Changemakers - T-Mobile Changemaker Challenge - Are You Ready for The Change?
The Change is a funk band empowering youth and transforming lives through the power of music.
Rocking against Bullying at The Myrtle Beach Speedway!
When I was in middle school, I experienced a lot of bullying. It caused me to experience severe depression and anxiety. A classmate of mine took his life when he was only 11 years old and I understood how he must have felt because I had contemplated suicide too. I wrote a song to try and express my pain and that is when I first recognized the power of music. Not only did it help other people cope, but it helped me heal as well. I started sharing my story and it was so powerful to realize I wasn't alone and even more powerful to educate other youth that they weren't alone either. I was blessed to start a band with some friends who had gone through some really tough times too. We started touring full time and sharing our stories with as many kids as possible. We encourage youth to use their pain for good. The power of this peer to peer mentorship is extraordinary!
Bullying is a serious problem in our country. 20% of high school students say they have seriously considered suicide within the last 12 months because of it. We believe Bullying exists because of lack of education, lack of support, and lack of empathy. We need to empower youth to stand up to bullying but we also need to reach those doing the bullying. Kids are DYING because of bullying and we won't stop until we have put an end to this epidemic.
My band, The Change, tours the East Coast spreading an anti-bullying message to schools, after-school programs, community centers and more. We aim to ignite passion in youth that inspires positive choices, self-respect, empathy, and accountability. During our concerts, we share stories of our struggles and stress to youth that they aren't alone, that being different is a strength, and that they too can overcome adversity. We believe the lack of empathy is the root of the bullying epidemic and we must instill empathy in this generation if we want to see change. We aim to unite others under the premise that we all hurt & that we can all heal. We believe everyone, even bullies deserve to feel safe and supported. We teach that no matter what you've done, no matter what someone has done to you, no matter what you're going through, you can rise above and you can use your experiences to help others. We believe this message empowers youth by giving a purpose to their pain. Empowered, empathetic, educated youth do not bully others and they are at a significantly smaller risk of taking their own life. We plan to empower as many youth as we can and won't stop until we see significant change.
We often walk into an auditorium filled with 1,000+ students expecting a musical performance who leave receiving a life-changing experience. We create a safe space where kids can interact and express themselves freely without judgment. We perform upbeat popular songs that bring instant engagement and share our struggles through powerful storytelling & passionate spoken word. We convey that you don't have to let your pain define you & your struggles can actually empower you. Each participant identifies what they personally stand for and we invite them to share it on the mic. Collectively, we unite and pledge kindness. We end with our empowerment song Be The Change & stress how important it is to reach out if you're struggling. At the conclusion, we meet one on one with students, provide empowerment packages & invite them to become difference makers in their community.
We uniquely combine music, relatable stories, peer to peer mentorship, under-resourced youth & a partnership with Free2Luv, a youth empowerment nonprofit. This combination is very powerful. Kids listen to us because it's easy for us to understand where they are coming from & they take us seriously because we speak their language. We use popular music to connect and share our deepest hurts & pain with them. We help them realize they're not alone & that they too can rise above. Our interaction doesn't end at the stage. Through Free2Luv, we provide resources to help students continue to thrive.
We have reached over 35,000 students in the past 4 years. We regularly have administration & teachers thank us because our program positively impacted their school. Students are inspired to reach out for help & they are inspired to help others. Some have even started clubs and asked how they could use their talent to make a difference. We have people tell us that we saved their lives or kept them from hurting themselves. Recently we had a counselor call us to tell us a student came to him after one of our shows. She had a note, and a plan to commit suicide but she was inspired by our program to come forward and ask for help. We have had approximately 10,000 people take the Free2Luv Kindness pledge with us with a goal to reach 1 million people. Together we pledge a zero tolerance for hate, to being the best me we can be, to choose kindness and stand up to bullying.
We joined forces with Free2Luv, a national nonprofit who shares our mission. Together we have created UNSTOPPABLE, a program specifically aimed at serving at-risk youth. Our goal is to instill hope in our peers who are often marginalized, bullied & afforded little opportunity to thrive and prosper. We hope to inspire these kids who haven't had a chance to experience pride or self worth & give them a platform to become a leader. We will form leadership alliances in each area we visit with our Unstoppable concert tour. These groups will be made up of diverse group of students who otherwise may not have realized their leadership potential. We plan to stay connected with these groups & help them make a long-lasting change in their communities.
More than ever we need to offer our empowerment programming free-of-charge, especially to underserved communities. However, it is very expensive to tour. Some of our expenses include hiring professional sound & lighting technicians, rental vans to haul equipment & hotel accommodations. In the past, we have done fundraisers and obtained sponsors but we have been limited in the number of tours we could afford to do. We hope to increase our outreach this next school year but in order to do so, we need funding. We volunteer our time & often have significant out of pocket expenses that aren't sustainable in the long run. We have all the pieces of the puzzle ready to go out and Be The Change and we are ready to take our program to the next level.
Our program is focused on encouraging others to make a difference. We believe that the empowering feeling you get from helping others changes your life while you are changing the lives of those around you. We hope to inspire others to Be The Change in their own communities and inspire youth to become leaders. Free2Luv has a youth advocacy program where youth can join hands and be a part of the movement alongside many famous celebrity ambassadors. There are advocates all over the United States and we are recruiting more each day. I was also instrumental in forming The One Voice Project, a group of youth musicians in my hometown who have made a commitment to use their music to make a difference in the world. They often tour with us bringing more diversity into our outreach. As we start the leadership alliance in each community we visit, the ripple effect will expand even more! Music unites people in a way that nothing else can. We also hope to inspire other musicians to spread the love. We have a unique platform to use our voice and we believe that is it our obligation to use that platform to make a difference every chance we get. Anyone Can Be The Change.


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Q: Bash to SSH copy Linux command output to remote Windows Host I need to take the output of some Linux commands and files (containing letters, numbers, symbols, white space and newlines), stored in a local Bash variable (dynamically generated) and then send the contents of the variable to a remote Windows host using SSH, appending to a specified file.
I've attempted many variations of the following command without any success:
 echo "ECHO $result >> $out_file" | ssh $remote_host
The special characters (<, > etc.) and newlines that exist in the input make it a mess, meaning the input is interpreted as many commands rather than a single chunk of input.
It is infeasible for me to change the format input itself, do I need to do some escaping?
Any tips on how I can achieve the desired result?
Edit: the commands themselves are hard coded, as are their arguments. The output of each command is concatenated to a variable, $result, and then the intention is to send the content of that variable via SSH and append it to the remote file.

redpajama_set_name: RedPajamaStackExchange


• Students will gain an understanding of different careers and fields such as: Pathologists' Assistant, Physical Therapy, Occupational Therapy, Radiation Therapy, Forensic Investigation, Mortuary Science, Doctor of Pharmacy, Pharmacology & Toxicology, and Physician Assistant.
The program will be offered from Monday, July 16 through Thursday, July 19, 2017 from 9:00 AM to 3:00 PM. All sessions will be held on Wayne State University's campus at the Eugene Applebaum Pharmacy and Health Sciences College.
For students attending one of the 15 C2 Pipeline schools there is no charge for this camp (for a list of schools visit our website). The charge for this program for non-C2 pipeline school students is $150 per student. Scholarship reductions are available based on a student's school lunch status; however, proof of eligibility must be submitted. This session is limited to 20 students.
Submissions for this form have closed on July 10, 2018 at 11:59 pm.


Do you know someone who spends far too much time on YouTube? Are they likely to spend countless hours watching videos of strangers dancing, monkeys falling from trees, and cats riding Roombas? Do they constantly forward videos to you, then harass you about whether or not you've watched them? Do they have YouTube hooked up to their television?
If you answered yes to any of these questions, your friend may have a clinical addiction to YouTube. Show them you recognize their video watching excellence with this free funny award. Download the certificate, type in their name, print it out, and post it on their office door or cubicle.


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Big Director wins against it! Steelman is over 3 years of victims of the United States
December 28th (Wen / ESPN Brooke Pryor Compile / Love) Although many people discuss the old four-dimensional Savage – Rodrisberg retired, but in this week's "04 Sanjie" In the conversation, his head covered the quartz of the Pony – Rivers Reeds. The 38-year-old quarter-saving battle in Pittsburgh 28-24, a wave of reversed offensive, and this victory also helped steel people locked the United States. This is what they won another partition champion after 2017.
Before the third quarter left, the steel man returned 17 points behind 7-24, but as the big passed to the outside Didtai – Johnson's 39 code, the steel man was blown up. Anti-tension angle. Within 11 minutes, Datinatedly completed twice to pass two times, found near-end Frieker Eric Ebron and the outer handlers Zhu Zhu – Smith Shrust completed the anti-superchard. This victory is also a third victory, which is 17 points above in large careers, and 17 points is also the biggest score of his career.
At the same time, the game is also the first time after the first half of the first half of the second half, the steel man is in the second half of the first half, and the steel man has fallen in the second half of the first half of the second half. With this victory, the steel man has locked the US Lianbei District Championship; more importantly, they stopped the three-game-losing battle, avoiding the first opening season 11 winning victory after NFL history, swallowing 4 The team, although their performance once, they think they may create embarrassing history.
In the first half, the steel man only took a decline in the anti-fall ball in the red area, other offensive performances were lacking, and half of the 5 times, they also explained their bad offensive status, once people think that this game is It was a turning version of the Tiger last week. And I opposing the characteristics of the small horse, the heat of the game, Rivers and the rookie run Wei Jonathan – Taylor's brave, Taylor 2 touches the ball to make the steel person's defense is very hard.
However, the next half of the fire has all been opened, and the whole game 49 pass 34, the 342 yards were promoted to 3 times, in which the second half of the 25 passed 25, the transfer of 244 yards was high. At the same time, the steel man's defensive group also retrieved in time, starting 3 consecutive times after the first half of the small horse, let the opponent abandoned, then Mike Hilton completed the key copy; then in the last wave of attack and defensive performance Rivers have passed the ball for 3 consecutive passes.
This game is also the first half of the first half of the black panther, and he has not been copied in this game, and he is not critical to the victory of steel people. Although the steel man's pavement is still poor, the status of the game seems to be a little slow, but at least they avoid the worst situation; now they can meet the arrival of the playoffs with the US Lianbei champion gesture.
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Love! On only 1 day of the winner of the baseball career, the most expensive ticket is the most expensive check.
Protest anthem NFL players play without the ball anchors often accompanied by his girlfriend around Jieyou Chou

redpajama_set_name: RedPajamaCommonCrawl


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El Clásico de Independencia es una rivalidad que enfrenta a dos de los equipos más laureados y tradicionales de Chile: Universidad Católica y Unión Española, ambos nacidos en la Comuna de Independencia.

