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Q: BigQuery stream to front-end with Express I'm trying to read a query from BigQuery and stream it to the front-end. In Node.js-land with Express, this would be:
app.get('/endpoint', (req, res) => {
  bigQuery.createQueryStream(query).pipe(res);
});

However, createQueryStream() does not create a Node.js stream, instead it's a custom stream object that returns table rows and as such it fails:

(node:21236) UnhandledPromiseRejectionWarning: TypeError [ERR_INVALID_ARG_TYPE]: The first argument must be one of type string or Buffer. Received type object

This is confirmed in the official documentation:
bigquery.createQueryStream(query)
  .on('data', function(row) {
    // row is a result from your query.
  })

So, is there a way to stream BigQuery data to the front-end? I've thought two potential solutions but wanted to know if anyone knows a better way:

*

*JSON.stringify() the row and return JSONL instead of plain JSON. This adds a front-end burden to decode it, but makes it fairly easy on both sides.

*Move to the REST API and do actual streaming with request like: request(url, { body: { query, params } }).pipe(res) (or whatever is the specific API, haven't dug there yet).

I was confused that a Node.js library that says that it does streaming doesn't work with Node.js native streams, but this seems to be the case.

A: BigQuery is intended to be used with a wide array of different client libraries written for different programming languages, and therefore, it does not return nodejs-specific data structures, but rather, more general structures which are common to mostly any structured programming language, such as objects. Answering to your questions, yes, there is a way to stream BigQuery data to the front-end, but this is a rather personal choice, because all it entails is converting from one data type to another. However, I would say the most straight-forward way to do this is by calling JSON.stringify(), which you have already mentioned.
I hope that helps.

A: We ended up making an implementation that stitched together the reply from BigQuery into a big JSON array:
exports.stream = (query, params, res) => {
  // Light testing for descriptive errors in the parameters
  for (let key in params) {
    if (typeof params[key] === "number" && isNaN(params[key])) {
      throw new TypeError(`The parameter "${key}" should be a number`);
    }
  }

  return new Promise((resolve, reject) => {
    let prev = false;

    const onData = row => {
      try {
        // Only handle it when there's a row
        if (!row) return;

        // There was a previous row written before, so add a comma
        if (prev) {
          res.write(",");
        }
        res.write(stringify(row));
        prev = true;
      } catch (error) {
        console.error("Cannot parse row:", error);
        // Just ignore it, don't write this frame
      }
    };

    const onEnd = () => {
      res.write("]");
      res.end();
      resolve();
    };

    res.writeHead(200, { "Content-Type": "application/json" });
    res.write("[");
    bigQuery
      .createQueryStream({ query, params })
      .on("error", reject)
      .on("data", onData)
      .on("end", onEnd);
  });
};

It will build a large JSON array by stitching together:
[   // <- First character sent
stringify(row1)   // <- First row
,   // <- add comma on second row iteration
stringify(row2)   // <- Second row
...
stringify(rowN)   // <- Last row
]   // <- Send the "]" character to close the array

This has the advantages:

*

*The data is sent as soon as available, so the bandwidth needs are lower.

*(depends on BigQuery implementation) lower memory needs on the server side since not all the data is hold at once in memory, only small chunks.


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Home / News / Michelle Avery Appointed to AICPA Financial Reporting Executive Committee
Michelle Avery Appointed to AICPA Financial Reporting Executive Committee
Veris Consulting, Inc. is pleased to announce the appointment of Michelle Avery to the AICPA Financial Reporting Executive Committee (FinREC).
FinREC is the senior technical committee of the AICPA for financial reporting and is authorized to make public statements on behalf of AICPA on financial reporting matters. The mission of FinREC is to determine the Institute's technical policies regarding financial reporting standards and to be the spokesbody on those matters.
Michelle Avery is President and Senior Managing Director at Veris Consulting, which specializes in providing forensic accounting and expert witness services to prominent law firms in high-profile domestic and international civil litigation involving complex accounting, financial reporting and economic damage issues. Michelle has extensive experience leading major dispute resolution engagements, frequently involving allegations of misstated financial statements, inadequate disclosures, auditor malpractice and director and officer liability.
Additionally, Michelle was recognized in November by the AICPA as a recipient of the AICPA Standing Ovation award. The award recognized Michelle as one of 25 CPAs age 40 and under who exhibited outstanding professional achievements in the areas of forensic accounting and business valuation. Michelle joins Jon Couchman, a managing director with Veris, as a recipient of this honor; Jon was recognized in November 2015.
Michelle has served as a member of the NAIC/AICPA Working Group Task Force and the International Association of Insurance Receivers Board of Directors. She is the Vice-Chair of the College of William & Mary Mason School of Accounting Programs Board and currently serves on the James Madison University School of Accounting Guiding Executive Board. Michelle is a Certified Public Accountant, licensed in Virginia, and is Certified in Financial Forensics. Michelle earned a Bachelor of Business Administration with a major in Accounting from the College of William & Mary.
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As I wrote this essay on the 12th anniversary of the U.S. invasion of Iraq (March 19-20), the news was filled with its violent repercussions across the Middle East and the world. The latest atrocity was a multiple suicide bombing at two mosques in Sanaa, the capital of Yemen, that have killed at least 137 people. Two days ago, 24 people, mostly foreign tourists, were killed in Tunis.
War rages on in Iraq, Syria, Afghanistan, Libya, Somalia and Nigeria. Our leaders seem paralyzed, unable to contain or control the seemingly endless and spreading storm of violence they have unleashed. Their stated reasons for their own use of violence – yes, Western use of military force is a form of violence too – ring increasingly hollow: security, stability, democracy, humanity. They have failed catastrophically to deliver any of these anywhere.
He promised a new mindset and was awarded a Nobel Peace Prize. Empty promises, hollow awards.
My third reason moves to an even deeper level of awareness, for it grows out of my experience in the ghettos of the North over the last three years, especially the last three summers. As I have walked among the desperate, rejected, and angry young men, I have told them that Molotov cocktails and rifles would not solve their problems. I have tried to offer them my deepest compassion while maintaining my conviction that social change comes most meaningfully through nonviolent action.
Today the dilemma Dr. King described has been globalized. If it was legitimate or effective for our government to use massive violence in pursuit of its political objectives, how would it be wrong for others to do likewise?
But this logic of violence is not so different from that of our own soldiers, generals and politicians. Once war is unleashed, the prescribed response to setbacks and defeats is to use even greater violence. America's unrivaled military budget and endless investment in more advanced and more destructive weapons is predicated on that assumption. We must have "military superiority." We must be able to militarily defeat any enemy. Anything less will leave us vulnerable. This is the same logic that draws young people to join the Islamic State, the strongest Islamist fighting force.
It is easy to see that this logic leads only to total, endless war on all sides. This is the very nightmare that world leaders confronted in 1945 when they stepped back from the abyss and signed the United Nations Charter, prohibiting the use of military force except in self-defense or at the request of the UN Security Council.
Will our leaders finally admit that they reneged on that commitment, seduced by the same mirage of security through strength and military supremacy as past aggressors like Nazi Germany?
Many Americans now understand that it has been a terrible folly to confuse the power to destroy with the power to build a better world. The two are quite distinct, and building a better world requires neither air strikes nor "boots on the ground" but working together, talking through problems and, even more important, listening to others.
That is still the kind of leadership we need from the President of the United States. Barack Obama's failure to provide it for the past six years has compounded the problem and spread untold violence and misery across even more of the world.
Proxy and covert war may be more politically palatable to President Obama and his colleagues, but death and violence are the same whether Americans blame them for it or not. A Houthi woman in Yemen told BBC reporter Safa AlAhmad how her three children were killed by artillery fire from the Yemeni armed forces that the U.S. armed, trained and conducted joint operations with.
She collected her children's bodies in small, bloody pieces and is still distraught that the only part of their faces she could find was a single ear. No wonder the Houthis, who now control Sanaa, write "Death to America" on walls all over the city.
Ending the mindset that unleashes such horrors requires a genuine renunciation of America's post-Cold War triumphalism, myths of military supremacy and record military budgets. In their place we need a new commitment to peace, diplomacy and international cooperation, and a serious recommitment to the letter and spirit of the UN Charter's prohibition on the use of force.
Article originally posted at Consortiumnews.com and used with the author's permission.
Nicolas J S Davies is the author of Blood On Our Hands: The American Invasion and Destruction of Iraq. Davies also wrote the chapter on "Obama At War" for the book, Grading the 44th President: A Report Card on Barack Obama's First Term as a Progressive Leader.
Provoking Wars: Is that what U.S. Foreign Policy Is About?
Hear! Hear! I would only add: What kind of "better world" is the US Ruling Class interested in cultivating? Only a world still more profitable for US corporations. This is the sole motivation for all their interventions overseas. And what a lovely mess we now see in Yemen after US meddling there. Funny, I didn't hear Obama declare victory as reason for withdrawing US Special Ops personnel. This can only mean one of two things: an unspoken admission that things are too out of control to leave US personnel on the ground there; or, more likely, US airstrikes are coming soon. More violence to beget still more violence. Is there any way we can petition the Nobel Committee to RETRACT that ridiculous Peace Prize??
" THE HORROR, the horror! ( Captain Kurtz in Apocalypse Now) That is the "Mindset of War".
In the winter of 1944-45 my old friend Mike was sitting in a foxhole in the Ardennes when the Germans surrounded his platoon's position. They shouted to the Americans to get out of their holes and surrender. Their platoon leader ordered them to surrender and the Germans machine gunned them all. Mike, had stayed in his hole and was taken prisoner. Because he was Jewish, in contravention of the Geneva Conventions, he was sent to a concentration camp called Berga rather than a POW camp. . He was 18 years old.
At the camp he was tortured by the Gestapo. As the Allied armies swept into Germany he escaped and crossed into American lines. Too weak from his ordeal in the camp he was given a machine gun and told to march a captured group of teen aged German "Werewolves" who were shooting American stragglers, back to a holding area. On the way there his prisoners began to sing the Nazi youth song and Mike shot them all. He told me this story when we reunited after the war. It's horrible what war does to youths.
I can understand Mike's story, but I must add to Nicholas's story above a much greater horror that our government has hidden from us. A U.N.related analysis has given a 'low' estimate that our war in Iraq killed at least one million people if not more! At least that many Vietnamese were also killed by us in that earlier war. I can understand an 18 year old doing what Mike did but I cannot comprehend a supposedly civilized nation killing millions for the false reasons we have been fed in Vietnam, Iraq, Afghanistan, Yemen, and on and on.
We keep killing and killing and as Nicholas says above all we leave behind is chaos and desolation. Some have said that this is our governments real purpose. To leave chaos and destabilization so that we can rule the world with raw power.


