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Isidore Geoffroy Saint-Hilaire (1805-1861)
Isidore Geoffroy Saint-Hilaire studied anatomy and congenital abnormalities in humans and other animals in nineteenth century France. Under the tutelage of his father, Etienne Geoffroy Saint-Hilaire, Isidore compiled and built on his father's studies of individuals with developmental malformations, then called monstrosities.
Subject: People, Disorders
Thesis: Leo Kanner and the Psychobiology of Autism
This thesis illustrates that Kanner held an explicitly descriptive frame of reference toward his eleven child patients, their parents, and autism. Adolf Meyer, his mentor at Johns Hopkins, trained him to make detailed life-charts under a clinical framework called psychobiology. By understanding that Kanner was a psychobiologist by training, I revisit the original definition of autism as a category of mental disorder and restate its terms.
Jeffrey Weinzweig's Experiments on In Utero Cleft Palate Repair in Goats (1999-2002)
Jeffrey Weinzweig and his team, in the US at the turn of the twenty-first century, performed a series of experiments on fetal goats to study the feasibility of repairing cleft palates on organisms still in the womb. Weinzweig , a plastic surgeon who specialized in cleft palate repair, and his team developed a method to cause cleft palates in fetal goats that are similar to clefts that occur in human fetuses. Using their goat congenital model, the team developed a method to repair a congenital cleft palate in utero, or in the womb.
Subject: Experiments, Disorders
Hermaphrodites and the Medical Invention of Sex (1998), by Alice Domurat Dreger
Hermaphrodites and the Medical Invention of Sex, by historian of science Alice Domurat Dreger, was published in 1998 by Harvard University Press. In the book, Dreger describes how many doctors and scientists treated human hermaphrodites from the late nineteenth century to the early twentieth century. She states that during this time period, many physicians and scientists struggled to determine the nature sex, and to support a classification of sex as male or female, many physicians and scientists resorted to viewing a person's gonads for identification of his or her sex.
Subject: Publications, Theories, Disorders
Truman William Brophy (1848–1928)
Truman William Brophy developed a cleft palate surgical repair, later called the Brophy Operation, in the late nineteenth century US. The procedure improved facial aesthetics and speech in cleft palate patients. A cleft palate occurs during development when the palatal bones in the roof of the mouth don't completely fuse, leaving an opening, or cleft, in the upper lip and mouth. Brophy's cleft repair used compression inside and outside of the mouth to push the palatal bones into normal alignment shortly after birth.
William Thornton Mustard (1914-1987)
William Thornton Mustard was a surgeon in Canada during the twentieth century who developed surgical techniques to treat children who had congenital heart defects. Mustard has two surgeries named after him, both of which he helped to develop. The first of these surgeries replaces damaged or paralyzed muscles in individuals who have polio, a virus that can cause paralysis. The other technique corrects a condition called the transposition of the great arteries (TGA) that is noticed at birth.
Subject: People, Disorders, Disorders
John Chassar Moir (1900–1977)
John Chassar Moir lived in Scotland during the twentieth century and helped develop techniques to improve the health of pregnant women. Moir helped to discover compounds that doctors could administer to women after childbirth to prevent life-threatening blood loss. Those compounds included the ergot alkaloid called ergometrine, also called ergonovine, and d-lysergic acid beta-propanolamide. Moir tested ergometrine in postpartum patients and documented that it helped prevent or manage postpartum hemorrhage in women.
Subject: People, Reproduction, Disorders
Early Infantile Autism and the Refrigerator Mother Theory (1943-1970)
In 1943, child psychiatrist Leo Kanner in the US gave the first account of Early Infantile Autism that encouraged psychiatrists to investigate what they called emotionally cold mothers, or refrigerator mothers. In 1949, Kanner published Problems of Nosology and Psychodynamics of Early Infantile Autism. In that article, Kanner described autistic children as reared in emotional refrigerators. US child psychiatrists claimed that some psychological or behavioral conditions might have origins in emotional or mental stress, meaning that they might be psychogenic.
Subject: Disorders, Theories
Emma Wolverton (1889–1978)
Emma Wolverton, also known as Deborah Kallikak, lived her entire life in an institution in New Jersey after psychologist Henry Goddard classified her as feeble-minded. He also wrote a book about Wolverton and her family that psychiatrists previously used to show that intellectual disability is hereditary. At the time, researchers in the psychology field, including Goddard, were working to understand differences in people's intellectual abilities. They used the term feeble-minded to refer to those they described as having lower intellectual functioning.
Martius Flap Procedure to Repair Obstetric Fistulas
The Martius flap procedure is a surgical procedure used to treat obstetric fistulas in women. Heinrich Martius developed the procedure in twentieth century Germany to treat women with urinary incontinence caused by stress, and later doctors used it to repair obstetric fistulas. Fistulas occur in pregnant women when a hole is torn between the vagina and the urinary tract (called vesicovaginal) or the vagina and the rectum (called rectovaginal). The hole, or fistula, occurs in the tissue separating two organs and therefore obstetric fistulas result in either urinary or fecal incontinence.
Subject: Technologies, Disorders
The Mustard Operation
The Mustard Operation is a surgical technique to correct a heart condition called the transposition of the great arteries (TGA). TGA is a birth defect in which the placement of the two arteries, the pulmonary artery, which supplies deoxygenated blood to the lungs, and the aorta, which takes oxygenated blood to the body are switched. William Thornton Mustard developed the operation later named for him and in 1963 operated on an infant with TGA, and ameliorated the condition, at the Hospital for Sick Children in Toronto, Canada.
"The Familial Factor in Toxemia of Pregnancy" (1968), by Leon C. Chesley, et al.
In the 1950s and 1960s, researchers Leon Chesley, John Annitto, and Robert Cosgrove investigated the possible familial factor for the conditions of preeclampsia and eclampsia in pregnant women. Preeclampsia and eclampsia, which are related to high blood pressure, have unknown causes and affect at least five percent of all pregnancies.
Subject: Reproduction, Experiments, Disorders
Transposition of the Great Arteries (TGA)
Transposition of the great arteries or TGA is a potentially fatal congenital heart malformation where the pulmonary artery and the aorta are switched. The switch means that the aorta, which normally carries oxygenated blood, carries deoxygenated blood. There are two types of the malformation, d-TGA where no oxygen reaches the body and l-TGA where some oxygenated blood circulates. In the US, the Centers for Disease Control estimate that about 1,901 infants are born each year with TGA, or about one for every 2,000 births.
"An Extended Family with a Dominantly Inherited Speech Disorder" (1990), by Jane A. Hurst et al.
In 1990, researcher Jane Hurst and her colleagues published "An Extended Family With a Dominantly Inherited Speech Disorder," in which they proposed that a single gene was responsible for a language disorder across three generations of a family. Affected individuals of the family, called the KE family, had difficulty producing, expressing and comprehending speech. Hurst and her team studied the KE family and the disorder at the Department of Clinical Genetics at the Great Ormond Street Hospital for Children in London, England.