Esta enemistad se fue diluyendo con el pasar de los años, debido a la mudanza de Universidad Católica a su actual estadio San Carlos de Apoquindo, ubicado en la Comuna de Las Condes. Sin embargo, en la actualidad se ha popularizado como uno de los partidos más llamativos del futbol chileno.

Los partidos entre la Unión y la UC siempre dejan cosas que nutren la rica historia de esta rivalidad, que comenzó a gestarse en 1939, cuando a propósito de la Guerra Civil de España el cuadro de Santa Laura sólo jugó un partido y entró en receso. El plantel, como era de esperarse, se desmanteló y los jugadores recalaron en diferentes clubes. Los principales elementos hispanos se integraron a la Universidad Católica.

Nombres como Fernando y Jaime Riera, Felipe Mediavilla y Luis Vidal, ex referentes de Unión, se transformaron en destacados jugadores de la UC. Fueron llamados "renegados", al igual que algunos dirigentes que siguieron el mismo camino. Además, Sergio Livingstone, que había realizado la etapa formativa en Unión Española, ya se había integrado a Católica luego de hacer un alto en su carrera deportiva debido a los estudios.

Historia y partidos destacados
El 21 de abril de 1946 se disputó el primer encuentro entre cruzados e hispanos en el Estadio Independencia. Se trataba de un significativo capítulo de una relación deportiva que se ha mantenido hasta hoy, con una rivalidad que ha crecido en el último tiempo.

Pero por momentos la rivalidad se escapó de las manos, incluso antes del primer partido jugado en el Estadio Independencia. En 1945, en el Estadio Nacional, y cuando el marcador favorecía a Católica, los jugadores y cuerpos técnicos protagonizaron incidentes, incluso con participación del público que por esos años podía apreciar el fútbol no sólo desde las gradas, sino que también sentado al borde de la cancha. Pero los incidentes más graves entre estos rivales se produjeron en 1960, y finalizaron con la primera suspensión de un partido por violencia en el estadio.

La relación entre los jugadores siempre fue buena por esos años en que compartieron el barrio, más allá de algún disgusto en el terreno de juego. Cuentan las crónicas que los jugadores de ambos equipos no tenían problema con llegar juntos a los campos de entrenamiento, o bien compartir con los compañeros del otro equipo cuando la jornada finalizaba.

Católica jugó en el viejo Estadio de Independencia entre 1946 y 1967. Pese a la mudanza, la relación se mantuvo.

La UC tuvo grandes jornadas en Santa Laura, estadio que albergó la localía cruzada para las campañas de 1984 y 1987, temporadas en que dio la vuelta olímpica. Por contrapartida, Unión Española también ha tenido éxitos en San Carlos, y no sólo enfrentando a la UC, sino que también por la obtención de la liguilla de 1993 ante Temuco, que le permitió realizar una gran Copa Libertadores en 1994.

El "Clásico de Independencia Retro"
Cuando parecía que la rivalidad entre hispanos y cruzados era cosa del pasado, a partir de 2013 los partidos entre estos equipos han ido más allá de lo estrictamente futbolístico. Incluso algunos jugadores –fuera de micrófono- reconocieron tenerle bronca al rival. La disputa del título de ese año, que quedó a manos de Unión en reñido desenlace ante la UC, colaboró para que los planteles se tuvieran sangre en el ojo en cada enfrentamiento.

En la Copa Chile de 2013, los jugadores se fueron a las manos en San Carlos de Apoquindo. Patricio Rubio, entonces delantero hispano, luego de convertir de los doce pasos en el partido de ida, con su mano derecho hizo la señal de la paz, que en este caso buscaba todo lo contrario: tocarle la oreja a los cruzados al mostrar el número dos, a propó- sito del mote de "segundones". En la revancha, el entrenador de Unión, José Luis Sierra, dedicó el mismo gesto a la barra estudiantil, esta vez Santa Laura, donde la discusión subió de tono y la cancha por minutos pareció más un ring de boxeo.

Pero esta relación tuvo otro capítulo. A mediados de 2014 la Universidad Católica anunciaba que Milovan Mirosevic, principal referente de los últimos años, no seguiría en la UC por una decisión directiva. Unión Española no tuvo problemas para abrirle las puertas al jugador más identificado con la franja.

Referencias

redpajama_set_name: RedPajamaWikipedia


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Don't Wake The Beast - An Interview with Kill The Beast
20 March 2017 | Eleanor Turney
"We're all friends because we're weirdos," says Clem Garritty, one fifth of theatre company Kill The Beast. "We just hope other people like the stuff that makes us laugh." Given that Soho Theatre has programmed a triple-bill of the company's work, it seems a reasonable bet. "We'd just foolishly come off the back of Edinburgh, where we did two shows a day in different venues. We sort of thought, well, if we can do that, and we're still standing and we just about still like each other, then we can probably do this."
The trilogy spans The Boy Who Kicked Pigs, which is an adaptation of a Tom Baker novel, original werewolf comedy He Had Hairy Hands and new sci-fi show Don't Wake The Damp. Garritty says: "We had always constructed them as a bit of a trilogy – each one is inspired by a different period of twentieth century cinema. TBWKP is early silent cinema and German Expressionism and James Whale horror; HHHH was really inspired by folk horror, Hammer Horror and stuff like that, and DWTD was inspired by 80s and 90s techno, body horror like The Thing and Alien."
"It's a dream to be putting our plays on in the main house at Soho," Garritty tells me. "We do keep trying to remind ourselves that and not get tied up in the panic of re-learning a show from two years ago. We're an oddball that sits somewhere between theatre and comedy, and Soho is the venue that we love going to, because they have that really good mix. We started out as a theatre company, and always will be a theatre company, but we have this love of comedy, too. They seem like the perfect little hub of both of those things."
Kill The Beast's madcap and macabre shows span comedy, theatre and horror in a way that's unlike anything else. The group met at Warwick University, where they were "encouraged to get together and make our own stuff". Now based between London and Manchester, the five members of the group work in an interestingly collaborative way:
"We have a WhatsApp group and we're always sending each other ideas. When something clicks, we go away and start writing scenes, come back together, read them out loud, and the stuff that we all laugh at stays in. Then we hand them over to the person to the left, who'll take it away and give it a re-jig. We're learning what each person is good at. We write the script, and then we have a team of exciting, talented designers and makers come on board. With DWTW, we've done projection mapping with moving screens, so we ended up working with a projection designer who usually does architecture."
Garritty directs the shows, and Natasha Hodgson, Zoe Roberts, Ollie Jones and David Cummings play every character, swapping hats, wigs and facial expressions with alarming rapidity. The group has a strong aesthetic and draw inspiration from a wide range of sources:
"In terms of theatre, it's companies like 1927, with their highly stylised work and storytelling, and smaller companies like Ridiculusmus, Theatre Ad Infinitum – people who use little in terms of props and set to tell huge stories. We want to have a strong visual identity like those guys do. In terms of other stuff, The League Of Gentlemen always pops up, but also things like Nighty Night and Alice Lowe who's just made Prevenge, which is amazing. We go and see a lot of sketch comedy, rather than stand-up. Lazy Susan are amazing and a group called Goose, Max and Ivan. We lean towards narrative-based comedy, and we like slightly odd stuff, obviously."
"We're massively inspired by film: when we're talking about the show or a scene we'll say 'we pan across a field, we crack through the window of the house into the room'. We write with film in mind, and then when we get it on its feet we have to work out how to show a camera panning across a motorway and then zooming into a Morris Minor, or whatever."
Having seen all three shows, it's clear that the company are developing their down style and voice. What can new audiences expect from their shows, I ask? "It's a fun night out. It's visually interesting and hopefully unlike stuff you've seen before. It's very silly and British, and slightly dark and twisted. There are original songs. The costumes are very old and smelly now, so it's very much a 4D experience…"
If that doesn't make you want to snap up the last few tickets, then Garritty also hints that this might be the last outing for these shows: "There's a nice feeling to doing them as a trilogy, and then we're thinking about cracking on with some other stuff… this feels like a good way to maybe pack them into the attic for a little while. So, if you want to see them, this is your chance."
Kill The Beast have three shows at Soho Theatre from 20th March: The Boy Who Kicked Pigs, He Had Hairy Hands and Don't Wake The Damp. Find out more and book tickets here.