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Biology 2e9.3 Response to the Signal
By the end of this section, you will be able to do the following:
Describe how signaling pathways direct protein expression, cellular metabolism, and cell growth
Identify the function of PKC in signal transduction pathways
Recognize the role of apoptosis in the development and maintenance of a healthy organism
Inside the cell, ligands bind to their internal receptors, allowing them to directly affect the cell's DNA and protein-producing machinery. Using signal transduction pathways, receptors in the plasma membrane produce a variety of effects on the cell. The results of signaling pathways are extremely varied and depend on the type of cell involved as well as the external and internal conditions. A small sampling of responses is described below.
Some signal transduction pathways regulate the transcription of RNA. Others regulate the translation of proteins from mRNA. An example of a protein that regulates translation in the nucleus is the MAP kinase ERK. The MAPK/ERK pathway (also known as the Ras-Raf-MEK-ERK pathway) is a chain of proteins in the cell that communicates a signal from a receptor on the surface of the cell to the nuclear DNA. ERK is activated in a phosphorylation cascade when epidermal growth factor (EGF) binds the EGF receptor (see Figure 9.10). Upon phosphorylation, ERK enters the nucleus and activates a protein kinase that, in turn, regulates protein translation (Figure 9.14).
Figure 9.14 ERK is a MAP kinase that activates translation when it is phosphorylated. ERK phosphorylates MNK1, which in turn phosphorylates eIF-4E, an elongation initiation factor that, with other initiation factors, is associated with mRNA. When eIF-4E becomes phosphorylated, the mRNA unfolds, allowing protein synthesis in the nucleus to begin. (See Figure 9.10 for the phosphorylation pathway that activates ERK.)
Another mechanism of gene regulation involves PKC, which can interact with is a protein that acts as an inhibitor. An inhibitor is a molecule that binds to a protein and prevents it from functioning or reduces its function. In this case, the inhibitor is a protein called Iκ-B, which binds to the regulatory protein NF-κB. (The symbol κ represents the Greek letter kappa.) When Iκ-B is bound to NF-κB, the complex cannot enter the nucleus of the cell, but when Iκ-B is phosphorylated by PKC, it can no longer bind NF-κB, and NF-κB (a transcription factor) can enter the nucleus and initiate RNA transcription. In this case, the effect of phosphorylation is to inactivate an inhibitor and thereby activate the process of transcription.
Increase in Cellular Metabolism
The result of another signaling pathway affects muscle cells. The activation of β-adrenergic receptors in muscle cells by adrenaline leads to an increase in cyclic AMP (cAMP) inside the cell. Also known as epinephrine, adrenaline is a hormone (produced by the adrenal gland located on top of the kidney) that readies the body for short-term emergencies. Cyclic AMP activates PKA (protein kinase A), which in turn phosphorylates two enzymes. The first enzyme promotes the degradation of glycogen by activating intermediate glycogen phosphorylase kinase (GPK) that in turn activates glycogen phosphorylase (GP) that catabolizes glycogen into its constituent glucose monomers. (Recall that your body converts excess glucose to glycogen for short-term storage. When energy is needed, glycogen is quickly reconverted to glucose.) Phosphorylation of the second enzyme, glycogen synthase (GS), inhibits its ability to form glycogen from glucose. In this manner, a muscle cell obtains a ready pool of glucose by activating its formation via glycogen degradation and by inhibiting the use of glucose to form glycogen, thus preventing a futile cycle of glycogen degradation and synthesis. The glucose is then available for use by the muscle cell in response to a sudden surge of adrenaline—the "fight or flight" reflex.
Cell Growth
Cell signaling pathways also play a major role in cell division. Cells do not normally divide unless they are stimulated by signals from other cells. The ligands that promote cell growth are called growth factors. Most growth factors bind to cell-surface receptors that are linked to tyrosine kinases. These cell-surface receptors are called receptor tyrosine kinases (RTKs). Activation of RTKs initiates a signaling pathway that includes a G-protein called RAS, which activates the MAP kinase pathway described earlier. The enzyme MAP kinase then stimulates the expression of proteins that interact with other cellular components to initiate cell division.
Cancer Biologist
Cancer biologists study the molecular origins of cancer with the goal of developing new prevention methods and treatment strategies that will inhibit the growth of tumors without harming the normal cells of the body. As mentioned earlier, signaling pathways control cell growth. These signaling pathways are controlled by signaling proteins, which are, in turn, expressed by genes. Mutations in these genes can result in malfunctioning signaling proteins. This prevents the cell from regulating its cell cycle, triggering unrestricted cell division and perhaps cancer. The genes that regulate the signaling proteins are one type of oncogene, which is a gene that has the potential to cause cancer. The gene encoding RAS is an oncogene that was originally discovered when mutations in the RAS protein were linked to cancer. Further studies have indicated that 30 percent of cancer cells have a mutation in the RAS gene that leads to uncontrolled growth. If left unchecked, uncontrolled cell division can lead to tumor formation and metastasis, the growth of cancer cells in new locations in the body.
Cancer biologists have been able to identify many other oncogenes that contribute to the development of cancer. For example, HER2 is a cell-surface receptor that is present in excessive amounts in 20 percent of human breast cancers. Cancer biologists realized that gene duplication led to HER2 overexpression in 25 percent of breast cancer patients and developed a drug called Herceptin (trastuzumab). Herceptin is a monoclonal antibody that targets HER2 for removal by the immune system. Herceptin therapy helps to control signaling through HER2. The use of Herceptin in combination with chemotherapy has helped to increase the overall survival rate of patients with metastatic breast cancer.
More information on cancer biology research can be found at the National Cancer Institute website (https://www.cancer.gov/research/areas/biology).
When a cell is damaged, superfluous, or potentially dangerous to an organism, a cell can initiate a mechanism to trigger programmed cell death, or apoptosis. Apoptosis allows a cell to die in a controlled manner that prevents the release of potentially damaging molecules from inside the cell. There are many internal checkpoints that monitor a cell's health; if abnormalities are observed, a cell can spontaneously initiate the process of apoptosis. However, in some cases, such as a viral infection or uncontrolled cell division due to cancer, the cell's normal checks and balances fail. External signaling can also initiate apoptosis. For example, most normal animal cells have receptors that interact with the extracellular matrix, a network of glycoproteins that provides structural support for cells in an organism. The binding of cellular receptors to the extracellular matrix initiates a signaling cascade within the cell. However, if the cell moves away from the extracellular matrix, the signaling ceases, and the cell undergoes apoptosis. This system keeps cells from traveling through the body and proliferating out of control, as happens with tumor cells that metastasize.
Another example of external signaling that leads to apoptosis occurs in T-cell development. T-cells are immune cells that bind to foreign macromolecules and particles, and target them for destruction by the immune system. Normally, T-cells do not target "self" proteins (those of their own organism), a process that can lead to autoimmune diseases. In order to develop the ability to discriminate between self and non-self, immature T-cells undergo screening to determine whether they bind to so-called self proteins. If the T-cell receptor binds to self proteins, the cell initiates apoptosis to remove the potentially dangerous cell.
Apoptosis is also essential for normal embryological development. In vertebrates, for example, early stages of development include the formation of web-like tissue between individual fingers and toes (Figure 9.15). During the course of normal development, these unneeded cells must be eliminated, enabling fully separated fingers and toes to form. A cell signaling mechanism triggers apoptosis, which destroys the cells between the developing digits.
Figure 9.15 The histological section of a foot of a 15-day-old mouse embryo, visualized using light microscopy, reveals areas of tissue between the toes, which apoptosis will eliminate before the mouse reaches its full gestational age at 27 days. (credit: modification of work by Michal Mañas)
Termination of the Signal Cascade
The aberrant signaling often seen in tumor cells is proof that the termination of a signal at the appropriate time can be just as important as the initiation of a signal. One method of stopping a specific signal is to degrade the ligand or remove it so that it can no longer access its receptor. One reason that hydrophobic hormones like estrogen and testosterone trigger long-lasting events is because they bind carrier proteins. These proteins allow the insoluble molecules to be soluble in blood, but they also protect the hormones from degradation by circulating enzymes.
Inside the cell, many different enzymes reverse the cellular modifications that result from signaling cascades. For example, phosphatases are enzymes that remove the phosphate group attached to proteins by kinases in a process called dephosphorylation. Cyclic AMP (cAMP) is degraded into AMP by phosphodiesterase, and the release of calcium stores is reversed by the Ca2+ pumps that are located in the external and internal membranes of the cell.
Section URL: https://openstax.org/books/biology-2e/pages/9-3-response-to-the-signal