Subject: Publications, Disorders
Agent Orange as a Cause of Spina Bifida
Spina bifida is a birth defect that affects the spines of developing fetuses and infants, and research in the 20th century indicated that chemicals in the herbicide Agent Orange likely lead to the birth defect. People with spina bifida can have nerve damage, paralysis, and mental disabilities. During the Vietnam War in the 1960s, the US military employed Agent Orange and other herbicides to destroy enemy crops and forest cover until 1970.
Teratogens
Teratogens are substances that may produce physical or functional defects in the human embryo or fetus after the pregnant woman is exposed to the substance. Alcohol and cocaine are examples of such substances. Exposure to the teratogen affects the fetus or embryo in a variety of ways, such as the duration of exposure, the amount of teratogenic substance, and the stage of development the embryo or fetus is in during the exposure.
Cystic fibrosis (CF) is a fatal, inherited disease found in humans and characterized by buildup of thick, sticky mucus, particularly in the respiratory and digestive tracts. The abnormally thick mucus prevents the pancreas from functioning normally; it often leads to digestive problems and chronic lung infections. Cystic fibrosis is most prevalent in Caucasian individuals, and approximately 1 in every 29 individuals in the US is a carrier for the mutated CF gene.
Agent Orange Birth Defects
Sprayed extensively by the US military in Vietnam, Agent Orange contained a dioxin contaminant later found to be toxic to humans. Despite reports by Vietnamese citizens and Vietnam War veterans of increased rates of stillbirths and birth defects in their children, studies in the 1980s showed conflicting evidence for an association between the two. In 1996, the US National Academy of Sciences reported that there was evidence that suggested dioxin and Agent Orange exposure caused spina bifida, a birth defect in which the spinal cord develops improperly.
Trisomy 18 (Edwards Syndrome)
John Hilton Edwards first described the symptoms of the genetic disorder known as Trisomy 18 - one of the most common forms of trisomy, which occurs when cells have an extra copy of a chromosome, in humans - in 1960. Trisomy 18, also known as Edwards Syndrome, occurs approximately once per 6000 live births and is second in frequency only to Trisomy 21, or Down's Syndrome, as an autosomal trisomy. Trisomy 18 causes substantial developmental problems in utero.
Angelman Syndrome
Angelman syndrome is a disorder in humans that causes neurological symptoms such as lack of speech, jerky movements, and insomnia. A human cell has two copies of twenty-three chromosomes for a total of forty-six-one copy from its mother and one from its father. But in the case of Angelman syndrome, the maternal chromosome numbered 15 has a mutation or deletion in its DNA and a gene on the paternal chromosome 15 is inactivated in some parts the brain. The result is the paternal gene is silenced during development of the sperm, which is called genetic imprinting.
Isotretinoin (Accutane) as a Teratogen
Isotretinoin is a molecule and a byproduct (metabolite) of vitamin A, and in greater than normal amounts in pregnant women, it can cause fetal abnormalities including cleft lips, ear and eye defects, and mental retardation. Isotretinoin is commonly called by its trade name Accutane, and it's a chemical compound derived from vitamin A, or retinoic acid. Doctors prescribe isotretinoin to treat severe acne. For pregnant women, too much vitamin A or isotretinoin can also cause greater than normal rates of stillbirths and fetal disintegrations after the ninth week of gestation.
VACTERL Association
VACTERL association is a term applied to a specific group of abnormalities involving structures derived from the mesoderm. Although the defects of this disorder are clearly linked, VACTERL is called an association rather than a syndrome because the exact genetic cause is unknown. "VACTERL" is an acronym, each letter standing for one of the defects associated with the condition: V for vertebral anomalies, A for anal atresia, C for cardiovascular anomalies, T for tracheoesophageal fistula, E for esophageal atresia, R for renal anomalies, and L for limb defects.
Tay-Sachs Disease
In 1881 British opthalmologist Warren Tay made an unusual observation. He reported a cherry-red spot on the retina of a one-year-old patient, a patient who was also showing signs of progressive degeneration of the central nervous system as manifested in the child's physical and mental retardation. This cherry-red spot is a characteristic that would eventually come to be associated with metabolic neurological disorders like Sandhoff, GM-1, Niemann-Pick, and, to the credit of Tay, the lysosomal storage disorder known as Tay-Sachs disease.


The Patriots are used to playing without receiver Julian Edelman. They did it all of last season (thanks to a torn ACL) and for seven games in 2015, and they finished in the top three in scoring both years. No team handles unexpected absences better.
The larger question, in the wake of Thursday's news that Edelman is facing a four-game suspension for violating the NFL's performance-enhancing substances policy, is what sort of player he will be when he rejoins the team.
Edelman emerged as one of the NFL's best slot receivers despite his size (5-foot-10, 200 pounds) because of his incredible quickness and ability to make plays after the catch. One easy way to annoy your local Patriots fan -- one of America's favorite pastimes -- is to tell him that Edelman at his peak was a more dynamic and valuable player than Wes Welker ever was. Now that Edelman is 32 years old and coming off a torn ACL, while also having a history of foot issues, it's unrealistic to expect him to reach those heights again. The Patriots just have to hope his decline is not as swift as that of Welker, who left the Patriots as he was about to turn ... 32 years old.
The expectation among many Patriots fans was that Edelman would have stepped in as the team's No. 1 wideout this season, soaking up a lot of the production that went to Brandin Cooks (who was traded to the Rams) and Danny Amendola (who signed in Miami) last season. I wasn't so sure that would happen even before this news, because of Edelman's age, his injury history and the reality that the only constant in New England is change.
The team is diminished at wide receiver overall, but no one in New England should panic, because it's a deep group. Third-year pro Malcolm Mitchell, who keyed the team's Super Bowl comeback over the Falcons, is back after spending 2017 on injured reserve with a knee injury. Chris Hogan and free-agent pickup Jordan Matthews will have roles. And there will be incredible competition for the final wideout spots among Phillip Dorsett, Kenny Britt, Cordarrelle Patterson and rookie slot receiver Braxton Berrios, who Miami Hurricane fans swear will be the next Edelman. Tight end Rob Gronkowski remains New England's true No. 1 receiver, and the team has a raft of running backs who catch the ball, including James White, Rex Burkhead and first-round pick Sony Michel.
Tom Brady has no shortage of options to throw to, and it's on Edelman to earn his place back on offense. Being suspended will undoubtedly make that more difficult, and there's no guarantee he'll have a primary role when he does return to the field.
The good news for Edelman and the Patriots, if there is any here, is they have time to plan for his absence, and he'll be back on the field when the Patriots' offense remains in the early stages of its development. The same New England offense that came from behind against Jacksonville in the playoffs and then gained 613 yards in the Super Bowl was very much a work in progress as late as December of last season, with the members of an ever-shuffling receiver crew still settling into their respective roles.
No team does a better job adapting on the fly and covering up weaknesses than the Patriots, which is how they won a Super Bowl without Rob Gronkowski and went 3-1 without Tom Brady to start the 2016 season. Nearly every receiver they have has the flexibility to play inside, where Brady likes to throw best, especially Matthews and Hogan. The offense figured to be more geared around their deep running back group this season; Edelman's suspension might only speed that process along.