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Global power rental market was valued at USD 10.02 billion in 2014 and is expected to reach USD 20.30 billion by 2020, growing at a CAGR of over 13.0% during 2015 and 2020.
Zion Market Research has published a new report titled "Power Rental Market by End-User (Oil & Gas, Industrial, Construction and Others) For Peak Shaving, Base Load/ Continuous Power and Standby Power Applications: Global Industry Perspective, Comprehensive Analysis and Forecast, 2014 - 2020" According to the report, The global power rental market was valued at approximately USD 10.02 billion in 2014 and is expected to reach approximately USD 20.30 billion by 2020, growing at a CAGR of over 13.0% between 2015 and 2020.
Power is generated by burning fossil fuels such coal, oil or gas to generate steam that drives large turbines that produce electricity. Power rental is referred as plant hire which provide flexibility, reliability, speed and cost-effectiveness. Power on rent can deliver complete operating power packages as well as provide scalable components within large power station installations to various industrial applications.
The global power rental market is expected to witness significant growth over the forecast period on account of increasing demand for power across the globe. The increasing power consumption, development of power infrastructure, and increasing construction & development is anticipated to boost the demand of power rental in the coming years. Furthermore, increasing demand for power from oil & gas industry is also a major driving force of the power rental market. However, presence of stringent regulations coupled with rising environmental awareness may hamper the market growth in the near future. Nonetheless, increasing demand for power in emerging economics is expected to open up new growth avenues for the power rental market in the years to come.
Oil & gas, industrial, construction and others are the key end-users of the power rental market. Industrial segment emerged as the leading application segment by accounting for over 22 % of the total revenue generated by the power rental market in 2014. Construction is another key outlet of power rental market and it held over 20% share of overall market in 2014. This is mainly due to growing infrastructural development in the emerging economics. Oil & gas is also expected to exhibit substantial demand within the forecast period.
Based on application, the power rental market can be segmented into peak shaving, base load/continuous power and standby power. Base load/continuous accounted for 45.56% share of total revenue generated in 2014. Base load/continuous segment are expected to exhibit a significant rise in light of the increasing demand from various sectors such as oil & gas, mining and construction and others. Peak shaving is another leading application segment owing to rising awareness among energy intensive industries in order to control the charged over high energy demand during peak hours.
In terms of geography, Middle East & Africa dominated the global power rental market and accounted for 31.63% of total revenue in 2014. This growth is mainly attributed to growing construction and developmental activities in this region. North America was the second largest regional market with 21.32% share of total revenue generated in 2014. Asia Pacific holds immense potential for the industry growth over the forecast period. Recently, emerging economies including India, Brazil, and Saudi Arabia have been contributing significantly to the demand for power rental in industrial sector owing to rapid industrialization.
Some of the key players include in power rental market such as Atlas Copco, United Rental, Cummins Inc, APR Energy Inc, Caterpillar Inc and Power Electrics.
Zion Market Research is a market intelligence company providing global business information reports and services. Our exclusive blend of quantitative forecasting and trends analysis provides forward-looking insight for thousands of decision makers. Zion Market Research experienced team of Analysts, Researchers, and Consultants uses proprietary data sources and various tools and techniques to gather, and analyze information. Our business offerings represent the latest and the most reliable information indispensable for businesses to sustain a competitive edge.


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"Why Married Women Are Frowned At When They Cheat" – Comedian Ali Baba Writes
Ben Murray Bruce shift birthday celebration till after election
February 18, 2019 Olumide Alake 0 Comment 2019 General Election, 2019 Presidemtial, 2019 Presidential Election, Senator Ben Murray Bruce
– Sen. Ben Murray Bruce has shifted his birthday celebration until after the election
– He stated that he can't celebrate while the country is in trouble
Nigerian Senator and businessman, Ben Murray Bruce clock 62 today and many have taken to various media to celebrate and congratulate him.
However, the Senator disclosed that he wouldn't be celebrating the birthday until after the elections.
Sen. Ben Bruce in his statement disclosed that all festivities will be postponed until after the elections.
He noted that he couldn't see himself celebrating when the country is going through challenging times.
Sen. Ben Bruce in a satement on Twitter wrote, "Thank you to all my friends and followers who have felicitated with me on my birthday. I do appreciate your warm greetings and show of love. However, any festivities will be postponed until after the elections. I cant see myself celebrating when the nation faces challenging times."
The Senator in a previous statement has also reacted to the postponed 2019 General Elections by the Independent National Electoral Commission INEC.
See More: Election Postponement Tragedies Claims The Lives Of 7 Across The Country
According to the statement released by INEC, the Presidential and National Assembly Election has now been shifted to the 23rd of February while the Governorship election has been shifted to the 8th of March.
Sen. Ben Murray Bruce was born on the 18th of February 1956, he is a politician registered under the Peoples Democratic Party PDP, he is also a business magnate and the founder of Silverbird Group.
The Senator is married to Evelyn Murray Bruce and has four children.
He was elected to the Nigerian Senate in March 2015 under the PDP, where he currently represents Bayelsa East constituency of Bayelsa state.
Sen. Ben Murray Bruce is also an advocate of the Common Sense initiative and he is a supporter of the PDP Presidential candidate, Atiku Abubakar.
← I'm now officially married to my baby daddy – Gifty Powers
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2019 General Election: NPF calls for the support of Nigerians
February 13, 2019 Olumide Alake 0
President Buhari says only God can punish the PDP
Kwara 2019 Governorship Election: Alh. Lai Mohammad urges voters to comes out says their security is guaranteed
March 8, 2019 Olumide Alake 0


Cool no worries mate, just keep in mind everything is slow in soil so easy to over feed mate, less is more and be patient to see the results of any changes you make.
Show BB your plants mate :) you would be wise to get his opinion mate!!
Issue reason: Good work keep it up..
Issue reason: Thank you for taking part in the 10th Birthday In It To Win It competition!!!
Issue reason: Organic and Amendments and Pests and Disease Specialist.


Since XenServer 7.2 is now available, we'll see how to upgrade.
Note: XenServer 7.1 is still in LTS. No emergency to upgrade on 7.2. Though, since it's a "small" revision, the risk to upgrade is low.
On Windows, use a dedicated program that can write ISO to USB drives.
You can upgrade your previous XenServer version directly to 7.2 since XenServer 6.2, 6.5, 7.0 and 7.1.
Are you upgrading from an older version than XenServer 7.0? Or do you have the old partition scheme? Please follow instructions in our previous blog post to switch to the new one.
You can always live migrate VMs from an older XenServer to a newer. The opposite IS NOT POSSIBLE.
Also, always check to empty your VM CD drives and disable HA during the operation.
Better backup reports to know everything on your scheduled jobs.


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Tags: Human rights
Vietnam – a country of safety: Algerian diplomat
With all the things it has done for its people, Vietnam can be proud of itself, said Algerian Ambassador to Vietnam Boubazine Abdelhamid, adding that he thinks Vietnamese people are very happy, and all must recognize that Vietnam is a very safe country.
Studies reveal that unlike Chinese and Southeast Asian villages during feudal time, the Vietnamese villages and communes applied a stricter self-rule regime with a fairly large scope covering various aspects of the community life, such as production organization (primarily irrigation), relations between different social strata, security protection, management of public property, public land division, learning promotion, social relief, organization of cultural and spiritual activities, and the implementation of tax and conscription duties.
Human rights protection and the use of new energies
The 2013 Constitution of Vietnam has clearly stated in its Article 43: "Everyone has the right to live in a clean environment and has the obligation to protect the environment". Against the backdrop of the Industrial Revolution 4.0, this principle must be strictly adhered to so as to ensure the human rights related to the living environment, especially with the use of green energy, clean energy and renewable energy for sustainable national development.
Vietnam – New potential, global position and aspirations
Vietnam is a friend, a reliable partner of all countries and a responsible member of the international community. Its stature has generated new potential toward rapid and sustainable development in the coming years.
Vietnam gathering momentum for sustainable development: Party leader
Party General Secretary Nguyen Phu Trong has highlighted Vietnam's achievements in 2022 and their impacts on the country's growth in 2023. He made the remark in an interview to the Vietnam News Agency on the occasion of the Lunar New Year Festival 2023 and the 93rd anniversary of the Communist Party of Vietnam (CPV) (February 3).
Official Gazette issues Nos 101-104/2022, issued on December 26, 2022
Prime Minister's tour of Europe highly successful: Foreign Minister
Prime Minister Pham Minh Chinh's official visits to Luxembourg, the Netherlands and Belgium, and attendance in the ASEAN - EU Commemorative Summit were highly successful, Minister of Foreign Affairs Bui Thanh Son told the media.
Vietnam rejects US' decision to place it on special watch list for religious freedom violations
Vietnam on December 16 denounced the United States' inclusion of the country in its special watch list for alleged engagement in and tolerance of religious freedom violations as a decision that is based on an "unobjective assessment" and incorrect information.
Vietnam prioritizes upholding international commitments to human rights: UN Resident Coordinator
Among Vietnam's pledges as part of its election to the United Nations Human Rights Council (UNHRC), it is encouraging that the country prioritized upholding its commitments and obligations under the international human rights treaties, of which Vietnam has ratified seven out of nine, according to UN Resident Coordinator in Vietnam Pauline Tamesis.
PM: Vietnam appreciates cooperation with Luxembourg
Vietnam always attaches importance to comprehensive cooperation and partnership with Luxembourg, a founding and important member of the European Union, said Prime Minister Pham Minh Chinh during talks with his Luxembourg counterpart Xavier Bettel on December 9 (local time).