Hello camp researchers and evaluators! Welcome again to Research 360: ACA's new full circle approach to research and evaluation. I promised in last week's post that I would dive deeper into ACA's 5-year research project, which is one element of Research 360 (local camp evaluation is the other).
Do camp experiences prepare campers and staff to persist and succeed by developing and transferring necessary skills, values, dispositions, and abilities leading to college/career readiness and lifelong learning; and, if so, what enables the development and transfer of these work and life skills for campers and staff?
With this group's task complete, a Research Advisory Group (again, all volunteer experts) was formed to oversee the research project that would explore this question. An advisory group such as this is critical to this study because it ensures accountability to the ACA community and trustworthiness of the research results, values that continue to make ACA's first large-scale study an important resource for camp professionals and researchers alike.
The Advisory Group's responsibilities include recommending a third-party research team, reviewing annual project reports, and working with ACA to share research results in ways that will improve the camp experience and advocate for camp to a board audience. Over the course of the 5-year project, the Research Advisory Group will make valuable contributions to this aspect of Research 360; so, with that in mind, let me introduce them to you.
Read more about the Research Advisory Group, and stay tuned for the next Research 360 post, Cast of Characters Part 2, which will introduce the University Research Team.
Laurie Browne, Ph.D., recently joined the Research Team at ACA. She specializes in ACA's Youth Outcomes Battery and supporting camps in their research and evaluation efforts. Prior to joining ACA, Laurie was an Assistant Professor in the Department of Recreation, Hospitality, and Parks Management at California State University-Chico. Laurie received her Ph.D. from the University of Utah, where she studied youth development and research methods.