Then again, the Patriots' ability to problem solve is often so apparent because they get out of the gates slowly. After an offseason full of roster change on both sides of the ball, Bill Belichick already figured to have his hands full in September. That challenge will only be more pronounced with Edelman out of the mix, especially when there is no guarantee that the Edelman New England used to know will ever return.


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Centre for Life: state-of-the-art science facility with an awe-inspiring planetarium.
Newcastle Castle: medieval fortification with excellent views of the city and river.
Laing Art Gallery: quirky collection of 18th- to 20th-century British paintings.
Angel of the North: a huge, winged statue and an unmissable feature of the northern landscape.
Metrocentre: Europe's largest shopping mall and a must-visit for shopaholics.
Discover more about your Newcastle journey with a read through our comprehensive travel guide, which covers everything that makes this city such a top destination.
What's the best way to get around Newcastle?
The Tyne and Wear Metro system offers visitors a great way of getting to the city's best spots, including the up-and-coming Jesmond district. Buses are plentiful as well. You can purchase tickets to use on both the metro and bus, which simplifies getting around the city.
What's the weather in Newcastle?
August in Newcastle sees the warmest weather with an average temperature of 14°C (57°F), whilst January's average temperature of 3°C (38°F) makes it the coldest. Whatever time of year you visit, make sure you prepare for rain as heavy showers can arrive without warning.


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Fernando Torres next club: Reunion among options as striker opts to leave Madrid
Mon, 9 April 2018, 14:24
If reports are true then we could soon be seeing former Liverpool and Chelsea striker Fernando Torres back in the Premier League. The Spanish star has announced that he will leave current club Atletico Madrid at the end of the season - with his contract expiring. Torres has said it's time up at his boyhood club but intends to keep playing for a few more seasons yet. What Are The Odds analyses the Fernando Torres next club odds from bookmaker BetVictor, as to what club could snap up experienced Spaniard in the summer transfer window on a free.
Numerous reports including ESPN have stated that Fernando Torres might be the answer to Newcastle United's need for a pure goalscorer, with Rafa Benitez seemingly keen on a reunion with the 34-year old. Both manager and player couldn't be more familiar with each other, with Benitez bringing the striker to England in the first place, going on to become a fans favourites at Liverpool before the two reunited later at Chelsea. Current odds of 4/1 (5.00) are currently on-offer, so Magpies fans best not start getting customised Torres shirts just yet, however there's seemingly a genuine interest and it would be quite conceivable that the striker would want to work with someone who knows his game and style like Rafa in the north east.
It would appear that if a move back to the Premier League isn't on the wishlist of the Champions League and Europa League winner Torres, then a move to high-flying Valencia in La Liga could be an appealing switch. Whether or not the striker would want to be part of direct competition against his boyhood club is up for debate, but Valencia are fighting strong at the end of this season - sitting just ahead of Real Madrid in third place. Odds of 8/1 (9.00) suggest it's unlikely, but then again there's still plenty of time for discussion to take place before the summer.
If it's one last pay off that Torres fancies then a move to the Chinese Super League looks the more likely option, backed at 6/1 (7.00) with the bookmaker. Former Atleti teammate Yannick Carrasco made the move to the Far East a couple of months ago and may just be putting in a word to Torres about making a move that so many players have opted for over the past few season.
Probability of Fernando Torres next club
Next Club
Fernando Torres Transfer Odds
Newcastle 4/1 20.00%
Any Chinese Club 6/1 14.29%
Valencia 8/1 11.11%
Sevilla 8/1 11.11%
Besiktas 10/1 9.09%
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31 Rosemount Viaduct, Aberdeen AB25 1NQ, UK
The Noose and Monkey
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About This Discount
The Noose and Monkey is one of Aberdeen's most loved bars, serving food & drink. Situated in the heart of the Rosemount area, right next to His Majesty's Theatre, it's a favourite destination for pre-show meals and drinks. The venue has been frequented by many famous stars performing at these shows with recently Samantha Womack, Alan McHugh and Greig Davies to name but a few. The Venue hosts Live Blues on the last Friday of the month, with guest DJ's spinning funk and soul on vinyl on occasional Saturdays throughout the year. With a newly added outside terrace and outside alcohol license granted late 2019, Rosemount has an added outside area for the summer months, being able to enjoy breakfast and brunch Saturday and Sunday from 10 am. The terrace can be enjoyed weekly until 10 pm. Established in 2000, The Noose and Monkey had previously been known as 'My Fathers Moustache' and 'The Silver Slipper'. She was given it's the unusual name by its previous owner and licensee who originated from Hartlepool. The story goes that during the Napoleonic Wars, fishermen from Hartlepool watched a French Warship founder off their coast. The only survivor was a monkey, dressed in French military uniform, presumably to amuse the officers on the ship. The fishermen assumed that this must be what Frenchmen looked like, and after a brief trial, they summarily executed the poor monkey by hanging.
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Wayne Mansfield
By Wayne Mansfield
Shane has never managed to catch a break. His mother died when he was a teen and his father left home the day he turned eighteen. Then he met a man he thought would fulfill all his dreams. Little does he know that Tyler will plunge him into a nightmare of both physical and verbal abuse.
With his self-esteem destroyed and no friends to turn to, Shane lives every day in despair. But when they're invited to a neighbour's house for dinner, things begin looking up. Michael, their host, is no stranger to Shane, for they have often said hello over the fence.
But is he the one who will come to Shane's rescue? Or will Tyler make sure their friendship goes no further than the dinner?
Shane occasionally had to stop and pinch himself. How could life change so dramatically, so rapidly? Of course, he knew it was entirely possible. His mother had developed a cough, taken herself off to the doctor for tests and the results had come back three days later -- positive for lung cancer. Six months later she was dead. She'd never smoked a day in her life, but for some reason, the universe had seen fit to curse her with it anyway. And his father. Always so loving and always there for his wife and children, had taken off the minute Shane had turned eighteen. He'd got home one day and found all his father's things gone. His clothes, his tools, and the car. His older brother had left home two years earlier. He had married a girl and taken off to live in Melbourne. But Shane, who at the time was flipping burgers at Hungry Jacks, couldn't afford to take on the burden of a rental property. Or pay the stack of unpaid bills his father had left behind.
There was no other choice but to pack his things and crash at a friend's house until he could afford to find a flat and some flatmates.
So, yes, he realised that life could change in the blink of an eye, but never for the better. And he felt guilty for thinking that Michael and the happiness he felt when he was with him, was all a dream. That at some time, maybe in the next five minutes or maybe tomorrow, it would all be taken away again, revealed as nothing more than a great joke.
Yet, despite this, there was also a small part of Shane that was revelling in the changes. A miniscule aspect of his spirit that dared to hope his current happy situation would stay a permanent part of his life.
Every day he went to work, and almost every day he would see Michael. They would make love and then go out for dinner, to some cheap little restaurant or other, before returning to Shane's motel room to make love again. Sometimes Michael would stay the night, and sometimes he wouldn't. And on those occasions, Shane made the most of a good night's sleep because whenever Michael was over, neither of them did very much sleeping at all.