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text:
167.221. Lunch programs in urban districts — managers and assistants, designation, duties — deposit and withdrawal of funds. — 1. The board of directors of any urban school district which has established facilities for supplying lunches to children attending any of the schools in the district in accordance with the provisions of section 167.211, may designate the secretary of the district or some other person as manager for any or all of the facilities in the schools and, by rule, prescribe his duties. The board may also designate and appoint submanagers, cashiers and other employees to staff the facilities and, by rule, prescribe their duties or the board may delegate to the manager the authority to employ the labor and assistance required to conduct the facilities; except that all persons who handle or are responsible for any moneys in connection with the operation of the facilities, or who have authority to sign or countersign any checks upon any of the funds or accounts shall be appointed by the board and shall be required to give bond to the district in the amount, and with such sureties as the board designates, conditioned to faithfully account for all funds coming into their possession, and for the faithful performance of their duties. The manager of the facilities shall keep full and accurate accounts of all receipts, expenditures and disbursements by the facilities under his direction and control, and shall on or before October first of each year deliver to the board a complete report in writing showing the operations of the facilities under his direction and control, and the receipts and disbursements on account thereof for the preceding fiscal year.
2. The board may authorize the deposit of funds received from the operations of the facilities in the banks or depositaries and in the amounts that the board determines, and may designate the persons by whom and the purposes for which checks may be drawn thereon. All deposits shall be secured in the manner provided in section 165.241. If the board designates depositaries for the receipts from the operation of the facilities, the treasurer of the school district shall not be required to take into his custody or control any funds so received and so deposited, and neither he nor his sureties shall be responsible for the funds not coming into his custody and control.
(L. 1963 p. 200 § 8-22)
(Source: RSMo 1959 § 165.550)


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always sweet dreams of Berlin ….
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After I returned home, I went right to work experimenting on Freezeout Hill, sand. There was one thing for sure, there was enough of it. Probably one of the largest deposits in the world. Most everyone discouraged me. "It's not worth a damn for anything." They would say. "It won't even make concrete or plaster. "In a few years, it falls apart." I took a shovel and a wash tub and brought a couple of hundred pounds of it down to the house. I gave it a thorough inspection. I got a hold of a set of testing screens. I made sure the material was good and dry, then I started sifting. I come up with about ninety percent pure white sand and ten percent just plain old clay. It was easy to see why it wouldn't make good concrete. That fine bauxite clay would dilute the cement. Make it rot away in a few years. And, the sand was far too course for the many things it would be used for. It would have to be crushed. I went back to the hill and brought back a pickup load of the stuff. I sifted out all the clay and put it in some used paper sugar bags. Then I looked around for a crusher.
One of my neighbors, Walt Smith, had a hammer mill. He did custom grinding. I talked him into running this batch through his hammer mill. At first, we had the thing going too fast. It was turning everything to dust. But after a bit, we found the right speed and salvaged most of the sand. Boy, it was now sure pretty and white. The stuff looked like table salt. I had brought back samples of Ottawa sand. I compared them the best I could. They looked quite a bit alike but the Ottawa sand was round like bird shot. This, was jagged and sharp.
Brother Dell and I always confided in each other. We shared with each other, like brothers should. He was building an apartment house in Boise. I went to see him. I told him about what I had done and showed him the sand. There was a fellow in Boise that was a professional sand blaster. His name was Ben Stadler. We went to see him. I had a small bottle of twenty mesh in my pocket. I handed it to him. He poured out some in the palm of his hand and looked it over carefully. "Sure is sharp." he said. "Boy, I'll bet that would sure cut. And if the price is anywhere near what I have been paying, I could use a hundred ton right now." I helped Dell finish the building and we took off for Portland. The west coast was where the big market would be, we decided.
At that time, wall board was not allowed in that area. Maybe not even yet. It would draw moisture and fall apart in that damp climate. We contacted Pacific Building Material co. The largest in that area. We had brought along several bags of thirty mesh. This was the size best suited for the finish coat. George Erwin, the manager, took one look and said. "One of our best plaster contractors is working on a building right down the street. We will let him give it a try." We held our breath as we watched the guy finish one room with our sand. When he was through, there was a big smile on his face.
"Boy, " he said. "That is the best sand that I have ever used. And it goes about twice as fast as that Del Monte sand from California. It isn't round and doesn't roll under the trowel like that beach sand. No chicken tracks." "And you would use it if it were available?" "Sure. And so would every other plasterer in this area." We explained to George that we weren't quite ready yet. But as soon as we were, we would get in touch with him. He gave us some good advice. "Some low cost items carry a special freight rate. And sand is one of them. Go to the railroad and apply for the same mile rate as Del Monte and Ottawa and you will be sitting pretty. You are much closer than either one of them." We thanked him and took off for home. Then we designed and built that first sand plant. No one else was involved. Just Dell and myself. We had a lot to learn about processing that sand. And no one to teach us. Naturally, we made mistakes. Controlling the dust was a big problem. And there were other things. One of them was, we had run out of money before we got going. But we borrowed from the bank and finally made it.
Dell run the plant. And I was on the road again. Selling sand. From the day that we started up that plant, we never shut down for a single day. I sold all the sand that it could produce. But we had lots of trouble. The old power shovel that we had bought to load the sand was old and kept breaking down. The trucks that hauled the sand were also about worn out. And everyone was yelling that we should be shut down on account of the dust and the noise. We sure shouldn't of built that close to town. But we were learning the business. And our sand was in big demand. We were shipping from one to two car loads a day. Fifty to a hundred ton.
Again, I could write a whole book about our experiences in those years that followed. But I will cut it as short as I can. We could see that we had a great thing going but we were in need of a new plant. This little pilot plant, had served it's purpose. Also we needed a new shovel and trucks. And a lot more things. Oh yes. There is something else that I must bring up. I made a lot of trips to Portland, Spokane, Seattle and other cities in the Northwest. I usually went through Baker and LaGrande. And of course, right through the town of Durkee Oregon. In this town, was a huge Cement plant. Oregon Portland Cement. They had been there a good many years and their mountain of limestone was about gone. The word was out that they were going to move.
One day when I had just returned home, I told Dell. "You haven't been away from here for a long time. I think you need a vacation. Take your wife and go up to Durkee. Then I told him what I knew. Also what I suspected. They took off. Well, I certainly didn't send a boy to do a man's job. About a week later, Dell returned. And with a handful of contracts. A guy by the name of Ivan Thompson and another guy had staked out that big mountain. They had dug some holes and found limestone. Dell had told them how we had started the sand plant in Emmett. He convinced them that we could help them. They signed a contract. What ever he could sell that mountain for, they would get half. The other half would be ours.
By this time, those two Dewey Bros., Ted and Dell, were getting quite will known. They had the reputation of being a couple of goers. I told Dell to finish up what he had started. He jumped in his car and went to Boise. The home of the famous contracting co. Morrison & Knudsen. He sure didn't start at the bottom. He made a good sales pitch. The cement co. was running out of limestone. All the sugar companies would need a new source. And the many paper companies in the north west would be out of limestone. The thing was right on the railroad and all it would take to find out if it was there, was a drilling crew for a couple of weeks. The next week they started core drilling that mountain. In about a month's time, they had proven over ten million tons of pure limestone. And for sure, there were millions more. They put in a railroad siding and giant crushers on the mountain. Soon they were shipping from twenty to thirty car loads a day. We and the old prospectors at Durkee divided twenty five cents a ton royalty. Thanks to brother Dell, we had done this one in a hurry.


The Mexican government estimates that 8 out of 10 women in Mexico have suffered abuse of some type.
Laws criminalizing domestic violence are difficult to apply. CCTV Correspondent Gerry Hadden travels to the Yucatan peninsula on the east coast of Mexico to speak to the victims of violence.