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www.yoroi.uk
鉄 Iron
革 Leather
威毛 Braid
漆 Lacquer
Testimonies & Contact
Odoshi-Ito: Lacing for Yoroi
Odoshi ito is the name for the silk or cotton-hemp lacing used to link samurai armour plates together. Sometimes leather is used which is referred to as kawa odoshi. When an armour was in service, it was common practice to have the lacing replaced at least every 10 years, or sooner should it have returned from a campaign. The Samurai were proud of their armours and after all, second to the sword, armour was a form of status. Shoddy appearances were frowned upon and allowing a suit to fall into disrepair was disrespectful. When the samurai class was abolished in 1873 armours became defunct as they only represented the past with no further requirement to maintain them. Many were sold off by their owners to dealers as export items for foreign customers.
Over the years UV Light damage fades the lacing and the original dyes damaged the silk fibres, this eventually degrades it to a level where it simply falls apart. There are methods of supporting the original lacing by backing each strand, but this type of conservation cannot be applied where the braiding is embedded between the odoshi-no-ana holes. There comes a time when it really has to be replaced. The issue with replacement is the cost of renewal, but this outlay can be recouped in time as the value of the armour increases.
Odoshi-Ito: Technical Correctness
Lacing an armour is not as simple as one may first imagine. There are a number of rules that must be adhered too in order to produce a professional finish as all errors made with the lacing will be visible to the viewer and will live with the piece until corrected. There are techniques that require special twists to form shapes, leather anchors are inserted into the holes to lock each section in place, cross knots have to be wetted and flattened, the tension has to be correct in passing through holes, cut strands have to be finished off in the right way, and most importantly the plates must hang naturally within the correct distance of each other.
I have been lacing armours for over 30 years, and have received training in Japan under the leading Katchushi masters such as Nishioka and Ogawa Sensei ensuring that my standard is equal to that of the Japanese studios.
All lacing commissions are conducted in my dedicated textile studio which has a clean and dust free environment equipped with all the custom frames needed to lace any type or armour. Katchushi have to order their lacing from a manufacturer based in Kyoto, therefore, maintaining a stock of all the colours and widths can become problematic. As all my braids are made in-house I can guarantee that the best-sized braid is selected for your armour. In some cases, I have decided to make a special size to ensure that the best possible finish was obtained. I can also offer Shikagawa odoshi 鹿革威
Making Odoshi outside of Japan
Silk Odoshi has a special weave, the strands are compacted tightly together, yet the depth is very thin. This weave prevents the braid from stretching and allows it to be fed through the small holes in the armour plates.
Using alternatives like synthetic rayon or cotton will never deliver the same results. During the lacing process the braid is wetted with water and pressed into shape, cross knots become flat, the tips of the strands adopt a triangular appearance. Once dry the silk retains the shape. Synthetics expel the water making it impossible to create the same finish. Cotten braid is also not acceptable as the fibres fray and becomes fluffy after being fed repeatedly through the holes of the armour. It has to be silk, and it has to be the correct weave.
For years the only place to obtain odoshi was from Japan. As a restorer, I could never pre-empt what commissions were going to turn up, or how much odoshi I should stock. Exchange rates, order levels, delivery times, communication issues and availability were becoming increasingly problematic.
Therefore in 2014, I decided to take on board a venture to produce odoshi in the UK. This was a massive and challenging undertaking for me as I knew absolutely nothing about the process For months I researched braiding machines on the net, youtube, talked to suppliers and manufacturers. By complete luck, I stumbled upon the right contact who could custom make the machines and provide the training. I also fell on my feet when sourcing the raw silk as one of Japan's leading silk manufacturers were so impressed with my ambition that they agreed to supply me with pre-dyed silk in authentic colours.
In 2016 I was able to produce the first-ever odoshi outside of Japan, made from authentic Japanese silk and weaved to exactly the same specification.
David Thatcher Katchushi Koubou
Towcester, Northants, UK
Email: dt@yoroi.uk
Birmingham International Arms Fairs
Hall 1 – The Shokunin Craftsman Table
The Token Society of Great Britain
The Association for the Research and Preservation of Japanese Helmets and Armor – Japan
Copyright 2021 © David Thatcher.


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Harman closed with two birdies and three tough pars for a 5-under 67 that gave him a 1-shot lead at 12-under 204 heading into Sunday's final round.
"Yeah, 12 under, I'd have about a 10-shot lead in most Opens," Harman said.
Erin Hills again lacked enough wind to be the stern test the U.S. Open wants, and it showed. There were 18 rounds in the 60s on Saturday. Going into the final round, 42 players were still under par.
"Usually when you shoot even in a U.S. Open and you're 1 off the lead, you're not 6 down afterward," J.B. Holmes said.
In the 116 previous U.S. Opens, only six players had ever reached 10-under par or better, never in the same tournament. By the end of a wild and wide-open Saturday, five players were double digits under par.
Brooks Koepka made three birdies over his last seven holes for a 68 and joined Thomas 1 shot out of the lead.
Also at 11 under was Tommy Fleetwood, who was poised to take the lead until his pitch came up short, his next shot went 60 feet down a slope on the back of the 18th green and he turned potential birdie into bogey and settled for a 68.
Rickie Fowler, on the verge of being left behind, rallied with three straight birdies and shot 68. He was 10 under, only 2 shots behind.
"It's going to be a really cool day for someone tomorrow," Fowler said. "I'm looking forward to my shot at it. I've been there a handful of times and had some good finishes. But I'm looking forward to getting the job done."
Thomas finished in style, and it had nothing to do with his hot pink pants. He had 310 yards to the hole when he hit 3-wood that could have led to big trouble if he went too far long or left. "Oh gosh, Jimmy, be good," he said to caddie Jimmy Johnson when the ball was in the air, and it was close to perfect.
Thomas poured in the eagle to become the 29th player with a 63 in a major championship.
"The finish was awesome. I'd love to have another one of those," Thomas said.
He might need one at this rate. About all that save Erin Hills from another onslaught was a forecast for the strongest wind of the week. Regardless of the weather, it didn't take long for the 24-year-old Thomas to understand the significance of his record round.
"That means I'm a part of history," he said. "It means I have a lot better chance to win the tournament than I did when the day started. I felt like I've been playing pretty well all week, and didn't have quite the numbers to show for it. Obviously, today I definitely had something to show for it."
Thomas, a major away from joining the young elite in golf, only added to a year of low numbers. He made an eagle on his final hole at the Sony Open in January to shoot 59, and he went on to break the PGA Tour's 72-hole scoring record.
The U.S. Open didn't seem to faze him, and he delivered a variety of big shots that led to his sensational finish.
He rolled in an 18-foot birdie putt on No. 5 from the edge of the green that broke so severely that he stood with his toes facing the hole and rapped the ball toward his left foot. It took a hard turn to the right and rolled in. In the hay left of the 12th fairway, he gouged it out with a 9-iron and watched it roll to 10 feet.
And with the tees moved up on the 15th hole to make it reachable, Thomas hit a 3-wood that rolled off the back slope of the green to 6 feet. He two-putted for birdie, and he rolled in a 25-foot birdie putt on the 17th.
"It doesn't matter how long, how whatever the course is," Thomas said about the longest course in major championship history. "When you give us soft greens, good greens and not much wind, you know there are going to be some good scores. I was just happy that I was the one that was able to take advantage of it today."
But the work is not done.
Of the five other players who shot a 63 in the third round at a major, none went on to win. Most of them had to come from far back going into the weekend, and it was difficult to put together two good rounds.
Then again, none of the top 16 on the leader board has ever won a major.
"Someone has a very good chance of ending up with their first major tomorrow," Fowler said.
Former KU fan favorite Julian Wright returns to Sprint Center on Saturday
By Gary Bedore
Julian Wright, a fan favorite during his two seasons at Kansas, will be playing for "Power" in Ice Cube's 3-on-3 BIG3 League on Saturday at the Sprint Center.
Columbia Rock Bridge guard Dajuan Harris commits to Kansas. He'll be in class of 2019
Altavilla, Carroll lead the way for AZL D-backs


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Croatia competed at the 2019 World Aquatics Championships in Gwangju, South Korea from 12 to 28 July.

Medalists

Diving

Croatia's diving team consisted of 2 athletes (1 male and 1 female).

Swimming

Croatia entered 11 swimmers.