It was a Wednesday. Shane had been home from work for an hour. He was showering in anticipation of Michael's visit in a couple of hours. He made sure he washed thoroughly, douching and soaping his foreskin. After drying himself, he brushed and flossed his teeth then dressed. With two hours to kill, he turned on the television and watched the five o'clock news.
There was a story about a mammoth bushfire currently burning in the hills. It had been deliberately lit. That explained the haze and the stench of smoke that hung over the city, although Shane wondered, with all the damage a bushfire caused, how anyone could possibly tell whether it had been deliberately lit or not.
There was a knock on the door.
Shane glanced at the clock and was initially confused. He was sure Michael had said seven. Then he smiled. Perhaps Michael couldn't wait. Perhaps he had skipped the gym because he knew he was going to get a work out with Shane. He hurried to the door, beaming. His cock, already stirring in anticipation.
He opened the door. "Hell ..."
It was Tyler.
"How ... how did you find me?"
Tyler had a self-satisfied smirk on his face.
"Aren't ya gonna invite me in?"
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Lyin' Ryan
The Artist's Mechanic
Tom Fleet's Machine
Family Bonds
He Ain't Heavy
The President's Son
Say You Love Me Like I Do


This collection contains 32 letters and postcards to and from Delbert Claire Brandt (Claire Brandt), who served with the 1st Cavalry in World War I, was wounded, and died on November 16, 1918.
University of South Carolina's Garnet and Black Yearbooks from 1899 to 1994, all available and keyword searchable.
Letters and photographs from Charleston-native George William Walker (b. 1894) who served as a lieutenant in the 52nd Infantry Regiment, World War I.
Several collections in SCPC contain material dating from the Great War.
Isaac Rosenberg, recognized as the first significant Jewish poet in English literature, was one of the major poets whose life was cut short by the Great War, and the only one who served in the ranks. He died on the Somme in 1918 at the age of 27.


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Flightless Birds Don't Form a Natural Group
Last week I wrote about how paleontology provided an insight into evolution that genetic methods so far have not anticipated. I am not biased towards paleontology or any other field. It is futile to debate whether "my field is better than yours". Clearly evolutionary science benefits from a variety of inputs. More complimentary data the better.
Even so here I am writing about another finding where this time genetic methods produced results that fossils and morphological comparisons alone did not anticipate. Large flightless birds of the southern continents like the emu, ostrich, rheas and the kiwi were believed to be more closely related to each other than to other flighted birds. A genetic analysis has apparently redrawn the family tree of these birds.
It was thought that flightlessness evolved just once. I have depicted this scenario in the figure below.
Here flightlessness evolves in a stem or ancestral species. This happens when the southern continents are still part of the super continent Gondwanaland. The stem species gives rise to several descendant species. The flightlessness in each of these descendant species is a plesiomorphy or an ancestral trait inherited from the stem species. As Gondwanaland breaks up different flightless descendant species end up on different continents and diverge in response to the unique conditions.
But the recent genetic analysis indicates a different scenario. There may have been flightless bird species on Gondwanaland but none of them were the common ancestor of all modern southern continent flightless birds. Rather:
Loss of flight evolved several times on different continents independently in different lineages of flying birds. The flightless trait of the modern flightless birds is said to be an apomorphy or a derived trait in each of these lineages.
Why am I not that surprised? Here is what Edward Braun, one of the researchers had to say:
Scientists assumed that a single flightless common ancestor of the ratites lived on the supercontinent of Gondwana, which slowly broke up into Africa, South America, Australia and New Zealand; once divided, the ancestor species evolved slightly in each new location to produce the differences among the present-day ratites
So, the hypothesis that flightless birds formed a monophyletic group with respect to flying birds was never tested using morphological criteria? It seems that a convenient paleo-geographic story was available and so the assumption was made that flightless birds are monophyletic. Does this speak to the limitation of morphological comparisons for understanding evolutionary relationships or is there something else going on? Why was convergence i.e. the independent evolution of similar features never considered a possibility?
Off course the most heretical suggestion is that we still carry an unconscious bias for typological thinking, a tradition of grouping organisms that goes back well before evolutionary thinking. Flightless birds form a group or a type different from flying birds not because we know they are evolutionarily more closely related to each other but simply because they share the distinct trait of losing flight. No evolutionary scientist thinks this way any more.
Posted by Suvrat Kher at 9:18 PM No comments:
Labels: evolution
Fishy Fingers And Why Paleontology Is Sooo Important
From my Science Daily Feed:
Tetrapods, the first four-legged land animals, are regarded as the first organisms that had fingers and toes. Now researchers at Uppsala University can show that this is wrong. Using medical x-rays, they found rudiments of fingers in the fins in fossil Panderichthys, the "transitional animal," which indicates that rudimentary fingers developed considerably earlier than was previously thought.
Source: Uppsala University
Taxa ancestral to tetrapods had already evolved digits. They were re-sculpted and molded by evolution to serve new functions in tetrapods. So far nothing too surprising. As Francois Jacob had long observed, evolution is a tinkerer, adept at rejigging available parts to serve a different function.
But this is what caught my eye:
When they examined genes that are necessary for the evolution of fins in zebrafish (a ray-finned fish that is a distant relative of coelacanth fishes) and compared them with the gene that regulates the development of limbs in mice, researchers found that zebrafish lacked the genetic mechanisms that are necessary for the development of fingers. It was therefore concluded that fingers appeared for the first time in tetrapods.
Palaeontology gave us an insight on how evolution works that genetic techniques did not anticipate. Some time back Olivia Judson wrote an article on how the new fields of molecular genetics and evolutionary developmental biology are providing insights into the details of evolution that fossils cannot provide. I agreed with much of what she had to say. These methods are revealing the innards of the evolutionary process. But I don't agree that this somehow relegates paleontology to a bit player, a support role to the main actors in evolutionary science. This finding is a case in point.
I like to think of it this way. Let's say Mr. Bill Gates writes an article on how he became rich. But for reasons of whimsy he provides only a log showing the important financial benchmarks of his career. How much money he had in his account at various stages, some selected details of his stock holdings etc. Now, in a technical sense he has told us how he became rich, and from this log we can infer up to a point on the important events of his career. But we don't get a complete picture. The evolution of Mr. Gates from a college dropout to a very successful businessman took place in the context of a human ecology. Who were his childhood influences and his mentors? What did he learn from his interactions with his peers? How did the existing computer industry environment influence his decisions? All this makes for deeper insights and a more complete story of how he became rich.
Paleontology and fossils provide just such a broad ecological context to understanding evolution. When did a particular morphological trait arise? Just how did one morphological form get transformed through successive stages and how does this correlate to functional and ecologic shifts? At what rates did morphological features change? What long term patterns of morphological stasis or changes do particular lineages show? What was the nature of faunal turnovers at mass extinctions? What patterns of evolution do the survivor taxa show? Myriad such details that genetic techniques have little to say about and where paleontology is the primary source for understanding the history of life.
Posted by Suvrat Kher at 11:32 AM No comments:
Labels: evolution, palaeontology
A Brief Return To Those Ancient Carbonates
I got an email from a faculty friend who teaches sedimentary geology and stratigraphy in the deep American south. Yes, they have Universities there too and daaawwg gon good ones I might add!