The prevalence rates of overweight and obesity are considered an important public issue in the United States, and both of these conditions are increasing among both children and adults. There is evidence of aberrations in the vitamin D-endocrine system in obese subjects. Vitamin D deficiency is highly prevalent in patients with obesity, and many studies have demonstrated the significant effect of calcitriol on adipocytes. Genetic studies have provided an opportunity to determine which proteins link vitamin D to obesity pathology, including the vitamin D receptor, toll-like receptors, the renin-angiotensin system, apolipoprotein E, vascular endothelial growth factor, and poly (ADP-ribose) polymerase-1. Vitamin D also exerts its effect on obesity through cell-signaling mechanisms, including matrix metalloproteinases, mitogen-activated protein kinase pathways, the reduced form of nicotinamide adenine dinucleotide phosphate, prostaglandins, reactive oxygen species, and nitric oxide synthase.
In conclusion, vitamin D may have a role in obesity. The best form of vitamin D for use in the obese individuals is calcitriol because it is the active form of the vitamin D3 metabolite, its receptors are present in adipocytes, and modulates inflammatory cytokine expression.
Obesity is an emerging health problem of growing importance. There is evidence of aberrations in the vitamin D-endocrine system in obese subjects , such as increases in serum parathyroid hormone (PTH), urinary cyclic adenosine 3,5′-monophosphate (cAMP), renal tubular reabsorption of calcium, and circulating 1α, 25-hydroxyvitamin D3 (1,25OHD3) and a decrease in serum 25-hydroxyvitamin D3 (25OHD) levels. In young adults, the dietary plus supplemental vitamin D intake was inversely related to the development of incident metabolic syndrome over 20 years of follow-up . Vitamin D deficiency is common in children in West Virginia and is associated with increasing age and obesity . Vitamin D deficiency is an independent risk factor for obesity and abdominal obesity in women . Obese women transfer less 25OHD to offspring than normal-weight women, despite similar serum levels; maternal obesity and vitamin D sufficiency are associated with cord-blood vitamin D insufficiency . Visceral adipose tissue is negatively associated with plasma 25OHD concentrations in South Asians . Total body fat has been shown to be a negative predictor of 25OHD levels in women even after controlling for age, lifestyle, and PTH in Germany . Vitamin D deficiency has been associated with obesity, visceral obesity, hypertriglyceridemia, and metabolic syndrome in Korean children . Body mass index (BMI) is inversely associated with the increase in the serum 25OHD levels in response to vitamin D supplementation . The expression of vitamin D-metabolizing enzymes has been demonstrated in human adipose tissue. Plasma 25OHD increased by 27% after weight loss in the obese individuals. The expression levels of the 25-hydroxylase CYP2J2 and the 1α-hydroxylase CYP27B1 were decreased by 71% and 49%, respectively, in the subcutaneous adipose tissue of obese subjects , suggesting that adipose tissue, which can be dynamically altered during obesity and weight loss, has the capacity to metabolize vitamin D locally. Furthermore, calcitriol directly regulates adipocyte 11β-hydroxysteroid dehydrogenase type 1 (11β-HSD-1), generating active cortisol from inactive cortisone, with expression and cortisol production in human adipocytes in vitro, suggesting a potential role for calcitriol in visceral adiposity. In a 12-week double-blind randomized clinical trial, cholecalciferol supplementation resulted in a statistically significant decrease in body fat mass in healthy and obese women compared with the placebo group . In addition, vitamin D3 supplementation also improved insulin sensitivity in apparently healthy, middle-aged, centrally obese men . In our previous publication, we discussed the role of vitamin D in obesity . These findings suggest that vitamin D may play a role in obesity. Herein, we further discuss the potential role of vitamin D in obesity, along with possible genetic and cell signaling mechanisms.
Genetic studies provide an opportunity to link molecular variations with epidemiological data. DNA sequence variations, such as polymorphisms, exert both modest and subtle biological effects. Vitamin D exhibits immune-modulatory and anti-proliferative effects through the vitamin D receptor (VDR) in disease states. The VDR is present in adipose tissue and may contribute to the action of vitamin D and its analogs in adipocytes. Specific calcitriol-binding is evident in pre-adipocyte 3T3-L1 cells, but there is no evidence of specific calcitriol-binding in mature adipocytes . This finding is consistent with the idea that any influence of vitamin D on adipogenesis would likely be exerted early in the pre-adipocyte to adipocyte transition when more VDR was available. However, Blumberg et al.  have further defined the molecular mechanism by which unliganded VDR and calcitriol-liganded VDR regulate adipogenesis. They also demonstrated that VDR is expressed early in adipogenesis. VDR were expressed sequentially at high, but short-lived, levels beginning at 30 min and lasting for only a few hours after adipogenic activation [17, 18]. A functional vitamin D response element was identified in the murine Insig-2 promoter, and its potential role is involved in the differentiation of 3T3-L1 preadipocytes . VDR-knockout mice have also displayed atrophy of adipose tissue surrounding the prostate and mammary glands [20, 21]. Under normocalcemic conditions, VDR-null mice displayed less body fat mass and lower plasma triglyceride and cholesterol levels compared with wild-type (WT) mice. When placed on a high-fat diet, VDR-null mice displayed a slower growth rate and accumulated less fat mass globally than WT mice, even when their food intake and intestinal lipid transport capacity were the same as WT mice [22–24]. In one study, transgenic mice with targeted expression of human VDR in the adipose tissue developed obesity due to reduced energy expenditure . Taken together, these data confirm an important role of the VDR in the control of adipocyte metabolism and the regulation of energy metabolism. Variations at the VDR locus have been associated with susceptibility and progression to several diseases. VDR gene polymorphisms have been linked to higher susceptibility to vitamin D deficiency in children and adolescents . The VDR TaqI allele is associated with obesity [27, 28]; BsmI and ApaI VDR genes are also significantly associated with overweight and obesity , and the BsmI VDR polymorphism appeared to influence body mass index . These observations suggested that alterations of VDR function may play a role in patients with obesity.
Toll-like receptors (TLRs) are a group of glycoproteins that function as surface trans-membrane receptors. These receptors are involved in the innate immune response to exogenous pathogenic microorganisms. Substantial evidence exists supporting the important role of TLRs in the pathogenesis of obesity. Both TLR-2 and TLR-4 expression are increased in adipose tissue in patients with obesity and type 2 diabetes mellitus . A high-fat diet- or leptin deficiency-induced obesity up-regulated the expression of TLR1-9 and TLR11-13 in murine adipose tissue. The extent of the obesity-induced up-regulation of most TLR genes and related pro-inflammatory signaling cascades was much greater in the epididymal adipose tissues than in the subcutaneous fat tissues of the mice with diet-induced obesity . These results suggest that TLRs and the related pro-inflammatory signaling molecules that are overexpressed in enlarged adipose tissues may play an important role in the obesity-associated phenomenon of meta-inflammation. In addition, adipose stores may play a dynamic role in the regulation of inflammation and innate immunity in human subjects via modulation of the TLR/Nuclear Factor-kappaB (NF-ĸB) regulatory pathway . TLR-4-deficient 10ScN mice were selectively protected against obesity-induced by diets high in saturated fat. Moreover, macrophage infiltration and monocyte chemotactic protein-1 (MCP-1) transcript abundance were lower in the adipose tissue of 10ScN mice fed an HFP diet . C3H/HeJ mice, which have a loss-of-function mutation in TLR-4, were protected against the development of diet-induced obesity  and were less susceptible to fat-induced inflammation and insulin resistance . The inhibition of TLR-2 expression improved insulin sensitivity and signaling in the muscle and white adipose tissue of mice fed a high-fat diet . These findings suggest that TLRs are a key modulator of the crosstalk between the inflammatory and metabolic pathways. Furthermore, there is an association between TLR-2 and TLR-4 polymorphisms and serum levels of tumor necrosis factor-alpha and its soluble receptors in obese children , suggesting that serum cytokine levels may be affected by TLR polymorphisms in obese children. In contrast, vitamin D deficiency increases the expression of the hepatic mRNA levels of TLR-2, TLR-4, and TLR-9 in obese rats . However, calcitriol suppresses the expression of TLR-2 and TLR-4 mRNA and protein in human monocytes and triggers hypo-responsiveness to pathogen-associated molecular patterns . Calcitriol has also been shown to down-regulate intracellular TLR-2, TLR-4 and TLR-9 expression in human monocytes . Interestingly, TLR activation up-regulates the expression of VDR and 1α-vitamin D hydroxylase in human monocytes . Taken together, these data indicate that vitamin D may have a role in obesity via modulating the TLR pathways.
The primary function of the renin-angiotensin system (RAS) is to maintain fluid homeostasis and to regulate blood pressure. Angiotensin-converting enzyme (ACE) is a key enzyme in the RAS and converts angiotensin (AT) I to the potent vasoconstrictor AT II . A local RAS has been described in adipose tissue. Human adipose tissue expresses angiotensinogen and RAS enzymes . Angiotensinogen, ACE and type 1 AT receptor genes are widely expressed, both in human adipose tissue and in cultured human adipocytes . Renin is synthesized and secreted by culture 3T3-L1 cells in a regulated manner . The renin receptor protein is specifically synthesized in the stromal portion of human adipose tissue in both isolated inter-adipocyte stromal cells and in stromal areas . Moderate weight loss reduces plasma renin and aldosterone levels , suggesting an effect of weight loss on the juxta-glomerular secretion of renin. The RAS has been implicated in a variety of adipose tissue function. AT II increases lipogenesis in cultured human and murine cells , and angiotensinogen-knockout mice display decreased weight gain and decreased adipocyte cell size . The deletion of the AT2 receptor reduces adipose cell size and protects from diet-induced obesity and insulin resistance . AT2 receptor deficiency attenuates adipocyte differentiation and decreases adipocyte number in atherosclerotic mice . These results suggest that AT2 receptor stimulation in adipose tissue is involved in the improvement of adipocyte differentiation and adipose tissue dysfunction in atherosclerotic models. Local renin angiotensin expression regulates human mesenchymal stem cell differentiation to adipocytes. Combined treatment with exogenous AT II and the AT II type 1 receptor blocker valsartan further inhibited adipogenesis . Obese Zucker rats exhibited a marked up-regulation of AT1 receptor mRNA expression . ACE inhibition and AT1 receptor blockade prevented fatty liver and fibrosis in obese Zucker rats . These findings suggest that the RAS plays a role in obesity. The RAS activity may be modified by variants of the genes coding functional proteins of this pathway. Among hypertensive subjects with metabolic syndrome, the presence of ACE polymorphisms, including TT235, MM174, DD, and CC1166 genotypes, could be a risk factor for central obesity and dyslipidemia . In response to low-energy diets in obese women, the reduction in body fat was significantly lower in patients with the ACE D/D genotype than the I/I plus I/D genotype . There is an association of ACE gene polymorphisms with BMI in the hypertensive Tunisian population . In the Olivetti Prospective Heart Study, the ACE I/D polymorphism was a significant predictor of overweight and abdominal adiposity in men, while DD homozygosity was associated with larger increases in body weight and blood pressure in aging persons, and with higher overweight incidence . There was a significant association of the I/D polymorphism with obesity, but only in Caucasian men . It is possible that hormonal differences between sexes may alter the influence that the ACE I/D polymorphism has on weight gain. However, there is also an interaction between vitamin D and the RAS. Lower 25OHD levels and higher BMI values have been associated with higher plasma renin and aldosterone concentrations in Indian subjects with hypertension . Vitamin D metabolites have been inversely associated with circulating renin . Genetic disruption of the VDR results in overstimulation of the RAS with increasing renin and angiotensin II productions, leading to high blood pressure and cardiac hypertrophy. Treatment with captopril reduced cardiac hypertrophy in VDR-knockout mice . The inactivation of 1α-hydroxylase, inhibiting calcitriol production, also leads to the development of hypertension, cardiac hypertrophy and impaired cardiac function, along with an up-regulation of the RAS in both renal and cardiac tissues, which could be reduced with calcitriol treatment . In contrast, VDR over-expression suppresses renin expression in the juxtaglomerular cells of mice independently of PTH and calcium . These findings suggest that calcitriol may function as an endocrine suppressor of renin biosynthesis. Calcitriol suppresses renin gene transcription by blocking the activity of the cyclic AMP response element in the renin core promoter  and decreases ACE activity in bovine endothelial cells . There was a positive association identified between 25OHD and the vascular sensitivity to AT II in obese Caucasians with hypertension . Vitamin D3 therapy in obese subjects with hypertension modified renal plasma flow, mean arterial pressure, and tissue sensitivity to AT II similar to ACE inhibition . Taken together, these findings indicate that the RAS is activated in obese patients and that vitamin D may play a role in patients with obesity by modulating the RAS.