Men

Women

Water polo

Men's tournament

Team roster

Marko Bijač
Marko Macan
Loren Fatović
Luka Lončar
Maro Joković
Hrvoje Benić
Ante Vukičević
Andro Bušlje
Lovre Miloš
Josip Vrlić
Anđelo Šetka
Javier Gadea Garcia
Ivan Marcelić
Coach: Ivica Tucak

Group B

Quarterfinals

Semifinals

Third place game

References

World Aquatics Championships
2019
Nations at the 2019 World Aquatics Championships

redpajama_set_name: RedPajamaWikipedia


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by Tony Van Gelder | Oct 8, 2021 | Coronavirus, Investing, Weekly Financial Thought
"Supply Chain Struggles"
This weeks thought, first appeared in Yahoo Finance's morning brief. With all else being equal, economic times are hard enough as we come out of the pandemic. However, as we do this, and demand spikes back up, we are seeing unprecedented supply chain issues playing out in the wider economy.
Global supply chains are badly stuck, and the timing couldn't be worse
A few short months ago when second quarter (Q2) earnings cranked up, stocks were on the rise, the mass vaccination campaign was going relatively smoothly, and Wall Street couldn't revise estimates upward quickly enough to compensate for what appeared to be a seemingly bottomless well of demand.
As the Q3 season gets underway, the tableau is starkly different. Investors are decidedly spooked about everything from soaring prices (which is hammering stocks and pushing up Treasury rates) to resurgent infection rates, and the latest public relations bombshells rocking Facebook (FB).
But the biggest storyline for markets to follow is corporate America's ongoing struggles with an economic nervous system (also known as the global supply chain) that, as Bank of America succinctly put it last month, is in "disarray."
As we've covered in the Morning Brief, COVID-19's aftershocks are making goods and services more scarce, and harder to deliver to the people who want them.
A global economy that's heavily reliant on goods manufactured abroad, and shipping them to lands far and wide, is still vulnerable to a virus that's running rampant, and creating strains in critical supply hubs. In fact, "supply chain" is becoming the hottest catchphrase of the earnings season — and hardly a good one.
As Bank of America stated in a September analysis, "a supply chain is only as strong as its weakest link. Seemingly small disruptions can snowball into a big economic loss. There is a strong case for diversification/localization of supply chains, but it will be hard to reduce dependence on Asia."
What that means is that inflation that Federal Reserve officials once believed would be temporary is looking "uncomfortably sticky" and is a drag on consumer demand. Loosely translated, that suggests consumer-facing companies may have some very unpleasant news for markets when Q3 results start to roll in — primarily in the form of high prices that could be hiked even further.
Playing out in Earnings Announcements
Already, the ratio of positive to negative earnings pre-announcements from S&P 500 corporations is narrowing. According to Refinitiv data, there's been 44 negative releases compared to 59 positive — a ratio of 0.7, well below the long-term average of 2.6 and lower than the four-quarter average of 0.8.
A taste of what could lie in store has already come from retail giants like Bed, Bath & Beyond (BBBY), Nike (NKE) and Kohl's (KSS), who have sent shockwaves down the market warning about supply strains that's making it harder to keep up with demand.
In a lengthy research note, Goldman Sachs' David Kostin identified strained supply chains as one "key risk" for earnings. He added that there's a chance that "supply chain normalization takes longer than expected and that unmet demand today is not fully recouped in later quarters." All this just when investors thought they had turned the page on a rough September.
Not coincidentally, October plays host to Halloween. Add in surging energy costs, an unresolved partisan food fight over the debt ceiling and risks popping up everywhere, things sound spooky!
While we certainly aren't proponents of a "doom & gloom" outlook, we are also viewing this time as a good opportunity to realize some of the great gains we have experienced over the past 18 months. Positioning investments for long-term growth means taking heed with current developments but not getting spooked to make a 180-degree course correction.


SPS Finquest SME IPO – Should you invest?
SPS Finquest Limited is coming up with an IPO to issue 33.44 Lakhs of shares @ Rs 75 per share aggregating to Rs 25.08 Crores. This NBFC company has been growing year on year. This SPS Finquest IPO has several positive points along with a few negative points. Should you invest in an SPS Finquest IPO? Do you think that your money could multiply in the next few years by investing in this SPS Finquest SME IPO?
SPS Finquest is a NBFC registered company with an RBI. It operates as a Non Deposit taking Non-Banking Finance Company engaged primarily in the business of advancing loans for investing/trading in securities. This company provides its shareholders with the opportunity to participate in the field of financing against equities and other financial instruments that provides a decent return on the capital deployed.
Also Read: How to check best blue chip stocks for investing?
The company has posted revenue of Rs 2.15 Lakhs for the year ended Mar-2009 and Rs 787 Lakhs for the year ended Mar-2013. First 9 months of this financial (FY 2013-14) it year made a revenue of Rs 598 Lakhs.
The company has posted a profit of Rs 38,205 for the year ended Mar-09 Vs profit of Rs 36 Lakhs for the year ended Mar-2013. First 9 months of this financial year (FY 2013-14) it made a profit of Rs 42.68 Lakhs.
Good revenue growth in last 4-5 years.
The company intends the issue proceeds of Rs 25 Crores to be repaid to the debt for single company named Shri Shapoor Mistry (shareholder) for Rs 24.73 Crores. This company has given this loan @ 11% interest. Means SPS Finquest is making Rs 2.47 Crores as interest on such loan every year. If this is repaid, the interest burden would be reduced and this amount would be straight away moving as profit. This would be a good move for the company to reduce debt.
Highly dependable on promoter performance.
NBFC companies face defaults and non payment would impact profits.
Loans held by the company are short term in nature which carries high interest rates.
Revenue would be highly volatile due to business nature.
The company posted a profit of Rs 36 Lakhs in FY 2012-13 which is 66% lower than FY 2011-12 profit.
Also Read: What you should learn from stock market rallies ?
EPS for the financial year ended Mar-2013 is Rs 9.36. Based on last financial year EPS of Rs 9.36, P/E ratio works out to be 8. This company has issued bonus shares to promoters and post bonus issue, P/E Ratio would be 24.11. The P/E ratio of its competitors BCB Finance Limited is 29.19 (Highest) and First Leasing Limited is 2.99 (Lowest). Hence the issue price is on the higher side.
Though company has been growing, considering all negative points and high issue price, I would advice investors to stay away from this IPO.
One off track question -My father is having shares in Hindalco from last 30 yrs. Should he keep it for some more time or sell it?
Hi Anjum, Markets are at peak. You can look to sell now.
I had never invested in any IPO. So, I would like to ask one question. My question may look silly as I don't have any idea about investing directly in share market.
I had read aprrox. all your analysis about SME-IPO. I found one thing common in all. Their profit % is quite low, less than 10% for any FY. For this SME-IPO, if I had purchased share worth 1.2 Lakhs & company had posted a porfit of 10%, then profit will be just 12,000 per year i.e. 1K/month. So, what's great in investing in share market as we get better returns in Mutual Funds & in some cases, in FD?
Even, there was one SME-IPO, something Biotech, you had recommended investors to invest in that IPO as it was great and at the same time, there were several other IPOs that you had recommended but profit % was far lower. Why did you recommended everyone to invest in those IPOs. I am sure that apart from profit%, there is something else that you consider before buying shares. So, what is that & why you had recommended those IPOs whose profit% are below 10%?
Mohit, Your understanding is wrong. I am not saying appreciation of share value. I am talking about what company is earning. If you invest Rs 1 lakh in IPO, company would use such proceeds for future growth or repaying debt. The profits are the turn-over / revenue of the company. Low profit means, high risk as they vanish anytime. Stable or high profits are indication that company is doing good and can sustain its performance.
Suresh KP, Thanks for your valuable suggestions. Really these kind of analysis helps many investors like me.
I appreciate for your excellent analysis on all the financial queries without any bias. Thank you.