Can you summarize your thesis work and send it to me by this week? He's teaching a course in sedimentary petrology over this semester and needed some material for the carbonate part of it. I've spent this past weekend writing up a summary of my research on calcite cements of Middle and Late Ordovician basins of the southern Appalachians. Cements are chemical and biochemical precipitates that form between pore spaces of sediments and help bind loose sediment into rock. Their morphology and chemistry can tell a lot about ambient conditions of temperature, pore fluid chemistry and basin history. It's been several years since I thought and wrote about carbonates and diving into my thesis was fun. Here's a snippet:
Both the Middle Ordovician Chickamuga Group and the Upper Ordovician Shellmound Formation were affected by meteoric diagenesis relatively early in their individual burial histories. Detailed examination of the diagenetic products using petrography (light and cathodoluminescence ) and geochemistry (trace and stable isotopes) has shown that the precipitation environments were very different. Late Ordovician carbonates of southern Tennessee and northeast Georgia were exposed to subaerial diagenesis during Richmondian sea-level falls associated with the formation of the Taconic unconformity, and show substantial vadose and phreatic zone cements and alteration.
The underlying Middle Ordovician limestones also contain abundant phreatic calcite cement, but cementation is not related to synsedimentary emergence, or to direct vertical infiltration of meteoric water sourced from the overlying Upper Ordovician unconformities. Instead, recharging meteoric water in basin-margin highlands to the southeast, entered Middle Ordovician limestones through confined aquifers during Late Ordovician to Early Silurian times. This meteoric cementation history suggests that patterns of groundwater flow in the basin were strongly influenced by regional shale and lime-mud facies that occur between these two units. These low-permeability strata compartmentalized the Mid-Late Ordovician basin fill into contemporaneous, but hydrologically isolated surficial and deeper aquifers.
I wrote incredibly dense and cryptic passages such as these, not too confuse anyone. That's just the way formal communication in science sometimes reads like. My work was a comparative study of early diagenetic patterns in Middle and Late Ordovician carbonates. The climate changed from a Middle Ordovician greenhouse to a Late Ordovician ice-house and I wanted to find out if this left any impact on the diagenetic patterns.
In many ways I regard the 5 years of graduate research as the most creative period of my life so far. I struck a good rapport with my adviser early on, and a lot of positive outcomes flowed naturally from there. The informal graduate lab atmosphere made work enjoyable. And then there was the bonus of finding treasures like the one below.
I wrote in an earlier post on sea-level fall and the formation of erosional unconformities. The image shows a thin section of a limestone magnified under a microscope. This rock was sampled below a Late Ordovician unconformity. As sea-level fell and exposed the basin a fresh water aquifer developed in the sediments below the unconformity. Meteoric (fresh) water seeping down from the exposed erosional surface into the underlying sediment precipitated the early zoned cement in the pore spaces present between skeletal grains. These cements are tiny, rarely more than a millimeter along the long axis and can be seen only under a microscope. Much later in the Silurian the sea-level rose again and the Late Ordovician sediments were buried under a thick cover of Silurian sediments. The deep burial cement was precipitated then from fluids that were expelled from adjacent iron rich mud.
The image is taken in a cathodoluminesence microscope chamber. When you place a calcite sample in a vacuum chamber and bombard it with cathode rays, the sample will turn luminescent depending upon the presence of certain trace elements like Mn and Fe. The intensity of luminescence in calcite cements has been found to be related to the concentrations of Mn+2 and Fe+2, which act as activator and quencher of luminescence respectively. Reduction of Mn and Fe to a divalent state is necessary for these elements to enter the calcite lattice. In oxidizing pore-fluids, neither Mn+4 or Fe+3 is incorporated into growing calcite crystals, and thus cements are black (non-luminescent). In pore fluids with progressively lower Eh , reduction of Mn first and then Fe leads to their incorporation into the growing cements, giving the crystals a bright to dull luminescence.
That's what the black-golden-brown cement bands in the image show. An initial phase of oxidizing conditions in the aquifer that developed below the Late Ordovician unconformity. And then progressively reducing conditions as the Ordovician sediments got buried under Silurian age deposits.
I obsessed over such stuff for several years. I've moved on to other work which I enjoy as much. Well.... almost.
What do they say? The first cut is the deepest :)
Labels: geology, research, stratigraphy
Some Good Evolution Talks On NPR
I listen to Science Friday on Sunday. It's an old habit. I usually keep aside an hour or so, select what I want to listen to and then make some hot chai in preparation. Not the insipid hot water you get at overpriced coffee shops but real chai with milk, sugar and ginger. Then its podcast time. This week three good talks on evolution.
The first one was a short summary of a pretty important discovery. A gene sequence named HACNS1 that regulates gene activity most notably in the thumb, wrist and ankle. James Noonan one of the researchers explained that a comparison of this sequence with other species showed evidence of considerable evolution in the human version. They got the human version to express itself in genetically engineered mice and it triggered gene activity in the thumb and wrist area. The image below shows expression in the thumb of transgenic mice. The blue color is due to a reporter gene inserted along with HACNS1 to make it easier to identify areas where HACNS1 was being expressed.
Source: Yale Univ.
This may be one of the genes that gives dexterity to our forelimbs and digits. A natural question that came to me was when in our history did HACNS1 diverge from the ancestral version shared with early chimpanzees. I wish they had invited an anthropologist to give a fossil perspective. I dug around some literature and found out that early Australopethicines around 3.5 million years ago already had opposable thumbs different than chimpanzees and gorillas do. So looks like HACNS1 had diverged from the version inherited from the common ancestor with chimps by at least 3.5 to 4 million years ago. Maybe even much earlier if HACNS1 influences ankle development as well. Does it play a role in bipedality? The earliest bipedal hominids go back almost 6 million years.
The second talk was about the release of the new evolution game Spore. The game creator Will Wright along with beta tester evolutionary biologist Richard Prum discussed the game and what we can learn about evolution. Here's how a NYTimes report describes it:
The game begins with a meteorite crashing into a planet, sowing its oceans with life and organic matter. Players control a simple creature that gobbles up bits of debris. They can choose to eat other creatures or eat vegetation or both. As the creature eats and grows, it gains DNA points, which the player can use to add parts like tails for swimming or spikes for defense. Once the creature has gotten big and complex enough, it is ready for the transition to land.
And so on... The games focuses on adaptation and natural selection and so has been criticized for leaving out many other important mechanisms of evolution. No random genetic drift, no migrations and demographic shifts for example. Will Wright defended his approach, in effect saying that the intent was to get people interested in evolution. There are then many other formal avenues for detailed study . I agree with him. It's a game. Have fun. The problem is not that this one game doesn't represent evolution accurately, but that evolution is not covered adequately in school and even college level around the world. Fix that first.
And finally for those who like to unwind with a tall cool one. A talk on the biology and evolution of yeast that are used to ferment ales and lagers. Gavin Sherlock a geneticist from Stanford University discusses these life saving yeasts. Ale was around earlier. Then at some point, two ale yeast strains hybridized to form a new lager yeast. Hurray for the monk who supervised this momentous transition (did anyone else make beer in those days? :)) Lots more in this talk about beer and yeast.