Apolipoprotein E (apoE) is a plasma protein and a key regulator of cholesterol and lipid metabolism. ApoE mRNA and protein can be found in human and rodent adipose tissue [70, 71]. The infusion of AT II into mice for 3 days significantly reduced apoE expression in adipocytes from freshly isolated adipose tissue. In isolated human adipocytes, treatment with AT II significantly reduced cellular and secreted apoE (by 20-60%). However, a specific AT1 receptor blocker, valsartan, eliminated the effect of AT II on adipocyte apoE expression . In in vivo and in vitro experiments, endogenous adipocyte apoE expression modulated adipocyte lipid metabolism and adipocyte gene expression . Endogenous adipocyte apoE expression has important implications for the acquisition of substrate from triglyceride-rich lipoprotein particles such as very-low-density-lipoprotein (VLDL) . High expression of apoE impairs lipid storage and promotes cell proliferation in human adipocytes . ApoE-knockout adipocytes are lipid-poor and triglyceride synthesis is lower compared to wild-type adipocytes in the absence and presence of extracellular VLDL. Macrophage-derived apoE ameliorates dyslipidemia and atherosclerosis in lean and obese apoE-deficient mice [75, 76], suggesting that apoE plays a role in regulating dyslipidemia. In comparison with ApoE3 mice, ApoE2 mice had elevated fasting plasma lipid and insulin levels and displayed prolonged postprandial hyperlipidemia accompanied by increased granulocyte number and inflammation 2 hours after being fed a lipid-rich meal. In comparison with ApoE3 mice, the ApoE2 mice also presented increased adiposity when maintained on a Western-type, high-fat, high-cholesterol diet . ApoE2-expressing mice were hyperlipidemic and had increased gonadal fat pad and adipocyte sizes compared with apoE3 mice. In isolated cells, however, the expression of the apoE2 isoform produced defective lipogenesis and increased triglyceride hydrolysis . The ApoE4 allele is associated with higher serum vitamin D levels . The ApoE4 allele is associated with a low bone mass in postmenopausal Japanese women . The common ApoE polymorphism has a complex effect on bone metabolism in peri-menopausal Danish women during five years follow-up. Namely, among women not receiving hormone replacement therapy (HRT), those with ApoE2 have a lower rate of bone mineral loss in the femoral neck and hip regions than other women, whilst among women receiving HRT, those with ApoE4 gained more bone mineral than other women . Calcitriol has been known to induce macrophages to exhibit specific saturable receptors for LDL and acetyl-LDL. The LDL receptor of calcitriol-induced macrophages has been found to exhibit specificity for apoB- and E-containing lipoproteins . In apoE-knockout mice, an animal model with dyslipidemia, high oxidative stress, and pronounced atherosclerosis after uninephrectomy, the animals developed reduced plaque growth and calcification with vitamin D analog treatment (paricalcitol) compared with control groups [83, 84]. Although vitamin D deficiency is associated with an unfavorable lipid profile in cross-sectional analyses; however, Ponda et al.  suggest that correcting for a deficiency might not translate into clinically meaningful changes in lipid concentrations. In addition, a systematic literature search revealed no statistically significant effects for vitamin D supplementation were observed for total cholesterol, HDL and triglycerides . Therefore, the lipid modulating effects of vitamin D supplementation should be further investigated. Taken together, these findings suggest that vitamin D may improve lipid profiles in ApoE allele obese patients.
Vascular endothelial growth factor (VEGF) is abundantly secreted from adipocytes and plays a key role in the process of fat tissue formation through the regulation of angiogenesis. A positive correlation between the concentrations of circulating VEGF levels and BMI was demonstrated in healthy male subjects under highly controlled conditions . Angiogenesis has been associated with visceral and subcutaneous adipose tissue in severe human obesity . The treatment of mice with TNP-470, an angiogenesis inhibitor, reduced blood flow from the recipient into the graft after subcutaneous transplantation of epididymal fat. The weight of transplanted tissues and the size of adipocytes in the grafts were significantly lower in mice treated with TNP-470 (TNP mice) than in the control mice . Different angiogenesis inhibitors have been shown to significantly decrease body and adipose tissue weights . These studies demonstrate that adipose tissue mass can be regulated through the vasculature and that metabolic changes accompany anti-angiogenic-induced weight loss, which may contribute to weight reduction. VEGF expression in visceral fat is enhanced during growth and is related to fat deposition . Serum VEGF concentrations have been positively correlated with BMI and visceral fat area . In one experiment, a high fat-intake increased the VEGF mRNA expression in visceral fat and the VEGF concentration in plasma, accompanied with the increase in the plasma free fatty acids concentration in mice . VEGF-B specifically controlled the endothelial uptake of fatty acids via transcriptional regulation of vascular fatty acid transport proteins . Angiogenesis was inhibited by blocking the VEGF receptor 2 in mice with diet-induced obesity. These treated mice had significantly lower body weights than control animals . The serum VEGF-A levels were significantly higher in obese patients than in lean controls, decreasing after weight loss with bariatric surgery . VEGF haplotypes confer susceptibility to obesity in children and adolescents . In contrast, calcitriol has been reported to inhibit angiogenesis in vitro and in vivo. Calcitriol significantly inhibits VEGF-induced endothelial cell spouting and elongation in a dose-dependent manner and decreases the production of VEGF . Calcitriol is a potent inhibitor of retinal neovascularization in a mouse model of oxygen-induced ischemic retinopathy . Vitamin D and its analogs also reduce the expression of VEGF in various cancer cell lines [101, 102]. Moreover, DBP-maf have been reported to inhibit angiogenesis and tumor growth in mice  and to inhibit VEGF signaling by decreasing VEGF-mediated phosphorylation of VEGF-2 and ERK1/2, a downstream target of the VEGF signaling cascade . These findings suggest that vitamin D modulates angiogenesis in obesity.
Poly(ADP-ribose) polymerases (PARPs) comprise a family of enzymes sharing a conserved catalytic domain that support mono- or poly(ADP-ribosyl)transferase activity using NAD+ as a donor of ADP-ribosyl units. PARPs are involved in a wide range of molecular and cellular processes, including maintenance of genome stability, regulation of chromatin structure and transcription, cell proliferation, and apoptosis . PARP-1 is a critical regulator of peroxisome proliferator-activated receptor gamma (PPARγ2)-dependent gene expression with implications in adipocyte function and obesity-related disease models. PARP-1 expression increases during adipocyte development . PARP-1 −/− mice with predominantly obesity-resistant backgrounds were more susceptible to age-related weight gain and diet-induced obesity than wild-type littermates [107, 108], while the decreased activity of PARP-1 in mice with obesity-prone background protected against high-fat diet-induced obesity [109–111]. These findings suggest that genetic determinants can modify the phenotypic outcomes of weight gain. PARP-2 −/− mice had defects in adipogenesis with reduced expression of adipogenic genes  and are also protected against diet-induced obesity . PARP-5 knockout mice also exhibited increased energy expenditure, increased fatty acid and glucose utilization, and reduced adiposity . In addition, increased levels of vitamin D seem to down-regulate PARP-1 expression; PARP-1 levels decrease following calcitriol treatment in NB4 cells, which are acute promyelocytic leukemia cells . Vitamin D exerts a concentration-dependent inhibitory effect on PARP-1 in human keratinocyte cells . Vitamin D-induced down-regulation of PARP is further enhanced by nicotinamide in human myeloblastic leukemia cells . Furthermore, PARP was attenuated in the hippocampi of rats that received dexamethasone and vitamin D , suggesting that the anti-inflammatory effects of dexamethasone and vitamin D are derived from their capacity to down-regulate microglial activation. These findings suggest that vitamin D may have a protective role in obesity by down-regulating PARP.
Matrix metalloproteinases (MMPs) are proteolytic enzymes that are responsible for remodeling the extracellular matrix and regulating leukocyte migration through the extracellular matrix. This migration is an important step in inflammatory and infectious pathophysiology. MMPs are produced by many cell types, including lymphocytes, granulocytes, astrocytes, and activated macrophages. There is growing evidence that MMPs play an important role in the pathogenesis of obesity. Bouloumié et al.  provided the first evidence that human adipose tissue releases MMP-2. Overweight/obese women had significantly higher plasma activity of MMP-2 than controls . MMP-9 levels are found increased in obese subjects [121, 122]. Significantly higher levels of MMP-9 have been reported in obese children with coexisting hypertension than in obese normotensive patients, and MMP-9 correlates with BMI . In one study, obese children and adolescents had higher circulating MMP-8 concentrations, lower plasma tissue inhibitor of metalloproteinase 1 (TIMP-1) concentrations, and higher MMP-8/TIMP-1 ratios than non-obese controls . An MMP-2 promoter haplotype was associated with percentage of body fat in childhood obesity in New Zealand . Functional MMP-9 gene polymorphism is strongly associated with obesity , and MMP-9 genotypes and haplotypes affect MMP-9 levels in obese children and adolescents . Moreover, VDR-knock-out mice have been shown to have an influx of inflammatory cells, phospho-acetylation of NF-κB, and up-regulated expression of MMP-2, MMP-9, and MMP-12 in the lungs . The VDR TaqI polymorphism is associated with the decreased production of TIMP-1, a natural MMP-9 inhibitor . In addition, calcitriol modulated tissue MMP expression under experimental conditions , down-regulated MMP-9 levels in keratinocytes, and may attenuate the deleterious effects of excessive TNF-α-induced proteolytic activity, which is associated with cutaneous inflammation . Calcitriol also inhibits both the basal levels and the staphylococcus-stimulated production of MMP-9 in human blood monocytes and alveolar macrophages . Moreover, a vitamin D analog has also been reported to reduce the expression of MMP-2, MMP-9, VEGF, and PTH-related peptide in Lewis lung carcinoma cells . Together, these studies suggest that calcitriol may play an important role in the pathological processes of obesity by down-regulating the level of MMPs and regulating the level of TIMPs.