Anglers can choose to support children's charities or reef building efforts by selecting which fishing tournament they want to compete in.
Anglers already enjoy the thrill of the catch. But who doesn't love the thrill of winning cash and prizes?
Tournament fans can combine the two thrills with the annual run of fishing tournaments along the Treasure Coast this year. What follows is an approximate roundup of summertime fishing tournaments scheduled to take place in and around salt waters of the Treasure Coast.
Some tournament operators might adjust dates based on weather or other circumstances, so always check with tournament organizers for complete details. This list does not include the winter run of sailfish tournaments.
Any organization planning to have a fishing tournament this summer, please send the information to Ed Killer at ed.killer@tcpalm.com.
Weigh-in: 3 p.m. June 8-9 at Fort Pierce Riverwalk. Awards ceremony and after-party will be at the Sailfish Brewery.
Fun fact: Divers who harvest lionfish, and apply for a saltwater products license, can sell their catches to a wholesale buyer on site during the event.
What else: Cook-off is 12-4 p.m. June 23. Tickets are $13 before June 15, $14 between June 16-22, and $15 at the door to enjoy tasting lionfish prepared by local chefs. Proceeds will benefit Coastal Connections.
Entry: $70 per diver by June 15, or $85 after; or $210 for diving team of four by June 15 or $250 after.
Fun fact: Last two years, event has removed 1,229 lionfish.
Entry: Non-members $275 early registration before May 3, then $325. Members $250 early registration before May 3.
Entry: $100 per boat for up to four anglers before Aug. 16. Kids 12 and younger fish for free.


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Alex Chalk
MP for Cheltenham
About Alex
News, Columns and Updates
Come to Parliament
Alex Chalk was first elected as Cheltenham's MP in 2015 and was re-elected in 2017.
Alex is a member of the Justice Select Committee which scrutinises the work of the Government in this area. In addition to this, Alex is a Chair of the All Party Parliamentary Group (APPG) on Pro Bono and Public Legal Education; Vice-Chair of the APPG on Cycling; Vice-Chair of the APPG on Bees; Vice-Chair of the APPG for Ancient Woodland and Veteran Trees; Vice-Chair of the APPG for Town & City Centres; and a member of the APPG for Highways Maintenance.
Alex was also made Parliamentary Private Secretary (PPS) to the Department of Education in early 2018.
You can read some of Alex's speeches and appearances in Parliament below. For a more comprehensive and up-to-date list of what Alex has raised in Parliament, please click HERE.
You can also find out more about what Alex has been doing in Parliament by recieving his Fortnightly Update.
Extended Schools | Department for Education | Written Answers
To ask the Secretary of State for Education, what progress the Government has made on providing additional funding to enable the extensio
To ask the Secretary of State for Education, what the process is to access the additional funding to extend secondary school hours with t
State Pension Age: Women | Commons debates
The WASPI women in Cheltenham I have spoken to broadly recognise that the SNP's plan to reverse the entire equalisation process, at a cos
Clean Air Zones | Department for Environment, Food and Rural Affairs | Written Answers
To ask the Secretary of State for Environment, Food and Rural Affairs, if she will make it her policy to use revenues from clean air zone
Pensions: Public Sector | HM Treasury | Written Answers
To ask Mr Chancellor of the Exchequer, whether the Government has made an assessment of the potential merits of changing the remit of the
National Health Service Funding | Commons debates
On financial management, does the Secretary of State recognise that in Labour-run Wales agency staff spend has increased 60% in the past
Topical Questions | Oral Answers to Questions - Education | Commons debates
Some parents and teachers in my constituency find it frustrating that if Cheltenham's schools simply received average funding per head, f
Parliament Milestones
Keep up-to-date in Westminster
Alex Chalk MP for Cheltenham
Promoted by J Double on behalf of A Chalk, both of 1143 Regents Court, Gloucester Business Park, GL3 4AD. In line with data protection regulations, Alex Chalk's office processes constituents' data for policy and casework purposes under the lawful basis of public task. In instances where this lawful basis is not sufficient and explicit consent is required, a member of staff will get in touch with you to establish consent. You can find our full privacy notice on our website at www.alexchalk.com/privacystatement
About Alex Chalk
Copyright 2019 Alex Chalk MP for Cheltenham. All rights reserved.


Charles Shrem, 24, left, speaks with another Bitcoin dealer, at a bar in New York. He will plead guilty to aiding unlicensed money transmitting.
A prominent supporter of Bitcoin, Charles Shrem, says he will plead guilty Thursday to resolve federal charges that he helped smooth the way for drug transactions on the online marketplace Silk Road.
He is to plead in New York federal court to one count of aiding and abetting the operation of an unlicensed money transmitting business.
The revelation came as the death of one of the inventors of Bitcoin was announced.
Shrem, one of the most vocal advocates of Bitcoin, was accused in January of using his company to convert dollars into Bitcoin for users of Silk Road, the now-defunct bazaar known for its wide selection of narcotics and drug paraphernalia.
Federal prosecutors contended that he operated the scheme in cooperation with Robert Faiella, known as BTCKing, who was also arrested in January, in Florida.
"They want a guilty plea on their books, and that's what they're going to get," Shrem said Saturday from his parents' home in Brooklyn.
Shrem's arrest at John F. Kennedy International Airport this year stirred consternation in the Bitcoin world, which continues to grapple with the perception that the digital currency provides an anonymous way to engage in illegal activity.
Shrem, 24, said his plea could include jail time, though he is optimistic that it will not.
Meanwhile, Hal Finney, a cryptographer and one of the earliest users and developers of the virtual currency, died on Thursday in Phoenix. He was 58.
Finney had been paralyzed by amyotrophic lateral sclerosis, or ALS, and was taken off life support at Paradise Valley Hospital, his wife, Fran Finney, said.
She said his body was immediately prepared for cryonic preservation by the Alcor Life Extension Foundation in Scottsdale, Ariz., according to his wishes.
In 1991, he began doing volunteer work for a new software project known as Pretty Good Privacy, or PGP, and immediately became one of the central players in developing the program.
PGP aimed to make it possible for people everywhere to encrypt electronic communication in a way that could not be read by anyone other than the intended recipient.
The program used relatively new innovations in encryption that are still thought to be invulnerable to code breakers.
While working on PGP, Finney became fascinated by the concept of digital currencies that could not be tracked by governments and banks.
He was involved in many experiments aimed at creating an anonymous form of digital money, including his own invention, in 2004, of reusable proofs of work.
Though that system never took off, he quickly saw the promise of the Bitcoin project when it was announced on an obscure email list in 2008 by a creator with the pseudonym Satoshi Nakamoto, who some thought was Finney, speculation that he denied.