Unfortunately I had had too much chai to switch to a frosty!
Posted by Suvrat Kher at 8:32 PM 1 comment:
Labels: evolution, great conversations, science outreach
Can Strata Above An Unconformity Be Older Than Strata Below It
I just could not resist this. A paper on sequence stratigraphy in the August issue of Journal of Sedimentary Research by Nikki Strong and Chris Paola discuss just how such stratal relations can develop during the evolution of a passive margin lowstand systems tract. That is a lot of terminology. Passive margins are continental margins that form when plates rift and separate as result of sea-floor spreading. The east coast of the U.S and the west coast of India are passive margins.
These margins are characterized by quite a broad continental shelf. Large thicknesses of sediment accumulates on these shelves over time. These deposits are not one homogeneous body of sediment. Rather they are arranged in packages or bundles of sediment known as sequences. The controlling factor on the deposition of these sequences is the rise and fall of sea-level. Of interest here are fluctuations of sea-level spanning roughly 1-10 million years. These are known as 3rd order cycles and the sequences deposited within are called third order sequences. 2nd and 1st order cycles span larger time frames and 4th and 5th order span much smaller periods.
3rd order sequences have become something of a "working unit" for sequence stratigraphers. They are readily recognized in outcrop and in the subsurface using seismic profiling of basins. They also span a long enough period to record different phases of global sea-level history and basin development. Hence the focus on these sequences.
Several types of 3rd order sequences are recognized depending on what phase of a sea-level cycle they are deposited in. Each exhibit distinctive facies arrangements and stratal relationships (should I call it architecture? :)). Sequences deposited during a sea-level rise are called transgressive systems tracts. Those deposited when the sea-level rise has peaked are called highstand systems tracts. And those deposited during a sea-level fall are called lowstand systems tracts. The study of how these sequences evolve and how they are distributed in time and space is known as sequence stratigraphy.
Right. So as sea-level drops, it eventually exposes the continental shelf and an erosional surface develops. When sea-level rises again drowning the shelf, sediment will get deposited on this erosional surface. Geologists recognize this type of surface as an erosional unconformity. It is also a sequence boundary since it separates older sediment of the underlying lowstand systems tract from the subsequent transgressive systems tract. Knowing this how then can strata above an unconformity be older than strata below it?
It is something of a trick question since the researchers were not concerned with the subsequent transgressive systems tract but the complex evolution of the erosional unconformity. The researchers used an experimental basin at the Experimental Earthscape facility at the Univ. of Minnesota. The basin has a subsiding basin floor and a sediment supply system and these were manipulated to recreate scaled versions of lowstand systems tracts.
When I was doing graduate research in carbonates, modeling of sequences to simulate stratigraphic evolution of basins had gained recognition as a powerful tool which complimented outcrop and subsurface evaluation. Now you can get experimental data by recreating scale models of sequences.
That's cool!
The somewhat counter-intuitive stratal relationships develop because sea-level drop across the continental shelf is not instantaneous. The entire shelf is not exposed to erosion all at once or very rapidly. Instead as sea-level starts dropping, the shallower parts of the basin (think near the coast) are exposed early and an erosional surface develops on the exposed sea-floor. At this time the deeper parts (think shelf edge) are still below sea-level and may be active centers of delta formation.
Eventually on the exposed part of the shelf, i.e on the erosion surface a river valley may form and a fluvial depositional system may develop. Sediment is now accumulating on the erosion surface, while towards the shelf edge marine conditions prevail and deltaic sedimentation continues.
As sea-level continues to drop the erosion surface will get extended and keep overriding younger and younger deltaic sediments in shelf edge areas. But sea-level fall and the gradual exposure of the shelf has spanned so much time (often 100's of thousands of years) that often the very initial fluvial sediments deposited on top of the erosion surface will be older than the youngest deltaic sediments that have been recently overridden by the migrating erosion surface.
This may not come as something terrible new to geologists working in sequence stratigraphy but I thought the use of experimental data combined with a theoretical understanding of sequence development is a pretty powerful way to analyze basin evolution. Will this have any practical relevance when it comes to say correlating strata based on their position relative to a sequence boundary? I don't work in this field so I can't say how important such a result will be. But a work like this does remind us that those squiggly lines you see in a geological column and dismiss as just an unconformity, a time of no deposition, a hiatus when nothing but erosion took place, often have complex, protracted and interesting geologic histories of their own.
Posted by Suvrat Kher at 1:42 PM 5 comments:
Labels: geology, sea-levels, stratigraphy
Shame That Apollo Doctor Didn't Say Evolution
I caught a CNN IBN show on the excessive use of medicinal drugs and the dangers of over medicating. One segment was on antibiotics and the bacterial resistance to antibiotics. You can listen to the short segment here.
Here's what the reporter said on the effect of excessive antibiotics and not completing the course: You are giving the bacteria a chance to survive and in time they become much stronger and the antibiotics cannot kill them
And Dr. Chatterjee from Apollo Hospital: Bacteria develop resistance to them (antibiotics). Then you have to go for a harder antibiotics and then over a few years you will have no medication for these ailments at all.
I may have missed a few words but what I did not miss was the complete absence of the word evolution.
There can be a real difference in meaning when you say "giving the bacteria a chance to survive and in time they become much stronger" or "bacteria develop resistance" and saying bacteria evolves resistance to antibiotics.
A lot of people I talk to have the impression that saying bacteria survive and develop resistance means that any particular bacterial cell over its lifetime will develop resistance or "adapt" to the antibiotic. Bacteria according to this view "get used to the antibiotic". But that is not what is happening. What is happening is evolution through natural selection.
Any population of bacteria in your body will initially be variable in its response to a particular antibiotic. Bacteria replicate at very short intervals and therefore a lot of mutations are arising in a bacterial population (most mutations are due to errors in copying DNA during replication). Under a strong selection environment such as antibiotic treatment, bacteria that happen to have the right mutation that enables it to resist the antibiotic will dominate the population. Bacteria that don't will die without leaving many descendants. The next generation therefore will be a population composed largely of bacteria which are resistant. Several such rounds of mutation and selection might lead to a population of bacteria fully resistant to the antibiotic. The schematic below shows this well.
Evolution is not a subject that only helps us reconstruct past events or make sense of fossils or explain the diversity of life. Understanding evolution has real and immediate relevance to several pressing issues we face such as agriculture, animal husbandry, origin and spread of diseases like AIDS and SARS and also how to best set up an antibiotic medication schedule. Understanding evolution let's us plan the antibiotic regimen with the best chance of curing us.
The media and the Apollo doctors missed out on an opportunity to explain that to the people.
Labels: evolution, media, Science and Society
Some Examples of Climate Change Illiteracy
Following on the heels of my previous post in which I attempted to put up arguments in favor of human induced global warming, a stark and scary reminder of what I am up against. A couple of example of climate change illiteracy I picked up from the blogosphere.
This one is from Climate Progress and an interview with Barb Davis White a republican who is running for the 5Th Congressional District in Minnesota.