The mitogen-activated protein kinase (MAPK) pathways provide a key link between the membrane-bound receptors that receive these cues and changes in gene expression patterns, including the extracellular signal-regulated kinase (ERK) cascade, the stress-activated protein kinases/c-jun N-terminal kinase (SAPK/JNK) cascade, and the p38 MAPK/RK/HOG cascade . p38MAP kinase activity is required for human primary adipocyte differentiation . Obese Zucker rats exhibit MAKP activity . MAPK phosphatase-1 (MKP-1) plays an essential physiological role in the negative regulation of the MAPKs, and MKP-1 plays an important role in the regulation of energy expenditure and facilitates the loss of oxidative myofibers associated with obesity in mice . Mice lacking MKP-1 expression are resistant to diet-induced obesity . The down-regulation of MKP-1 is critical for increased production of MCP-1 during the course of adipocyte hypertrophy . The exposure of obese mice to UV-B resulted in the phosphorylation of ERK1/2, JNK, and p38 proteins of the MAPK family. Compared to wild-type mice, obese mice exhibited higher levels of phosphorylation of these proteins, greater activation of NF-ĸB/p65, and higher levels of circulating proinflammatory cytokines , suggesting that obesity in mice is associated with greater susceptibility to UVB-induced oxidative stress. In both dietary and genetically (ob/ob) obese mice, adipose tissues displayed a marked decrease in p38MAPK activity compared with the same tissues from lean mice. Furthermore, p38MAPK activity is significantly higher in pre-adipocytes than in adipocytes , suggesting that p38MAPK activity decreases during adipocyte differentiation. The MAP2K5-linked single nucleotide polymorphism rs2241423 was associated with BMI and obesity in two cohorts of Swedish and Greek children , suggesting a role for MAP2K5 in early weight regulation. By regulating VDR mRNA expression, the p38 MAPK pathway participates in the mediation of calcium signals and affects lipid accumulation in murine pre-adipocytes . Pretreatment with calcitriol has been shown to inhibit JNK activation by all stressors and also to inhibit p38 activation in keratocytes . Zhang et al.  demonstrated that the up-regulation of MKP-1 by vitamin D inhibited lipopolysaccharide (LPS)-induced p38 activation and cytokine production in monocytes and macrophages. In another study, the vitamin D analog (24R)-1,24-dihydroxycholecalciferol prevented neuronal damage caused by hydrogen peroxide-induced toxicity in the SH-SY5Y cell line . Interestingly, the neurotoxic effects of hydrogen peroxide were dependent on JNK and p38 MAPK. In addition, the long-term actions of vitamin D in MCF-7 and LNCaP cancer cells can suppress the estradiol-induced activity of ERK-1 MAPK and inhibit cell growth .
The reduced form of the nicotinamide adenine dinucleotide phosphate (NADPH) oxidase (NOX) enzyme complex mediates critical physiological and pathological processes, including cell signaling, inflammation, and mitogenesis, by generating reactive oxygen species (ROS) from molecular oxygen. The up-regulations of p22phox and p47phox in adipose and Nox4, p22phox, and p47phox in the kidneys has been observed in obese subjects . Overweight and obese adults have increased vascular endothelial expression levels of NOX-p47phox and evidence of endothelial oxidative stress, with selective compensatory upregulation of antioxidant enzymes and Ser1177-phosphorylated endothelial nitric oxide synthase . The reduced expression of the NADPH oxidase NOX4 is a hallmark of adipocyte differentiation . NOX4 acts as a switch between differentiation and proliferation in pre-adipocytes . NOX4-deficient mice display latent adipose tissue accumulation and are susceptible to diet-induced obesity and early onset insulin resistance . The knockdown of NOX4 by RNA interference inhibits reactive oxygen species production and adipocyte differentiation by differentiation-inducing agents . The suppression of NOX2 may restore free fatty acids-induced dysfunction and apoptosis in β cells . However, vitamin D deprivation in rats decreased the activity of cytosolic NADPH-dependent 3,5,3′-triodo-L-thyronine (T3) binding in the liver. This decrease can be restored by administering calcitriol . In heart mitochondria, NAD+-dependent isocitrate dehydrogenase decreased notably in vitamin D-deficient rats, but calcitriol subsequently restored normal values . In rat centrilobular hepatocytes, a vitamin D-deficient diet induced a significant increase in NADPH . Husain et al.  reported that cardiac NOX activity increased by 300% in uremic rats compared with the normal controls. Treatment with paricalcitol protected the uremic rats from cardiac oxidative stress by inhibiting NOX activity (by 50%), thus lowering superoxide production in the heart. Taken together, these findings and results indicate that vitamin D may have a role in obesity via the suppression of NADPH expression.
Prostaglandins (PGs) play a role in inflammatory processes. Cyclooxygenase (COX) participates in the conversion of arachidonic acid into PGs. PGE2 and PGD2/PGJ2 have shown to promote adiposity in mice through the inhibition of lipolysis and the induction of adipogenesis, respectively [157, 158]. PGF2α is a potent inhibitor of adipocyte differentiation in vitro[159–161]. PGE2 enhanced lipid accumulation in hepatocytes and contributed to the development of hepatic steatosis in in vivo models of obesity . Obesity-associated inflammatory foci in the human breast are associated with elevated COX-2 levels and activation of the PGE2. In one study, a COX-2 genetic deficiency produced in a significant reduction in total body weight and percentage of body fat. In adipose tissue, the production of the precursor required for 15d-PGJ2 formation, PGD2, was significantly reduced; macrophage-dependent inflammation was also significantly reduced . Moreover, calcitriol has been reported to regulate the expression of several key genes involved in the PG pathway, causing a decrease in PG synthesis . Calcitriol and its analogs have been shown to selectively inhibit the activity of COX-2 . These findings suggest that vitamin D may play a role in modulating the inflammatory process in obesity.
Reactive oxygen species (ROS) are produced by activated phagocytes as a part of their microbicidal activities. An association between ROS and metabolic syndrome has been demonstrated in asymptomatic Japanese men . Diet-induced obesity increases the levels of total and individual ROS in the brain and highlights a direct relationship between the amount of adiposity and the level of oxidative stress within the brain , suggesting that obesity increases cerebro-cortical ROS and impairs brain function. High-fat diets (HFDs) induce obesity and result in an increase in oxidative stress in adipose tissue. One study reported an increase in body weight after 90 days of HFD and observed that exercise training prevented greater gain. Lipid peroxidation and protein carbonylation increased in fat tissue after HFD and decreased significantly after exercise training , suggesting an interaction between ROS and lipolysis. Obesity has been linked to a low-grade pro-inflammatory state, in which impairments in the oxidative stress and antioxidant mechanisms are involved [169, 170]. The over-expression of manganese superoxide dismutase (MnSOD) ameliorated high-fat diet-induced insulin resistance in rat skeletal muscle . Obese adult rats experienced greater mitochondrial hydrogen peroxide release compared with lean adult rats . Glutathione S-transferase P1 gene polymorphisms increase the susceptibility to and risk of type 2 diabetes mellitus and obesity . Significant impairment in glutathione peroxidase has been observed in response to diet-induced obesity . The suppression of glutathione peroxidase activity was demonstrated in diabetic patients compared to healthy controls, and suppression was observed to occur in a greater degree in obese vs. non-obese diabetics. There were higher levels of oxidative stress in the obese diabetics even after control of hyperglycemia by insulin treatment, suggesting the importance of obesity in contributing to oxidative stress . A significant decrease of all glutathione forms, including the content of total glutathione (GSH) and glutathionylated proteins, has been demonstrated in obese and type 1 diabetic children . Similarly, calcitriol has been reported to exert a receptor-mediated effect on the secretion of H2O2 by human monocytes . Human monocytes in culture gradually lose their capacity to produce superoxide when stimulated. The addition of calcitriol, lipopolysaccharide or lipoteichoic acid restored the capacity of stimulated monocytes to produce superoxide and increased their oxidative capacity compared with unstimulated monocytes . Calcitriol can also protect nonmalignant prostate cells from oxidative stress-induced cell death by eliminating ROS-induced cellular injuries . Vitamin D metabolites and vitamin D analogs have been reported to induce lipoxygenase mRNA expression, lipoxygenase activity and ROS in a human bone cell line . In another study, the vitamin D analog (24R)-1,24-dihydroxycholecalciferol prevented neuronal damage caused by hydrogen peroxide-induced toxicity in the SH-SY5Y cell line . Vitamin D can also reduce the extent of lipid peroxidation and can induce SOD activity in a hepatic anti-oxidant system in rats . Astrocytes play a pivotal role in the CNS detoxification pathways in which GSH is involved in eliminating oxygen and nitrogen reactive species, such as NO. Calcitriol affects this pathway by enhancing intracellular GSH pools and significantly reduces the nitrite production that is induced by LPS . These findings suggest that vitamin D modulates oxidative stress in obesity.
Nitric oxide synthase (NOS) is an enzyme that is involved in the synthesis of nitric oxide (NO), which regulates a variety of important physiological responses, including cell migration, the immune response, and apoptosis. High fat diets modulate nitric oxide biosynthesis and antioxidant defense in red blood cells in C57BL/6 mice , which lead to increasde NO sensitivity in rat coronary arterioles . Platelet NO production has been significantly correlated with BMI, waist circumference, and triglyceride concentrations, thus suggesting an association between increased platelet NO production, obesity, and hypertriglyceridemia, independent of the degree of insulin-resistance . Chronic NOS blockade by L-NAME in mice ameliorated high-fat diet-induced adiposity and glucose intolerance, accompanied by reduced adipose inflammation and improved insulin signaling in skeletal muscle, suggesting that endogenous NO plays a modulatory role in the development of obesity-related insulin resistance . Mice deprived of the eNOS and/or nNOS gene exhibit metabolic syndrome, including insulin resistance, hypertension, and dyslipidemia . Inducible NOS (iNOS)-null mice, although protected from obesity-related insulin resistance, exhibit increased adiposity . Conversely, the activation of 1α-hydroxylase in macrophages increases the level of calcitriol, which inhibits the iNOS expression and reduces NO production within LPS-stimulated macrophages . Thus, calcitriol production by macrophages may provide protection against the oxidative injuries that are caused by the NO burst. Calcitriol is known to inhibit LPS-induced immune activation in human endothelial cells . In experimental allergic encephalomyelitis, calcitriol inhibits the expression of iNOS in the rat CNS .
This paper reviewed the relationship between vitamin D and obesity. Genetic studies provide opportunities to determine which proteins link vitamin D to obesity pathology. Vitamin D is also able to act through numerous non-genomic mechanisms, including protein expression, oxidative stress, inflammation, and cellular metabolism. These findings suggest that vitamin D plays a role in obesity. However, some studies demonstrated no effect of vitamin D on weight change and energy expenditure and little improvement in cardiovascular risks with cholecalciferol [191–194]. Interestingly, Vitamin D is fat-soluble and readily stored in adipose tissue; it is sequestered after absorption and stored in tissues such as fat and muscle . This fate of vitamin D has been demonstrated by injecting radio-labeled vitamin D3 into individuals and monitoring the highest levels of biological activity and radioactivity in the fat tissue . In addition, cholecalciferol supplementation has no effect on cytokines and markers of inflammation in obese subjects . Calcitriol, inversely, modulates adipokine expression, inhibits anti-inflammatory cytokine expression, reduces monocyte recruitment by human pre-adipocytes, and restores glucose uptake in adipocytes [198–200]. Calcitriol has a role in human adipose tissue because it is the active form of vitamin D3 metabolite, and VDR receptors present in adipocytes allow the suppression of PTH levels. The PTH excess observed in elderly subjects with both primary and secondary hyperparathyroidism may promote weight gain by impeding catecholamine-induced lipolysis . However, monitoring of serum 25OHD after calcitriol intake is not necessary because calcitriol inhibits the production of serum 25OHD in the liver [202, 203]. Taken together, there is widespread evidence from laboratory, animal and genetic studies that calcitriol is active in obesity. Calcitriol therapy in obesity has not been reported whilst the results of trials of cholecalciferol supplementation have so far been limited. Therefore, further investigation of calcitriol in obese patients is needed.
This work was carried out in collaboration between both authors. Author KL designed the study and wrote the protocol. Author LN managed the literature searches. All authors read and approved the final manuscript.