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Immunotherapy Research
PLGA Fund at PBTF
Opportunity Grants
Early Career Development
Research Advisory Network
Pediatric Brain Tumor Foundation Names Awardees of Early Career Development Research Grants
The Pediatric Brain Tumor Foundation is excited to announce three new Early Career Development research grants.
The PBTF's multi-year Early Career Development program funds talented pediatric brain tumor researchers who are in the first five years of their first faculty position, with the expectation that this funding will facilitate grantees' transition to becoming fully independent investigators.
Each grant awardee will receive $300,000 over the next three years to support their research project. The projects will study blood-brain barrier permeability and drug penetration in diffuse intrinsic pontine gliomas, viral mimicry as a potential therapeutic strategy against H3K27M-driven midline high-grade gliomas, and the use of ATM inhibition to radiosensitize brainstem gliomas.
"It is critically important to keep fueling the pediatric brain tumor field with talented and dedicated researchers who have a long-term commitment to moving the needle forward and carving out new lines of investigation," says Dr. Joanne Salcido, PBTF's vice president of research and advocacy. "Our Early Career Development awardees' work today and over the years ahead offers hope for changing the future for the good of all children and teens diagnosed with a brain tumor."
The following researchers and three-year projects were awarded a $300,000 Early Career Development grant:
Timothy Phoenix, PhD, University of Cincinnati
Co-mentors: Q. Richard Lu, PhD, and Maryam Fouladi, MD, Cincinnati Children's Hospital Medical Center
Project title: Maintenance of DIPG Blood-Brain Barrier Integrity by Angiopoietin1
Stephen C. Mack, PhD, Texas Children's Hospital
Primary mentor: Donald W. Parsons, MD, PhD, Baylor College of Medicine, Texas Children's Hospital
Co-mentor: Nada Jabado, MD, PhD, McGill University
Project title: Harnessing Viral Mimicry to Target H3K27M-Driven Pediatric Glioma
Zachary Reitman, PhD, Duke University
Co-mentors: David G. Kirsch, MD, PhD, and David M. Ashley, MBBS, PhD, Duke University
Project title: Identifying Brainstem Glioma Subtypes That Can Be Radiosensitized by ATM Inhibition
View Early Career Development project summaries at www.curethekids.org/early-career-grants. Proposals were evaluated by a scientific review team on the merit of researchers' mentorship and research environment, researchers' productivity to date and career plan, and the proposed research projects.
"Given the significant and ever-increasing challenge of establishing a successful research program as a junior investigator, grant mechanisms such as the Pediatric Brain Tumor Foundation's Early Career Development grants awarded to Drs. Mack, Phoenix and Reitman are critical to support the development of the next generation of brain tumor researchers," says Dr. Parsons, who will serve as Dr. Mack's primary grant mentor. "Equally importantly, the collaborative research community fostered through the PBTF will serve to provide valuable guidance and mentorship as these researchers pursue scientific advancements and novel therapies for children with brain tumors."
The PBTF started its Early Career Development program in 2014 to support rising stars in research and seed the field for future pediatric brain tumor discovery. Previous awards have led to progress in medulloblastoma and neuro-epidemiology research and helped recipients leverage additional research funding to establish their work in the pediatric brain tumor field. Following receipt of an Early Career Development grant, Pratiti (Mimi) Bandopadhayay, MBBS, PhD, was named Assistant Professor of Pediatrics at Harvard Medical School and launched the Bandopadhayay Lab at Dana-Farber Cancer Institute.
"The Pediatric Brain Tumor Foundation's Early Career Development program was key in supporting me in the very early stages of my career," says Dr. Bandopadhayay. "The research results generated through this award allowed me to get more funding from the National Institutes of Health and allowed me to get the training and mentorship that was essential in helping me set up my own independent lab which is dedicated to pediatric brain tumors. Just as important, the Early Career Development grant allowed me to become part of the PBTF family, introducing me to other scientists with whom I now collaborate. Thank you so much to the PBTF for supporting me with an Early Career Development award."
As part of our continued commitment to foster innovation in the pediatric brain tumor research field, the PBTF is also sponsoring the Young Investigators portion of the 2019 Neurobiology of Disease in Children (NDC) conference. This year's NDC on Oct. 23 and 24 will focus on pediatric brain tumors. The Young Investigators program highlights the research of up-and-coming scientists and engages them in discussion of future directions and research priorities.
"The Pediatric Brain Tumor Foundation is honored to provide support for the NDC symposium's high-quality program. It assembles preeminent investigators and medical practitioners who tend to the impaired neurological function pediatric brain tumor patients can experience as a result of their tumor or treatment," says Dr. Salcido. "The program's emphasis on patient-focused research aligns with the PBTF's dual-mission to fuel lifesaving cures and life-changing family support, with this forum also giving us a platform to increase the medical community's awareness of the family support resources we offer."
As the world's leading nonprofit dedicated to children and teens with brain tumors, the PBTF has invested and partnered with organizations to invest more than $44 million in scientific discovery at more than 50 centers worldwide. Visit www.curethekids.org/research to learn more about the PBTF's Early Career Development grants and other research programs.