WHITE: My name is Barb Davis White and I'm running for the 5Th Congressional District against Keith Ellison for the United States House of Representatives, which is called Congress.
ROMM: Where are you on global warming?
WHITE: Well, global warming really has not been proven. There are 30,000 scientists, including Al Gore's professor, from Princeton, who says that we are now in a cooling stage. And ev-every — also every other climate that has been warmed had better grapes.
ROMM: So you don't believe in global warming and you don't think that people caused it.
WHITE: No, I think global warming is a scam. I think it's a scam to put taxes — more taxes on us, and it's called carbon taxes. Our environment has never been so clean, and if we want to push global warming, let's push it on China, where the smog is so thick that you almost need a helmet to breathe. Let's push it on Africa and see how they adapt to it, because they're not going to.
And from Pharyngula who got it from Diatomaceous Earth. A letter sent to the local paper in Fargo, North Dakota. Is Fargo really this eerie?!!
When God sent the rain on this Earth for 40 days and nights, all this water had to go someplace so the Earth would be dry again.
Remember, God is the Creator and controls the universe.
God tilted the Earth from its original position and caused all the excess water to rush to the poles, and there he instantly froze the water into the ice formations that exist today. Time is ticking down on God's time clock.
With all the nuclear bombs that are made and stored for the fast-emerging last battle, this Earth would burn up when these nuclear bombs are set off.
We are not creating global warming - God is tipping the Earth back to its original position on its axis and thus getting all this ice to get ready to move and extinguish the nuclear destructive fires man will create.
Is this being taught in church or at home.? Where do people learn this? Unbelievable! I have come across the first type of illiteracy i.e. the Barb Davis White type in India, but so far not the second. We can find humor in this but it is a rather depressing example of how a religious fundamentalist education can warp your world view. Have you experienced such extreme views in India?
Posted by Suvrat Kher at 11:15 AM 4 comments:
Labels: global warming, humour, Science and Society
How Should I Convince Friends About Global Warming
I learned about climate change in geology classes but that was about changes over geological time, about the Pleistocene ice ages and the Eocene -Paleocene Thermal maxima and the Mesozoic warm period and so on. All these had natural causes but I am persuaded by the collective scientific evidence that humans are causing the earth to warm up and will contribute increasingly to global warming over the next few decades. But if someone asks me a very specific question, let's say "Can you tell me if there have been errors in the measurement of CO2 recovered from air bubbles trapped in ice", I won't be able to answer that.
A few days ago a friend emailed me just such an article which accuses scientists of mishandling, misrepresenting and even deceit in measurements and the presentation of climate data. The article is written by a Polish radiologist and well know climate change skeptic Zbigniew Jaworowski. I read through the article realized that a layperson who is not aware of the data favoring human induced climate change might get persuaded that human induced global warming is a fraud. How should I go about making a convincing case that the article misrepresents climate science?
Can I leave aside the specific questions about ice-core CO2 levels, maybe give references that point to original work and then still make broad arguments that show that Jaworowski himself has made very unreasonable assumptions and omitted important facts and that the scientific data is trustworthy. I am going to try doing that. This is a bit like the evolution-creationism debate. I can't answer very specific biological questions like maybe how a particular protein evolved but over the years I have gathered broad arguments in favor of evolution. I have persuaded some of my friends using these broad level explanations of evolution and I am hoping I can use the same kind of arguments to explain climate change.
Right. So here is what I came up with. Feel free to add you own insights and suggestions on how to improve such arguments.
1) Combating the conspiracy theory charge: At the outset Jaworowski plays on people's suspicion of big organizations and huge bureaucracies to imply that the IPCC report on climate change is a political document. He invokes a world wide conspiracy involving the U.N, large global warming research lobbies (which he does not identify) and the U.S government. Yes, the U.S government wanting to politicize climate science to show bias in favor of human induced global warming! This has to be one of the greatest perverse jokes of all. This is a government who for the last 8 years has muzzled its own scientists from presenting the true picture about global warming, has edited scientific reports from its research organizations to convey the impression that the science is still uncertain, and had to be dragged kicking and screaming to admit finally that anthropogenic warming is true. Has Jaworowski been sleeping in a cave?
But is it possible that the report has been manipulated? I have found that many people have the perception that it is the IPCC which was responsible for the science as well. This is simply not true. Research about climate change has been going on for decades in hundreds of labs, universities and research organizations. Scientists have been working on their pet projects, often on very specific topics, sometimes not even asking directly whether humans are causing warming or not. Their research for example may involve improving methods of estimating past atmosphere composition using tree ring data as proxy. As a result we have measurements of past atmosphere CO2 which some other researcher could use to ask a different question. Our understanding of climate change has proceeded by just such hundreds of individual efforts of data collection, measurements and sharing. The IPCC has never commissioned any research. It simply appointed a committee to collate evidence into a report.
2) Meeting charges of deceit and omission head on: Throughout the report Jaworowski accuses scientists of manipulating data, being selective in their presentations and sometimes outright deceit. But I found Jaworowski does pretty much the same. Here is just one example. He says that "It is true that CO2 is the most important anthropogenic [trace] greenhouse gas, but a much more important greenhouse factor is the water naturally present in the atmosphere, which contributes some 95% to the total greenhouse effect." Now water vapor may be contributing a lot to the total greenhouse effect but this does not explain why the current warming is taking place. The atmosphere is not a homogeneous mixture of gases. It has a layered structure and water vapor is more common in the lower atmosphere. With altitude the atmosphere become drier and colder. The concentration of CO2 is increasing in these high cold drier layers. These layers are absorbing more heat and warming up. The earth is taking in more energy than it is giving out since these cold layers don't radiate energy much. Eventually though some of that heat radiates back to the lower levels of the earth causing warming.
In an effort to convince that the human contribution to warming is at best negligible he fires the following statistics; 97% of emissions of CO2 in the atmosphere come from natural source, humans account for just 3% of emissions. This 3% of emissions are responsible for just 0.12% of the total greenhouse effect. Again reading the figure 3% and 0.12% is enough to persuade many people that human contributions are at best minimal. But that is not how the figure should be read.
What is important is the carbon budget and exchange of carbon between the three big reservoirs, the ocean, the terrestrial biosphere and the atmosphere. The ocean emits about 330 billion tons of CO2 and reabsorbs a little more than that per year. The terrestrial biosphere emits through organic material decay and plant respiration about 440 billion tons of CO2 but absorbs an equal amount through photosynthesis per year. We know that the oceans are gaining CO2 every year and cannot be a net source of CO2. Ditto for the terrestrial biosphere. Humans emit about 27 billion tons per year out of which about 40% are reabsorbed in the oceanic sink. Which means that the only excess CO2 left in the atmosphere is from human emissions. We know this because we can fingerprint human emissions in a variety of ways. This means the correct question to ask is how much of the increase in atmospheric CO2 observed over the last few decades is due to human activity and the answer is not 3% but 100%.
