatres. This other film is yet to make its trainer known to the fans. Fans are already expressing their interest as to how the Star Wars story will turn out to be for DeadPool 2 in the second weekend.
At the moment, ComicBook.com's anticipation ranking puts DeadPool 2 at 4.09 out of 5.
The rescheduling of the Hollywood movies that mainly affected DeadPool 2, Gambit, and The New Mutants seems to be a deliberate marketing strategy. The studio Management is making an effort to avoid unnecessary competition of the three films. Recent Hollywood updates reveal that the changes raised emotions amongst the Management to the extent that the director Gore Verbinski left the project.
Inside sources have cited one of the reasons for the change of the release dates as an effort to avoid an overlap with X-Men movies in market places abroad. The Management foresaw Mutants and DeadPool 2 being in the theatres at the same time. They noted that this was not going to be good for the studio and for the fans.
There is no doubt that Hollywood movies are dominant in the industry. Any matter relating to the movies such as determining and altering the release dates is expected to be sensitive. It is also worth noting that such a simple matter as release date can determine the survival of the movies in the market. This requires a careful and tactical approach on the part of the Management when handling them. Otherwise, a fall out among the stakeholders is likely to occur.
Stay tuned with FilmyWhisky for more Hollywood Updates.


The funny thing about mayo is: like many other now tasty to me foodstuffs, I used to dislike it. It probably didn't help that the first memory I have of it is on warm bologna sandwiches where it was gloopy and just plain weird. I think the first time I started liking mayo was when it was cold and used as more of a dipping sauce as opposed to a spread. For the record, extra crispy fries dipped into garlic mayo is a resounding YES in my books.
If you're not a mayo fan, you have to try Kewpie mayo: the ubiquitous clear, red-topped squeeze bottle of mayo found in practically every Japanese household. It's tangy from rice vinegar, slightly sweet, and extra creamy. It is absolutely delicious and found on a bunch of classic Japanese dishes like takoyaki and okonomiyaki. It works especially well as a stand in for anywhere your might use mayo, like this tuna salad.
This salad is a little umami bomb and absolutely the perfect moreish no-carb snack. To a can of drained tuna, I stirred in a little bit of kewpie mayo, white miso instead of salt, celery for crunch, and green onions for bite. Instead of bread or crackers, I scooped it onto crispy seaweed. Super oishii!
Mix together the miso and kewpie until smooth. Add the drained tuna, celery and green onions. Mix throughly and season to taste. Enjoy on it's own or on top of crackers, toast, or seaweed.
Notes: Adjust the miso content to taste, miso pastes vary greatly in terms of saltiness. Also, feel free to add more kewpie mayo if needed.
i love the flavor of miso, and korean seaweed can make anything taste good! will have to try this on a hot, lazy day.
If you don't have access to kewpie mayonnaise, would regular mayonnaise work ok here, too?
yes, for sure! you might want to add a touch of rice vinegar and a pinch of sugar for a bit more of a kewpie mayo taste, but it'll taste just as good with plain mayo.
I'm with you – I used to be a big time mayo hater but I'm slowly coming around to it. I still don't like super mayo heavy pasta or potato salads, but when I was in Chile (where they are mayo-obsessed) I had hot dogs slathered in it and they were actually delicious! I keep meaning to buy kewpie mayo – I pretty much love any Japanese food so I'm pretty sure I'll be a fan!
This sounds really great. Add a little rehydrated seaweed, some cucumber for crunch, a shallot, or???
I use Miso all the time in soups, stews, vegetables, etc. Keeps forever in fridge because it is so salty. It also comes in a lower sodium formulation which is what I use.
Mayo-lover over here! Definitely want to try this, though I'd probably end up putting it all between a couple slices of bread.
omg I LOVE this!! You make miso magic, lady — still not over that miso + mustard genius in your beautiful green bean salad, and now miso + MAYO (my favorite thing ever, lololol #fattylife). This is so great.
I made this for lunch twice this week! I didn't have kewpie mayo so I used light mayo and subbed the celery for pickled jalapeños. I made a basic tuna sandwich with it and it was SO EFFING GOOD. I'll be making this all the time now. My tuna game has been forever changed.
This was amazing! I hate canned tuna and am always looking for ways to make it good. This is exceptional.
I had to make my own Kewpie Mayo from a recipe I found on serious eats and frankly had to sub a few items… I'd say straight Mayo with rice vinegar and Miso would recreate what I came up with.
The Kewpie Mayo on its own was super unique and tasty but it didn't stand up to the white Miso at all… Which is odd because it's a spicier stinger flavor than American Mayo.
Anyhow I just ate 3 cans of tuna and wanted to offer a big thanks and kudos for a great creation.
This sound so good. I have never used miso paste for any meals that aren't soups. I'll have to try it soon!


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Q: Does a HIVE table get duplicated across all nodes of a Hadoop cluster When I create a HIVE table, does the table get created on all nodes of the Hadoop Cluster? When I load the data into the table, does the data get replicated across all these nodes?
My guess is yes?

A: Hive is just a metadata layer on top of hdfs. So to answer your question you would want to simply think about how replication works within hdfs.
Your question about "on each node" is just not the correct mental model of how hdfs replication works. hdfs replication knows/cares nothing at all about hive tables. It only knows about data blocks.
There will be K copies of each data block of the hive table - where k is the replication factor configured for the given hive table.  The association of the data blocks to particular nodes is driven by the local/rack/cluster location policy of hdfs.

A:
When I create a HIVE table, does the table get created on all nodes of
  the Hadoop Cluster?

No.
Hadoop datanodes are not mirror images of each other. These are just data storage units whose the storage and meta is managed by NameNode.
Your hive table will be replicated based on the replication factor set. If your replication factor is one, there will only be one copy.

When I load the data into the table, does the data get replicated
  across all these nodes?

No.
As I specified earlier, hadoop datanodes are not mirror images of each other so they all don't contain all the data. They just contain the defined number of copies of data in the cluster(based on the replication factor set).
The data loaded by you gets replicated based on the replication factor (on nodes decided by the NameNode).