Tired of studying from textbooks for hours and coming up with little to no results? Already tried audiobooks in the hopes of learning English and that didn't work out? Why not make learning a new language fun and have an experience similar to immersing yourself with natives speakers from the comfort of your home? Try watching movies in English!
Why learning English through movies just works?
Learning English through movies is effective and fun!
In a classroom words are often learned out of context. You are sitting at your desk, looking at your student book and completing exercises as directed by your teacher. And you have to rely on your imagination when practicing phrases related to grocery shopping and that airport visit. That's difficult and not very effective. Even if you learn that dictionary definition you forget it in no time.
Learning English through videos such as English movies engages more of your senses. You hear the dialogs and background sounds, see facial expressions and the whole movie set. As a result you learn effectively without even noticing and as a bonus you remember better what you learn by associations.
"Hello. How do you do?" is one of those English expressions you may find in your textbook or hear in your English class. And while that's proper English..
You will not hear native speakers say "Hello. How do you do?" in a casual conversation. Surprised?
In fact, when I did a survey on English phrases which are too formal to be used in a casual conversation, one of the native speakers respondents said: "When I hear hello, I can tell I'm being greeted by non-native speakers". There is nothing wrong with saying "hello" or "goodbye" however English speakers just find these words to be a little too formal. But it's hard to find that out from your English textbook. Movies, on another hand, provide you with the same natural language that would actually be used by native speakers.
English and most languages is more than just the words we speak. It is also about how they are said. Two words may appear to have the same meaning in the dictionary however it is common to hear them used in different contexts. Tone can also have a major effect on how a sentence is perceived. "How is it going?" you may ask your native speaker friend (now that you know that how do you do won't work). She may answer good or OK. But does good mean good and ok mean ok? To be able to communicate well you have to learn to read between the lines.
And so you will learn that for some reason when it comes to how things are going – great means good, good means ok and OK may means so-so or not good (but I don't want to complain). You won't learn that from your textbook. But movies can help. And the good news is – that you will be able to learn from the world's best actors how to use the tone of voice and the body language to covey the message. As long as they are English native speakers you can also use them as an excellent source of native English pronunciation. After all they all received great voice training.
Motivation is key when it comes to language learning. Does studying from a textbook put you to sleep? Do you really have enough of boring English grammar exercises and cramming those endless word lists? Just rent an good movie, relax and learn English not even noticing that you're learning new words and phrases, grammatical structures and pronunciation .. while having fun!
Improve your listening skils, vocabulary, pronunciation and more through English movies.
What's the most challenging thing for students learning English through a movie that can actually be beneficial?
Many English learners find the movies difficult to follow along with because words are spoken too fast.
However, this can actually be very beneficial because it prepares you well for interactions with native speakers. English textbooks and recordings are spoken unnaturally clearly and slowly. Also, in your typical language learning environment words and expressions are chosen very carefully, often with the use of high frequency word lists.
And that can handicap you instead of strengthening your skills. Movies provide you with the ability to learn commonly used informal expressions, slang, or scum words and phrases that, while quite common, are rarely taught in textbooks or dictionaries. All, spoken at natural speed.
Improving speaking skills through listening? Wait a second..
Yes, you can improve your speaking skills by listening. After all that's exactly how babies learn their first language. Speaking and listening go hand in hand. And when it comes to language learning – most is learned through imitation.
We spend 45% of our active daily time listening and 30% speaking found Leroy Lane in 1987. Studies also confirm that most of us are poor and inefficient listeners.
Listening to native speakers will help you speak better and more fluently. You will hear how to put words together and how to use intonation to link them and to emphasize the important parts of the message. Best part is – with movies you can replay everything and practice until you get it exactly right.
English pronunciation no matter if British or American is challenging for most learners.
Attempting to learn the English language through reading alone can build poor habits. Listening to English spoken in movies naturally helps you learn how to pronounce words properly. You hear how the world's best actors speak and you can see the position of their lips and mouth which can help you pronouncing the vowels. By turning on subtitles you can also see how these words are spelled.
Good news. There is a such a way! It's called incidental learning. Here's how it works. You learn new words without even knowing guessing their meaning from the context. The more telling the context and the higher your attention the more effective the incidental learning. Watching can work miracles since you are exposed to many visual cues that help you guess the meaning of unknown words. Movies are also entertaining which keeps your attention high. You just need to select a movie with the level of English that is not to difficult to follow for you.
Through courses you will learn a lot of vocabulary and grammar but you may not understand how to apply this in real life. By watching movies you will understand how to actually apply what you have learning in everyday situations.
Some learners find it difficult to learn English through watching movies. This may be due to the level of English being too high for you to understand or the movie may be too long or just boring. Some movies feature characters with heavy accents that make it difficult to follow along.
Those shouldn't discourage you because for every problem on that list there is a solution which you can leverage.
Solution: Choose a movie that matches your language proficiency goal. Your goal ideally should be one level higher than your current level of English. If you are just starting out focus on movies that are appropriate to your level and what you want to achieve. Children's shows, movies and cartoons are great for this.
Solution: Good news is, it doesn't have to be. It is totally up to you to make it interactive. For example: you can make notes while watching and reading up on the topic featured in the movie after watching. You could also talk to a friend about what you watched or even watch the movie with someone and exchange remarks as you are both watching.
Solution: Deliberately decide on a movie with mainly American, British or other English accents. Choose only one accent of English that you want to practice and stick to it. The differences in pronunciation are significant and mixing up accents will confuse you. Finally, avoid movies where a lead actor has to fake an accent as it won't be authentic and you would want to learn from the natives speakers.
Solution: Select movies that are interesting for you. Read movie description and decide on a theme that sounds fun and is at least somewhat familar. Don't start with very long movies e.g. Titanic.
1. Choose an interesting movie.
Don't wast time on boring movies. If you are bored it will be difficult to pay attention. And no attention equals no incidental learning. Avoid movies that are vague, overly philosophical, or don't have a solid story line . Choose films that you would normally watch in your native language. When in doubt check out movie reviews. Sites such as Rotten Tomatoes and IMBD have reviews for tens of thousands of movies. For quick suggestions check out the following sections.
2. Choose a movie that matches your level of English.
For beginners, I highly recommend choosing animated movies. The main reason is that the English used is very friendly, enjoyable and easy to understand. Children's movies are the lest likely to include rarely used and difficult English words. Attempting to use movies based on authors such as Shakespeare (for example "West Side Story (1961)") can lead to confusion. Shakespearian English has not been used for centuries and even advanced and native English speakers have difficulty understanding it.
3. Pick a movie in your target accent.
You would be surprised to learn how many differences there are between British English and American English. Especially when it comes to pronunciation. Pick movies that will help you polish your target accent. If you don't have any preference, pick an American movie due. You will have an excellent choice of Hollywood movies to choose from.
Subtitles can help you big time. They will let learn spelling. They will make it easy to look up the new words in a dictionary.
English learners who activated English subtitles performed 17% better in listening comprehension – found a study by J. Birulés-Muntané and S. Soto-Faraco.
After watching a movie for the first time you may want to watch it again without subtitles. This will help you to focus on the conversation and dialogue as a whole rather than looking at individual words. You will be able to focus better on the story. You can see how much of the movie you understand without subtitles and perhaps recognize any weaknesses in your understanding of English.
Practice and repeat short sentences after learning what they sound like. Doing this will help you to ingrain it in your brain and remember it longer. The exercise can be fun especially when you know that you are doing it properly and you understand what you're saying.
6. Watch English movie with friends.
You may have friends that want to learn English or improve their skills. It might be difficult to get them to join you in a study session however they are unlikely to turn down an opportunity to watch an enjoyable movie. Your friends can help you with parts you don not understand. After the movie you and your friends can practice what you learned and discuss the movie (in English of course).
7. Keep a movie journal.
Did you hear a sentence or a word that you liked or that popped out at you? Would you like to use some of these in the future? Write it down! If you watch your movies, stop the video if you hear something that might be useful and write it down in a notebook. This way, you can collect real-life phrases that you can practice and use with your English friends in the future (so long as they're not offensive, of course).
8. Just have fun and don't try to understand every word.
You will find that many of the words you have learned are actually almost never used in day to day life. It is fine to begin putting less emphasis on these words in your studying and to begin using words that are actually used.
If you decided that you are going for the American accent you will have a lot of movies to choose from. In fact, the American film industry (often called Hollywood) produces more than 800 files a year in American English. Hollywood is the oldest film industry in the world and grosses more money every year than that of any other country.
Finding Nemo is an animated movie about a clownfish named Marlin who loses his son Nemo. In his search for his son he meets another fish named Dory. Together, the two make an incredible adventure across the ocean in order to find Nemo. The movie is a classic and recognized as one of the best animated films ever.
Zootopia tells a story of the unlikely partnership between a rabbit police officer and a red fox con artist, as they uncover a mystery causing the disappearance of inhabitants from a big city of anthropomorphic animals. You will like the movie for the spectacular animations, sense of humour and enchanting music and stunning acting. Adult viewers will find a lot of parallels to the real world.
This movie is the story of young Mark Zuckerberg, a student at Harvard who went on the create Facebook and revolutionize how we communicate with other. Zuckerberg finds fame and becomes a billionare but at a great price. The movie will help introduce you to some more technical terms relating to computers, technology, and social media without being too difficult to understand.
Colin Firth plays a drama like King George at this time, struggling to overcome his speech obstacle. He wishes to give an inspirational speech to inspire his soldiers to fight. The movie focuses especially on pronunciation and therefore is a great choice for those wishing to learn English.
The best way to learn "Queen's English" is to hear the Queen. This film, played by Helen Mirren, is a historical drama documenting the reaction of the British royal family to the death of Princess Diana. Not only does it give a fascinating insight into how the institution truly works, but the spoken English language is incredibly clear and easy to understand. You will learn how the upper-class accent differs from the other accents in the UK.
Some dialogue in this movie is easy to follow however others can be quite difficult to understand. However, as with "King's Speech", you will learn a lot of tips to improve your pronunciation, distinguish different British accents and vocabulary, thanks to its excellent classical songs.
This movie is a better choice for those that are more experienced. The strong north-English accent of its protagonists can make it difficult due to the many different expressions and slang terms used.


2001-02 House Bill 3874. An Act Requiring Identification Of Parents For Children Receiving Benefits From The Department Of Transitional Assistance.
Title 2001-02 House Bill 3874. An Act Requiring Identification Of Parents For Children Receiving Benefits From The Department Of Transitional Assistance.