Jaworowski's claim that humans are responsible for just 0.12% of the greenhouse effect is also misleading. The earth is at a comfortable livable temperature today due to the building up of the greenhouse effect by natural emissions of greenhouse gases over the past 4 billion years. Humans had nothing to do with this historic building of the greenhouse effect. So naturally our contribution today to the total greenhouse effect is going to be very small. What is worrying is that our CO2 emissions henceforth over the next few decades will be making the majority contribution to the greenhouse effect. I won't say 100% because future variations in solar radiation may contribute, but our influence on the greenhouse effect is increasing.
95%, 3%, 0.12% all these numbers can easily confuse but they really mean something different than what Jaworowski is letting on. How is that Jaworowski omits these real implications? And what does that say about his credibility?
3) The past is not always a key to the present: A commonly heard argument and one that Jaworowski also uses is that past climatic changes for example the ice ages were triggered not by changing CO2 levels but by changes in solar radiation and so CO2 is not really an important causative factor in climate change. It is true that the Pleistocene ice ages were likely triggered by natural oscillations of solar radiation operating on a Milankovitch frequency of a 100,000 years or so. But that does not mean that CO2 had no role to play in amplifying and maintaining interglacial warm temperatures. This misunderstanding arises primarily because in ice core data, CO2 increase lags the temperature signal by 800-1000 years. Skeptics play on this and claim that therefore historically CO2 has not been an important forcing mechanism for climate change.
Let's assume that this is true and CO2 had no role to play in the Pleistocene ice age and interglacial warm periods. Does that somehow change the physics of thermal infrared absorption and radiation? If the ice ages were triggered by changes in solar radiation and did not involve the warming effects of CO2 will certain laws of physics become invalid through disuse? Maybe genes can become nonfunctional through disuse but the laws of physics don't change. The fact is that increases in CO2 in the atmosphere today will inevitable lead to a more potent greenhouse effect. That is just basic physics and it is not influenced by which forcing mechanism controlled past warming. Climate change can take place through a variety of mechanisms. You have to evaluate episodes on a case by case basis.
4) Can we trust scientists? Why do I believe one group of scientists (a large group) and not a loner like Jaworowski? My belief has nothing to do with whether I believe personally in the IPCC or whether I am chummy with some of the scientists or whether I find them to be virtuous honest citizens. What I believe in is the process of science. Scientific ideas are criticized and rethought, data collected and recollected, experiments replicated, instruments re-calibrated when errors are pointed out. The ideas and data we have today about climate change have withstood the test of peer review and elimination. That is why I believe when the vast majority of scientists tell me that humans have and will continue to contribute significantly to global warming.
But Jaworowski and his ilk don't participate in this free marketplace of ideas. They prefer ambush marketing. Resort to pot-shots at scientists, circulate documents by chain mail over the internet and confuse people with X Files conspiracy theory yarns. Take a look at Jaworowski's publication record. All in pretty obscure journals and as the caption says "Two of his papers on climate appear on the website of 21st Century Science & Technology magazine!! And I am supposed to believe him over 2 decades of peer reviewed science?
This is all I have for the moment. There are a few other points I have left out regarding past sea-level changes. Maybe I will tackle those in a later post. If you can point to more references and better explanations do leave behind a comment. As I said these are broad explanations for some misleading statements I noticed in Jaworowski's paper and I am sure climate experts can point out many other problems with his paper. But for now I have a list of references below for some of the specific points regarding climate change.
The gripe that the IPCC report ignores water vapour
The gripe about the lag between temperature and CO2 in ice cores
The gripe about the Mann hockey stick curve
Blog on debunking Jaworowski's criticism of ice core data
Some more articles about ice core data
More on Human Fingerprints of Global Warming
Posted by Suvrat Kher at 10:51 PM 3 comments:
Labels: climate change, global warming, Science and Society
I Finally Made It On The BBC
I mentioned earlier that in May as I munched my way through hot dogs on that mother of all outdoorsy day, Memorial Day, the BBC tried contacting me to take part in World Have Your Say. I missed out on that one. But you cannot keep a good man out for ever. Last night I got a call again and this time I was prepared. Be ready at 6.00 pm GMT I was told. We will call anytime during the next hour. The topic was "Are there places we just shouldn't live" referring to risky places like in the path of hurricanes, earthquake prone zones and flood plains.
I remember during graduate studies in the U.S. in the nineties, soccer was not very high on the list of sports news. ESPN Sportscentre used to announce that highlights of soccer will follow after the break and I used to stay glued to the TV hoping to catch them. Inevitably they used to come on last, almost as an afterthought. Me having wasted an hour, then got to catch a glimpse for a few seconds of some smuck hitting a goal. Total frustration! That's what happened to me yesterday night. The BBC admin told me I would be on soon, and I kept waiting and waiting until 6.56 pm GMT and then I was told to say my 2 cents worth.
My spiel lasted for a grand total of 1 minute something and then the show was over. No response from other viewers no debate no nothing! Bollocks, damn and damn!! The podcast is available for any sucker interested in this sort of thing. It's a long download about 22 Mb. I come on last. I finally got on the BBC. I have spoken.
Can Strata Above An Unconformity Be Older Than Str...


Vancouver Rowing Club (VRC) is one of Canada's leading rowing clubs for para/adaptive rowing possibilities. We strive to provide recreational and competitive rowing opportunities to anyone who wants to be out on the water rowing! The physcial health benefits of rowing are many, plus, VRC offers a thriving social component to all those involved in the club. We're equipped for AS (trunks and arms) and LTA (Legs,Trunk, and Arms) athletes.
Whether you've never rowed before, or are an experienced Para athlete, please contact the Rowing Coordinator at row@vancouverrowingclub.ca for current information on learning to row or continuing your rowing here.


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StarStyle BTSYA Radio
Cynthia Brian
Site Coordinators
Radio Express UrSelf
It's Just Not Fair
empowering women, families, and youth-at-risk through improved literacy, increased positive media, and tools for living.
By Morgan Hunter
Sometimes, as a teenager, it's easy to feel like material possessions are the center of our world, especially in high-income towns like Moraga, Lafayette, and Orinda. It's easy to feel left out when you don't have the newest phone or the nicest clothes. It's especially easy to forget that not everyone is on a level playing field in terms of wealth. And it's easy to feel that life is unfair.
For my senior year in high school, I will share a single, rented room with my mother. It's not what I would choose, but I don't have an option. "What a lot of people don't understand," says Campolindo student, Suzie (not her real name), "is that not everyone in our community is from a high-income family. A lot of kids don't have the money for fancy cars and other expensive things, and it really bugs me when people aren't considerate of that." While monetary status appears to dominate lives in this community, for teens, it is critical to remember that a parent's bank balance does not determine what our individual future will be.
John, a Lamorinda student states that "sometimes I look at other kids and think to myself: 'Why can't I have that? Why does life have to be so hard for me?' I know so many kids who have a license and a car that was given to them "just because". I have a job and I still can't afford a car."
Although Lamorinda is considered a wealthy area, there are low-income families living here as well. Not everyone is fortunate enough to drive a BMW and in fact, some people may not be able to even afford a bike. Having money is great, yet many of us go without. Fair or not fair, this is life.
Morgan is a Campolindo senior with a passion for art, writing, and birds. She enjoys long walks on the beach, obscure Internet jokes, and red pandas.
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