Each of the candidates running in the Judo Ontario elections on June 15th were offered the opportunity to record a short video campaign message and have it posted to the Judo Ontario Facebook page. Two videos were submitted. To watch the videos, click the button below to visit the Judo Ontario Facebook page (and don't forget to "Like" the page while you're there!).


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Q: CodeIgniter Flashdata doesn't work on server I'm using CodeIgniter's flashdata to store my captcha word for the next request. It works fine on the local site, but not on the server. There is no difference between the two codes.
I'm wondering wether the flashdata feature requires any additional plugin or configuration for apache/php.
Here's how I set the data:
$this->session->set_flashdata('captchaWord', $cap['word']);

and this is how I retrieve it:
if (strcasecmp($this->input->post('captchaWord'), $this->session->flashdata('captchaWord')) != 0)

Any ideas?

A: It doesn't. Something strange is going on with your server.
Check that that a database connection is being made if you're using database sessions.
Check that what you're redirecting to is actually the same server.
Check that you're using flashdata correctly. (Post code?)
Check that session variables are working at all.
Update:
// Again, be sure that 'session' is autoloaded in config.php
class Test_Controller {
    public function page1()
    {
        $this->session->set_flashdata('test', 'HELLO WORLD');
        redirect('test/page2');
    }
    public function page2()
    {
        echo $this->session->flashdata('test');
    }
}

Add this controller and visit http://yourserver.com/index.php/test/page1
It should redirect to page2 and display 'HELLO WORLD'.
If it does, then it's a problem with your code. If it doesn't, then I would suggest loading up a virtual machine with an OS and installing a new copy of a webserver and trying again.

A: Check to see if Codeigniter is calling a controller twice, in which case it is clearing the flashdata between the first and second calls (See an answer to Codeigniter flash data not working in certain browsers).
There are Codeigniter cases where a controller may be called twice. For example, the GoCart shopping cart package, built on Codeigniter, has a helper function to force SSL by changing http to https and doing a redirect to the new URL. If the function is used in the constructor, the controller ends up being called twice, once without and once with SSL. Any flashdata is lost with the second call.
Per another similar question (CodeIgniter: setting flash data not working), you can use $CI->session->keep_flashdata() to preserve flashdata across additional requests.
The answer on this page does a good job of explaining the issue and the workaround: CodeIgniter "flashdata" doesn't work


Motiv Power Systems (Motiv), provider of all-electric medium-duty fleet chassis, has appointed current Board Chairman Matt O'Leary as CEO. Jim Castelaz, Motiv founder and former CEO, has assumed the role of CTO and will maintain a seat on Motiv's board. O'Leary will continue to serve as interim board chairman and plans to relocate to the San Francisco Bay Area in the coming weeks.
Before joining the Motiv board, O'Leary served in various capacities in his 38 years at Ford Motor Co., including, most recently holding the title of Vehicle Line Director - North America Trucks, SUVs, and Commercial Vehicles. During his extensive career at Ford Motor Company, O'Leary co-led the launch of the aluminum-intensive 2015 F150 at two assembly plants.
In January, Motiv had announced a battery supply agreement with BMW AG for its new line of lithium-ion batteries and earlier this month, a new deployment with CALSTART and the United States Postal Service.
As Director of Corporate Strategy, he led development of a corporate strategy to significantly grow Ford's presence in the Asia Pacific. As Director of Product Development for South America, O'Leary led the historical transformation of Ford South America from 100% legacy to 100% global products in three years, including local design and development of two global vehicles - EcoSport and Ka. O'Leary also led from inception to launch the successful all-new 2009 F150 which won many awards including Motor Trend and North American Truck-of-the-Year awards, as well as Truck of Texas.


text:
Folketingsvalget den 16. november 1932. Danmarks Kommunistiske Parti bliver for første gang repræsenteret i Folketinget. DNSAP stiller også op, men får et elendigt resultat.

(+/-) – Forskellen i antal pladser i Folketinget i forhold til fordelingen ved forrige valg.

1932
Politiske valg i 1932


text:
Tag Archives: owncloud media streaming
Posts tagged "owncloud media streaming"
admin Streaming Services internet streaming media, owncloud media streaming, pure stream media
Which is better: streaming or streaming on your own?
New York's ownCloud is one of the most popular cloud-based media streaming platforms for users of the New York Times, Forbes and many others.
But, while it's been popular with some of its users for a few years now, the company is facing a new problem: It's not available in the United States.
New York was the only place where it was offered, and the company announced that the only way to access it is to pay an extra $2 per month for a license.
The company has offered a similar plan for a year, but this time, it's only available for subscribers to the Times and Forbes websites.
The cost is $9 per month, but users of other news sources, including the Associated Press, must pay $2.50 per month or wait until a month after the end of the first quarter.
It's unclear why New York has no streaming service, but New York may be doing it out of frustration.
After all, the Times has more than 1 million subscribers, and Forbes has more then 1.4 million.
New Yorkers also have to deal with an additional $2 fee per month when using a third-party app.
The problem is even worse for users who don't own an Apple TV or an Android device.
Apple TV owners can't stream from the New Yorkers app because the company doesn't allow streaming from the iPad, Mac, Roku, Apple TV, Android TV, and Windows Phones.
While it's a pain for those who have the devices, it also forces users to switch to Apple's own app for other devices, which may cost them a lot more money.
New Jersey's owncloud is also available on iOS devices, and it works well for those users who want to access their owncloud.
It also has a much higher download speed than New York, which means users can download and watch hundreds of millions of movies and TV shows.
Newyork's own Cloud is also the only app that allows users to watch a movie in progress or to view a video while streaming.
But when New York tries to stream the movie or TV show on its own servers, it doesn't work.
That means that users can't watch the same episode again and again, or play the same movie in multiple viewing positions.
And Newyorks users can only watch the movies in their home.
There's also a $2 premium fee to access the Newyron cloud.
Users have to pay $10 a month to use the NewYron cloud and another $10 to watch and stream the same movies and shows.
When it comes to streaming, users have to have the same subscription to the NewYorkTimes.com or Forbes.com servers, or to have a third party that can stream them.
Users can stream to the cloud for $9.99 a month for the Times, and $9 a month or $9 to access Forbes.
The $9 cost is for streaming on New Yorks own servers.
But the company has plans to expand the offering in the coming months.
admin Special Offers owncloud media streaming, uva streaming media
The FBI's new streaming site is the latest step in its war on piracy
The FBI is rolling out a new online streaming service, uva streaming, to fight online piracy.
The service is the FBI's latest move in its ongoing war on online piracy and is one of a series of initiatives to combat piracy in the United States, which is already under increased pressure due to the Great Recession.
"The streaming video market is worth $100 billion, and it is a growing opportunity," U.S. Attorney Carmen Ortiz said in a news conference announcing the new streaming service on Wednesday.
"We have an opportunity to make sure that that opportunity is available to our citizens."UVA is the new name for the FBI streaming service.
The bureau is now known as the Federal Bureau of Investigation.
It launched its new streaming website, uvabox.com, in March 2017, and the FBI is now using the service to help combat online piracy, according to a federal court filing.
"It is vital that we ensure that the federal government has the resources to provide the federal courts with the tools to enforce our nation's laws," Ortiz said.
The FBI is also using its streaming site to help fight online child pornography.
UVA offers a tool that users can use to report and block the download of child pornography on the site.
The service has been available since January and has been used by many law enforcement agencies across the country.
The FBI said that since the service launched, it has made an estimated $50 million in revenue through the service, according the filing.
admin Help & Guides dane media streamer, ez stream media, ionic streaming media, new stream media, owncloud media streaming
OwnCloud vs. Google Play Movies and TV: What's in it for you?
We've already covered the big differences between the two streaming platforms, but what's the difference between the newer platforms and the old ones?
What do you get when you cross over?